ライフサイエンスコーパス: eminence

1 he ventricular zone of the medial ganglionic eminence.

2 the cell body and DA secretion at the median eminence.

3 he internal and external zones of the median eminence.

4 migration from the MGE and caudal ganglionic eminence.

5 pulation derived from the lateral ganglionic eminence.

6 es in spontaneous GnRH release in the median eminence.

7 hin their birthplace, the lateral ganglionic eminence.

8 NK3R-expressing GnRH terminals in the median eminence.

9 y to the dorsal vagal complex and the median eminence.

10 ry vesicles in nerve terminals in the median eminence.

11 he release of GnRH from fibers in the median eminence.

12 ons with extensive projections to the median eminence.

13 GnRH terminal microenvironment in the median eminence.

14 es not enhance CRH storage within the median eminence.

15 ate developmentally in the caudal ganglionic eminence.

16 nt and extension of GnRH axons to the median eminence.

17 ntricular zone and not the medial ganglionic eminence.

18 the internal and external zone of the median eminence.

19 ugh the tuberal hypothalamus into the median eminence.

20 rk of nerve fibers was present in the median eminence.

21 urons that derive from the medial ganglionic eminence.

22 d elicit papilla formation on the intermolar eminence.

23 r optic probe was placed over the hypothenar eminence.

24 the third ventricle to the caudal ganglionic eminence.

25 of interneuron progenitors in the ganglionic eminence.

26 ctions to the exterior portion of the median eminence.

27 2-1 leads to a loss of the medial ganglionic eminence.

28 derived from the embryonic medial ganglionic eminence.

29 activation of GnRH projections in the median eminence.

30 s originating in the more distant ganglionic eminences.

31 in cortical ventricular zones and ganglionic eminences.

32 medial (MGE) or the caudal (CGE) ganglionic eminences.

33 er p-H3 showed down-regulation in ganglionic eminences.

34 ithelium but not medial or caudal ganglionic eminences.

35 as the medial, lateral, or caudal ganglionic eminences.

36 genetic evidence that the caudal ganglionic eminence, a distinct subpallial progenitor zone, contrib

37 is, Gsh2 expression in the medial ganglionic eminence after E10.5 may negatively regulate Nkx2.1 depe

38 the axons of the internal zone of the median eminence and a marked reduction in IL-6-like material in

39 pression of markers specific to the thalamic eminence and anterodorsal hypothalamus.

40 Thenar eminence and brain tissue oxygenation and side-stream da

41 IPe, arcuate nucleus of hypothalamus, median eminence and dorsal hypothalamic area in the diencephalo

42 generated from the ventral medial ganglionic eminence and dorsal preoptic area based on fate mapping

43 entrally by the Tbr-1-expressing prethalamic eminence and dorsally by the Gbx-2-expressing part of th

44 ory cells derived from the medial ganglionic eminence and few expressed VGAT, found in GABAergic inte

45 ne (BrdU) labeling in the lateral ganglionic eminence and frontal cortical neuroepithelium but not me

46 that are normally expressed in the thalamic eminence and in the anterodorsal hypothalamus in a porti

47 The lack of RFRP-3 fibers in the median eminence and of Fluoro-Gold uptake from the periphery im

48 y nuclei and in cell processes of the median eminence and pituitary stalk.

49 ogenitors in the embryonic medial ganglionic eminence and preoptic area preferentially develop electr

50 domains equivalent to the medial ganglionic eminence and rhombic lip, resembling the gnathostome bra

51 The lateral ganglionic eminence and rostral migratory stream developed normally

52 in cell death within the lateral ganglionic eminence and rostral migratory stream.

53 as well as the attachment between the tibial eminence and the anterior cruciate ligament, the latter

54 eoptic region, caudally with the prethalamic eminence and the prethalamus, and ventrally with the bas

55 the arcuate nucleus extending to the median eminence and throughout the periventricular zone of the

56 s are found in the dorsal lateral ganglionic eminence and ventrolateral palliumembryonic rudiments of

57 lx1/2 genes in the ventral medial ganglionic eminences and adjacent regions of the septum, resulting

58 haracterized the developing human ganglionic eminences and found that the subventricular zone (SVZ) e

59 K signaling in progenitors of the ganglionic eminences and had fewer SST(+) and VIP(+) interneurons.

60 arts of the hypothalamus, and the ganglionic eminences and their derivatives in the subpallial telenc

61 lar organs such as subfornical organ, median eminence, and area postrema.

