ライフサイエンスコーパス: electrocardiographic

1 0% vs. 51%, p < 0.0001) and to have baseline electrocardiographic abnormalities (50% vs. 17%, p < 0.0

2 longitudinal strain impairment (P=0.005) and electrocardiographic abnormalities (long PR, complete bu

3 nary heart disease (CHD), diabetes, or major electrocardiographic abnormalities at baseline.

4 carotid intimal medial thickness, and major electrocardiographic abnormalities only modestly attenua

5 ed long QT syndrome (acLQTS), which produces electrocardiographic abnormalities that have been associ

6 Electrocardiographic abnormalities were 2-fold more comm

7 No clinically relevant laboratory or electrocardiographic abnormalities were reported.

8 s, left ventricular contractile dysfunction, electrocardiographic abnormalities).

9 vascular risk factors, severity of diabetes, electrocardiographic abnormalities, and coronary anatomy

10 njury, those with myocardial injury had more electrocardiographic abnormalities, higher inflammatory

11 from its main features; that is, lentigines, electrocardiographic abnormalities, ocular hypertelorism

12 We performed electrocardiographic activation mapping in 33 consecutiv

13 dy sought to investigate whether noninvasive electrocardiographic activation mapping is a useful meth

14 We recorded facial electromyographic and electrocardiographic activity while participants watched

15 Using a stepwise electrocardiographic algorithm, the accuracy rates for t

16 itude spectrum area values, representing the electrocardiographic amplitude frequency spectral area c

17 chest compressions to avoid artifacts during electrocardiographic analyses and to minimize the risk o

18 Electrocardiographic analyses show that older R21C mice

19 Electrocardiographic analysis of 1,959 elite male athlet

20 Electrocardiographic and 24-h Holter monitoring findings

21 Electrocardiographic and clinical parameters of 382 LQT1

22 This study investigated the prevalence and electrocardiographic and electrophysiologic characterist

23 This study investigated the prevalence and electrocardiographic and electrophysiological characteri

24 This study investigated the electrocardiographic and electrophysiological characteri

25 The electrocardiographic and electrophysiological characteri

26 The electrocardiographic and electrophysiological characteri

27 study investigated the efficacy of RFCA and electrocardiographic and electrophysiological characteri

28 Demographic data and electrocardiographic and electrophysiological findings w

29 Electrocardiographic and mapping data were collected to

30 The aim of the study was to define the electrocardiographic and morphological features of femal

31 tentially arrhythmia-inducing drug, enhanced electrocardiographic and other monitoring strategies may

32 sk stratification, specified time points for electrocardiographic and serial troponin testing within

33 n beyond ejection fraction and commonly used electrocardiographic and serum biomarkers.

34 ctions for combining risk stratification and electrocardiographic and troponin testing in an accelera

35 Clinical, electrocardiographic, and arrhythmic outcome (composite

36 Clinical, electrocardiographic, and cardiopulmonary exercise test

37 J-point elevation, the associated clinical, electrocardiographic, and echocardiographic characterist

38 Clinical, electrocardiographic, and echocardiographic data from 90

39 series of demographic, clinical, laboratory, electrocardiographic, and echocardiographic measures in

40 We sought to identify clinical, electrocardiographic, and electrophysiological features

41 Echocardiographic, electrocardiographic, and invasive hemodynamic data were

42 Detailed baseline clinical, angiographic, electrocardiographic, and revascularization data were co

43 Structural, histological, electrocardiographic, arrhythmic, and familial features

44 ach of classifying structural, histological, electrocardiographic, arrhythmic, and genetic features o

45 SAAP can effect ROSC from hemorrhage-induced electrocardiographic asystole in large swine.

46 The etiopathogenesis of electrocardiographic bundle branch and atrioventricular

47 A prospective continuous electrocardiographic (cECG) assessment was therefore per

48 ship between ischemia detected on continuous electrocardiographic (cECG) recording and cardiovascular

49 th persistent severe hypotension (68.2%) and electrocardiographic changes (56.8%).

