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1 ufficient in the egg-laying muscles to limit egg laying.
2 ficantly in their influence on the timing of egg laying.
3 n decreased sexual receptivity and increased egg laying.
4  SP retained in the female did not influence egg laying.
5 P in females and its quantitative effects on egg laying.
6 d pupariation occurring only at day 14 after egg laying.
7 at mediate this clozapine-specific effect on egg laying.
8 ey events in female reproduction: mating and egg laying.
9 gnal imparted to the egg during ovulation or egg laying.
10 rootworm that had lost fidelity to maize for egg laying.
11 he onset of bursting activity 17 hours after egg laying.
12 serotonergic inputs that combine to modulate egg laying.
13 he serotonergic HSN motor neurons to control egg laying.
14 l for a proper uterine-vulval connection and egg laying.
15 ndocrine cells but has no detectable role in egg laying.
16 dant heteromeric channels prevents premature egg laying.
17 ges are likely to be related to the onset of egg laying.
18 ly couple to a serotonin receptor to mediate egg laying.
19 two dominant mutations that severely inhibit egg laying.
20 e signal (attractin) that is released during egg laying.
21 s that egl-30 requires serotonin to regulate egg laying.
22 eromone attractin), which is released during egg laying.
23 neurons could also disrupt the inhibition of egg laying.
24 egulating one or both of ovariole number and egg laying.
25 movement, and minimal, if any, resistance in egg laying.
26 etry (GC/MS), also correlated with decreased egg laying.
27 rease the value of the sucrose substrate for egg laying.
28 movement and pharyngeal pumping, and delayed egg-laying.
29 ns redundantly with acetylcholine to inhibit egg-laying.
30 ng, and queens that mated once and initiated egg-laying.
31  PDZ domain 10 also inhibits 5-HT stimulated egg-laying.
32 reduce their sexual receptivity and increase egg laying [1, 2].
33 hanges in the female, including the onset of egg laying(1).
34 olfactory learning, aggression, feeding, and egg laying [4-14].
35                                          The egg-laying abnormal-9 (EGLN) prolyl hydroxylases have be
36  protein pheromones that are released during egg laying act in concert to stimulate mate attraction.
37     Subsequently, development of larvae into egg-laying adult hookworms (patency) coincided with a sw
38 e reproductive tract after mating and female egg laying after copulating with virgin males.
39 Ns, sex peptide disinhibits oviDNs to enable egg laying after mating.
40  Like many behaviors, Caenorhabditis elegans egg laying alternates between inactive and active states
41                   Birds stand out from other egg-laying amniotes by producing relatively small number
42 y to determine incubation periods in extinct egg-laying amniotes has implications for dinosaurian emb
43 showed defects in locomotion, body size, and egg laying and an inability to form dauer larvae.
44 tion mutations in clh-3 lead to misregulated egg laying and an increase in HSN excitability, indicati
45 esponsible for a dramatic increase in female egg laying and decrease in female receptivity after copu
46 s post-mating responses, including increased egg laying and decreased sexual receptivity, in the mate
47                              5-HT-stimulated egg laying and egg retention correlated well with differ
48 rolled by species-specific physiology, e.g., egg laying and grazing.
49                       This mutation inhibits egg laying and inhibits HSN activity by decreasing its e
50 morphic behaviors of Caenorhabditis elegans, egg laying and male ventral tail curling.
51 ing effects on many behaviors in addition to egg laying and may generally act, as they do in the egg-
52                 Interestingly, 5-HT inhibits egg laying and pharyngeal pumping in ser-7 null mutants
53 s essential for the 5-HT stimulation of both egg laying and pharyngeal pumping, but that other signal
54 ed to water-filled human-made containers for egg laying and production of progeny.
55                           It also shows that egg laying and refractory period response to SP is at le
56        Male-derived Sex-peptide (SP) elicits egg laying and rejection of courting males in mated Dros
57 ch a disproportionate number were related to egg laying and were expressed in neurons and/or muscle.
58 idence of males) gene partially suppress the egg-laying and connection-of-gonad morphology defects ca
59 elayed insect development and reduced female egg-laying and egg hatching.
