ライフサイエンスコーパス: ejaculation

1 al stimulation (e.g., mounts, intromissions, ejaculations).

2 rons is triggered by stimuli associated with ejaculation.

3 of defined spinal segments in the control of ejaculation.

4 o become receptive within 30 min and mate to ejaculation.

5 y and length of intromission, and latency of ejaculation.

6 the medial preoptic area (MPOA), facilitates ejaculation.

7 ocus must result from events following sperm ejaculation.

8 up along this attractor, peaking just before ejaculation.

9 ndent predictor for failure of PVS to induce ejaculation.

10 l motivation acutely after experiencing male ejaculation.

11 iminishes the occurrence of intromission and ejaculation.

12 ir, with activity best predicted by rates of ejaculation.

13 their activity late after the onset of male ejaculation.

14 known as capacitation that takes place after ejaculation.

15 ation state after the female mice experience ejaculation.

16 nt (ADC) values was applied before and after ejaculation.

17 olinergic circuits used for intromission and ejaculation.

18 gy is coupled to cellular events involved in ejaculation.

19 on T2-weighted images, both before and after ejaculation.

20 R imaging while masturbating to the point of ejaculation.

21 he vas deferens during the emission phase of ejaculation.

22 ng copulation, and especially at the time of ejaculation.

23 eurons that express Fos after mating reflect ejaculation.

24 of its neurons participate in the control of ejaculation.

25 rugs are approved for treatment of premature ejaculation.

26 nt for men with moderate-to-severe premature ejaculation.

27 dapoxetine in patients with severe premature ejaculation.

28 ehavior, but failed to rescue the deficit in ejaculation.

29 current understanding of central control of ejaculation.

30 n evoked as unconditional responses (URs) to ejaculation.

31 The latter could help to trigger ejaculation.

32 neurons signals information associated with ejaculation.

33 achieved less intromissions and virtually no ejaculations.

34 g the amount of sperm in a similar number of ejaculations.

35 erectile dysfunction (0%-3%), and retrograde ejaculation (0%-3%) but are associated with increased ne

36 d intromissions, peaked 2-3 min after male's ejaculation (0.2-0.4 degrees C), and abruptly dropped un

37 dial preoptic area (MPOA) of male rats after ejaculation; (2) whether increasing 5-HT in these sites,

38 , oxymetholone generally failed to stimulate ejaculation above the levels of the oil group.

39 hese percentages are combined with those for ejaculation-activated cells involved in the PdPN and lat

40 Ejaculation-activated cells participate in the PdPN and

41 To identify other potential targets of ejaculation-activated cells, we traced PdPN and lateral

42 Ejaculation-activated PdPN cells project to the AVPv (43

43 Male ejaculation acutely suppresses sexual motivation in male

44 t, relatively little is known about how male ejaculation affects sexual motivation and sexual behavio

45 adly to social cues, respond strongly during ejaculation and are not essential for male aggression.

46 Upon ejaculation and during their transit through the female

47 f the bulbospongiosus muscle, which mediates ejaculation and ejaculatory behavior, is markedly dimini

48 Distal penis expansion ("penile cupping") at ejaculation and forceful expulsion of ejaculatory fluid

49 eurons were activated in males by display of ejaculation and in females by vaginocervical stimulation

50 fective methods of treatment of disorders of ejaculation and orgasm in men.

51 adulthood, resulting in increased latency to ejaculation and postejaculatory intromission and longer

52 However, the neural substrates mediating ejaculation and processing ejaculation-related signals r

53 mating females peaked specifically upon male ejaculation and remained elevated above baseline until d

54 In mammals, sperm cells become motile during ejaculation and swim up the female reproductive tract.

55 ivation of PdPN and lateral MeApd neurons at ejaculation and that NOS in PdPN and MPNm cells is regul

56 In rats, these neurons are activated with ejaculation and their lesion selectively abolishes ejacu

57 nsignificantly increased risk for retrograde ejaculation and urogenital problems.

58 uprapubic discomfort or pain during or after ejaculation and voiding complaints such as irritative an

59 return latencies following intromissions and ejaculations and increase withdrawal from the male follo

60 creases during copulation, especially during ejaculation, and increased glutamate facilitates copulat

61 cause its levels in the preoptic area affect ejaculation, and it could synchronize clustered neurons

62 LHAA; no 5-HT increases were observed before ejaculation, and levels were decreased toward basal valu

63 reuptake inhibitors (SSRIs) impair erection, ejaculation, and libido.

