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1 he epidermal basal layer, to hair follicles, eccrine and sebaceous glands as well as to endothelium o
2 ionate epidermal transcriptomes in the early eccrine anlagen: one that is shared with hair follicles
3 ajor sweat-producing glands present in skin; eccrine, apocrine, and apoeccrine glands.
4                                   In humans, eccrine appendages express DCD and CAMP, genes encoding
5 natures of eccrine identity and uncovers the eccrine dermal niche, setting the stage for targeted reg
6                          We demonstrate that eccrine development requires the induction of a dermal n
7 ratinocytes, melanocytes, mononuclear cells, eccrine ducts, and nerves.
8  epidermal keratinocytes (four of five), and eccrine epithelia (two of four).
9 compare the expression content of wild-type, eccrine-forming mouse skin to that of mice harboring a s
10              LL-37 was localized to both the eccrine gland and sweat ductal epithelial cells, whereas
11 use Chromosome 1 that controls both hair and eccrine gland densities.
12  reciprocal determinant of hair follicle and eccrine gland density and identify a pathway that could
13 lthough reciprocal changes in hair cover and eccrine gland density are required for efficient thermor
14  a major determinant of natural variation in eccrine gland density between strains, and increased En1
15                                 Humans' high eccrine gland density has long been recognized as a hall
16 rmal niche that is responsible for promoting eccrine gland developmental progression.
17  confounded by a lack of knowledge about how eccrine gland fate and density are specified during deve
18  (En1), a transcription factor that promotes eccrine gland formation in humans and mice.
19 specifically on the human lineage to promote eccrine gland formation.
20 cription factor correlates with the onset of eccrine gland formation.
21                                          The eccrine gland is one of the major cutaneous appendages a
22 hair follicle, in myoepithelial cells of the eccrine gland, and in the basement membrane/basal lamina
23  including the dental lamina, mammary gland, eccrine gland, and primary and secondary HFs.
24 hways controlling the relative patterning of eccrine glands and hair follicles, we exploited natural
25 En1 activity directs the relative numbers of eccrine glands and hair follicles.
26 ssociated with an increased number of active eccrine glands in the Han Chinese.
27 ent study was designed to assess the role of eccrine glands in the repair of wounded human skin.
28                                              Eccrine glands secrete water onto the surface of human s
29 sarcoma (KS), and in the epithelial cells of eccrine glands within KS lesions.
30 d appendages, including the sebaceous gland, eccrine glands, and apocrine glands, as well as low leve
31  the transcriptional signature of developing eccrine glands, and they also uncover a unique dermal ni
32 in 5 receptor localized to sebaceous glands, eccrine glands, hair follicles, and epidermis in human s
33 man and mouse epidermis, hair follicles, and eccrine glands, indicating that these channels are found
34  and multiway cross-talk with adipocytes and eccrine glands, perpetuating the sinister thrombotic mil
35 ected by the mutation, including mammary and eccrine glands.
36  secretory cells of sebaceous, apocrine, and eccrine glands.
37  observed in secretory cells of apocrine and eccrine glands.
38  in the skin to induce the formation of more eccrine glands.
39 xpression promotes the specification of more eccrine glands.
40          Our study defines the signatures of eccrine identity and uncovers the eccrine dermal niche,
41  that were present in affected porokeratotic eccrine ostial and dermal duct nevus (PEODDN) tissue but
42                          Both passive (e.g., eccrine/sebaceous secretions, proteolytic) and active (e
43  follicles and one that is En1 dependent and eccrine specific.
44 mors (1.1%) (an adenoid cystic carcinoma, an eccrine spiradenoma and small B cell lymphoma) were diag
45 l canthal tendon, which was identified as an eccrine spiradenoma and small B cell lymphoma.
46       LL-37 protein and mRNA was seen in the eccrine structures of normal human skin by immunohistoch
47                                              Eccrine sweat can serve as a source of biomarkers for as
48 ool their bodies and have by far the highest eccrine sweat gland density among primates.
49 is transport regulator as densely as did the eccrine sweat gland when three monoclonal antibodies for
50 in receptors are also expressed on the human eccrine sweat gland, although it remains unclear whether
51 nsport in the reabsorptive duct of the human eccrine sweat gland, which most likely represents a sodi
52 y isolated human epidermal fragments and the eccrine sweat glands amplified the cystic fibrosis trans
53                                              Eccrine sweat glands are essential for sweating and ther
54                                              Eccrine sweat glands are indispensable for human thermor
55                                              Eccrine sweat glands are skin-associated epithelial stru
56 ths that ultimately form new epidermis; (ii) eccrine sweat glands are the most abundant appendages in
57 vel appreciation of the unique importance of eccrine sweat glands for epidermal repair may be exploit
58 ated with mixed tumor formation arising from eccrine sweat glands found only in the foot pads of mice
59                       More specifically, (i) eccrine sweat glands generate keratinocyte outgrowths th
60                                  The gain of eccrine sweat glands in hairy body skin has empowered hu
61          Our data demonstrate a key role for eccrine sweat glands in reconstituting the epidermis aft
62 sured in isolated nonperfused ducts of human eccrine sweat glands in vitro to investigate basolateral
63 of expansion of keratinocyte outgrowths from eccrine sweat glands parallels the rate of reepitheliali
64  increase in the density and distribution of eccrine sweat glands relative to other mammals and a con
65 an important regulatory function of corin in eccrine sweat glands to promote sweat and salt excretion
66  appendage organ class, which includes hair, eccrine sweat glands, and mammary glands.
67                           In human and mouse eccrine sweat glands, corin and ANP are expressed in the
68 l that contains an approved drug to activate eccrine sweat glands, electrodes and a simple circuit an
69 es and is characterized by reduced or absent eccrine sweat glands, hair follicles and teeth, and defe
70   Hyperhidrosis, excessive sweating from the eccrine sweat glands, is caused by overactivity of the s
71 ), a congenital disorder of teeth, hair, and eccrine sweat glands, is usually inherited as an X-linke
72 by defective development of hair, teeth, and eccrine sweat glands, is usually inherited as an X-linke
73 well as in a subset of cells in the bone and eccrine sweat glands.
74 lated anhidrosis, but morphologically normal eccrine sweat glands.
75 dition characterised by over-activity of the eccrine sweat glands.
76 in abnormal morphogenesis of teeth, hair and eccrine sweat glands.
77 the abnormal development of teeth, hair, and eccrine sweat glands.
78 the features of hypoplastic hair, teeth, and eccrine sweat glands.
79 und, and in the duct and proximal tubules of eccrine sweat glands.
80 igned to enhance fluid and salt loss via the eccrine sweat glands.
81 r cells of the sweat glands is important for eccrine sweat production.
82 oncentrations from passively expressed human eccrine sweat using electrochemical impedance spectrosco
83 ose reporting from passively expressed human eccrine sweat.
84           Due to the high rate of secretion, eccrine sweating is a vital regulator of body temperatur