60 d adverse effects of imidacloprid on queens (egg-laying and locomotor activity), worker bees (foragin
61 treetlights influence timing of dawn chorus, egg-laying and male success in siring extra-pair young,
62 tic manipulations of the retina suggest that egg-laying and movement aversion of UV are both mediated
63 em to an artificial mated status, triggering egg-laying and reducing susceptibility to copulation.
64 gnificantly impairs embryonic morphogenesis, egg laying, and egg hatching even in mutants lacking the
65 is a central pathway for driving locomotion, egg laying, and growth in Caenorhabditis elegans, where
66 pha(q) signaling that drives the locomotion, egg laying, and growth of the animal.
67     piRNA production commenced shortly after egg laying, and inactivation of the more abundant piRNA
68 ons (oviDNs) is necessary and sufficient for egg laying, and is equally potent in virgin and mated fe
69         Since taste is essential to feeding, egg laying, and mating decisions in insects, improved un
70 , including embryogenesis, cuticle turnover, egg laying, and oocyte maturation.
71 ht activity, swarming, mating, host seeking, egg laying, and sugar feeding.
72 cus on the circuits that control locomotion, egg-laying, and male mating behaviors and their modulati
73  queens that mated once but did not initiate egg-laying, and queens that mated once and initiated egg
74 ells, and severer impairments in locomotion, egg-laying, and survival in Caenorhabditis elegans.
75 rodites also require functional germline and egg-laying apparatus.
76                                   Mating and egg laying are tightly cooordinated events in the reprod
77 eurons (HSNs), the executive neurons driving egg-laying, are tonically inhibited when CO2 is elevated
78 tors mediate avoidance of LPS in feeding and egg laying assays.
79 endent pathway is responsible for inhibiting egg-laying at high-salt concentrations.
80 This suggests that ppk301 is instructive for egg-laying at low-salt concentrations, but that a ppk301
81  outputs, we propose that the interaction of egg-laying attraction and positional aversion for AA pro
82 contrast, pharyngeal taste cells mediate the egg-laying attraction to lobeline, as determined by anal
83  UV spectral preference, are dispensable for egg-laying aversion but essential for movement aversion
84                       First, females exhibit egg-laying aversion of UV: they prefer to lay eggs on da
85 prove understanding of the chemical basis of egg laying behavior of Ae. aegypti, and the kairomones w
86 (HSNs) that stimulate Caenorhabditis elegans egg-laying behavior and discovered mutations in a potass
87 ablished serotonergic response in C. elegans egg-laying behavior as a paradigm, we show that action o
88 ating induces changes in the receptivity and egg-laying behavior in Drosophila females, primarily due
89                       Caenorhabditis elegans egg-laying behavior is inhibited by neurotransmitter sig
90                 However, the neural basis of egg-laying behavior is still not well understood; in par
91                                              Egg-laying behavior of the Caenorhabditis elegans hermap
92                      Effects of gliotoxin on egg-laying behavior required the G protein-coupled serot
93 e HSN neurons play a central role in driving egg-laying behavior through direct excitation of the vul
94  to act in octopaminergic neurons to control egg-laying behavior.
95 e-specific neurons (HSNs), which control the egg-laying behavior.
96 cts cell autonomously in the HSNs to inhibit egg-laying behavior.
97  lack of HSN inhibition, resulting in normal egg-laying behavior.
98 n the mosquito and wAnga influenced mosquito egg-laying behavior.
99 n tph-1 expression were sufficient to affect egg-laying behavior.
100 of serotonergic motor neurons that stimulate egg-laying behavior.
101 ically in the HSNs was sufficient to inhibit egg-laying behavior.
102 nervous system and in the vulval muscles for egg-laying behavior.
103 s, including EGL-47, to set the frequency of egg-laying behavior.
104 lemma and neurons leads to ethanol-resistant egg-laying behavior.
105 ommunity influences Drosophila olfactory and egg-laying behaviors differently than individual members
106 of neural serotonin signaling on feeding and egg-laying behaviors.
107  profound ethanol resistance in movement and egg-laying behaviors.
108 rwise associated with <20% reduction in post-egg-laying breeding success.