64 hieve and maintain an erection, frequency of ejaculation, and orgasm frequency than did patients rece

65 mes were age at menarche, voice break, first ejaculation, and Tanner stages 2 to 5 for pubic hair, br

66 y large increase in samples collected during ejaculation (approximately 300% of BL).

67 ain cells that are activated specifically at ejaculation as assessed by Fos expression.

68 Testosterone cypionate sustained ejaculation at all doses tested.

69 Stanozolol failed to maintain ejaculation at any dose tested.

70 s induction was specifically associated with ejaculation, because mounts or intromissions did not tri

71 ar trends for reductions in intromission and ejaculation behavior.

72 ocin receptor antagonist not only attenuates ejaculation but also affects pre-ejaculatory behavior du

73 ation and their lesion selectively abolishes ejaculation but not other mating behaviors.

74 ponds significantly and specifically to male ejaculation but not to female-to-male sniffing or to mal

75 Mammalian spermatozoa become motile at ejaculation, but before they can fertilize the egg, they

76 ular neuron damage; these males copulated to ejaculation, but they had lower intromission ratios and

77 oes not occur by augmenting the frequency of ejaculations, but by increasing the amount of sperm in a

78 sexual function-desire, arousal, orgasm, and ejaculation-can be affected.

79 on) and consummatory (mounts, intromissions, ejaculations) components of male sexual behavior were me

80 .99, 3.63-4.39), sneezing (2.77, 1.40-5.50), ejaculation difficulty (2.63, 1.61-4.28) and reduced lib

81 Sexual dysfunction like ejaculation discomfort is described as a symptom of CP/C

82 l contact and others were not, revealed that ejaculation duration was the key factor in the induction

83 The incidence of ejaculation dysfunction was lower in the Exp-group than

84 that alteration of NTPDase1 activity affects ejaculation efficacy and male fertility.

85 Successful dynamic MR imaging of ejaculation events and the ability to visualize internal

86 ial (first mount or intromission) and final (ejaculation) events of each copulatory cycle, suggesting

87 and age at menarche or voice break and first ejaculation-every 6 months from 11 years of age until fu

88 similarly to the microinjection, increasing ejaculation frequency and decreasing ejaculation latency

89 blocked 8-OH-DPAT's facilitative effects on ejaculation frequency and latency, while the 5-HT1A anta

90 No association was observed between ejaculation frequency in early adulthood and LUTS.

91 Our results suggest that ejaculation frequency is not related to increased risk o

92 Ejaculation frequency was assessed by asking participant

93 Ejaculation frequency was not statistically significantl

94 However, high ejaculation frequency was related to decreased risk of t

95 Most categories of ejaculation frequency were unrelated to risk of prostate

96 ieve erection, ability to maintain erection, ejaculation frequency, orgasm frequency, and sexual desi

97 exually transmitted infections, prostatitis, ejaculation frequency, surgery for an enlarged prostate,

98 the defects and recover penile erectile and ejaculation function successfully.

99 A spinal ejaculation generator (SEG) has been identified in the r

100 that are commonly referred to as the "spinal ejaculation generator (SEG)." We have examined the funct

101 ulatory behavior and may be part of a spinal ejaculation generator.

102 onal Index of Erectile Function (IIEF-5) and ejaculation grading.

103 ncrease in research with regard to premature ejaculation has led to a significant number of new paper

104 dence-based definition of lifelong premature ejaculation has set a model in the evaluation and treatm

105 rm from copulating for several minutes after ejaculation have been identified.

106 ndings in the physiology and neurobiology of ejaculation have expanded our understanding of male sexu

107 cies and intervals between intromissions and ejaculation, higher lordosis quotients and ratings, more

108 areas of the gerbil brain are activated with ejaculation, i.e., the posterodorsal preoptic nucleus (P

109 Activating these Crz INs caused rapid ejaculation in isolated males, a phenotype mimicked by i

110 sal medial amygdala (MeApd) express Fos with ejaculation in male gerbils.

111 sal medial amygdala (MeApd)-are activated at ejaculation in male rats and gerbils as seen with Fos im

112 othesis that these cells are activated after ejaculation in male rats and vaginocervical stimulation

113 ng mounts and pelvic thrusts in females, and ejaculation in males.

