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1 n (both experimentally and clinically, as in electroconvulsive therapy).
2 patients hospitalized preoperatively (e.g., electroconvulsive therapy).
3 ties, psychotherapy, psychotropic drugs, and electroconvulsive therapy.
4 network aberrations following risperidone or electroconvulsive therapy.
5 effectively treated with antidepressants and electroconvulsive therapy.
6 order and resistant to other medications and electroconvulsive therapy.
7 consultation and treatment changes including electroconvulsive therapy.
8 itive or behavioural therapy), and sometimes electroconvulsive therapy.
9 ulation, a well-established animal model for electroconvulsive therapy.
10 nders, and clozapine nonresponders following electroconvulsive therapy.
11 categories: (1) seizure therapies, including electroconvulsive therapy and magnetic seizure therapy,
12 thin days or a few weeks), such as ketamine, electroconvulsive therapy and non-invasive neurostimulat
13 metabolism, which can explain the effects of electroconvulsive therapy and the beneficial and side ef
14 arbamazepine, oxcarbazepine, antipsychotics, electroconvulsive therapy and various light intervention
15 stroke and epileptic seizures, and following electroconvulsive therapy, and AHN is diminished in neur
16 zodiazepines such as lorazepam and diazepam, electroconvulsive therapy, and N-methyl-D-aspartate anta
17 ceptor modulators (ketamine, D-cycloserine), electroconvulsive therapy, and transcranial brain stimul
18 t of catatonia relies on benzodiazepines and electroconvulsive therapy-both effective, yet unspecific
19 eased in rodent brains by antidepressants or electroconvulsive therapy, but decreased in an animal mo
21 apy, ketogenic diet, emergency neurosurgery, electroconvulsive therapy, cerebrospinal fluid drainage,
22 l nerve stimulation, deep brain stimulation, electroconvulsive therapy, drainage of the cerebrospinal
25 itive side effects of right unilateral (RUL) electroconvulsive therapy (ECT) are reported to depend o
26 investigate if the neuronal underpinnings of electroconvulsive therapy (ECT) are similarly associated
31 fficacy of right unilateral ultrabrief pulse electroconvulsive therapy (ECT) combined with venlafaxin
32 mmendations to tighten federal regulation of electroconvulsive therapy (ECT) devices have focused att
34 de SNP genotyping of 2725 cases who received electroconvulsive therapy (ECT) for a major depressive e
35 TMS) has been reported to be as effective as electroconvulsive therapy (ECT) for major depression.
36 nd ethical controversy concerning the use of electroconvulsive therapy (ECT) for psychiatric disorder
37 and individualizing the current amplitude in electroconvulsive therapy (ECT) has been proposed as a m
43 istance is the leading indication for use of electroconvulsive therapy (ECT) in major depression.
65 fying biomarkers associated with response to electroconvulsive therapy (ECT) may aid clinical decisio
66 observational study examined the effects of electroconvulsive therapy (ECT) on suicide and all-cause
67 ic medication but that responded promptly to electroconvulsive therapy (ECT) on two separate occasion
70 of RSFC patterns associated with response to electroconvulsive therapy (ECT) remain limited, and focu
73 ng to the rapid and robust clinical effects, electroconvulsive therapy (ECT) represents an optimal mo
75 and default mode networks, while studies of electroconvulsive therapy (ECT) were restricted mainly t
77 persistent cognitive defects associated with electroconvulsive therapy (ECT), a highly effective and
78 Effects of antidepressant medication and electroconvulsive therapy (ECT), and other potential mod
79 for depression, receipt of antidepressants, electroconvulsive therapy (ECT), and suicide, as well as
80 mulation (ECS), the experimental analogue of electroconvulsive therapy (ECT), has been shown to produ
82 ponse rate versus placebo or sham treatment: electroconvulsive therapy (ECT), minocycline, theta-burs
85 e shock (ECS), which is the animal analog of electroconvulsive therapy (ECT), the most effective shor
86 severely depressed patients before and after electroconvulsive therapy (ECT), we measured cerebrospin
89 was treated with a five-treatment course of electroconvulsive therapy (ECT), which resulted in a com
95 most persistent cognitive adverse effect of electroconvulsive therapy (ECT); however, it is not know
96 therapy with ultra-brief pulse continuation electroconvulsive therapy (ECT-arm), or no add-on therap
97 reatment, hospital admission for depression, electroconvulsive therapy, emergency department visit fo
98 being evaluated as a possible alternative to electroconvulsive therapy for the treatment of refractor
99 cal agents were effective for depression and electroconvulsive therapy for treatment-resistant bipola
100 ranscranial magnetic stimulation (instead of electroconvulsive therapy) for drug-resistant major depr
101 lar disorder, as well as antidepressants and electroconvulsive therapy have recently been shown to ac
103 mptoms; 2) treatment response to lithium and electroconvulsive therapy is excellent; 3) in half of th
110 uently used device (10 studies), followed by electroconvulsive therapy (one study) and direct-current
113 to explore other therapeutic options such as electroconvulsive therapy, transcranial magnetic stimula
115 ticholinergic exposure, lifetime exposure to electroconvulsive therapy, vascular illness, and incipie