ライフサイエンスコーパス: electroretinogram

1 ement of retinal function as measured by the electroretinogram.

2 .9 +/- 5.6 to 45.8 +/- 15.2 muV for photopic electroretinogram.

3 eld photopic negative response (PhNR) of the electroretinogram.

4 asis for cone dystrophy with supernormal rod electroretinogram.

5 s and decreased retinal function measured by electroretinogram.

6 that was reflected in the slow decay of the electroretinogram.

7 ing S- and M-opsins and a preserved photopic electroretinogram.

8 g in abnormal visual function as measured by electroretinogram.

9 ons to this generalization, for example, the electroretinogram.

10 ear identical derived rod responses from the electroretinogram.

11 tern blot analysis, histologic analysis, and electroretinogram.

12 demonstrated in rAAV.sFLT-1-treated eyes by electroretinogram.

13 unction can be assessed using the multifocal electroretinogram.

14 electro-oculogram light rise, and a reduced electroretinogram.

15 syndrome (SARDS) exhibiting a nonrecordable electroretinogram.

16 eservation of visual function as measured by electroretinogram.

17 and increased the amplitude of the photopic electroretinogram.

18 pharmacological treatment through multifocal-electroretinogram.

19 d increased a-wave amplitude of the scotopic electroretinogram.

20 at reported for visual fields and full-field electroretinograms.

21 educed amplitudes and prolonged latencies in electroretinograms.

22 s to estimates of rod function from in vitro electroretinograms.

23 ones, concomitant with reduced cone-mediated electroretinograms.

24 y ranges from structural defects to abnormal electroretinograms.

25 plete loss of visual function as measured by electroretinograms.

26 compromised maximal voltage response in the electroretinograms.

27 aturated fatty acids and severely attenuated electroretinograms.

28 change in the a and b waves of bright-flash electroretinograms.

29 Analysis of the electroretinogram a-wave indicates that rescued rod cell

30 and Rs1-KO mice have early reduction in the electroretinogram a-wave.

31 Electroretinogram abnormalities observed in b-wave and o

32 electron microscopy, rhodopsin measurement, electroretinogram activity, and visual acuity, indicatin

33 Retinal function measured by electroretinogram after light exposure was also delayed

34 95% confidence interval [CI] 5.6-10.6), cone electroretinogram amplitude declined exponentially at a

35 mplitudes and implicit times, and multifocal electroretinogram amplitude distribution.

36 USH2A patients lose visual field and cone electroretinogram amplitude faster than patients with RH

37 hout the study duration and exhibited higher electroretinogram amplitude, thicker photoreceptor layer

38 ite test light), and 30-Hz (cone) full-field electroretinogram amplitude.

39 y, 7.0% for visual field area, and 13.2% for electroretinogram amplitude.

40 nd tritan thresholds, pattern and full-field electroretinogram amplitudes and implicit times, and mul

41 nths of pioglitazone significantly increased electroretinogram amplitudes in type 2 diabetic obese ra

42 The electroretinogram amplitudes of double heterozygotes are

43 onsyndromic USH2A patients had 30 Hz-flicker electroretinogram amplitudes that were significantly hig

44 isplay scattered OS disorganization, reduced electroretinogram amplitudes, and progressive photorecep

45 tor apoptosis and to a sustained increase in electroretinogram amplitudes.

46 Electroretinogram analyses revealed reduced cone light r

47 Electroretinogram analyses showed decreased B-wave and o

48 function was evaluated 8 weeks later by the electroretinogram and compared with photoreceptor cell l

49 While case 1 had an undetectable electroretinogram and features supporting a diagnosis of

50 ed retinal stimulus processing by full-field electroretinogram and found impaired photoreceptor funct

51 9.4 +/- 4.6 to 57.6 +/- 8.8 muV for scotopic electroretinogram and from 10.9 +/- 5.6 to 45.8 +/- 15.2

52 umbers of degenerate capillaries, as well as electroretinogram and heart morphology.

53 litazone's effects on retinal function using electroretinogram and markers of apoptosis.

