ライフサイエンスコーパス: effective refractory period

1 eriod; the inner limit, however, was not its effective refractory period.

2 d preexcited cycle length, or anterograde AP effective refractory period.

3 generate localised synthetic measurements of effective refractory period.

4 namic measurements or changes in ventricular effective refractory period.

5 ) 50 micromol/L had no significant effect on effective refractory period.

6 n=30) by an S2 at intervals shorter than the effective refractory period.

7 properties, and atrial, AV, and ventricular effective refractory periods.

8 Patients with DWR had a shorter effective refractory period (138.8+/-13.4 versus 163.8+/

9 , and rate-adaptive shortening of the atrial effective refractory periods (14+/-13 versus 12+/-14 ms;

10 10% increases in noninfarct zone ventricular effective refractory period, 3% to 5% increases in infar

11 1 +/- 28 ms; p = 0.05) and ventriculo-atrial effective refractory periods (AC(VI): 97 +/- 21 ms; cont

12 Vagal stimulation shortens the atrial effective refractory period (AERP) and maintains atrial

13 In pace/controls, atrial effective refractory period (AERP) at a drive cycle leng

14 nt efficacy in the preclinical rabbit atrial effective refractory period (AERP) model.

15 brillation (AF)-induced shortening of atrial effective refractory period (AERP), we examined the pote

16 mil has rate-dependent effects on the atrial effective refractory period (AERP).

17 Atrial effective refractory period, AF inducibility, and durati

18 ardioversion to sinus rhythm included atrial effective refractory periods, AF cycle lengths, left atr

19 tential duration and conduction time and the effective refractory period after delivery of the basic

20 ially excitable EBZ, pinacidil shortened the effective refractory period and abolished conduction blo

21 Atrial effective refractory period and AF inducibility on singl

22 Effective refractory period and APD are closely related

23 sms are mediated by increases in ventricular effective refractory period and ARIs, decreases in S(max

24 autoantibodies significantly reduced atrial effective refractory period and predisposed animals to a

25 In AF, both sexes showed reduced effective refractory period and wavelength and depressed

26 he effects of AP14145 and vernakalant on the effective refractory periods and acute burst pacing-indu

27 sic cardiac neural activity, and ventricular effective refractory periods and slope of restitution (S

28 matic children had similar accessory pathway effective refractory periods and supraventricular tachyc

29 revealed that both sexes exhibited shortened effective refractory periods and wavelengths in cAF vs.

30 to 5% increases in infarct zone ventricular effective refractory period, and 4% to 6% increases in Q

31 The changes in AVNW-CL, AV nodal effective refractory period, and ventricular response du

32 oupling interval, from 2 to 45 ms beyond the effective refractory period, and was associated with uni

33 he atria to investigate conduction patterns, effective refractory periods, and inducibility of AF.

34 the pacing site and the other MAPs, and PRR (effective refractory period-APD90=PRR) and related to th

35 g to shortened action potential duration and effective refractory period, as well as the loss of thei

36 urately reproduced AP shortening and reduced effective refractory period associated with altered IKs

37 with organized atrial electrograms and long effective refractory periods associated with disorganize

38 rial effective refractory period, with short effective refractory periods associated with organized a

39 The effective refractory period at the high right atrium rem

40 ed, an effect not related to a change in the effective refractory period at the site of block.

41 /-554 versus 376 +/- 466 ms; P=0.86), atrial effective refractory periods at 90 bpm (250+/-32 versus

42 ing AF prevention was prolongation of atrial effective refractory periods, at least in part attributa

43 The difference in the effective refractory period between the high right atriu

44 siological changes in heart rates and atrial effective refractory period, but both significantly incr

45 Ibutilide prolonged atrial and ventricular effective refractory period by 15% and 8%, respectively,

46 ne was associated with a prolongation of the effective refractory period by 18 +/- 2 ms (P < .05), an

47 he combination of the 2 drugs lengthened the effective refractory period by 42% in atria (P<0.01) but

48 Current treatments extend the atrial effective refractory period by nonselective blockade of

49 by 17%, and APD(-61 mV) (reflecting cellular effective refractory period) by 22% (P < 0.05 for each).

