ライフサイエンスコーパス: ectasia

1 h abnormal coronary dilatation (aneurysm and ectasia).

2 coherence tomography (AS-OCT)-based stage of ectasia.

3 oing PTK, in an effort to prevent iatrogenic ectasia.

4 to stabilize and sphericize the cornea with ectasia.

5 uality of vision without the risk of corneal ectasia.

6 ia and to assess the risk of post-refractive ectasia.

7 aimed at halting the progression of corneal ectasia.

8 corneal steepening, which may mimic corneal ectasia.

9 ost common noninflammatory bilateral corneal ectasia.

10 d testing samples for normal versus clinical ectasia.

11 ication, ulceration, neovascularization, and ectasia.

12 tentially help reduce the risk of post-LASIK ectasia.

13 lafoy's lesions, and gastric antral vascular ectasia.

14 aser-assisted in situ keratomileusis (LASIK) ectasia.

15 the association with gastric antral vascular ectasia.

16 AV disease with and without ascending aortic ectasia.

17 There was no ectasia.

18 blished epithelium-off technique for corneal ectasia.

19 agen cross-linking in postrefractive surgery ectasia.

20 sler-Krumeich classification for severity of ectasia.

21 and epithelium-off cross-linking for corneal ectasia.

22 toconus and postlaser in-situ keratomileusis ectasia.

23 ts (ICRS) for treating patients with corneal ectasia.

24 treatment of both keratoconus and post-LASIK ectasia.

25 potentially decreased risk of postprocedure ectasia.

26 merged as a novel approach for management of ectasia.

27 he non-ectatic eye in bilaterally asymmetric ectasia.

28 on of keratoconus and postrefractive surgery ectasia.

29 ssible refractive surgery that could produce ectasias.

30 tive and structural abnormalities of corneal ectasias.

31 K value, CDVA, and UDVA in eyes with corneal ectasia 1 year after treatment, with an excellent safety

32 5 studies) and post-laser refractive surgery ectasia (1 study), with a mean postoperative follow-up o

33 ce of Chiari I malformation (6.4%) and dural ectasia (42.6%); and physical examination findings of a

34 ), carcinoma (13), colitis (5), and vascular ectasia (5).

35 ic eye from 57 patients with very asymmetric ectasia (57 eyes, VAE-E group), and the nonoperated fell

36 More eyes with stage 4 ectasia achieved 20/25 visual acuity after PROSE than af

37 ost recent management strategies for corneal ectasia after keratorefractive surgery.

38 Management options for ectasia after laser in situ keratomileusis include intra

39 , and future research directions for corneal ectasia after laser in situ keratomileusis.

40 gation has resulted in increased interest in ectasia after laser in situ keratomileusis.

41 facilitates recognition of risk factors for ectasia after laser in-situ keratomileusis.

42 sslinking (CXL) for the treatment of corneal ectasia after laser refractive surgery.

43 ients (18 male, 8 female) with postoperative ectasia after LASIK (23 eyes) and PRK (3 eyes) were incl

44 cido-based corneal topography that developed ectasia after LASIK (ectasia group) and 174 eyes from 88

45 Ectasia after LASIK and PRK was arrested by CXL with sta

46 ve patients treated with CXL for progressive ectasia after LASIK or PRK at the Institute for Refracti

47 xteen eyes of 14 patients with postoperative ectasia after LASIK were enrolled.

48 e hundred seventy-nine subjects with corneal ectasia after previous refractive surgery.

49 Patients with progressive keratoconus or ectasia after refractive surgery (n = 510).

50 comparable representation of keratoconus and ectasia after refractive surgery in the 2 treatment arms

51 coronary disease, vascular atherosclerosis, ectasia and aneurysm, collateral vessel development and

52 he most frequent vascular abnormalities were ectasia and aneurysm.

53 esions, such as optic pathway gliomas, dural ectasia and aqueduct stenosis.