54 Vision was assessed using the electroretinogram and optokinetic response and retinal m

55 resulting in complete absence of a photopic electroretinogram and progressive cone degeneration.

56 Electroretinogram and visual evoked potential tests show

57 Electroretinograms and behavioral tests showed decreased

58 Electroretinograms and electro-oculograms were recorded

59 To measure changes in rod physiology, electroretinograms and intracellular Ca(2+) recording we

60 attenuation of visual function with abnormal electroretinograms and reduced retinal rhodopsin levels.

61 dly disrupts retinal function as assessed by electroretinograms and vision as assessed by optomotor b

62 ation of Mfn2 and Mlh1 and retinal function (electroretinogram), and the retinopathy continued to pro

63 Intraocular pressure (IOP), pattern electroretinogram, and optical coherence tomography meas

64 ity, automated visual field testing, pattern electroretinogram, and spectral-domain optical coherence

65 onal data were obtained using the full-field electroretinogram, and static or kinetic perimetry.

66 receptor potential (ERP), a component of the electroretinogram arising from photoisomerization-induce

67 The pattern electroretinogram assesses retinal ganglion cell functio

68 All eyes underwent standard full-field electroretinogram at baseline and 8 weeks after surgery.

69 ight adaptation, as evidenced by an impaired electroretinogram b-wave from cones, whereas a dopamine

70 RGS11 produced delays in the ON-BPC-derived electroretinogram b-wave, but no changes in the photorec

71 photopsias, and a selective reduction of the electroretinogram b-wave.

72 ice is associated with an attenuation of the electroretinogram b-wave.

73 dystroglycan was sufficient to attenuate the electroretinogram b-wave.

74 nsmission as indicated by a reduction in the electroretinogram b-wave.

75 sing homozygous Nmnat1(V9M) mutant mice, the electroretinogram becomes undetectable and the pupillary

76 oducible and reversible changes in the flash electroretinogram between daylight and mel-low.

77 Multifocal electroretinogram can also help in early screening of fo

78 ce restricted peripheral vision and scotopic electroretinogram confirmed the diagnosis of retinitis p

79 as indicated by the loss of ON transients in electroretinograms, consistent with a neurotransmission

80 ice showed nonprogressive b-wave deficits on electroretinograms, consistent with compromised BP cell

81 Based on single-photoreceptor recordings, electroretinograms, cortical recordings, and visual beha

82 while Prph2Y/+ animals exhibited a cone-rod electroretinogram defect, Prph2Y/+/Rom1-/- animals displ

83 racteristic of RPE and choroidal defects and electroretinogram defects.

84 Electroretinograms demonstrated that affected mice were

85 Electroretinograms demonstrated that the organ responds

86 nd/or macular pigment mottling, and abnormal electroretinograms demonstrating mixed cone and rod dysf

87 retinal function, measured by the multifocal electroretinogram, differs between males and females wit

88 ch as early visual impairment as assessed by electroretinogram, disorganization of lamination and api

89 g with retinal function as determined by the electroretinogram (ERG) a- and b-waves.

90 As a group, CSNB1 patients have a normal electroretinogram (ERG) a-wave, indicative of photorecep

91 d and cone function declines, accompanied by electroretinogram (ERG) abnormalities.

92 erited retinal disorders with characteristic electroretinogram (ERG) abnormalities.

93 rea (V4e white test light) and in full-field electroretinogram (ERG) amplitudes to 0.5- and 30-Hz whi

94 expression, endogenous retinoid levels, and electroretinogram (ERG) analyses were performed on Lrat(

95 Morphometric and electroretinogram (ERG) analyses were used to assess the

96 Microperimetry 1 (MP-1) mapping and electroretinogram (ERG) analysis were performed on the p

97 uter nuclear layer (ONL) and functionally by electroretinogram (ERG) analysis, 5 to 7 days after expo

98 Electroretinogram (ERG) and optical coherence tomography

99 Recordings of the electroretinogram (ERG) and spiking activity of single r

100 n the etiology of psychiatric disorders, and electroretinogram (ERG) anomalies have been reported in

101 Electroretinogram (ERG) anomalies occur in patients with

102 ess, in which the a- and b-wave responses of electroretinogram (ERG) are abolished.

