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1              This study examined the fate of embryonated and unembryonated Ascaris suum ova in six la
2 ed exclusively in Vero cells, MDCK cells, or embryonated chicken eggs (hereafter referred to as eggs)
3 S1] restores impaired growth to wt levels in embryonated chicken eggs and CEFs.
4       These rNDV mutants grow poorly in both embryonated chicken eggs and chicken embryo fibroblasts
5 /02 virus lineages did not replicate well in embryonated chicken eggs and had to be isolated original
6 ent system to produce cost-effective VLPs in embryonated chicken eggs and has the potential to be use
7  of influenza virus: virus isolation (VI) in embryonated chicken eggs and hemagglutinin subtyping by
8              This virus was propagated using embryonated chicken eggs and inoculated into a duck via
9  generated and evaluated for their growth in embryonated chicken eggs and their immunogenicity and pr
10 e neuraminidase (NA) stalk, does not grow in embryonated chicken eggs because of defective NA functio
11 as wild-type (WT) virus in MDCK cells and in embryonated chicken eggs but is highly attenuated in mic
12 nt viruses grew to high titers in 10-day-old embryonated chicken eggs but were attenuated in mammalia
13 with inactivated virus vaccine produced from embryonated chicken eggs is the most prevalent method to
14 rescued viruses grew well and were stable in embryonated chicken eggs over multiple passages.
15 otein in DF1 cells and in allantoic fluid of embryonated chicken eggs than did the conventional vecto
16               The vaccine can be produced in embryonated chicken eggs using the same process as that
17  of the recombinant virus after passage into embryonated chicken eggs was identical to that of the in
18 creased A/Fujian/411/02 virus replication in embryonated chicken eggs were found to have no significa
19 ire an alternative host cell system, because embryonated chicken eggs will likely be insufficient and
20 n of human subtype H3N2 influenza viruses in embryonated chicken eggs yields viruses with amino acid
21 analysis of viral pathogenicity in 9-day-old embryonated chicken eggs, 1-day-old and 2-week-old chick
22 analysis of viral pathogenicity in 9-day-old embryonated chicken eggs, 1-day-old chicks, and 2-week-o
23 ogenicity of mutant viruses was evaluated in embryonated chicken eggs, 1-day-old chicks, and 6-week-o
24    This PR8 backbone also improves titres in embryonated chicken eggs, a common propagation system fo
25 t on the neutralization of NDV purified from embryonated chicken eggs, a common source for virus prod
26 ally related strains that replicated well in embryonated chicken eggs, A/Sendai-H/F4962/02 and A/Wyom
27 absence of trypsin, caused death in mice and embryonated chicken eggs, and displayed a high-growth ph
28 ruses were viable, grew to similar titers in embryonated chicken eggs, and expressed Gag in a stable
29 6/98, which yields relatively high titers in embryonated chicken eggs, between RNA polymerase I and R
30                                           In embryonated chicken eggs, the parental virus grew to hig
31  cell culture and in experimentally infected embryonated chicken eggs.
32 or-binding domain on the HA globular head in embryonated chicken eggs.
33 te of replication of the parental strains in embryonated chicken eggs.
34 tant viruses replicated relatively poorly in embryonated chicken eggs.
35 on HA for a reassortant H5N1 strain grown in embryonated chicken eggs.
36  to the same titer in MDCK (canine) cells or embryonated chicken eggs.
37 ct on virus replication in MDCK cells and in embryonated chicken eggs.
38  following several passages in MDCK cells or embryonated chicken eggs.
39 hogen were available, and the isolate killed embryonated chicken eggs.
40 hown to be stable after multiple passages in embryonated chicken eggs.
41 enza virus production in both cell lines and embryonated chicken eggs.
42 rulent because of a short mean death time in embryonated chicken eggs.
43 rus in Madin-Darby canine kidney cells or in embryonated chicken eggs.
44 ed by serial passage of a virulent strain in embryonated chicken eggs; however, the molecular mechani
45 Nine Mile phase I (NMI) strain purified from embryonated egg yolk sac preparations.
46 ethods could provide benefits over classical embryonated-egg technology, including a higher productio
47 3 adjuvant system or 15 microg of plain HA), embryonated-egg-derived vaccines (3.75 microg of HA with
48 opment of influenza vaccines that do not use embryonated eggs as the substrate for vaccine production
49 rently being considered as an alternative to embryonated eggs for influenza virus propagation and hem
50 containing a fibronectin-binding domain into embryonated eggs increased the survival rate of virus-in
51          We found that passage adaptation in embryonated eggs is driven by repeated convergent evolut
52 AD and VE implies that passage adaptation in embryonated eggs may be a strong contributor to the rece
53 poration of chicken RCA into NDV produced in embryonated eggs similarly provided species specificity
54 d by serial passage of a virulent isolate in embryonated eggs until attenuation is achieved.
55  and the maintenance of a constant supply of embryonated eggs would be difficult in a pandemic.
56 ons accumulated during vaccine production in embryonated eggs) have been implicated in reduced vaccin
57 As in the same group from recombinant hosts, embryonated eggs, and commercial vaccine lots.
58 ed in Vero or CEF cells but was recovered in embryonated eggs, suggesting that VP2 contains the deter
59 improved vaccine yield by 10-fold in chicken embryonated eggs, the substrate for vaccine manufacture.
60 ammalian cells and mice, yet it grew well in embryonated eggs.
61 decreasing the pathogenicity of the virus in embryonated eggs.
62 f recently isolated viruses, particularly in embryonated eggs.
63 ent to the amount of HA obtained from 10,000 embryonated eggs.
64  potential problems with the availability of embryonated eggs.
65 vel and is overexpressed in dauer larvae and embryonated eggs.
66  of which was recovered after propagation in embryonated eggs.
67  in frequency during passage of the virus in embryonated eggs.
68 ) that can be manufactured at high yields in embryonated eggs.
69 re, we demonstrate that infection of chicken embryonated fibroblasts (CEFs) with highly pathogenic MD
70 ivirus (GPgV) can be propagated in goslings, embryonated goose eggs, and primary goose embryo fibrobl
71 ssed and active in rickettsiae isolated from embryonated hen egg yolk sacs.
72 ansplants on the chorioallantoic membrane of embryonated hen eggs showed reduced tumor-induced angiog
73 iently as the parental recombinant strain in embryonated hen eggs, in MDCK cells, or in vivo in a mou
74                                           In embryonated hen's eggs and leucopenic mice, the outcome
75 ed by serial passage of a virulent strain on embryonated hen's eggs until attenuation; however, littl
76 ines are generated through serial passage in embryonated hens' eggs, an empirical process which achie
77  passage of a virulent field isolate through embryonated hens' eggs, typically 80-100 times.
78 ssage of virulent IBV field isolates through embryonated hens' eggs.
79 ultiple passages of a virulent virus through embryonated hens' eggs.
80  passage of a virulent field isolate through embryonated hens' eggs.
81 7:1 reassortants with T/E segment 3 grown in embryonated hens' eggs.
82  results support the idea that partridge and embryonated partridge egg can be utilized as appropriate
83 ion of systemic candidiasis in partridge and embryonated partridge egg.
84 for systemic candidiasis using partridge and embryonated partridge egg.
85 ay was used to monitor gene expression in de-embryonated rice grains.