50 ore normal areas of the EBZ, nor was the EBZ effective refractory period changed.

51 107 ms) and ventricular (117 versus 77.5 ms) effective refractory periods, compared with controls.

52 ogy study for 45 minutes to determine atrial effective refractory periods, conduction velocity, condu

53 electrograms (type I) and the longest atrial effective refractory period corresponding to disorganize

54 refractory period, with the shortest atrial effective refractory period corresponding to organized a

55 The effective refractory period data were used to determine

56 Concurrently, the right effective refractory period decreased.

57 dent prolonged action potential duration and effective refractory period, decreased LSG function were

58 The effective refractory period difference between the sites

59 applied to the site with the shortest atrial effective refractory period, disorganized atrial electro

60 e were associated with an abbreviated atrial effective refractory period, enlarged atria, and atrial

61 rolonged the antegrade atrioventricular node effective refractory period (ERP) (from 252+/-60 to 303+

62 heterogeneity (p < 0.001); no change in the effective refractory period (ERP) (p > 0.8) or ERP heter

63 t ventricular (RV) and left ventricular (LV) effective refractory period (ERP) and absolute refractor

64 30 minutes, and their effects on ventricular effective refractory period (ERP) and arrhythmia develop

65 otential duration (APD90), right ventricular effective refractory period (ERP) and blood pressure mea

66 Atrial fibrillation (AF) shortens the atrial effective refractory period (ERP) and predisposes to fur

67 Standard restitution, effective refractory period (ERP) and VF threshold (VFT)

68 rdings provide a surrogate for measuring the effective refractory period (ERP) in human ventricle.

69 ct of atrial fibrillation (AF) on the atrial effective refractory period (ERP) in humans is unknown.

70 ced blockade of membrane currents on APD and effective refractory period (ERP) in rat endocardial and

71 ing cycle length, obese patients had shorter effective refractory period (ERP) in the left atrium (25

72 tion of the action potential duration and/or effective refractory period (ERP) is thought to decrease

73 Because the effective refractory period (ERP) of the working myocard

74 n AP profile, AP duration (APD) restitution, effective refractory period (ERP) restitution, and condu

75 The effect of effective refractory period (ERP) shortening on the vuln

76 imals had repeat attempts at inducing AF and effective refractory period (ERP) testing.

77 shorter action potential duration (APD) and effective refractory period (ERP) than a noninducing sit

78 LAA effective refractory period (ERP) was measured before an

79 maintaining AF and the width, area, weight, effective refractory period (ERP), and wavelength in atr

80 rff-perfused hearts were assessed for atrial effective refractory period (ERP), conduction velocity,

81 Atrial effective refractory period (ERP), conduction velocity,

82 Action potential durations (APD(50,75,90)), effective refractory period (ERP), post repolarization r

83 While approaching the effective refractory period (ERP), the tissue response i

84 AP duration (APD), conduction velocity (CV), effective refractory period (ERP), tissue excitation thr

85 atrial fibrillation interval (AFI) and local effective refractory period (ERP).

86 as associated with attenuation of the atrial effective refractory period (ERP).

87 I, we constructed restitution curves for the effective refractory period (ERP).

88 n potentials (APs) at 90% repolarization and effective refractory periods (ERPs) (60 +/- 1 ms vs. 44

89 Effective refractory periods (ERPs) were determined at 5

90 The effective refractory periods (ERPs) were measured in the

91 /kg) and propranolol (0.1 mg/kg), and atrial effective refractory periods (ERPs) were obtained at bas

92 r limit of the AF vulnerability zone and the effective refractory period for a BCL, decreased as BCL

93 ular action potentials, resulting in shorter effective refractory periods, greater beat-to-beat varia

94 ular action potentials, resulting in shorter effective refractory periods, greater beat-to-beat varia