103 n optical coherence tomography (SD-OCT), and electroretinogram (ERG) assessment.

104 Electroretinogram (ERG) b-wave implicit time in young RG

105 In the Gbeta5(-/-) mouse, the electroretinogram (ERG) b-wave is absent, and the R7 sub

106 e impairment of night vision, absence of the electroretinogram (ERG) b-wave, and variable degrees of

107 sensitivity and a selective reduction of the electroretinogram (ERG) b-wave.

108 Amplitude reduction of >50% in the relevant electroretinogram (ERG) component or a peak time shift o

109 mune retinopathy based on clinical features, electroretinogram (ERG) findings, and serum antiretinal

110 Goldmann perimetry, and full-field standard electroretinogram (ERG) from all patients were registere

111 s were classified on the basis of full-field electroretinogram (ERG) Fundus autofluorescence (FAF) an

112 The electroretinogram (ERG) has proved to be a valuable tool

113 wed nonspecific retinal inflammation, and an electroretinogram (ERG) illustrated decreased amplitude

114 the impact of KCNJ10 mutations on the human electroretinogram (ERG) in four unrelated patients with

115 The electroretinogram (ERG) is a non-invasive method used to

116 Electroretinogram (ERG) measurements were used to assay

117 and photopic negative response (PhNR) of the electroretinogram (ERG) noninvasively with an electrode

118 The electroretinogram (ERG) of the super p53 mouse exhibited

119 The electroretinogram (ERG) of TRPM1-deficient (TRPM1(-/-))

120 Multifocal electroretinogram (ERG) or VEP can provide an objective

121 Recordings of electroretinogram (ERG) oscillatory potentials and scoto

122 The visual symptoms and electroretinogram (ERG) phenotype characteristic of MAR

123 ouse, which lacks GPR179 and has a no b-wave electroretinogram (ERG) phenotype, to demonstrate that d

124 disrupts signal transduction and reveals an electroretinogram (ERG) phenotype.

125 To evaluate the diagnostic potential of the electroretinogram (ERG) photostress test and the focal c

126 Scotopic electroretinogram (ERG) recording was used to investigat

127 S males underwent ophthalmic examination and electroretinogram (ERG) recording.

128 (OCT), fundus autofluorescence imaging, and electroretinogram (ERG) recording.

129 The bright flash response in electroretinogram (ERG) recordings recovered quickly in

130 Electroretinogram (ERG) response amplitudes were recorde

131 eal injection (IVI) of 25 pmol MTX increased electroretinogram (ERG) response and rhodopsin level in

132 localization of cone opsin, loss of photopic electroretinogram (ERG) responses and loss of cone cells

133 Electroretinogram (ERG) responses to a 1.8-log-unit rang

134 Electroretinogram (ERG) responses were obtained from ten

135 Electroretinogram (ERG) responses were recorded in both

136 Cone electroretinogram (ERG) responses were reduced in Irbp(-

137 y biochemically, by recording single rod and electroretinogram (ERG) responses, by intracellular free

138 An electroretinogram (ERG) screen identified a mouse with a

139 The electroretinogram (ERG) undergoes parallel changes.

140 e known to contribute to the mammalian flash electroretinogram (ERG) via activity of third-order reti

141 An electroretinogram (ERG) was done at baseline and thereaf

142 The electroretinogram (ERG) was measured prior to ischemia a

143 A standard electroretinogram (ERG) was obtained before injection an

144 Electroretinogram (ERG) was recorded from MPS IIIB and w

145 group were investigated by monocular pattern electroretinogram (ERG), L&M (long and medium wavelength

146 assessed by restoration of the cone-mediated electroretinogram (ERG), optomotor responses, and cone o

147 d examining gene message and protein levels, electroretinogram (ERG), retinal morphology and ultrastr

148 ent in normal numbers, and the a-wave of the electroretinogram (ERG)--reflecting their physiological

149 , brain, and male reproductive organs and by electroretinogram (ERG)-based studies of the retina and

150 and luminance specific signals in the human electroretinogram (ERG).

