ライフサイエンスコーパス: ectopic beat

1 mally helps protect against the formation of ectopic beats.

2 ectopic beats compared with those with <0.1% ectopic beats.

3 hythmias were mainly single supraventricular ectopic beats.

4 In the pre-implantation Holter recording, ectopic beats accounted for a mean 3.2 +/- 5.5% of all b

5 50; p < 0.001) in patients with 0.1% to 1.5% ectopic beats and 13-fold (odds ratio: 13.42; 95% confid

6 ed with late interval count decreases due to ectopic beats and 9% with erratic count changes due to a

7 dings showed no change in mean heart rate or ectopic beats and no arrhythmias.

8 K2P1 leak cation channels induce ventricular ectopic beats and sudden death of transgenic mice with h

9 Adenosine reduced ectopic beats and the incidence of ventricular tachycard

10 olume, and dP/dtmax were obtained during all ectopic beats and ventricular pacing.

11 an evoke graded depolarizations, propagating ectopic beats, and if timed appropriately, spiral reentr

12 hm, constant pacing, spontaneous ventricular ectopic beats, and premature stimulation at intermediate

13 It is often initiated by ectopic beats arising from the pulmonary veins and atriu

14 AF), but also frequent atrial or ventricular ectopic beats, cause or aggravate HF.

15 to 25.66; p < 0.001) in patients with >1.5% ectopic beats compared with those with <0.1% ectopic bea

16 chanism-targeted AF treatment, and malignant ectopic beat detection with likely AF progression.

17 ), with a reduction in number of ventricular ectopic beats during the ischaemic phase compared with a

18 Ectopic beats (EBs) are cellular arrhythmias that can tr

19 Relatively low frequencies of ectopic beats (>/=0.1%) dramatically increase the probab

20 n sleep-disordered breathing and ventricular ectopic beats/h (p<0.0003) considered as a continuous ou

21 Patients with >/=0.1% ectopic beats had significantly less reverse remodeling

22 hancing dispersion of refractoriness so that ectopic beats have a high probability of inducing reentr

23 : 3.13 and 1.84, respectively) and for >1.5% ectopic beats (hazard ratio: 2.38 and 2.74, respectively

24 sed significantly in those with 0.1% to 1.5% ectopic beats (hazard ratio: 3.13 and 1.84, respectively

25 ckade of those currents and the emergence of ectopic beats in the action potential.

26 th negligible early afterdepolarizations and ectopic beats in untreated controls.

27 ice results in multiple types of ventricular ectopic beats including single and multiple ventricular

28 Epicardial origin of the ectopic beats increases transmural dispersion of repolar

29 nderlies the degradation of a pulmonary vein ectopic beat into AF.

30 lecular mechanisms alters the probability of ectopic beats is not understood.

31 ernans in arrhythmogenesis and suggests that ectopic beats may not be required.

32 or who are predisposed to magnesium loss or ectopic beats may require more dietary magnesium than wo

33 rrhythmias ranging from frequent ventricular ectopic beats, nonsustained and sustained ventricular ta

34 ardiograms of Kir2.1 (-/-) neonates, neither ectopic beats nor re-entry arrhythmias were observed.

35 models were used to assess the influence of ectopic beats on the outcomes of heart failure (HF) or d

36 ic regression, we estimated the influence of ectopic beats on the percentage of biventricular pacing.

37 nization therapy (CRT), but the influence of ectopic beats on the success of biventricular pacing has

38 There was no increase in either ventricular ectopic beats or complex ventricular arrhythmias as a re

39 as similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479

40 s per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricul

41 n LVESV 31 +/- 15%) than patients with <0.1% ectopic beats (percent reduction in LVESV 39 +/- 14%; p

42 xia-induced QTc prolongation and ventricular ectopic beats persists, especially during physical activ

43 such as conduction velocity restitution and ectopic beats, promote spatially discordant alternans.

44 = 0.841, p = 0.007) but not total number of ectopic beats (r = 0.383, p = 0.313).

45 This study sought to determine if increased ectopic beats reduce the chance of high biventricular pa

46 Without removing any outliers due to ectopic beats, the method is able to detect a degradatio

47 rning heart rate variability parameters, and ectopic beats throughout the recording (20%).

48 Non-PV triggers were defined as non-PV ectopic beats triggering AF or sustained focal atrial ta

49 Non-PV triggers were defined as non-PV ectopic beats triggering AF or sustained focal atrial ta

50 Ventricular ectopic beats (VEBs) are frequently observed in athletes

51 nimal models, early afterdepolarizations and ectopic beats were observed in 33% and 40% of embryoid b