109 at these infections significantly accelerate egg laying but do not induce cytoplasmic incompatibility
110 er-7 null mutants and the 5-HT inhibition of egg laying, but not pumping, is abolished in ser-7(tm132
111    Our results suggest that EGL-47 regulates egg laying by activating Galpha(o) in the HSN motor neur
112             Caenorhabditis elegans regulates egg laying by alternating between an inactive phase and
113 rimental outcomes than did oligidic food for egg laying by females.
114 ity, indicating that these channels modulate egg laying by limiting HSN excitability.
115 fected host larvae were still acceptable for egg laying by parasitoids, and the parasitoids thereafte
116 ee swallows have continuously advanced their egg laying by ~3 d per decade.
117 rrored these changes, concurrently advancing egg-laying by 11 days.
118 to the blood, where they are acquired during egg-laying by parasitic wasps that transmit the virus.
119 ied side-by-side in three different lines of egg-laying chickens.
120 ogenetic analyses of the worm motor circuit, egg-laying circuit and mechanosensory circuits that have
121  of a neural circuit, we analyzed the simple egg-laying circuit in the model organism C. elegans We i
122                                    Using the egg-laying circuit of C. elegans as a model, we mapped w
123 nsmitter GPCR expression and function in the egg-laying circuit provides a model for understanding GP
124 al nature of the synaptic connections in the egg-laying circuit remain uncharacterized.
125       The VC neurons play a dual role in the egg-laying circuit, exciting the vulval muscles while fe
126 t on the timing of breeding, being higher if egg-laying commenced before the median date, and substan
127 tion by B. megaterium was linked to impaired egg laying, corresponding to a known trade-off between f
128                      The 5-HT sensitivity of egg laying could be restored by ser-5 muscle expression.
129 ists have traditionally assumed that in each egg-laying cycle mosquitoes take a single bloodmeal that
130 ogy through development of a simple model of egg-laying date evolution, parameterized using data from
131 nk food-odor perception with aggregation and egg-laying decisions.
132                                          The egg laying defect could be rescued by the expression of
133 hibition of 5-HT metabolism also rescued the egg laying defect.
134 w genes from a screen for suppressors of the egg-laying defect associated with elevated lin-12 activi
135 dentified in a screen for suppressors of the egg-laying defect associated with hypermorphic alleles o
136 of the N'3 variant leads to paralysis and an egg-laying defect in the adult, suggesting a deficit in
137 ic transmission and suppressed the lethargy, egg-laying defect, and deficient neurotransmitter releas
138 e stack, resulting in a blocked lumen and an egg-laying defect.
139 units OCR-1 and -4, resulting in a premature egg-laying defect.
140 xylase domain protein 2 (PHD2, also known as Egg Laying Defective Nine homolog 1) is a key oxygen-sen
141                               We obtained an egg-laying defective (Egl) mutant in which the AC fails
142     We isolated cog-3(ku212) as a C. elegans egg-laying defective mutant that is associated with a co
143 s die by necrosis and the mutant animals are egg-laying defective.
144 rate that grk-2 loss-of-function strains are egg laying-defective and contain low levels of serotonin
145                        Here we show that the egg laying-defective mutant egl-6(n592) carries an activ
146 HD, also known as HIF prolyl hydroxylase and egg laying-defective nine protein) site specifically hyd
147 clear migration results in uncoordinated and egg-laying-defective animals.
148                                  Analysis of egg-laying-defective mutants has provided insight into a
149  conserved prolyl residues by members of the egg-laying-defective nine (EGLN) family.
150                             The eggshell and egg-laying defects of cuff mutants are suppressed by a m
151                                          The egg-laying defects of unc-4, cha-1, and unc-17 were resc
152 ct NAD(+), whereas the uv1 cell necrosis and egg-laying defects result from accumulation of the subst
153                Receptor knockouts reveal few egg-laying defects under standard laboratory conditions,
154 tion of the egl-47 gene caused no detectable egg-laying defects, suggesting that EGL-47 functions red
155 hat are defective in HLH-17 via RNAi display egg-laying defects, while those carrying null mutations
156 earance, while hsf-1 and sup-45 mutants have egg-laying defects.