62 ng Calb2 and Nkx2.2 ISH (for the prethalamic eminence, and derivatives of the rostral zona limitans s

63 um had hemorrhages in the cortex, ganglionic eminence, and thalamus, as well as abnormal vascular mor

64 pressed in the subfornical organ, the median eminence, and the area postrema.

65 Z) is expressed in the developing ganglionic eminences, and their derivatives.

66 pothalamic areas: the arcuate nucleus-median eminence (ARC-ME) and the paraventricular nucleus (PVN).

67 d CVOs include the subfornical organ, median eminence, area postrema and choroid plexus, and accumula

68 that are derived from the medial ganglionic eminence, as most studies have examined this interneuron

69 the cytoarchitectural features of the median eminence became disorganized with aging.

70 urons originate in the subpallial ganglionic eminences, but their developmental origins in humans are

71 in, reducing innervation of the adult median eminence by GnRH-positive neurites.

72 nglionic eminence (MGE) or caudal ganglionic eminence (CGE) progenitors.

73 s lacking nNOS arises from caudal ganglionic eminence (CGE) progenitors.

74 er, so far the role of the caudal ganglionic eminence (CGE), a posterior subpallial domain, in telenc

75 ganglionic eminence (LGE) caudal ganglionic eminence (CGE), and septum, including loss of GAD1 expre

76 In the hippocampal CA1, caudal ganglionic eminence (CGE)-derived interneurons are recruited by act

77 ence (MGE)-derived but not caudal ganglionic eminence (CGE)-derived interneurons.

78 ventral telencephalon, the caudal ganglionic eminence (CGE).

79 ns that originate from the caudal ganglionic eminence (CGE).

80 on of INs derived from the caudal ganglionic eminence (CGE).

81 to derive mainly from the caudal ganglionic eminence (CGE).

82 from either the caudal or medial ganglionic eminences (CGE and MGE).

83 onic eminences (LGEs), and caudal ganglionic eminences (CGEs) between preterm-born [born on embryonic

84 The hypothalamic arcuate-median eminence complex (Arc-ME) controls energy balance, ferti

85 After median eminence compression to produce axonal injury, unilatera

86 ificantly different (P < or = 0.01) near the eminence crest, but joint load minimization was consiste

87 Our findings establish ganglionic eminence-dependent rules for early synaptic integration

88 , conditionally deleting Arx from ganglionic eminence derived neurons including cortical interneurons

89 ich labels a subset of GABAergic, ganglionic eminence derived neurons.

90 ed in the prethalamus and lateral ganglionic eminence-derived corridor and on corticofugal axons, but

91 that Satb1 is required for medial ganglionic eminence-derived interneuron differentiation, connectivi

92 fferentiation of two major medial ganglionic eminence-derived interneuron populations and defines the

93 at O-LM cells parse into a caudal ganglionic eminence-derived subpopulation expressing 5-HT(3A) recep

94 ceptors (5-HT(3A)Rs) and a medial ganglionic eminence-derived subpopulation lacking 5-HT(3A)Rs.

95 entral neuronal specification and ganglionic eminence development in the Shh(N/-) telencephalon were

96 TCs arise from the dorsal lateral ganglionic eminence (dLGE) and migrate in the lateral migratory str

97 We found that dorsal lateral ganglionic eminence (dLGE)-derived olfactory bulb interneurons are

98 We measured oxygen consumption in the thenar eminence during brachial artery occlusion in sickle cell

99 recursors generated in the medial ganglionic eminence during embryogenesis.

100 ng cells, are born in the ventral ganglionic eminences during mid-gestation and then migrate tangenti

101 6ei) in embryonic day 13.5 medial ganglionic eminence (E13.5 MGE).

102 Very few fibers were observed in the median eminence, especially in the external zone.

103 The medial ganglionic eminence exhibited unique patterns of progenitor cell or

104 pus as well as lateral and medial ganglionic eminences exhibited a 20-30% reduction in mitotic neural

105 ons originate from lateral/caudal ganglionic eminences, express the transcription factor FoxP2, fire

106 frontal arc, a moderately large juxtamastoid eminence, extremely large molars that become progressive

107 euroendocrine with projections to the median eminence for controlling anterior pituitary hormone secr

108 Although tibial eminence fractures are uncommon, their importance cannot

109 a gold standard modality in treating tibial eminence fractures, most studies agreed on several issue

110 progenitors of the dorsal lateral ganglionic eminence from Pax6 mutant Small Eye (Pax6(Sey/Sey)) mice

