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1 ions that are degraded prior to shedding and ecdysis.
2 at prevented entry into cells also prevented ecdysis.
3 t peptide eclosion hormone (EH) before pupal ecdysis.
4 sis I, while ETH activates preecdysis II and ecdysis.
5 of EcR-B1 that is evident at 3 d after pupal ecdysis.
6 H has a significant but nonessential role in ecdysis.
7 reactivity decreases centrally during normal ecdysis.
8 on hormone (EH) is a key regulator of insect ecdysis.
9 R, although none showed increases in cGMP at ecdysis.
10 eecdysis within 2 to 10 minutes, followed by ecdysis.
11 ting corresponding to in vivo preecdysis and ecdysis.
12  and neurotransmitters in lepidopteran pupal ecdysis.
13 e putative nAChR subunits that mediate pupal ecdysis.
14 quent 20E pulse responsible for larval-pupal ecdysis.
15 in pupal ecdysis following successful larval ecdysis.
16 ential role of mAChRs in regulation of pupal ecdysis.
17 d it to investigate the role of EH in larval ecdysis.
18 ile directly expressing the motor program of ecdysis.
19 functions of CCAP and bursicon in Drosophila ecdysis.
20 for the function of CCAP efferent neurons in ecdysis.
21 rogression and results in a failure of pupal ecdysis.
22 val life with trace amounts seen just before ecdysis.
23 ior cuticle and is shed in the cuticle after ecdysis.
24 ht time to degrade the cuticle to facilitate ecdysis.
25 protease gene, nas-37, that cause incomplete ecdysis.
26 regulate hatching, larval growth, and larval ecdysis.
27 in the execution and circadian regulation of ecdysis.
28 ents and dendrite severing peak during pupal ecdysis, a period of strong abdominal contractions, and
29 al weight larvae [5 hours after third instar ecdysis (AL3E)] fed on sucrose alone showed suppressed W
30 e neurons did not show detectable cGMP-IR at ecdysis, although the neurons were CCAP-IR.
31                       The molt culminates in ecdysis, an ordered sequence of behaviors that causes th
32 ned-up, which is characterized by incomplete ecdysis and 98% mortality at the transition from first t
33 lure to complete larval-pupal or pupal-adult ecdysis and abnormal wing development are among the most
34 the failure of pupal ecdysis, whereas larval ecdysis and adult eclosion behaviors showed only subtle
35 licit the first two motor behaviors, the pre-ecdysis and ecdysis behaviors, respectively.
36 resulted in the sequential appearance of pre-ecdysis and ecdysis motor outputs.
37                  Effects on the execution of ecdysis and its circadian regulation indicate that CCAP
38 injected Mas-ETH appears much earlier before ecdysis and occurs with shorter latency than that report
39 tants in CCAP express no apparent defects in ecdysis and postecdysis, producing normal adults.
40 ains are maintained at similar numbers after ecdysis and subsequent feeding.
41 are the polymerization of melanin during the ecdysis and the change in the layer spacing during the s
42 aried with respect to their cGMP response at ecdysis and their CCAP-IR.
43 e expression of only EcR-A just before pupal ecdysis and then with the expression of low levels of bo
44 c enzyme degrading cuticular proteins before ecdysis and/or indirectly by processing other proteins s
45  increases in cGMP immunoreactivity (-IR) at ecdysis, and all were immunopositive for crustacean card
46 vior consisting of three steps: pre-ecdysis, ecdysis, and postecdysis.
47 P/MIP neurons are active prior to and during ecdysis; and activity of CCAP/MIP/bursicon neurons coinc
48 nce, and immunohistochemical staining during ecdysis, at which time Mas-ETH is found in the hemolymph
49                                              Ecdysis behavior allows insects to shed their old exoske
50 hort of Ap-let interneurons regulates larval ecdysis behavior by secretion of an unidentified amidate
51 important and heretofore undescribed role in ecdysis behavior itself.
52  key targets of ETH and control the onset of ecdysis behavior, depends fundamentally on the actions o
53      Here, we focus on the endocrine control ecdysis behavior, which is used by arthropods to shed th
54 ir old cuticle by performing the stereotyped ecdysis behavior.
55 t in the central nervous system to evoke the ecdysis behavioral sequence, a stereotype behavior durin
56 atory system on schedule, do not perform the ecdysis behavioral sequence, and exhibit the phenotype b
57  of corazonin signaling in initiation of the ecdysis behavioral sequence.
58  as well as its importance in the control of ecdysis behaviors may change during the postembryonic de
59 aracterized by double mouthparts, absence of ecdysis behaviors, and failure to shed the old cuticle.
60 rst two motor behaviors, the pre-ecdysis and ecdysis behaviors, respectively.
61 the old cuticle by performing preecdysis and ecdysis behaviors.
62  discussed in terms of their highly modified ecdysis behaviors.
63 ells, which induce precocious preecdysis and ecdysis behaviors.
64 ntribute to the mechanisms underlying insect ecdysis behaviors.
65 ide (CCAP), which is involved in stereotyped ecdysis behaviour.