50 The mechanisms of the electrocardiographic changes and arrhythmias in Brugada

51 by chest pain associated often with peculiar electrocardiographic changes and, at times, accompanied

52 No subject had chest pain or electrocardiographic changes during the stressor.

53 T interval in order to improve assessment of electrocardiographic changes in the treatment and preven

54 However, interpretation of electrocardiographic changes is confounded by the coinci

55 tive cardiac biomarkers and either ischaemic electrocardiographic changes or an atherosclerotic culpr

56 cute coronary syndromes but without ischemic electrocardiographic changes or an initial positive trop

57 003, presenting with chest pain and positive electrocardiographic changes or cardiac biomarkers consi

58 Patients with unstable angina and electrocardiographic changes or non-Q-wave myocardial in

59 involvement, including chest discomfort with electrocardiographic changes, acute hemodynamic instabil

60 on, reduced exercise capacity, nondiagnostic electrocardiographic changes, and balanced ischemia from

61 ar spasm (reproduction of symptoms, ischemic electrocardiographic changes, and no epicardial spasm).

62 ria including chest pain, a pericardial rub, electrocardiographic changes, and pericardial effusion.

63 infarction (MI) combines ischemic symptoms, electrocardiographic changes, and troponin rather than c

64 Hypothermia is known to result in similar electrocardiographic changes.

65 Electrocardiographic characteristics at the first clinic

66 Electrocardiographic characteristics differ depending on

67 fic injury marker, and an improvement in the electrocardiographic characteristics during the chronic

68 er pace mapping or some algorithms using the electrocardiographic characteristics less reliable.

69 urpose of this study was to evaluate whether electrocardiographic characteristics of ventricular arrh

70 There is an urgent need to identify electrocardiographic characteristics that differentiate

71 Electrocardiographic characteristics were specific for e

72 Specific electrocardiographic characteristics, including QRS morp

73 s had similar baseline echocardiographic and electrocardiographic characteristics.

74 LQTS patients relies upon a constellation of electrocardiographic, clinical, and genetic factors.

75 Baseline electrocardiographic, computed tomography, and procedura

76 LEOPARD syndrome (multiple Lentigines, Electrocardiographic conduction abnormalities, Ocular hy

77 If at least 1 parent had any electrocardiographic conduction defect or pacemaker inse

78 Here, we review the pathophysiologic and electrocardiographic correlations that inform ablation s

79 a subgroup of 113 patients with intermediate electrocardiographic criteria (QRS duration of 120-149 m

80 Several electrocardiographic criteria can help distinguish right

81 Electrocardiographic criteria used as risk predictors fo

82 from CRT, including those with intermediate electrocardiographic criteria, where CRT response is les

83 from 3 apical views add prognostic value to electrocardiographic criteria.

84 eat-to-beat analysis of up to 4 full days of electrocardiographic data per participant was performed

85 QRS morphology is a more important baseline electrocardiographic determinant of CRT response than QR

86 nd type qualitatively compared the automated electrocardiographic diagnostic statements generated by

87 Electrocardiographic early repolarization (ER) pattern h

88 Electrocardiographic (ECG) abnormalities are common in o

89 ndent prognostic value of minor and/or major electrocardiographic (ECG) abnormalities in asymptomatic

90 stematic investigations on the prevalence of electrocardiographic (ECG) abnormalities in these patien

91 The goal of this study was to define electrocardiographic (ECG) and echocardiographic charact

92 sociation between structural progression and electrocardiographic (ECG) changes in patients with ARVD

93 This study sought to examine whether electrocardiographic (ECG) changes provide prognostic in

94 aimed to assess the diagnostic properties of electrocardiographic (ECG) criteria for right ventricula

95 Current electrocardiographic (ECG) criteria for the diagnosis of

96 This study sought to identify electrocardiographic (ECG) criteria that are associated

97 reased P-wave terminal force in lead V1 , an electrocardiographic (ECG) marker of left atrial abnorma

98 nt guidelines recommend at least 24 hours of electrocardiographic (ECG) monitoring after an ischemic

99 Patients were on continuous electrocardiographic (ECG) monitoring during hospitaliza

100 Although continuous electrocardiographic (ECG) monitoring is ubiquitous in h

101 singly, this is supplemented with continuous electrocardiographic (ECG) monitoring.