111 ncy of embryonic day 14.5 (E14.5) ganglionic eminence (GE) progenitors that grew into neurospheres in

112 lls isolated from the subpallium (ganglionic eminence) generate CalR(+) or GABA(+) cells, whereas thi

113 markers revealed that the caudal ganglionic eminence generated a greater proportion of cortical inte

114 the mammalian medial and lateral ganglionic eminences generated medium spiny neurons found in Area X

115 erneurons (CINs) originate in the ganglionic eminences (GEs) and migrate tangentially to the cortex g

116 ilage, abnormal development of the articular eminence/glenoid fossa in the TMJ, and fusion of the art

117 rostral and caudal domains and a prethalamic eminence histogenetic domain in zebrafish.

118 termediate zones of the thalamus, ganglionic eminence, hypothalamus, and cortical preplate.

119 evious study reported that tongue intermolar eminence (IE) oral papillae of Follistatin (a BMP antago

120 CVS did not alter CRH median eminence immunoreactivity, indicating that CVS does not

121 imitations of traditional procedures and the eminence impacts of the advanced methods with clinical a

122 tical interneurons arise from the ganglionic eminences in the ventral telencephalon and migrate tange

123 interneurons are produced in the ganglionic eminences, including an enormous contribution from non-e

124 the volumes of basal ganglia and ganglionic eminence increase along with that of the whole brain, wh

125 eal gland, medial mammillary nucleus, median eminence, infundibular stem, periaqueductal gray, area p

126 Transplanting medial ganglionic eminence interneuron progenitors to introduce new GABAer

127 onic interneurons from the medial ganglionic eminence into the prefrontal cortex of neonatal Pten mut

128 ed into the portal circulation at the median eminence, is known to prime downstream hormone release,

129 h is expressed only in the medial ganglionic eminence, is not.

130 e of the human SVZ at the lateral ganglionic eminence late in the second trimester of development (23

131 achiasmatic nucleus, arcuate nucleus, median eminence, lateral hypothalamic area, thalamus, hippocamp

132 tor zones in the pallium, lateral ganglionic eminence (LGE) and medial ganglionic eminence (MGE) in t

133 ubventricular zone of the lateral ganglionic eminence (LGE) at embryonic day 13.5 may underlie such d

134 ypes, particularly in the lateral ganglionic eminence (LGE) caudal ganglionic eminence (CGE), and sep

135 rt that activin A induces lateral ganglionic eminence (LGE) characteristics in nascent neural progeni

136 he size of the Tlx mutant lateral ganglionic eminence (LGE) from embryonic day 14.5 onward.

137 uronal progenitors of the lateral ganglionic eminence (LGE) in the ventral telencephalon.

138 The lateral ganglionic eminence (LGE) is known to give rise to striatal project

139 The embryonic lateral ganglionic eminence (LGE) is thought to be the site of origin of po

140 dh1a3), is reduced in the lateral ganglionic eminence (LGE) of Gsh2 mutants.

141 nal subtypes derived from lateral ganglionic eminence (LGE) progenitors at specific embryonic time po

142 rogenitor identity in the lateral ganglionic eminence (LGE), despite upregulating the neurogenic fact

143 f progenitor cells in the lateral ganglionic eminence (LGE), the neuroepithelial precursor of the neo

144 eurons (GABA INs), or the lateral ganglionic eminence (LGE), which generate GABA INs that normally mi

145 they acquire a subset of lateral ganglionic eminence (LGE)-specific properties at the expense of MGE

146 ways are generated in the lateral ganglionic eminence (LGE).

147 by their position in the lateral ganglionic eminence (LGE).

148 neurons derived from the lateral ganglionic eminence (LGE).

149 Ps from the lateral and/or caudal ganglionic eminences (LGE and CGE).

150 enitors in the lateral and medial ganglionic eminences (LGE and MGE).

151 in the forebrain itself (lateral ganglionic eminence; LGE) starting at E12.5, suggesting a later rol

152 glionic eminences (MGEs), lateral ganglionic eminences (LGEs), and caudal ganglionic eminences (CGEs)

153 some MGE progenitors to a caudal ganglionic eminence-like, bipolar calretinin-expressing cell fate t

154 persistence of the dorsal lateral ganglionic eminence marker Sp8.