114 Men with moderate-to-severe premature ejaculation in stable, heterosexual relationships took p

115 g the gender-specific outcome of copulation: ejaculation in the male and sperm transport in the femal

116 imately 280% of BL), increased the number of ejaculations in the 40 min test, decreased ejaculation l

117 ring subsequent retention testing, and after ejaculation) in experimental rats [that received electro

118 Sexual activity, and especially ejaculation, increased levels of glutamate in the MPOA.

119 In particular, ejaculation-induced Fos expression is expressed in the m

120 ons were all inhibitory indicating that male ejaculation induces a prolonged inhibitory activity in t

121 nd mating-specific (such as for erection and ejaculation) inputs.

122 Ejaculation is an integral part of normal sexual functio

123 ons playing an pivotal role in expression of ejaculation is reviewed.

124 as been reported for silodosin, but abnormal ejaculation is the most commonly reported adverse effect

125 Ejaculation is the most reinforcing component of sexual

126 A crucial role in ejaculation is thought to be played by a population of l

127 es found in the caput are destroyed prior to ejaculation, is a newly discovered function for the epid

128 produce a change in mount, intromission, and ejaculation latency or in mount and intromission frequen

129 f ejaculations in the 40 min test, decreased ejaculation latency, and decreased the postejaculatory l

130 reasing ejaculation frequency and decreasing ejaculation latency, postejaculatory interval and mount

131 Prior to ejaculation, mammalian sperm are stored in the epididymi

132 us, every PdPN and MeApd cell activated with ejaculation may participate in one of these projections.

133 has been suggested that 5-HT released after ejaculation may promote the sexual quiescence of the pos

134 uctive behaviors (mounts, intromissions, and ejaculations) more quickly than their control counterpar

135 Krause corpuscles impaired intromission and ejaculation of males and reduced sexual receptivity of f

136 Similarly, ejaculation of more sperm when males are paired with fem

137 males were removed after the first or second ejaculation or left in the test chamber.

138 Erectile function, arousal, ejaculation, orgasm, and overall satisfaction domain mea

139 CREB activity, however, has no influence on ejaculation parameters.

140 per month compared with men reporting 4 to 7 ejaculations per month at ages 20 to 29 years were 0.89

141 relative risks for men reporting 21 or more ejaculations per month compared with men reporting 4 to

142 In females, only ejaculation preceded by multiple intromissions induced a

143 ncrease in Fos-IR; multiple intromissions or ejaculation preceded by only 0-1 intromission did not af

144 Thus, we determined whether the ejaculation-related cells produce NO by assessing Fos co

145 s minimal in gerbils, involving few, if any, ejaculation-related cells.

146 These cells are positioned to relay ejaculation-related signals from reproductive organs to

147 strates mediating ejaculation and processing ejaculation-related signals remain poorly understood.

148 viors, including mounting, intromission, and ejaculation, remain largely unexplored.

149 males tested immediately after receiving two ejaculations showed analgesia.

150 Our study identifies a female ejaculation-specific signal in a major neuromodulatory s

151 e spinothalamic pathway involved in relay of ejaculation-specific signals is discussed.

152 Upon ejaculation, sperm must alter their metabolism to genera

153 ed for sexual desire, arousal, and orgasm or ejaculation stages of sexual responding.

154 s arousal, orgasm and the expulsion phase of ejaculation such as functional MRI, dynamic pelvic ultra

155 ired more intromissions at a faster pace per ejaculation than did bLR males.

156 urons that showed increased response to male ejaculation, the response magnitude as well as the propo

157 turn to the male after an intromission or an ejaculation, thereby decreasing the percentage of time s

158 ar 5-HT in the MPOA, yet both can facilitate ejaculation, these data suggest that moderate changes in

159 participants to report the average number of ejaculations they had per month during the ages of 20 to

160 apable of fertilizing eggs immediately after ejaculation; they acquire fertilization capacity after r

161 nable to fertilize the egg immediately after ejaculation; they acquire this capacity during migration

162 animals, male seminal fluid coagulates upon ejaculation to form a hardened structure known as a copu

163 ntromission and facilitates the intromission-ejaculation transition by inducing a slowdown in DA rhyt

164 Ejaculation was correlated with enhanced 5-HT release fr

165 No changes before and after ejaculation were observed in T1 values or in T2 and ADC

166 s, young-onset prostatitis, and frequency of ejaculation, were investigated in relation to lower urin

167 penile vibratory stimulation (PVS) to elicit ejaculation when the concerned spinal segments were inju

168 howed reduced levels of intromissions and no ejaculations whereas simple mounting behavior was not af

169 ibute to a multifactorial model of premature ejaculation with some neurobiological vulnerability.

170 estigation for the possibility of retrograde ejaculation with urine cytology, the results of which we

171 nd lateral MeApd cells that express Fos with ejaculation would be retrogradely labeled.