102 long-term outcome and if this is additive to electrocardiographic (ECG) morphology and duration.

103 rdial diffuse fibrosis and scar with surface electrocardiographic (ECG) parameters in individuals fre

104 igate the prevalence of potentially abnormal electrocardiographic (ECG) patterns in young individuals

105 Arrhythmia or electrocardiographic (ECG) phenotypes defined by Interna

106 he purpose of this study was to establish an electrocardiographic (ECG) profile in a biracial populat

107 olonging action potential repolarisation and electrocardiographic (ECG) QT interval, associated with

108 During this time, standard vital signs, electrocardiographic (ECG) readings, and blood sample va

109 The analysis of equine electrocardiographic (ECG) recordings is complicated by

110 y sought to estimate the costs of a national electrocardiographic (ECG) screening of athletes in the

111 additional investigations are an obstacle to electrocardiographic (ECG) screening of young athletes f

112 (RR) and tidal volume (TV) from analysis of electrocardiographic (ECG) signals only.

113 It is unclear whether using fetal electrocardiographic (ECG) ST-segment analysis as an adj

114 this study was to validate existing 12-lead electrocardiographic (ECG) ST-segment elevation myocardi

115 pressure; and cardiac morbidity (defined by electrocardiographic [ECG] abnormalities).

116 t precordial leads (V1 to V3; type 1 Brugada electrocardiographic [ECG] pattern) and the presence of

117 Clinical, electrocardiographic, echocardiographic, and cardiac MRI

118 NP), and troponin I (TnI) concentrations and electrocardiographic, echocardiographic, and clinical ch

119 Electrocardiographic, echocardiographic, and serum bioma

120 or (TF) gene NKX2-5 has been associated with electrocardiographic (EKG) traits through genome-wide as

121 previous paper, we considered the different electrocardiographic elements of the early repolarizatio

122 ne transthoracic echocardiographic (TTE) and electrocardiographic evaluation during their index hospi

123 eterozygous fish manifest overt cellular and electrocardiographic evidence for delayed ventricular re

124 on myocardial infarction do not present with electrocardiographic evidence of active ischemia.

125 , both of which required CK-MB elevation and electrocardiographic evidence of permanent myocardial da

126 as documented in 40% of study patients, with electrocardiographic evidence of Q waves corresponding t

127 frequently results in an overlap in surface electrocardiographic features of ventricular arrhythmias

128 Compared with electrocardiographic findings alone, a point score based

129 ession to electromechanical dissociation and electrocardiographic findings consistent with acute hype

130 ly stable patients with LBBB who do not have electrocardiographic findings highly specific for ST-seg

131 picture of retrosternal chest pain, aided by electrocardiographic findings of ST segment deviations a

132 Echocardiographic and electrocardiographic findings were consistent with cardi

133 Electrocardiographic findings were unremarkable.

134 There were no changes in vital signs, electrocardiographic findings, or laboratory values that

135 h spontaneous or drug-induced Brugada type 1 electrocardiographic findings, who underwent ICD implant

136 ng history, serum creatine phosphokinase, or electrocardiographic findings.

137 minimized scan range, heart rate reduction, electrocardiographic-gated tube current modulation, and

138 omputed tomography and 15 studies (79%) were electrocardiographic-gated.

139 computed tomography (CT) with retrospective electrocardiographic gating (one examination per patient

140 Electrocardiographic gating is increasingly used for (82

141 The validity of electrocardiographic gating using (82)Rb for assessment

142 ion was used in 104 (97.2%), and prospective electrocardiographic gating was used in 106 (99.1%).

143 rasonography at 8000 frames per second (with electrocardiographic gating).

144 Patients underwent 48-h electrocardiographic Holter monitoring quarterly to dete

145 ients with a broad spectrum of disease using electrocardiographic imaging (a method for noninvasive c

146 Noninvasive electrocardiographic imaging (ECGi) is used clinically t

147 sequences of AF in humans using noninvasive electrocardiographic imaging (ECGI).