54 The biliary ductual ectasia and hepatic portal fibrosis associated with ARPK

55 s been documented to stop the progression of ectasia and in some cases may cause regression.

56 Mucinous ductal ectasia and IPN differed histopathologically only in deg

57 Mucinous ductal ectasia and IPNs represent newly recognized categories o

58 able lenses for the visual rehabilitation of ectasia and irregular astigmatism, and an update on scle

59 lens design, predominantly in patients with ectasia and postkeratoplasty astigmatism.

60 ad arteriopathy, the combination of arterial ectasia and stenosis and, importantly, absence of the ty

61 detection of keratoconus and very asymmetric ectasia and to assess the risk of post-refractive ectasi

62 Decreased lesional vascular ectasia and tortuosity were also observed and were accom

63 coronary disease characterized by remodeling ectasia and unusual plaque morphology, the relative high

64 (LVC) due to the risk of progressive corneal ectasia and vision decline post-surgery.

65 Aortic dilations (ectasias and aneurysms) may occur on any segment of the

66 ation of g-C(3)N(4) QDs in A-CXL for corneal ectasias and other corneal diseases.

67 III antibodies with gastric antral vascular ectasia, and a temporal association between SSc onset an

68 days, including jeopardy score and coronary ectasia, and at 1 year, including previous percutaneous

69 y cases can improve visual acuity, stabilize ectasia, and delay or even prevent the need for more inv

70 on of tumor-associated macrophages, vascular ectasia, and hemorrhage.

71 onary intervention, jeopardy score, coronary ectasia, and increasing number of diseased vessels.

72 romising results for keratoconus, post-LASIK ectasia, and pellucid marginal degeneration.

73 he right eye was unavailable due to advanced ectasia, and that of the left eye revealed central steep

74 osis, diffuse or focal; segmental dilatation/ectasia; and tortuosity.

75 bnormalities of vessel calibre, aneurysm and ectasia, are challenging to quantify and are often overl

76 Findings included aneurysms, ectasia, arteriovenous fistulas, and anomalous origins.

77 considered in patients with advanced corneal ectasia before proceeding to keratoplasty, especially if

78 odality for both angiodysplasia and vascular ectasia bleeding.

79 greater for angiodysplasia than for vascular ectasia bleeding.

80 otentially reducing the incidence of corneal ectasia but seems to be associated with an increased inc

81 Mucinous ductal ectasia, but not IPN, was characteristically mucin-hyper

82 t the progression of keratoconus and corneal ectasia by inducing corneal stiffening.

83 The presence of coronary artery ectasia (CAE) is influenced by genetic factors and relat

84 Ectasia can also rarely occur in patients without curren

85 various changes in keratometry data implying ectasia can be observed in patients with PCOS.

86 Eyes with advanced corneal ectasia can be successfully fitted with the PROSE device

87 lafoy's lesions, and gastric antral vascular ectasia, constitutes a significant and increasing propor

88 egments, and photorefractive keratectomy for ectasia, corneal edema, and infectious keratitis.

89 hlighting the potential of BAD-D v4 in early ectasia detection, without altering the index scale or t

90 Coronary artery aneurysms or ectasia develop in approximately 15 to 25% of untreated

91 When ectasia develops, early recognition and proper managemen

92 ch were genes implicated in glaucoma, aortic ectasia, diabetes mellitus, muscular dystrophy and heari

93 shift following RK may be a sign of corneal ectasia disorders such as keratoconus.

94 on difference map at Belin/Ambrosio enhanced ectasia display (BAD) at the Pentacam.

95 ain outcome measure: Belin/Ambrosio enhanced ectasia display (BAD-D) score and keratoconus, defined a

96 tometry (Kmean), and Belin/Ambrosio enhanced ectasia display deviation (BAD-D).