157 oduced a pleiotropic phenotype that included egg-laying defects.
158                     This study suggests that egg-laying demand can temporarily convert UV into an ave
159 nal tracking of single females suggests that egg-laying demand increases their tendency to turn away
160 ies exhibit UV aversion in response to their egg-laying demand.
161 rve cord octopaminergic neurons, triggers an egg-laying drop in response to infection.
162 irst established at approximately 23 h after egg laying during Drosophila testis morphogenesis.
163                            In a field study, egg-laying E. solidaginis females discriminated against
164 ion period, number of eggs laid, duration of egg laying, egg hatchability, and adult longevity in P.
165  TGF-beta Dauer pathway that is masked by an egg-laying (Egl) phenotype; mutants in the pathway displ
166 the sucrose substrate frequently before most egg-laying events, suggesting that they actively suppres
167 st, the 3rd instar larvae (90-94 hours after egg laying) expressing a mutated form of human alpha-Syn
168 y was detected when using homogenates of ten egg-laying females per well.
169                                          How egg-laying females respond to UV light is not known, how
170 nly two known cases in existence of normally egg-laying fish species retaining embryos within the ova
171 ified assumptions that (1) workers take over egg-laying following queen death and (2) productivity in
172  estimation of time since oviposition (i.e., egg laying) for post mortem interval determination, or f
173                         Mutations that alter egg-laying frequency have identified genes encoding a nu
174                                Male birds of egg-laying hen strains have no commercial value and are
175  focuses on sex-specific neurons such as the egg laying hermaphrodite-specific neurons (HSNs), VCs, a
176 in pairs and were associated with successful egg-laying, highlighting a potential fitness relevance o
177                                          The egg-laying hormone ovulin (Acp26Aa) is among the most ra
178 ge of action potentials and the secretion of egg-laying hormone.
179 ollect and concentrate peptide release, with egg-laying hormones and acidic peptide detected.
180 dies reveal that 5-HIAA functions to inhibit egg laying in a manner dependent on the 5-HT receptor SE
181                         Clozapine stimulated egg laying in C. elegans in a dose-dependent manner.
182                                              Egg laying in Caenorhabditis elegans has been well studi
183 5-HT) stimulates both pharyngeal pumping and egg laying in Caenorhabditis elegans.
184    Loss of ser-1 function leads to decreased egg laying in hermaphrodites and defects in the turning
185                       With a model on female egg laying in insects, we demonstrate how variation in b
186                                     Communal egg laying in nonsocial species minimizes egg predation.
187  choose a plain (sucrose-free) substrate for egg laying in our sucrose versus plain decision assay.
188          For example, 5-HT did not stimulate egg laying in ser-1, ser-7, or ser-7 ser-1 null animals,
189                 In contrast, 5-HT-stimulated egg laying in ser-4;mod-1 animals was greater than in wi
190                 5-HT also fails to stimulate egg laying in ser-7(tm1325), ser-1(ok345), and ser-7(tm1
191 lencing the JNK phosphatase puckered induces egg laying in virgins.
192              Serotonin (5-HT) stimulation of egg-laying in Caenorhabditis elegans is abolished in ser
193 ariability in induced responses to stemborer egg-laying in maize Zea mays (L.) (Poaceae).
194 n, vitellogenin (Vg), which is necessary for egg-laying in queens, brood food production in workers,
195  mpz-1 RNAi has no effect on 5-HT stimulated egg-laying in ser-1 mutants rescued by expression of a t
196 Ns), a homologous pair of cells required for egg-laying in the hermaphrodite.
197           mpz-1 RNAi reduces 5-HT stimulated egg-laying in wild type animals and in ser-1 mutants res
198 osomes (viability) and adult worms (pairing, egg laying) in culture without general toxicity to human
199        Indeed, body touch appears to inhibit egg laying, in part by interfering with HSN calcium osci
200 gh the vulva and release tyramine to inhibit egg laying, in part via the LGC-55 tyramine-gated Cl(-)
201 rent downstream targets to control different egg-laying-induced behavioral modifications.
202 nd sensory neurons expressing ppk301 control egg-laying initiation and choice in Ae. aegypti.
