ライフサイエンスコーパス: ejection time

1 ic fluid content index, and left ventricular ejection time).

2 and 0.390+/-0.051 (P<0.023 versus beta of LV ejection time).

3 e plus isovolumic relaxation time divided by ejection time).

4 ia due to excessive prolongation of systolic ejection time.

5 of the pre-ejection period/right ventricular ejection time.

6 e find a greater than 1000-fold variation in ejection times.

7 determinations of left ventricular systolic ejection times.

8 Hg) and backward transit time normalized to ejection time (1.7; 0.4% to 3.0%) increased; Zc (-0.01;

9 1.2 versus 4.9 +/- 1.2 cm(2)/m(2)), systolic ejection times (214.0 +/- 29.4 versus 231.3 +/- 28.6 ms)

10 e mean differences were as follows: systolic ejection time 25 ms (95% CI 18-32, p<0.0001), stroke vol

11 inute(-)(1) (-13, -3; P=0.003) and increased ejection time 26 ms (2, 50; P=0.03).

12 ter backward wave transit time normalized to ejection time: -3.5; -6.5% to -0.5%), and higher wave re

13 olume relationship, and a prolonged systolic ejection time adjusted for heart rate, which reverses po

14 tion time(PAT) and PAT/PET, while (3) Aortic ejection time(AET) and velocity time integral(VTI) were

15 In both groups, systolic ejection time and ejection fraction increased after OM (

16 novel inotropic agent that prolongs systolic ejection time and increases ejection fraction through my

17 beta values of the relation between LV ejection time and LV end-diastolic volume and mass were

18 ymptomatic AS, metoprolol increases systolic ejection time and reduces aortic valve gradients, global

19 erally well tolerated, it increased systolic ejection time, and it may have improved dyspnea in the h

20 een MA peak velocity, MA displacement and LV ejection time, and LV end-diastolic volume (and mass) we

21 uce ventricular systolic velocity, elongates ejection time, and sustains stroke volume.

22 ed less severe abnormalities of the systolic ejection times, and the patients without infarction were

23 heart rate, cardiac pre-ejection period and ejection timing, aortic opening mechanics, heart rate va

24 n rate and the ratio of acceleration time to ejection time (AT/ET).

25 Right ventricular ejection time correlated with hemodynamics and survival

26 Acceleration time (AT)/ejection time (ET) ratio is a marker of aortic valve ste

27 The LV dimensions, corrected ejection time (ETc), percent fractional shortening (%FS)

28 Ejection times (ETs) correlate with stroke volumes and c

29 Thus, nascent chain ejection times from the ribosome can vary greatly betwee

30 HF myocardium, which may extend the systolic ejection time in vivo.

31 ect measurement of left ventricular systolic ejection time is a valuable adjunct in the bedside asses

32 concentration-related increases in systolic ejection time (mean increase from baseline at maximum to

33 or bacteriophage lambda, we establish a mean ejection time of roughly 5 min with significant cell-to-

34 (the first mass dimension) and also with the ejection time of the product ions, allowing the correlat

35 Correspondingly, the left ventricular rapid-ejection time of the transgenic mouse hearts was signifi

36 fect of flow rate (ratio of stroke volume to ejection time) on prognostic value of AVA <=1.0 cm(2) fo

37 lected-wave transit time to left ventricular ejection time (P < .001), which contributed to early and

38 lated increases in left ventricular systolic ejection time (p < 0.0001) and decreases in end-systolic

39 onary artery(PA) flow-velocity and pulmonary ejection time(PET) (2) Pulmonary acceleration time(PAT)

40 a high pre-ejection period/right ventricular ejection time ratio (p < .0001).

41 en the pre-ejection period/right ventricular ejection time ratio and the slope of the right ventricul

42 The right ventricular preejection period to ejection time ratio was normal in all subjects.

43 on the pre-ejection period/right ventricular ejection time ratio, the slope of the right ventricular

44 op area was determined by cardiac output, LV ejection time, tau, and early transmitral flow.

45 icular (LV) filling times and 25% shorter LV ejection times than healthy participants because of tach

46 ing sodium) reduces the pressure, increasing ejection time to 8-11 s.

47 total IVT (s/min; calculated as: 60 - [total ejection time + total filling time] ) and CO were measur

48 rdiac output, left ventricular filling time, ejection time, total isovolumic time, mitral regurgitati

49 tion-dependent increases in left ventricular ejection time (up to an 80 ms increase from baseline) an

50 Controlling the ion trap ejection time was found to result in efficient removal o

51 Right ventricular ejection time was measured from the rapid upstroke of th

52 RV ejection time was prolonged and correspondingly filling

53 tion and relaxation times were prolonged and ejection time was shortened (p < 0.001) in patients with

54 and isovolumetric relaxation time divided by ejection time, was measured from left ventricular outflo

55 ectromechanical systole and left ventricular ejection time were shortened in acute myocardial infarct

56 mal-mean area, opening and closing rates and ejection times were obtained and compared with Doppler-d

57 Left ventricular systolic ejection times were significantly higher and inversely r

58 ocity, peak velocity, acceleration time, and ejection time, were measured in each patient by investig

59 n without excessive prolongation of systolic ejection time, which may compromise diastolic filling.

60 sulting product ions are identified by their ejection time within a repeating orthogonally applied no

61 n equation (stroke volume = left ventricular ejection time x volume of electrically participating tis