66         We also report a mechanism of insect ecdysis between the first- and second-instar larva, whil
67   CCAP exposure also actively terminated pre-ecdysis bursts from the abdominal CNS, even in the conti
68 In our experiments, the ETH-induced onset of ecdysis bursts was always associated with a rise in intr
69 a responded to CCAP by generating rhythmical ecdysis bursts.
70  resulted in the generation of sustained pre-ecdysis bursts.
71 iter begins to decline and again just before ecdysis, but in the developing adult wings is expressed
72 etwork is remodeled immediately before pupal ecdysis by the emergence of 12 late CCAP neurons.
73 trong abdominal contractions, and abolishing ecdysis causes non-cell autonomous dendrite pruning defe
74 -C was first detected earlier 1.5 days after ecdysis, coincident with the pupal commitment of the win
75 , and DLGR2 transcripts are increased before ecdysis, consistent with their role in postecdysial cuti
76 lease could not be detected, the lack of pre-ecdysis could not be rescued by injections of ETH, sugge
77                     All larvae with arrested ecdysis developed pupal cuticle, but with incomplete she
78 involved in controlling growth, development, ecdysis, digestion, diuresis, and many more physiologica
79 t acetylcholine regulates lepidopteran pupal ecdysis directly through CCAP neurons or by activating t
80 expressed in hypodermal cells 4 hours before ecdysis during all larval stages.
81 nate behavior consisting of three steps: pre-ecdysis, ecdysis, and postecdysis.
82 , FMRFamide neurons are activated during pre-ecdysis; EH, CCAP, and CCAP/MIP neurons are active prior
83 o be entirely sufficient for wild-type pupal ecdysis, even after targeted ablation of all other 42 CC
84 the cuticle between larval stages, and pupal ecdysis externalizes and expands the head and appendages
85  They grow through moulting the exoskeleton (ecdysis) facilitated by breaking along lines of weakness
86  neonicotinoid insecticides: arrest in pupal ecdysis following successful larval ecdysis.
87 network that directs the essential behavior, ecdysis, generates a distinct behavioral sequence at eac
88                                              Ecdysis (i.e., the shedding of the exoskeleton) in insec
89 or viability through its regulation of pupal ecdysis in a type II receptor Wishful thinking (Wit)-dep
90     In this paper, we explore arrested pupal ecdysis in greater detail and propose adverse outcome pa
91 e to preservation biases, making evidence of ecdysis in soft-bodied organisms extremely rare.
92                                  Molting and ecdysis in vitro required entry of the parasite into cel
93 role in the Drosophila CNS in the control of ecdysis, in addition to its known role in the periphery
94 ons that produce neuropeptides implicated in ecdysis, including Eclosion hormone (EH), Crustacean car
95 n placed upstream of CCAP, may also regulate ecdysis independently of CCAP.
96                                       Insect ecdysis is a hormonally programmed physiological sequenc
97          In the month, Manduca sexta, larval ecdysis is accompanied by increases in intracellular cyc
98                                   Crustacean ecdysis is controlled by at least three neuropeptides: m
99                                              Ecdysis is triggered by ETH (Ecdysis triggering hormone)
100 sects at the end of a molt (a process called ecdysis) is typically followed by the expansion and tann
101          This sequence, which mediates pupal ecdysis, is governed by the serial release of several ke
102 plays a critical role in regulation of pupal ecdysis, likely by modulating the release of peptides fr
103 h toxicokinetic analyses, suggest that pupal ecdysis may be mediated by the alpha1, beta1, and beta2
104                                        These ecdysis motor bursts persisted as long as CCAP was prese
105 the sequential appearance of pre-ecdysis and ecdysis motor outputs.
106 P and MIPs, whose joint actions initiate the ecdysis motor program.
107 P) plays a key role in the initiation of the ecdysis motor program.
108 an cardioactive peptide (CCAP) activates the ecdysis motor program; the hormone bursicon is believed
109                     The results suggest that ecdysis (moulting) arose once and support the idea of a
110                                              Ecdysis (moulting) is the defining character of Ecdysoza
111                               An analysis of ecdysis movements showed that neonicotinoid-treated lepi
112 a stereotypic behavioral sequence leading to ecdysis of the old cuticle.
113 opeptide-mediated developmental transitions (ecdysis or wing expansion) when crossed to the panneurop
114    Despite more than 40 years of research on ecdysis, our understanding of the precise roles of these
115 uct the evolution of major components of the ecdysis pathway.
116 mmed cell death (PCD) 24 to 48 h after pupal ecdysis (PE).
117 nt six [APR(6)s] die by 48 hours after pupal ecdysis (PE; entry into the pupal stage), whereas APR(4)
118 deficient in PC2 revealed an abnormal larval ecdysis phenotype.
119 dysis; their targeted ablation alters larval ecdysis progression and results in a failure of pupal ec
120 N(CCAP) mediates environmentally insensitive ecdysis-related behaviors in Drosophila development befo
121                           Both the timing of ecdysis relative to the molt and the actual execution of
122 s of these signaling molecules in regulating ecdysis remain unclear.