151 asuring outer nuclear layer thickness and by electroretinogram (ERG).

152 re central macular dysfunction on multifocal electroretinogram (ERG).

153 to a decline in visual acuity as detected by electroretinogram (ERG).

154 retinal light response was measured with the electroretinogram (ERG).

155 delayed falling phase of photopic b-wave of electroretinogram (ERG).

156 utcome was a change in the parameters of the electroretinogram (ERG).

157 involuntary eye movement, and nonrecordable electroretinogram (ERG).

158 ing Gng13(-/-) knockout (KO) mice, recording electroretinograms (ERG) and performing immunocytochemic

159 in the retina, and the scotopic and photopic electroretinograms (ERG) and retinal morphology in wild-

160 athy were recruited, and photopic full-field electroretinograms (ERG) were performed at baseline and

161 no adverse effects on the eye as assessed by electroretinograms (ERG), corneal and retinal tomography

162 -digital sign and markedly reduced or absent electroretinograms (ERG).

163 sual acuity, constricted fields, and reduced electroretinograms (ERGs) 5 years before death correlate

164 Bsg(-/-) mice have severely reduced electroretinograms (ERGs) and progressive photoreceptor

165 These mice had normal fundi, electroretinograms (ERGs) and retinal histology at 6 mon

166 Electroretinograms (ERGs) can detect the associated ligh

167 Weekly electroretinograms (ERGs) followed by retinal histology

168 The electroretinograms (ERGs) of both eyes from all rats wer

169 nction was assessed with serial dark-adapted electroretinograms (ERGs) optimized for detection of the

170 However, we find further variability in cone electroretinograms (ERGs) ranging from normal to absent

171 In addition, electroretinograms (ERGs) recorded in the same groups we

172 Electroretinograms (ERGs) were measured to assess retina

173 Scotopic electroretinograms (ERGs) were performed 3, 7, and 14 da

174 Whole-field electroretinograms (ERGs) were performed on dynein morph

175 Electroretinograms (ERGs) were performed on transplanted

176 Electroretinograms (ERGs) were recorded 7 weeks after th

177 Electroretinograms (ERGs) were recorded and the retinas

178 Full-field scotopic electroretinograms (ERGs) were recorded from 44 male hem

179 Full-field electroretinograms (ERGs) were recorded from wild-type (

180 Electroretinograms (ERGs) were recorded to measure outer

181 ual acuity loss early in life, nondetectable electroretinograms (ERGs), and little or no detectable v

182 On the basis of standard electroretinograms (ERGs), patients were diagnosed with

183 ver, each patient had recordable and similar electroretinograms (ERGs), which demonstrated absent con

184 heir respective cellular contributions using electroretinograms (ERGs).

185 erturbed retinal function as demonstrated by electroretinograms (ERGs).

186 common cause of inherited PD) on Drosophila electroretinograms (ERGs).

187 evaluated by retinal histologic analyses and electroretinograms (ERGs).

188 ith dark-adapted electroretinograms (monthly electroretinograms [ERGs]), and cataract formation with

189 The photopic flash electroretinogram (FERG) and visual evoked potential (FV

190 uded kinetic widefield perimetry, full-field electroretinogram (ffERG), and visual acuity (VA).

191 ative autofluorescence (qAF), and full-field electroretinogram (ffERG).

192 n kinetic visual field (GVF), and full-field electroretinogram (ffERG).

193 At end of the study, electroretinogram findings, retinal ganglion cell (RGC)

194 ng the development of ametropia based on the electroretinogram findings.

195 lides or through physiological testing using electroretinogram flicker photometry.

196 fect on the b-wave was confirmed by in vitro electroretinograms from the inner retina.

197 Unlike visual fields and electroretinograms, however, the repeat variability is l

198 e were no other causes of an electronegative electroretinogram identified in any of the affected pati

199 Full-field electroretinogram identified mildly reduced scotopic and

200 unction as determined by almost extinguished electroretinogram in 2 of 3 siblings.