155 ed ventrally in the telencephalic ganglionic eminences (Mash1, Dlx2 and Gsh2) are upregulated cell au

156 tanycytes are stem cells and, in the median eminence, may be stimulated by diet to generate new neur

157 ed animals restored PC1 levels in the median eminence (ME) and the PVN to approximately the level fou

158 Placement of CFMEs in the median eminence (ME) near GnRH terminals allowed detection of b

159 e their neuroendocrine signals at the median eminence (ME) to control long-lasting pituitary hormone

160 ose axons project to the hypothalamic median eminence (ME) where they release gonadotropin-releasing

161 uid (CSF) barrier at the level of the median eminence (ME), a circumventricular organ (CVO) located i

162 dantly expressed in astrocytes of the median eminence (ME), and its enzymatic activity increases sele

163 mine (TIDA) neurons projecting to the median eminence (ME).

164 irst OLPs originate in the medial ganglionic eminence (MGE) and anterior entopeduncular area (AEP) in

165 --including those from the medial ganglionic eminence (MGE) and OB--fail to generate neuroblasts with

166 Progenitor cells in the medial ganglionic eminence (MGE) and preoptic area (PoA) give rise to GABA

167 lencephalon, including the medial ganglionic eminence (MGE) and preoptic area.

168 neuron neurogenesis in the medial ganglionic eminence (MGE) and, more importantly, that estrogen trea

169 x2.1(+) progenitors in the medial ganglionic eminence (MGE) are misspecified such that they acquire a

170 recursors derived from the medial ganglionic eminence (MGE) can induce a second period of functional

171 c precursor cells from the medial ganglionic eminence (MGE) can migrate and differentiate into mature

172 tor cells derived from the medial ganglionic eminence (MGE) can reverse mechanical hypersensitivity i

173 Embryonic medial ganglionic eminence (MGE) cells transplanted into the adult brain c

174 d, a system modeling human medial ganglionic eminence (MGE) development, a critical ventral brain dom

175 neuron precursors from the medial ganglionic eminence (MGE) enhances GABAergic signaling in the brain

176 eloping telencephalon, the medial ganglionic eminence (MGE) generates many cortical and virtually all

177 The medial ganglionic eminence (MGE) gives rise to the majority of mouse foreb

178 glionic eminence (LGE) and medial ganglionic eminence (MGE) in the subpallium has been well studied;

179 GABAergic neurons from the medial ganglionic eminence (MGE) into adult mouse spinal cord ameliorates

180 r cells from the embryonic medial ganglionic eminence (MGE) into early postnatal neocortex generates

181 nhibitory neurons from the medial ganglionic eminence (MGE) into postnatal visual cortex makes it los

182 nterneurons from the mouse medial ganglionic eminence (MGE) into the adult mouse spinal cord complete

183 ventral telencephalon, the medial ganglionic eminence (MGE) is a major source of cortical interneuron

184 The medial ganglionic eminence (MGE) is an embryonic forebrain structure that

185 Young neurons born in the medial ganglionic eminence (MGE) migrate a long distance dorsally, giving

186 mosaic elimination in the medial ganglionic eminence (MGE) of Smo, a key effector of SHH signaling,

187 inate primarily within the medial ganglionic eminence (MGE) of the subcortical telencephalon, whereas

188 ting cells by lineage from medial ganglionic eminence (MGE) or caudal ganglionic eminence (CGE) proge

189 Prdm16 expression in mouse medial ganglionic eminence (MGE) progenitors is required for maintaining t

190 Caudally situated medial ganglionic eminence (MGE) progenitors receive high levels of Wnt si

191 NGCs are both derived from medial ganglionic eminence (MGE) progenitors under control of the transcri

192 ajor subtypes generated by medial ganglionic eminence (MGE) progenitors.

193 ted in a partial rescue of medial ganglionic eminence (MGE) properties, including interneuron migrati

194 rneuron (IN) production in medial ganglionic eminence (MGE) secondary progenitors in mice.

195 n GABAergic neurons of the medial ganglionic eminence (MGE) showed marked deficits in distinct subpop

196 ntricular zone (VZ) of the medial ganglionic eminence (MGE) using Olig2-Cre mice causes moderate or s

197 N subtypes derive from the medial ganglionic eminence (MGE), a transient ventral telencephalic struct

198 nic structure known as the medial ganglionic eminence (MGE), but how the remarkable diversity is spec

199 euron progenitors from the medial ganglionic eminence (MGE), can overcome the mechanical hypersensiti

200 interneurons, derived from medial ganglionic eminence (MGE), is implicated in disorders of learning a

201 ing on the mouse embryonic medial ganglionic eminence (MGE), the major birthplace for CINs, and on MG