203                                              Egg-laying involves a simple motor program involving a s
204                                              Egg laying is inhibited by the G protein Galphao and act
205 SER-1-dependent withdrawal response in which egg laying is significantly decreased.
206 e genomes of two species of mongooses and an egg-laying mammal called an echidna show that a virus cu
207  the embryonic pattern in modern monotremes (egg-laying mammals) and placental mammals, but is a paed
208 ting states," with two extremes (virgins and egg-laying, mated queens) that differ dramatically in th
209 e absence of synaptic input, suggesting that egg laying may be controlled through modulation of auton
210  that both genes are required maternally for egg laying, mitotic progression in early embryos, and em
211 ject via the anterior commissure, similar to egg-laying monotreme mammals.
212 is also present in the duckbill platypus, an egg-laying monotreme, consistent with TCRmu being ancien
213                                          The egg-laying motor circuit comprises a simple three-compon
214 in-coupled receptor that inhibits C. elegans egg-laying motor neurons in a G(o)-dependent manner.
215 its to it, but by actively suppressing their egg-laying motor program during their visits.
216 rgic VC motor neurons slow locomotion during egg-laying muscle contraction and egg release.
217 annels localize to postsynaptic sites in the egg-laying muscle, and mutants lacking ERG have more fre
218 ns, which synapse onto both the HSNs and the egg-laying muscles and are thus the third cell type comp
219 t calcium transients and contractions of the egg-laying muscles even during the inactive phase.
220 speed video recording and calcium imaging of egg-laying muscles in behaving animals, we found that th
221  function is necessary and sufficient in the egg-laying muscles to limit egg laying.
222 e-specific neurons (HSNs) that innervate the egg-laying muscles to stimulate contraction.
223 ate behavior by limiting excitability of the egg-laying muscles.
224 kes calcium transients or contraction of the egg-laying muscles.
225  drives twitching or full contraction of the egg-laying muscles.
226 kdown results in a defect in the function of egg-laying muscles.
227 the schistosome larvae and markedly impaired egg laying of adult worm pairs maintained in culture.
228 e receptors function redundantly or regulate egg-laying only in specific conditions; however, increas
229 lity" of the sucrose-containing medium as an egg-laying option to the reproductive system depends on
230 that the value of a sucrose substrate- as an egg-laying option-can be adjusted by the activities of a
231 whether male SP expression influences female egg laying or if any effect of SP is mediated by SP rete
232 ions in parallel with egl-30 with respect to egg laying or is not the major effector of EGL-30.
233 iety of species, mismatches in the timing of egg laying or numbers of offspring have had relatively l
234 , but found no additional Acps that affected egg laying or receptivity upon ectopic expression.
235 with some individuals specialized for either egg-laying or foraging and 'generalists' that perform bo
236 f either unmated queens (n = 8) with induced egg laying, or queens (n = 12) mated in isolation with d
237 lation between male SP expression levels and egg laying, or the amount of SP in the female reproducti
238                In birds, maternal effects on egg-laying order and offspring growth, two proximate det
239 ck coloration increases with position in the egg-laying order, but parasitic eggs are usually the fir
240 f female mammals, with much less known about egg-laying organisms or paternally-mediated effects.
241 w that as acylsucrose amounts increased, WFT egg-laying (oviposition) decreased and TSWV inoculation
242 gy from 280 plant and insect species and the egg-laying phenology of 21 British songbird species, we
243 strinae): a helper's chance of inheriting an egg-laying position, and the workforce available to rear
244 neurons compete to either enhance or inhibit egg-laying preference for ethanol-containing food.
245   However, our observations reveal that this egg-laying preference is a complex process, as it direct
246                                              Egg-laying preference provided a context-dependent fitne
247 rning and decision center, affected females' egg-laying preferences in our sucrose versus plain assay
248 n (Acp26Aa), stimulates an early step in the egg-laying process, the release of oocytes by the ovary.
249 lie the transition from a virgin to a mated, egg-laying queen.
250 queens than those in workers and virgin, non-egg-laying queens, even if methoprene or precocene treat
251 than mortality of bee larvae or reduction in egg laying rate.
252 r of ovarioles per ovary strongly influences egg-laying rate and fecundity.