148 Electrocardiographic imaging accurately identifies areas

149 The results suggest a potential role for electrocardiographic imaging and late gadolinium enhance

150 invasive mapping of cardiac arrhythmias with electrocardiographic imaging and noninvasive delivery of

151 Electrocardiographic imaging colocalizes the epicardial

152 Electrocardiographic imaging constructs epicardial elect

153 Electrocardiographic imaging depicted salient features o

154 ombining anatomical imaging with noninvasive electrocardiographic imaging during ventricular tachycar

155 Electrocardiographic imaging is a noninvasive method for

156 Nevertheless, raw electrocardiographic imaging phase maps presented reentr

157 AND EGM, body surface potential mapping, and electrocardiographic imaging phase maps were obtained fr

158 Electrocardiographic imaging reveals electrophysiologica

159 m (EGM), body surface potential mapping, and electrocardiographic imaging signals.

160 This review summarizes results from electrocardiographic imaging studies of arrhythmogenic s

161 Noninvasive electrocardiographic imaging was used before and after 1

162 Here, we use a noninvasive imaging modality (electrocardiographic imaging) to study normal activation

163 Electrocardiographic imaging, a noninvasive functional E

164 t activity in AF from the body surface using electrocardiographic imaging, calibrated to panoramic in

165 In electrocardiographic imaging, HDF filtering allowed to i

166 enerated phase maps and activation maps from electrocardiographic imaging-reconstructed epicardial un

167 Reduction of electrocardiographic left ventricular hypertrophy (LVH)

168 Although the presence and severity of electrocardiographic left ventricular hypertrophy (LVH)

169 evaluated in 8831 hypertensive patients with electrocardiographic left ventricular hypertrophy with n

170 index, smoking, valvular disease, diabetes, electrocardiographic left ventricular hypertrophy, hyper

171 ned phenotype testing did not identify novel electrocardiographic loci unapparent using traditional u

172 Change in Cornell product electrocardiographic LVH between baseline and in-study e

173 n, aged 55-80 years (median, 67 years), with electrocardiographic LVH by Cornell voltage-duration pro

174 New-onset AF in relation to electrocardiographic LVH determined at baseline and subs

175 Reduction in Cornell product electrocardiographic LVH during antihypertensive therapy

176 Lower Cornell product electrocardiographic LVH during antihypertensive therapy

177 ict development of AF, whether regression of electrocardiographic LVH is associated with a decreased

178 However, whether reduction of electrocardiographic LVH is associated with decreased he

179 rapy targeted at regression or prevention of electrocardiographic LVH may reduce the incidence of new

180 Use of electrocardiographic LVH to select patients may have inc

181 Electrocardiographic LVH was measured using sex-adjusted

182 ent blood pressure, and baseline severity of electrocardiographic LVH, in-treatment decrease of Corne

183 ent blood pressure, and baseline severity of electrocardiographic LVH, lower in-treatment Cornell pro

184 In order to more clearly understand the electrocardiographic manifestations of early transmural

185 Using an electrocardiographic mapping (ECM) system in 27 patients

186 l, noninvasive, beat-by-beat mapping system, Electrocardiographic Mapping (ECM), in facilitating the

187 entricular electrical uncoupling measured by electrocardiographic mapping predicted clinical CRT resp

188 patients using a noninvasive multielectrode electrocardiographic mapping technique.

189 ive imaging techniques based on body surface electrocardiographic mapping to elucidate the mechanisms

190 Electrocardiographic maps revealed homogeneous patterns

191 atrial physiology and anatomy, chemical and electrocardiographic markers) and other competing clinic

192 ical dyssynchrony compared with conventional electrocardiographic markers.

193 The QT interval, an electrocardiographic measure reflecting myocardial repol

194 The V(2) transition ratio is a novel electrocardiographic measure that reliably distinguishes

195 The P-wave duration (PWD) is an electrocardiographic measurement that represents cardiac

196 In individual subjects, 1,256 +/- 220 electrocardiographic measurements of QT, JTp, and JT50 i

197 ations between usual dietary fish intake and electrocardiographic measures of heart rate, atrioventri

198 iors and estimated arousal fluctuations from electrocardiographic measures of heart rate.