202 lation was examined in the medial ganglionic eminence (MGE), the major source of PV interneurons in m

203 cursors from the embryonic medial ganglionic eminence (MGE), the source of neocortical parvalbumin- (

204 erneurons originate in the medial ganglionic eminence (MGE), where the signaling molecule sonic hedge

205 not by progenitors in the medial ganglionic eminence (MGE), which generate cortical GABAergic intern

206 is mediated by inputs from medial ganglionic eminence (MGE)-derived but not caudal ganglionic eminenc

207 oring tdTomato-fluorescent medial ganglionic eminence (MGE)-derived cortical GABAergic interneurons w

208 terestingly, compared with medial ganglionic eminence (MGE)-derived cortical interneuron populations,

209 Medial ganglionic eminence (MGE)-derived GABAergic cortical interneurons (

210 erentiation of a subset of medial ganglionic eminence (MGE)-derived neurons, but are dispensable for

211 Medial ganglionic eminence (MGE)-derived somatostatin (SST)+ and parvalbum

212 alic excitatory neurons or medial ganglionic eminence (MGE)-like inhibitory neurons.

213 Medial ganglionic eminence (MGE)-like interneuron precursors derived from

214 ferentiation of hPSCs into medial ganglionic eminence (MGE)-like progenitors and their maturation int

215 e neurons derived from the medial ganglionic eminence (MGE).

216 subpallium, including the medial ganglionic eminence (MGE).

217 erneurons generated in the medial ganglionic eminence (MGE).

218 t primarily resides in the medial ganglionic eminence (MGE).

219 g those originating in the medial ganglionic eminence (MGE).

220 INs) that originate in the medial ganglionic eminence (MGE).

221 s from embryonic medial or caudal ganglionic eminence (MGE, CGE) were made in a well-characterized mo

222 embryo from the medial or caudal ganglionic eminences (MGE and CGE).

223 from either the medial or caudal ganglionic eminences (MGE and CGE).

224 ng of the mouse medial and caudal ganglionic eminences (MGE and CGE, respectively), from which most c

225 euronal progenitors in the medial ganglionic eminences (MGEs), lateral ganglionic eminences (LGEs), a

226 as examined: embryonic neocortex, ganglionic eminence, midbrain, retina, hindbrain, and spinal cord;

227 derived from the embryonic medial ganglionic eminence, migrate long distances following transplantati

228 aring adeno-associated virus into the median eminence of adult GnRH-Cre mice resulted in the selectiv

229 Our results highlight the pre-eminence of cis-acting variants on transcription factor

230 The pre-eminence of EPPGE over mercury electrodes has been prove

231 These results call into question the pre-eminence of escape as the primary advantage of dispersal

232 entopeduncular area-basal medial ganglionic eminence of mammals).

233 and resembled structures seen in the median eminence of rats.

234 GnRH release detected by FSCV in the median eminence of slices from adults with previous reports of

235 T(H)-17 lineage appeared to supplant the pre-eminence of T(H)1 cells in promoting autoimmunity, the n

236 In addition, unlike in the ganglionic eminence of the embryonic forebrain where Olig2 is mostl

237 Pe neurons derive from the medial ganglionic eminence of the embryonic subpallium and express the tra

238 elivered to a central location on the thenar eminence of the hand.

239 ted by infusion of letrozole into the median eminence of the hypothalamus.

240 ar neurons has been observed, the ganglionic eminence of the telencephalon.

241 nterneurons that originate in the ganglionic eminence of the ventral forebrain and incorporate into t

242 al investigations, although recently the pre-eminence of this system in nicotine dependence has been

243 r surrounding microenvironment in the median eminence of young (4-5 months) and old (22-24 months) ov

244 rons within the medial and caudal ganglionic eminences of the developing telencephalon.

245 mulation applied independently to the thenar eminence on each hand and also to bilateral (simultaneou

246 othalamic area (MBA), arcuate nucleus/median eminence, paraventricular nuclei and piriform cortex.

247 the caudate nucleus adjoining the ganglionic eminence, potentially a waiting compartment.