253 ostmating response (PMR) comprises increased egg-laying rate and reduced sexual receptivity and is co
254         We show that peptidoglycan regulates egg-laying rate by activating NF-kappaB signaling pathwa
255 ve workers, and foragers, as well as reduced egg-laying rate, could impact colony dynamics over multi
256 as developmental progression, body size, and egg-laying rate.
257 ion to AA-containing food for the purpose of egg-laying relies on the gustatory system, while positio
258 well as the physiological mechanism blocking egg-laying, remained unknown.
259 ractoriness to further mating and triggering egg-laying, remains elusive.
260                           This inhibition of egg laying requires MOD-1, a 5-HT-gated chloride channel
261  we found that clozapine-induced increase in egg laying requires tyramine biosynthesis.
262  between repulsive positional and attractive egg-laying responses toward the bitter-tasting compound
263                   We found that selection of egg-laying site in Drosophila melanogaster is a suitable
264     These findings suggest that selection of egg-laying site involves a simple decision-making proces
265                      Drosophila melanogaster egg-laying site selection offers a genetic model to stud
266  flies, geosmin is not aversive but mediates egg-laying site selection.
267 tive about the chemosensory quality of their egg-laying sites, an important trait that promotes the s
268 ence of food, potential mates, or attractive egg-laying sites.
269 wing that inbreeding cost is reduced in some egg-laying species by postcopulatory mechanisms that fav
270                This constraint is relaxed in egg-laying species, which are sensitive to hormones duri
271 nce of the two commonest wasp species at the egg-laying stage and favours the pollinators.
272  no significant effect upon blood feeding or egg laying success.
273 ive queue and that PQs take over the role of egg-laying, successively, without overt conflict, as the
274 lar mechanisms of presynaptic development in egg-laying synapses of Caenorhabditis elegans, demonstra
275                            CO2 modulates the egg-laying system partly through the AWC olfactory neuro
276                                 A functional egg-laying system requires cell fate specification event
277 ays multiple roles in the development of the egg-laying system, acting to both promote cell fate and
278 ing and may generally act, as they do in the egg-laying system, to integrate multiple signals and con
279 n coordinating development of the C. elegans egg-laying system.
280  are thus the third cell type comprising the egg-laying system.
281               Three weeks after the onset of egg laying, T-cell responsiveness began to increase and
282 eromones are predicted to be released during egg laying that act in concert with albumen gland attrac
283 also reveals an inhibitory effect of 5-HT on egg laying that is normally masked by SER-1-dependent st
284 ls and exhibited significant 5-HT-stimulated egg laying that was dependent on a previously uncharacte
285 e plays a specific role in the inhibition of egg laying, the modulation of reversal behavior, and the
286          Among the best studied behaviors is egg-laying, the process by which hermaphrodites deposit
287  we identify the neural circuitry that links egg laying to mating status in Drosophila melanogaster.
288                              The switch from egg-laying to retaining and giving birth to live young i
289 ior of the roundworm Caenorhabditis elegans, egg laying, to perform a behavior-based screen for natur
290 role for dopamine in coupling locomotion and egg-laying together across states.
291 EGL-47 functions redundantly, or it inhibits egg laying under specific circumstances as yet unidentif
292 mote release of neurotransmitters that block egg laying until eggs filling the uterus deform the neur
293  or indirectly to the risk of Se toxicity to egg-laying vertebrates (fish and piscivorous birds) in S
294 mes available to toxicologically susceptible egg-laying vertebrates.
295            Further, fluoxetine can stimulate egg laying via the Gq protein EGL-30, independent of SER
296                              5-HT-stimulated egg laying was absent in ser-5;ser-4;mod-1;ser-7 ser-1 a
297 s that contributed to the 5-HT modulation of egg laying were identified in Caenorhabditis elegans.
298 vior of the nematode Caenorhabditis elegans, egg laying, which is driven by a pair of serotonergic ne
299 animals prolong the duration of reproductive egg laying, which may enable the subsequent development
300 ned the behavior and physiology of queenless egg-laying workers.
301 single egg layer (queen) and a number of non-egg-laying workers.

 
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