199 ation pattern were reviewed to delineate the electrocardiographic measures to be used when defining t

200 phenotypes relating to metabolic traits and electrocardiographic measures, along with another 8 prev

201 cardiac mode at four pitch values and three electrocardiographic modulation temporal windows.

202 be initiated or reinitiated with continuous electrocardiographic monitoring and in the presence of t

203 The clinical utility of routine electrocardiographic monitoring following percutaneous c

204 ommendations on indications and duration for electrocardiographic monitoring in accordance with the A

205 SCAF is frequently detected by continuous electrocardiographic monitoring in older patients withou

206 Thus, electrocardiographic monitoring is required to minimize

207 commendations for Indication and Duration of Electrocardiographic Monitoring presented by patient pop

208 Long-term continuous electrocardiographic monitoring shows a substantial prev

209 (AT), verified by monthly visits, ambulatory electrocardiographic monitoring, and implantable loop re

210 echocardiography, optical mapping, telemetry electrocardiographic monitoring, and inducibility studie

211 5 days after surgery, as assessed by Holter electrocardiographic monitoring, and myocardial injury w

212 with the use of echocardiography, ambulatory electrocardiographic monitoring, exercise stress testing

213 on formulas should be considered for routine electrocardiographic monitoring.

214 in the intensive care unit under continuous electrocardiographic monitoring.

215 We implanted subcutaneous electrocardiographic monitors (St. Jude CONFIRM-AF) in p

216 ate, ST segment behavior, and arrhythmias in electrocardiographic monitors may lead to inappropriate

217 Patients were followed with implanted electrocardiographic monitors when possible (85.2% of FI

218 disease, low T1 values correlate with early electrocardiographic, morphological cardiac changes, and

219 We compared the electrocardiographic morphology of ventricular tachycard

220 In 18 patients, the 12-lead electrocardiographic morphology was left bundle branch b

221 were ICU mortality, readmission to ICU, new electrocardiographic or cardiac enzyme changes suggestiv

222 r fibrillation (VF) and sudden death without electrocardiographic or echocardiographic abnormalities

223 f abnormalities were observed in laboratory, electrocardiographic, or Holter monitoring assessments.

224 at clinically relevant levels) prolonged the electrocardiographic P wave and increased susceptibility

225 Electrocardiographic P-wave duration agreed with the dur

226 ardiogram was recorded through defibrillator/electrocardiographic pads oriented in the standard cardi

227 tive prognostic value to routine clinical or electrocardiographic parameters (P<0.001).

228 ks models evaluated associations between GEH electrocardiographic parameters and SCD.

229 is study sought to analyze the usefulness of electrocardiographic parameters as markers of sudden car

230 ors, presence of coronary heart disease, and electrocardiographic parameters as time-varying factors.

231 entricular function, heart ultrastructure or electrocardiographic parameters except for slower heart

232 ween dietary fish intake and several cardiac electrocardiographic parameters in humans relevant to ar

233 Electrocardiographic parameters of sympathetic nervous s

234 Electrocardiographic parameters were measured from the s

235 sence of diabetes mellitus, body mass index, electrocardiographic parameters, B-type natriuretic pept

236 deled the effects of ion channel activity on electrocardiographic parameters, estimating the change i

237 ciation studies conducted in recent years on electrocardiographic parameters, highlighting their pote

238 Electrocardiographic parameters, including QT intervals,

239 oung patients with the Wolff-Parkinson-White electrocardiographic pattern are scarce.

240 by using the Valsalva maneuver to reveal the electrocardiographic pattern in family members.

241 a defibrillator (CRT-D) in patients with an electrocardiographic pattern showing left bundle-branch

242 This electrocardiographic pattern was considered benign until

243 PP1 did not change infarct size, electrocardiographic pattern, or cardiac function.

244 It discusses the electrocardiographic phenomenon of T-wave alternans (TWA

245 cell, 2D, and 3D) accurately reproduced the electrocardiographic phenotype of the proband, including

246 Early repolarization (ER), a common electrocardiographic phenotype, has been associated with

247 The electrocardiographic PR interval increases with aging, d

248 The electrocardiographic PR interval reflects atrioventricul

249 notypes: higher resting heart rate (HR), the electrocardiographic PR interval, atrial fibrillation an

250 Prolongation of the electrocardiographic PR interval, known as first-degree

251 In RV and biventricular disease, electrocardiographic preceded imaging features, whereas

252 ividuals from a primary care population with electrocardiographic preexcitation.