248 n precursors in the developing telencephalic eminences predicts the intrinsic physiological propertie

249 erned to NKX2.1-expressing medial ganglionic eminence progenitors by simple treatment with sonic hedg

250 mine whether hypocretin modulates the median eminence-projecting proopiomelanocortin (POMC) neurons i

251 onpainful punctate stimulation of the thenar eminence provoked more diffuse activity but was still ip

252 m the medial basal hypothalamus (MBH)/median eminence region (S-ME) is essential for normal reproduct

253 es, an ependymoglial cell type of the median eminence, regulate LHRH release during the estrous cycle

254 nhibitory neurons from the medial ganglionic eminence reinstate ocular dominance plasticity in adult

255 erneurons derived from the medial ganglionic eminence represent the largest cohort of GABAergic neuro

256 ch in Nkx2.1 expression, and the prethalamic eminence, rich in Tbr1 expression.

257 joint load minimization was consistent with eminence shape for x < 3.0 mm.

258 ypothesis that temporomandibular joint (TMJ) eminence shapes develop ideally to minimize joint loads.

259 dict, and jaw-tracking data used to measure, eminence shapes.

260 only in the subfornical organ and the median eminence, situated outside the blood brain barrier, but

261 GABA neurons identified by antidromic median eminence stimulation.

262 the cortical anlage (CTXOE03) and ganglionic eminence (STROC05), as well as an adult EC line (D3) der

263 us, amygdala, thalamus, hypothalamus, medium eminence, substantia nigra, ventral tegmental area, ocul

264 , in GABAergic nerve terminals in the median eminence suggested that rather than a functional redunda

265 e in GABAergic nerve terminals in the median eminence suggests that only the non-GABAergic dopamine n

266 he cortex, most stem cells in the ganglionic eminence SVZ did not maintain radial fibers or orientati

267 s, most of which originate in the ganglionic eminences, take distinct tangential migratory trajectori

268 g genetic fate mapping, we found that median eminence tanycytes generate newborn neurons.

269 within the infundibular stalk/caudal median eminence, termed here gamma tanycytes, and a subset of c

270 to neural progenitor cells in the ganglionic eminence that are fated to differentiate into GABAergic

271 omologue of the mammalian lateral ganglionic eminence (the adult caudatoputamen in mammals).

272 homologue of the mammalian medial ganglionic eminence (the adult pallidum in mammals).

273 f cells from the embryonic medial ganglionic eminence (the major origin of cerebral cortical interneu

274 In the medial ganglionic eminence, the NKX2-1 transcription factor controls regio

275 ellular level, we show that, similar to bone eminences, the patella is formed secondarily by a distin

276 eniculate nucleus from the caudal ganglionic eminence, there is no obvious new source of proliferatin

277 migration from the dorsal lateral ganglionic eminence through maturity.

278 namic variables and also cerebral and thenar eminence tissue oxygenation and side-stream dark-field i

279 Median eminence tissues were freeze-plunge embedded and serial

280 hat the GnRH neuron projection to the median eminence to control pituitary hormone secretion possesse

281 the brain, where they project to the median eminence to release GnRH.

282 rneurons migrate from the ventral ganglionic eminence to the cerebral cortex within several migratory

283 ransported in varicose fibres via the median eminence to the posterior pituitary gland.

284 How circulating signals enter the median eminence to trigger homeostatic hypothalamic responses i

285 ration from the medial and caudal ganglionic eminences to the cerebral cortex in slice preparations o

286 from their place of birth in the ganglionic eminences to their place of terminal differentiation in

287 ors normally found in the lateral ganglionic eminence, to prevent precocious differentiation and depl

288 enitors of the lateral and median ganglionic eminences, two protrusions of the ventral telencephalon

289 om their generation in the medial ganglionic eminence up to their settlement in the AC, express two c

290 rogenitors are located within the ganglionic eminences, using Dlx5/6-Cre-ires-EGFP (Dlx5/6-CIE) mice.

291 Enforced collapse of ganglionic eminence vessels and resultant periventricular neural ap

292 se in target brain regions and in the median eminence via a direct inhibition of vGluT2-GnRH neurons.

293 rs located in the ventral lateral ganglionic eminence (vLGE).

294 ced secretion of dopamine (DA) at the median eminence was strongly blunted during lactation, at least

295 wn to innervate the middle finger and thenar eminence were also transected.

296 those that project their axons to the median eminence, were robustly activated by hypocretin in a dos

297 icular-subventricular zone of the ganglionic eminences, whereas at midgestation (20 GW), they were al

298 he PL develops next to the caudal ganglionic eminence, which generates inhibitory interneurons, yet m

299 deleted mouse Nf1 from the medial ganglionic eminence, which gives rise to both oligodendrocytes and

300 aired proliferation in the medial ganglionic eminence without grossly altering differentiated fate.