201 o sign of vitreous inflammation and abnormal electroretinogram in at least 1 eye), and a negative fam

202 recording electrodes; the maturation of the electroretinogram in preterm infants and in the first ye

203 s the modifications needed for performing an electroretinogram in young children.

204 cal modifications for performing a pediatric electroretinogram, including the possible need for sedat

205 red at 4 weeks of age, although a full-field electroretinogram indicated a visual response was still

206 Pde6d(-/-) scotopic paired-flash electroretinograms indicated a delay in recovery of the

207 Differences in the electroretinograms, intraocular pressure, and histopatho

208 The pattern electroretinogram is a noninvasive, direct, objective me

209 The electroretinogram is an essential tool in the evaluation

210 The pattern electroretinogram may also optimize treatment strategies

211 Electroretinogram measurements of the frequency response

212 croscopy parameters, retina cell counts, and electroretinogram measurements.

213 ity (BCVA), foveal threshold, and multifocal electroretinogram (mfERG) amplitude and timing.

214 functional recovery evidenced by multifocal-electroretinogram (mfERG) and microperimetry (MP1) after

215 The multifocal electroretinogram (mfERG) can provide objective corrobor

216 nship between DE development and: multifocal electroretinogram (mfERG) implicit time (IT) Z-score, mf

217 Local first-order multifocal electroretinogram (mfERG) implicit time (K1-IT) delays h

218 Multifocal electroretinogram (mfERG) provides evidence of focal ret

219 l parameters of surgical success, multifocal electroretinogram (mfERG), and histopathologic analyses

220 ithout clinical retinopathy using multifocal electroretinogram (mfERG).

221 charts, 10-2 visual fields (VFs), multifocal electroretinograms (mfERG), and spectral-domain optical

222 Photopic and scotopic multifocal electroretinograms (mfERGs) were recorded.

223 sitivity, retinal function with dark-adapted electroretinograms (monthly electroretinograms [ERGs]),

224 Although the technique for performing an electroretinogram must be modified for young children, w

225 ematic forward genetic Drosophila screen for electroretinogram mutants lacking synaptic transients id

226 statistically significant differences in the electroretinograms obtained between the linezolid-inject

227 We found that the electroretinogram of Gbeta5-/- mice lacks the b-wave com

228 Contrary to expectation, electroretinograms of CFH(-/-).C3(-/-) mice displayed mo

229 at flash responses measured by trans-retinal electroretinogram or single-cell suction electrode recor

230 croscopy parameters, retina cell counts, and electroretinogram parameters were compared between the g

231 Variants of the electroretinogram (pattern ERG and long-pulse ERG) were

232 Flash electroretinograms performed at 2, 7, and 10 months of a

233 mice were longitudinally tested with pattern electroretinogram (PERG) and spectral-domain optical coh

234 Pattern electroretinogram (PERG) and visual evoked potentials (V

235 eliability, and dynamic range of the pattern electroretinogram (PERG) as a tool to monitor progressiv

236 ical response of RGC was measured by pattern electroretinogram (PERG) in 43 C57BL/6J mice 4 to 6 mont

237 optical coherence tomography (OCT), pattern electroretinogram (PERG) or frequency-doubling technolog

238 IOP and pattern electroretinogram (PERG) were sequentially measured with

239 ommon inbred mouse strains using the pattern electroretinogram (PERG), a sensitive measure of RGC fun

240 avioral abnormalities was made using pattern electroretinogram (PERG), magnetic resonance imaging (MR

241 d structure were evaluated using the pattern electroretinogram (pERG), spectral domain optical cohere

242 ical coherence tomography (OCT), and pattern electroretinogram (PERG).

243 The authors retrospectively analyzed pattern electroretinograms (PERG) recorded twice a year in 32 gl

244 Serial pattern electroretinograms (PERGs) and IOPs measures were obtain

245 Pattern-electroretinograms (PERGs) were obtained in response to

246 pamine in vision using electrophysiological (electroretinogram), psychophysical (optokinetic tracking

247 Electroretinograms recorded from larval zebrafish show l

248 p2 mutant but also functionally improved the electroretinogram recording (ERG).