97 nus was defined as a Belin/Ambrosio enhanced ectasia display score of 2.6 or more in either eye based

98 mized version of the Belin/Ambrosio Enhanced Ectasia Display version 4 (BAD-D v4) was developed and v

99 egarding the increasing incidence of corneal ectasia following laser in situ keratomileusis procedure

100 Twenty-eight cysts and ectasia, frequently biliary, were seen in 22 of 44 Prote

101 topathology, topographic pseudokeratectasia, ectasia from transient raised intraocular pressure, poor

102 ectasias, including gastric antral vascular ectasia (GAVE) and angiodysplasia, are increasingly reco

103 (GI) bleeding due to gastric antral vascular ectasia (GAVE).

104 rent hemorrhage from gastric antral vascular ectasias (GAVE).

105 pography that developed ectasia after LASIK (ectasia group) and 174 eyes from 88 consecutive patients

106 In the ectasia group, percent tissue altered >/=40 was the most

107 nsive gastropathy (PHG) and gastric vascular ectasia (GVE), to transjugular intrahepatic portosystemi

108 families with a combination of SRNS, tubular ectasia, haematuria and facultative neurological involve

109 Gastric antral vascular ectasias have strongly been associated with the presence

110 The 46 patients with UGI vascular ectasia hemorrhage included 27 patients with angiodyspla

111 th stenosis in 19%, and segmental dilatation/ectasia in 56%.

112 cluding arterial stenoses, interruptions and ectasia in 7.6%.

113 XL stabilized primary and iatrogenic corneal ectasia in 89% of the patients.

114 in slowing, halting or reversing progressive ectasia in both keratoconus and progressive post-LASIK k

115 rrated eyes, including treatment for corneal ectasia in conjunction with collagen cross-linking (CXL)

116 a safe and effective new technique to reduce ectasia in eyes with advanced keratoconus, potentially a

117 wman layer graft is a new approach to reduce ectasia in eyes with advanced keratoconus.

118 ificantly associated with the development of ectasia in eyes with normal preoperative topography and

119 r generating topographic features of corneal ectasia in human tissue is demonstrated.

120 ion of KCN and post-laser refractive surgery ectasia in most treated patients with an acceptable safe

121 ial genetic and mechanical basis for corneal ectasia in patients with congenital eyelid anomalies.

122 Keratoconus can be a debilitating corneal ectasia in which the cornea thins, bulges, and steepens

123 llucid marginal degeneration, and post-LASIK ectasia, in addition to potentially decreasing or delayi

124 portal gastropathy but not gastric vascular ectasias.In the area of gastric cancer, management revol

125 Management options for postoperative ectasia include conservative management with various typ

126 Vascular ectasias, including gastric antral vascular ectasia (GAV

127 g all of the individual indices, the maximum ectasia index for epithelium had the highest ability to

128 The discriminant function containing maximum ectasia indices of epithelium and Bowman's layer further

129 All Pentacam ectasia indices significantly differed between Groups 1

130 se in having erythrocytic sickling, vascular ectasia, intravascular hemolysis, exuberant hematopoiesi

131 Gastric antral vascular ectasia is a vascular manifestation, and bleeding may be

132 roceeding to keratoplasty, especially if the ectasia is deemed stable.

133 Early management of ectasia is essential to prevent its progression and to p

134 Our knowledge about ectasia is still in evolution.

135 Keratoconus, a progressive corneal ectasia, is a complex disease with both genetic and envi

136 clude scoliosis, chest wall deformity, dural ectasia, joint hypermobility, and acetabular protrusion.

137 ous drainage (kappa = 1), presence of venous ectasia (kappa = 1), and final Cognard classification of

138 ith more distal ductal obstruction or ductal ectasia may benefit from pancreaticojejunostomy.

139 Because ectasia may occur in the absence of risk factors, there

140 nstitutional experience with mucinous ductal ectasia (MDE) and intraductal papillary neoplasms (IPNs)

141 utcomes observed in eyes treated for corneal ectasia (mean 0.40 +/- 0.32 logMAR, Snellen equivalent ~

142 1), and laser in situ keratomileusis-induced ectasia (n = 12).