123                                       Larval ecdysis replaces the cuticle between larval stages, and
124 nimals invariably died at around the time of ecdysis, revealing an essential role in its control.
125 cts shed their old cuticle by performing the ecdysis sequence, an innate behavior consisting of three
126 ach developmental stage, insects perform the ecdysis sequence, an innate behavior necessary for shedd
127 o elucidate neural substrates underlying the ecdysis sequence, we identified neurons expressing ETH r
128 products in initiation and regulation of the ecdysis sequence.
129 nal in insects through its regulation of the ecdysis sequence.
130  layers of the network controlling the pupal ecdysis sequence: a modular input layer, an intermediate
131 f a new insect peptide hormone that triggers ecdysis - shedding of an old cuticle - has revealed hidd
132 nd bursicon show much more severe defects at ecdysis than do animals null for either neuropeptide alo
133                     Every molt culminates in ecdysis, the shedding of the remains of the old cuticle.
134 ptide hormones they secrete are critical for ecdysis; their targeted ablation alters larval ecdysis p
135 le by means of a stereotyped behavior called ecdysis, thought to be initiated by the brain peptide ec
136  may be an immediate blood-borne trigger for ecdysis through a direct action on the nervous system.
137  this cGMP response has been associated with ecdysis throughout most of insect evolution.
138 ) instar nymph, between a blood meal and the ecdysis to 5(th) instar.
139 re was required at least 26 h prior to pupal ecdysis to disrupt the molt.
140 ETH1 restores all deficits and allows normal ecdysis to occur.
141 g discs, the BRC RNAs appeared shortly after ecdysis to the fifth instar and coincided with the onset
142 interactions among several peptide hormones: ecdysis triggering hormone (ETH), eclosion hormone (EH),
143                This sequence is activated by Ecdysis triggering hormone (ETH), which acts on the CNS
144 activating hormone (PK2/PBAN); PKr-C, and 4) ecdysis triggering hormone (ETH); ETHr-b.
145                 Ecdysis is triggered by ETH (Ecdysis triggering hormone), and we show that the respon
146 amide), Cap2b-like peptides (-FPRXamide), or ecdysis triggering hormones (-PRXamide).
147                                              Ecdysis triggering hormones (ETHs) initiate these behavi
148 tence in endocrine Inka cells, the source of ecdysis triggering hormones (ETHs).
149                                  Blood-borne ecdysis-triggering hormone (ETH) activates the behaviora
150 stem of Manduca sexta show that the peptides ecdysis-triggering hormone (ETH) and crustacean cardioac
151 r to the cnidarian-bilaterian split, whereas ecdysis-triggering hormone (ETH) and crustacean cardioac
152                                              Ecdysis-triggering hormone (ETH) is essential for courts
153                                        While ecdysis-triggering hormone (ETH) is sufficient to trigge
154 ated in thoracic ganglia of larvae expresses ecdysis-triggering hormone (ETH) receptors, suggesting t
155                                              Ecdysis-triggering hormone (ETH) was originally discover
156 ing preecdysis-triggering hormone (PETH) and ecdysis-triggering hormone (ETH), which activate these b
157 orazonin (25-100 pM) induces preecdysis- and ecdysis-triggering hormone secretion.
158                                      Here an ecdysis-triggering hormone, Mas-ETH, is described from t
159 coordinated eclosion and an insensitivity to ecdysis-triggering hormone.
160 ecdysis, which are initiated upon release of ecdysis-triggering hormones (ETH) into the bloodstream,
161 tracheal glands (EGs) secrete preecdysis and ecdysis-triggering hormones (PETH and ETH) at the end of
162  and CG5911b) that respond preferentially to ecdysis-triggering hormones of flies and moths.
163 ne Inka cells, the source of preecdysis- and ecdysis-triggering hormones.
164 onal mechanical properties-immediately after ecdysis until the formation of the fully functional club
165                  Delayed initiation of pupal ecdysis was also observed with treated larvae.
166 , including the periods preceding and during ecdysis when genetic analysis suggests that pqn-47 funct
167 at the pupal stage from the failure of pupal ecdysis, whereas larval ecdysis and adult eclosion behav
168 derstanding the neuroendocrine regulation of ecdysis, which appears to be largely conserved across in
169              The immediate events leading to ecdysis, which are initiated upon release of ecdysis-tri
170 anidin, and thiamethoxam had arrest in pupal ecdysis, which is a novel adverse outcome for neonicotin
171 d to express the preparatory behavior of pre-ecdysis while directly expressing the motor program of e
172 showed high susceptibility to arrested pupal ecdysis while painted ladies (Vanessa cardui) and red ad
173 ursicon gene (pburs)] show severe defects in ecdysis, with escaper adults exhibiting the expected fai
174 phylogenetically earliest direct evidence of ecdysis within total-group Euarthropoda, and represents
175   CCAP KO animals showed specific defects at ecdysis, yet the severity and nature of the defects vari

 
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