249 ETDRS chart), MP-1 microperimetry, and focal electroretinogram recording (fERG).

250 al function as shown by light microscopy and electroretinogram recording, respectively.

251 mination, retinal thickness measurement, and electroretinogram recording.

252 t of functional roles in the photoreceptors, electroretinogram recordings demonstrate impaired respon

253 Electroretinogram recordings from PCP2-null mice showed

254 tion in NCKX2-deficient (Nckx2(-/-)) mice by electroretinogram recordings revealed normal photopic b-

255 Consistently, electroretinogram recordings show age-progressive loss o

256 Electroretinogram recordings suggest that retinal bipola

257 Electroretinogram recordings under scotopic conditions s

258 s but display reduced "on-off" transients in electroretinogram recordings, indicating a failure to ev

259 rget cones and rescue both the cone-mediated electroretinogram response and visual acuity in the Gnat

260 Multifocal electroretinogram response did not improve, yet peaks we

261 ular toxicity, second cancer development and electroretinogram response were all evaluated.

262 restored rod morphology and the rod-derived electroretinogram response, but cone photoreceptors were

263 ght-blindness models with an electronegative electroretinogram response, which is also characteristic

264 enously expressed rod or cone Talpha rescued electroretinogram responses (ERGs) in mice lacking funct

265 ubretinal delivery of PDE6alpha' rescues rod electroretinogram responses and preserves retinal struct

266 Scotopic and photopic electroretinogram responses declined progressively from

267 ent, one fourth of the mice showed increased electroretinogram responses in the transplanted eyes.

268 The electroretinogram responses of both rod and cone photore

269 se of vitreo-macular traction and multifocal electroretinogram responses showed a significant increas

270 However, cone electroretinogram responses were decreased by 40% at 6 m

271 Electroretinogram responses were recorded using conducti

272 e exhibited absence of scotopic and photopic electroretinogram responses, a phenotype that resembles

273 nd no statistically significant influence on electroretinogram responses, and used in conjunction wit

274 nmf223 homozygotes also have reduced electroretinogram responses, which are coupled histologi

275 h statistically significant reduction in the electroretinogram responses.

276 sion testing, light sensitivity testing, and electroretinograms (retinal imaging and fundus photograp

277 Electroretinograms revealed abnormal cone photoreceptor

278 Electroretinograms revealed photoreceptor dysfunction pr

279 examination, dilated fundus examination, and electroretinograms showed no evidence of vitritis, uveit

280 Electroretinograms showed significantly decreased functi

281 Also, we present electroretinogram studies that have added to our underst

282 In the retina, similarly, an abnormal electroretinogram suggested reduced transmission at the

283 induced negative-masking behavior and normal electroretinogram, suggesting an intact retina light res

284 e was bilateral and in the context of normal electroretinograms therefore indicates generalized dysfu

285 and the size and density of RPE melanosomes, electroretinograms to study retinal function, and retrog

286 cted individuals, nonrecordable rod-specific electroretinogram traces were recovered after prolonged

287 Small, but robust electroretinogram type responses are routinely detected

288 gatively affected in glaucoma, including the electroretinogram, visual evoked potential, visual spati

289 The visual field was severely decreased and electroretinogram was undetectable in most cases; howeve

290 Electroretinogram was used to evaluate visual function.

291 ts in visual impairment assessed by abnormal electroretinogram waveforms.

292 Focal electroretinograms were recorded in response to a sinuso

293 Electroretinograms were recorded, and the outer nuclear

294 Photopic and scotopic electroretinograms were reviewed.

295 of neurophysiological responses (multifocal electroretinogram) were decreased in all eccentricity ri

296 f retinal degeneration, and a non-recordable electroretinogram with negligible amplitudes in both eye

297 d photophobia, reduced amplitude of the cone electroretinogram with normal rod responses, normal fund

298 ng to abnormalities of scotopic and photopic electroretinograms with decreased b-wave amplitude as th

299 Compound 49b maintained a normal electroretinogram, with no changes in blood pressure, in

300 nd N1-P1 amplitudes from photopic multifocal electroretinograms within the central 45 degrees.