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1 proliferation varies by location rather than embryonic age.
2  Galpha(i2) promoter activity independent of embryonic age.
3 an dorsal forebrain stem cells from the same embryonic age.
4 uctive interactions changes as a function of embryonic age.
5 ursors from neural tissue derived from early embryonic ages.
6 ction system in the MA-Ao region starting at embryonic age 11.5.
7 f LHRH neurons from rhesus monkey embryos at embryonic ages 35-37 were dissected out and cultured on
8 opment examined, at Carnegie stage 15 or 16 (embryonic age 5-6 weeks).
9 d in vivo function-blocking of GDNF at early embryonic ages almost entirely suppresses ciliary axon o
10                                       At all embryonic ages analyzed, the distribution of SPNs and PP
11 ences in permeability among RPE of different embryonic age and culture conditions.
12 ing chick long bones changes with increasing embryonic age and that syndecan-3 gene expression change
13 lls (iPSCs) resets their identity back to an embryonic age and, thus, presents a significant hurdle f
14 gal (TCF optimal promoter) reporter mouse at embryonic ages and compared to Axin2 mRNA expression, an
15 se transcription factors emerges at distinct embryonic ages and only in postmitotic cells.
16                            The difference in embryonic age between nodes inversely correlated with th
17 auditory system, initial connections form at embryonic ages, but the functional characteristics of th
18 ptors and ephrins in the VIIIth nerve during embryonic ages corresponding to the initial innervation
19             We have found that, during early embryonic ages (E12-E16), both glutamic acid decarboxyla
20                                     At later embryonic ages EphA4 expression is symmetric around NL,
21                                 At the three embryonic ages examined, E70, E90, and E120, alpha 2A re
22 promoted an increase in neuron number at all embryonic ages examined, there was a developmental shift
23 ility, especially across data from different embryonic age groups.
24 use embryonic SC, this activity starts at an embryonic age of approximately 12 d and is characterized
25 Cs with reduced neurogenic behavior at early embryonic ages presenting a particular molecular signatu
26 nalysis revealed strong associations between embryonic age, structure-function centrality, and the ex
27 ncephalic morphology was abnormal at all the embryonic ages studied (days 10.5, 12.5 and 14.5).
28  The incidence of each subunit declined with embryonic age, suggesting a role in early development.
29                                     At young embryonic ages there was significant overlap in sensitiv
30  visual cortex of cats and ferrets from late embryonic ages to adulthood.
31 that various immune cells arise at different embryonic ages via multiple waves of hematopoiesis from
32 tiated thymidine ([(3)H]dT, or TdR) at early embryonic ages were killed at different intervals postin
33 (Pu.1 WT)) mouse (Mus musculus) brainstem at embryonic ages when the respiratory networks are known t