253 The electrocardiographic QRS duration, a measure of ventricu

254 Prolongation of the electrocardiographic QT interval is a risk factor for su

255 Electrocardiographic QT interval prolongation is the mos

256 Prolongation of the electrocardiographic QT interval, a measure of cardiac r

257 Extremes of the electrocardiographic QT interval, a measure of cardiac r

258 eterminant of arrhythmia, as measured by the electrocardiographic QT interval.

259 cal hypokalaemia is associated with acquired electrocardiographic QT prolongation and arrhythmic acti

260 nitrogen and creatinine levels and a longer electrocardiographic QTc interval than did the sham grou

261 y letters outlining the phenotype (prolonged electrocardiographic QTc interval).

262 e intensive care unit is a relatively common electrocardiographic reading both by standard interpreta

263 tion and history, the characteristics of the electrocardiographic recording at rest and during exerci

264 33a levels increased QT intervals in surface electrocardiographic recordings and action potential dur

265 iability was calculated in 5-min sections of electrocardiographic recordings at baseline and 4 hrs af

266 Heart period variability was analyzed from electrocardiographic recordings collected from 159 preho

267 or characterization of non-linear aspects of electrocardiographic recordings.

268 (10-15 mg/dL) clamps for 3 h with continuous electrocardiographic recordings.

269 changes in heart rate variability (HRV) and electrocardiographic repolarization changes measured bef

270 value over traditional risk factors, stress electrocardiographic, rest echocardiographic, and SE var

271 ardiopulmonary arrests regardless of initial electrocardiographic rhythm with return of spontaneous c

272 g debate about the role of pre-participation electrocardiographic screening for the prevention of sud

273 Adding electrocardiographic screening is being considered by so

274 ive prognostic value to routine clinical and electrocardiographic selection criteria for cardiac resy

275 uce, from digital files, the original analog electrocardiographic signals of previously instrumented

276 a convolutional neural network to detect the electrocardiographic signature of atrial fibrillation pr

277 onin-based criteria were less likely to have electrocardiographic ST-segment elevation and had better

278 the final study population of 46 cases with electrocardiographic ST-segment elevation myocardial inf

279 it patients who present with chest pain, the electrocardiographic ST-segment elevation myocardial inf

280 roposed features of LD disease, encompassing electrocardiographic, structural, histological, and arrh

281 To facilitate the precision of clinical electrocardiographic studies of J-to-Tpeak (JTp) and Tpe

282 The genesis of the electrocardiographic T wave is incompletely understood a

283 on studies have found an association between electrocardiographic T-wave morphology parameters and ca

284 We assessed the predictive value of electrocardiographic T-wave morphology parameters and TP

285 Electrocardiographic T-wave morphology parameters descri

286 up studies assessing the association between electrocardiographic T-wave peak to T-wave end interval

287 A) reflects beat-to-beat fluctuations in the electrocardiographic T-wave, and is associated with disp

288 rried out a genome-wide association study of electrocardiographic time intervals in 6,543 Indian Asia

289 one-third as many participants as published electrocardiographic trait genome-wide association studi

290 We examined how expanding electrocardiographic trait genome-wide association studi

291 llumina HumanCVD Beadchip and 4 quantitative electrocardiographic traits (PR interval, QRS axis, QRS

292 Electrocardiographic traits are important, substantially

293 ated genome-wide association data for AF and electrocardiographic traits to link disease-related vari

294 d univariate results across the 6 continuous electrocardiographic traits using the combined phenotype

295 s genome-wide association study findings for electrocardiographic traits, while the expression analys

296 luating the contiguous but not the composite electrocardiographic traits.

297 hythmia suppression is essential for optimal electrocardiographic triggering and image acquisition.

298 for ARVC when applied to athletes exhibiting electrocardiographic TWI and to identify discriminators

299 The mother had electrocardiographic U-wave changes consistent with Ande

300 Electrocardiographic variables that significantly increa