143 Indications included corneal ectasia (n = 15), failed graft (n = 15), corneal perfora

144 Coronary artery aneurysms (CAAs) and ectasia occur in 0.2% to 5.3% of patients referred for a

145 nking was a safe and effective treatment for ectasia occurring after LASIK.

146 same-day corneal collagen cross-linking for ectasia occurring after LASIK.

147 cterized by symmetrically large kidneys with ectasia of collecting ducts.

148 by thinning of the posterior cornea without ectasia of the anterior cornea.

149 dentify patients at high risk of postsurgery ectasia or those who may benefit most from keratoconus i

150 ed in a patient with gastric antral vascular ectasia or watermelon stomach, a disorder that is increa

151 1) and when including only eyes with stage 4 ectasia (P < .001).

152 ablation map patterns in postoperative LASIK ectasia (POE) and to examine correlations between newly

153 erosclerosis: stenosis-producing plaques and ectasia-producing abdominal aortic aneurysm (AAA).

154 The rate of postoperative ectasia progression was comparable between both groups.

155 scharge, with fibrocystic disease and ductal ectasia providing the next most common causes.

156 ed incidence from 0.66 to 0.033%, iatrogenic ectasia remains a concern due to the severe vision loss

157 , residual stromal bed </=300 mum (57%), and ectasia risk score >/= 3 (43%) (P < .001 for all).

158 by residual stromal bed </= 300 mum (74) and ectasia risk score >/= 4 (8).

159 ral corneal thickness, residual stromal bed, Ectasia Risk Score System scores, and percent tissue alt

160 orneal thinning without a pattern of corneal ectasia (specific), and characteristic features on confo

161 al elevation; keratoconus screening; corneal ectasia; subclinical keratoconus; keratoconus suspect; a

162 The TBI may epitomize ectasia susceptibility and distinguish cases with fruste

163 Accurate detection of post-refractive ectasia susceptibility is essential during preoperative

164 by providing a good sensitivity in detecting ectasia-susceptible corneas.

165 Tubular ectasia (TE) was shown to be associated with UOC in expe

166 es in the keratoplasty group had more severe ectasia than eyes in the PROSE group (P = .038).

167 n being less in patients with postrefractive ectasia than keratoconus.

168 charts of consecutive patients with corneal ectasia that were evaluated for PROSE or underwent kerat

169 For patients with gastric antral vascular ectasia, the panel suggested endoscopic band ligation ov

170 hown to be an effective modality for corneal ectasia, the regression being less in patients with post

171 nase for generating an experimental model of ectasia to evaluate the topographic effects of CXL inter

172 ines, the AUC of the Belin/Ambrosio enhanced ectasia total derivation (BAD-D) and the inferior-superi

173 ed progressive primary or iatrogenic corneal ectasia underwent CXL following the Siena protocol.

174 KCE cohort), 9 subjects with very asymmetric ectasia (VAE cohort) with and without their Rigid Gas Pe

175 toconus (KC), and cases with very asymmetric ectasia (VAE) categories, having one eye with normal top

176 phy (VAE-NT and the fellow eye with clinical ectasia (VAE-E).

177 tion, reduction and stabilization of corneal ectasia was achieved in eyes with progressive, advanced

178 e, and the visual acuity outcome for stage 4 ectasia was better and more rapid compared to keratoplas

179 eratoconus (KCN) and post-refractive surgery ectasia were included.

180 n excluding eyes with early signs of corneal ectasia when screening patients for excimer laser surger

181 in expansive vascular remodeling and luminal ectasia, whereas Th1 immune responses cause intimal hype

182 ients underwent PROSE evaluation for corneal ectasia while 37 patients underwent keratoplasty for the

183 eratoconus (KC) is a multi-factorial corneal ectasia with unknown etiology affecting approximately 1:

184 esults in halting the progression of corneal ectasia, with significant improvement in CDVA and long-t