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1 erse dialysis of 5-HT into the MPOA impaired ejaculatory ability and attenuated glutamate release.
2 evelopment of new instruments to investigate ejaculatory and orgasmic disorders.
3 formed a purely psychoanalytical approach to ejaculatory and orgasmic function into a novel multidisc
4 athophysiology, neuroscience and genetics of ejaculatory and orgasmic function will eventually lead t
5  cells plays a pivotal role in generation of ejaculatory behavior and may be part of a spinal ejacula
6  attenuates ejaculation but also affects pre-ejaculatory behavior during normal sexual activity.
7 iosus muscle, which mediates ejaculation and ejaculatory behavior, is markedly diminished in mice wit
8 saporin resulted in a complete disruption of ejaculatory behavior.
9  In males, cVA colocalizes at the tip of the ejaculatory bulb with a second acetylated hydrocarbon na
10 specific sex pheromones were detected in the ejaculatory bulb, a specialized site of pheromone produc
11 s are secreted exclusively by males from the ejaculatory bulb, transferred to females during mating,
12 ge, likely due to its expression in the male ejaculatory bulb.
13 geted to treat neurogenic and non-neurogenic ejaculatory disorders.
14                 One of these pairs, the male ejaculatory duct and female oviduct, are known to be dev
15 el of the evolution of Gld expression in the ejaculatory duct and oviduct is presented.
16 ye injection revealed a blockage between the ejaculatory duct and the urethra, which is rare in mice
17 mal aplasia) or extrinsic compression of the ejaculatory duct by Mullerian structures.
18        We surveyed the growing literature on ejaculatory duct obstruction and provide suggestions reg
19                                              Ejaculatory duct obstruction is a rare but surgically co
20                                              Ejaculatory duct obstruction is a rare cause of male inf
21 re no pathognomonic findings associated with ejaculatory duct obstruction, the diagnosis should be su
22 ricle (not in the patient with hypospadias), ejaculatory duct reflux, internal urachal sinus, and lip
23 s in males causes atresia of the homolateral ejaculatory duct that results in obstruction of the prox
24 tivates a heterologous hsp70 promoter in the ejaculatory duct, but not in other reproductive tract ti
25                                          The ejaculatory duct/oviduct enhancer retains the ability to
26 61-nt region of the SREC that is involved in ejaculatory duct/oviduct-specific expression.
27 luation of infertility in men with suspected ejaculatory-duct obstruction.
28 spond best to transurethral resection of the ejaculatory ducts (TURED).
29 n-positive areas frequently located close to ejaculatory ducts and negative for prostatic-specific an
30 n of the vas deferens, seminal vesicles, and ejaculatory ducts by calcification or fibrosis in 43 (15
31                   The steno-occlusion of the ejaculatory ducts was diagnosed, which was resolved thro
32 ctive tract (vas deferens, seminal vesicles, ejaculatory ducts).
33        Epithelial cells of prostatic glands, ejaculatory ducts, and seminal vesicles expressed E-cadh
34 cysts of the seminal vesicles, vas deferens, ejaculatory ducts, or prostate in 26 (9.4%) patients; an
35 ot detected in the basal cell layer, stroma, ejaculatory ducts, seminal vesicles, or transitional epi
36 epithelial cells of the seminal vesicles and ejaculatory ducts.
37                                              Ejaculatory dysfunction is common and may cause substant
38  an independent risk factor for erectile and ejaculatory dysfunction.
39 ation between LUTS, erectile dysfunction and ejaculatory dysfunction.
40 g") at ejaculation and forceful expulsion of ejaculatory fluid each provided sufficient mechanical st
41 sed 5-HT neural activity but the delivery of ejaculatory fluids was not.
42 ire, sexual function, including erectile and ejaculatory function, and sexual satisfaction before, du
43 % reported impairment in erectile and 22% in ejaculatory function.
44 dala was positively correlated with the post-ejaculatory interval.
45 hey had lower intromission ratios and longer ejaculatory latencies.
46        The primary endpoint was intravaginal ejaculatory latency time (IELT) measured by stopwatch.
47                             The intravaginal ejaculatory latency time remains the primary measure of
48 ing cardiovascular function, bowel motility, ejaculatory latency, and bladder control.
49   The urethrogenital (UG) reflex is a spinal ejaculatory-like reflex.
50 mental intraspinal circuitry responsible for ejaculatory-like responses and demonstrate the potential
51 rea neurons that were active during the post-ejaculatory period, but not during appetitive or consumm
52 in region strongly activated during the post-ejaculatory period.
53 e of men reporting posttreatment erectile or ejaculatory problems remained higher than baseline, alth
54 is marked by a mixture of pain, urinary, and ejaculatory symptoms with no uniformly effective therapy
55 the medial preoptic area (MPOA), reduces the ejaculatory threshold in male rats.
56  latency time remains the primary measure of ejaculatory time although increasing bother and distress
57  in normal sperm morphology (- 50%), and the ejaculatory volume (- 7.4%), indicating a progressive de