95 Flecainide increased the effective refractory period in atria by 27% (P<0.01) but

96 c mechanism of STAR driven by increasing the effective refractory period in locally treated areas, co

97 dial APD90, endocardial APD90 or ventricular effective refractory period in Scn5a+/Delta and WT heart

98 We found a significantly longer local effective refractory period in the left ventricular apex

99 VIP shortens atrial effective refractory periods in dogs.

100 n5a+/Delta hearts, and prolonged ventricular effective refractory periods in initially non-arrhythmog

101 cantly prolonged atrial and atrioventricular effective refractory periods in rat isolated hearts and

102 At the first-degree AV block dose, AVN effective refractory period increased from 186+/-37 to 2

103 blood pressure during apnea were abolished, effective refractory period increased to 126.7+/-26.9 ms

104 Effective refractory periods increased from 149+/-16 to

105 ned by measuring prolongation of ventricular effective refractory period induced by bilateral vagal s

106 Effective refractory period is most closely reflected by

107 Given its association with a reduced effective refractory period, it may contribute to the su

108 type I ECG, history of syncope, ventricular effective refractory period <200 ms, and QRS fragmentati

109 n/rapid atrial pacing</=250 ms (or antegrade effective refractory period</=250 ms if shortest preexci

110 dicting VF identified an optimal anterograde effective refractory period of the accessory pathway cut

111 ersus 432 +/- 104 ms, P < .0001), as did the effective refractory period of the AV node (279 +/- 60 v

112 The relative and effective refractory period of the His-Purkinje system i

113 ersistent AF had shorter left atrial APD and effective refractory period (p = 0.01).

114 is demonstrated that short accessory-pathway effective refractory period (P<0.001) and atrioventricul

115 arrhythmias showed shorter accessory-pathway effective refractory period (P<0.001) and more often exh

116 pressure (P<0.0003), and reduction in atrial effective refractory periods (P<0.0001) compared with co

117 Ventricular effective refractory periods prolonged significantly at

118 on potential upstroke, a prolongation of the effective refractory period secondary to the development

119 The atrial effective refractory period shortened in ATR and CAF gro

120 Atrial effective refractory period shortened progressively from

121 Apnea-induced effective refractory period shortening from 110.20+/-31.

122 After establishing chronic AF, atrial effective refractory period shortening, increases in spo

123 Using vagally induced atrial effective refractory period shortening, slowing of spont

124 reentrant circuit, the resulting changes in effective refractory periods tend to stabilize reentry i

125 with programmed extra stimuli at 10 ms above effective refractory period than with stable pacing (13.

126 d to the site of shortest and longest atrial effective refractory periods until atrial fibrillation i

127 We also measured ventricular effective refractory period (V-ERP) and QT interval in s

128 g ventricular fibrillation (VF), ventricular effective refractory period (VERP) and defibrillation th

129 tivity via a prolongation of the ventricular effective refractory period (VERP) in the models, althou

130 c Scn5a+/- hearts, and prolonged ventricular effective refractory periods (VERPs) in non-arrhythmogen

131 with programmed extra stimuli at 10 ms above effective refractory period versus 66.1 +/- 22.9 ms with

132 n of atrial electrogram type with the atrial effective refractory period was further demonstrated by

133 terminal open-chest study, left-atrial (LA) effective refractory period was reduced similarly with A

134 wave duration, but not differences in atrial effective refractory periods, was associated with the de

135 that AV nodal function and right ventricular effective refractory period were impaired in the mutant

136 rogram of atrial fibrillation and the atrial effective refractory period were obtained from multiple

137 Resetting response curves and atrial effective refractory periods were determined with single

138 Effective refractory periods were increased homogeneousl

139 re observed at sites with the longest atrial effective refractory period, whereas 1:1 atrial capture

140 specific location are related to the atrial effective refractory period, with short effective refrac

141 am types closely followed that of the atrial effective refractory period, with the shortest atrial ef