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1 rs in a follow-up study of patients with VZV encephalitis.
2 iated protein-like 2 (CASPR2)-IgG associated encephalitis.
3 cause severe diseases, such as blindness and encephalitis.
4 ral and genital lesions, ocular disease, and encephalitis.
5 itis virus (JEV) is a leading cause of viral encephalitis.
6 is a novel therapeutic target to prevent JEV encephalitis.
7 nsensus criteria for definite NMDAR-antibody encephalitis.
8 keratitis, corneal scarring, blindness, and encephalitis.
9 tal brain injury and experimental autoimmune encephalitis.
10 in severity from benign cold sores to fatal encephalitis.
11 the activation of astrocytes during reovirus encephalitis.
12 g)P cells, prevented experimental autoimmune encephalitis.
13 ng TLR3 deficiency with herpes simplex virus encephalitis.
14 ts of checkpoint inhibitor-associated immune encephalitis.
15 responsible for oral lesions and herpesviral encephalitis.
16 d 1100 patients in total with NMDAR-antibody encephalitis.
17 of EEEV as a cause of transplant-associated encephalitis.
18 the diagnosis, management, and prognosis of encephalitis.
19 that the CXCR6(+) T cells are the drivers of encephalitis.
20 methyl-D-aspartate receptor (NMDAR)-antibody encephalitis.
21 itted flavivirus that can cause debilitating encephalitis.
22 rove aetiological diagnosis in children with encephalitis.
23 ith the virus spreading to the brain causing encephalitis.
24 ase, neonatal infection, and, in rare cases, encephalitis.
25 en with signs and symptoms of meningitis and encephalitis.
26 ted and includes demyelinating syndromes and encephalitis.
27 al control during acute and persistent MuPyV encephalitis.
28 ever, this was less pronounced than in viral encephalitis.
29 argely fall under the umbrella of autoimmune encephalitis.
30 developed an akinetic mutism attributable to encephalitis.
31 h outbreaks of acute respiratory disease and encephalitis.
32 nations of 19 patients with confirmed BoDV-1 encephalitis.
33 ARs: schizophrenia and autoimmune anti-NMDAR encephalitis.
34 toimmune neurological diseases or with viral encephalitis.
35 disorders and their mechanisms in autoimmune encephalitis.
36 e; 25% (95% CI, 20%-30%) had immune-mediated encephalitis, 18% acute disseminated encephalomyelitis,
38 tis, 287 children met criteria for confirmed encephalitis: 57% (95% confidence interval [CI], 52%-63%
39 lso recapitulated in experimental autoimmune encephalitis, a mouse model of multiple sclerosis and th
40 ient with adult onset epilepsy and suspected encephalitis, a strong signal at ~70kDa was detected by
41 of patients (n = 38) after autoimmune limbic encephalitis, a syndrome associated with focal structura
42 identification of anti-NMDA receptor (NMDAR) encephalitis about 12 years ago made it possible to reco
44 COVID-19 CNS disease include encephalopathy, encephalitis, acute disseminated encephalomyelitis, meni
45 ly exclusive criteria, including meningitis, encephalitis, acute flaccid myelitis, and seizures) and
46 ncluding hepatitis, vascular shock syndrome, encephalitis, acute flaccid paralysis, congenital abnorm
47 isease in Kozhikode in May 2018 presented as encephalitis, acute respiratory distress and myocarditis
50 if cerebrospinal fluid (CSF) from autoimmune encephalitis (AE) patients regulates in vitro neuronal n
51 ceptors (NMDARs) in patients with anti-NMDAR encephalitis alter neuronal synaptic function and plasti
52 h anti-N-methyl-d-aspartate receptor (NMDAR) encephalitis alter the levels of dopamine 1 receptor (D1
53 ntral nervous system infections, meningitis, encephalitis, amyotrophic lateral sclerosis, Huntington
57 ith flu-like symptoms, which can progress to encephalitis and cause permanent neurological sequelae i
59 rrhages, diffuse cerebral edema, necrotizing encephalitis and decreased bone size (Alizarin red stain
60 pose that the differences in the severity of encephalitis and demyelination between RSA59 (PP) and RS
63 ted the induction of experimental autoimmune encephalitis and enabled long-term skin allograft surviv
66 between SARS-CoV-2 infection and autoimmune encephalitis and for the use of (18)F-FDG PET in such a
67 dren worldwide and have been associated with encephalitis and meningitis in immunocompromised individ
68 to anti-N-methyl-D-aspartate receptor [NMDA] encephalitis and multiple sclerosis), and noninflamed co
69 hese include the relevance of herpes simplex encephalitis and of epilepsy to AD, the action of IFN, a
70 squito-borne zoonotic flavivirus that causes encephalitis and reproductive disorders in mammalian spe
71 (NiV) continues to cause fatal outbreaks of encephalitis and respiratory disease in Bangladesh and I
73 aviruses (HNVs) responsible for outbreaks of encephalitis and respiratory illness with fatality rates
74 f a 72-y-old man with concomitant autoimmune encephalitis and severe acute respiratory syndrome coron
76 ually similar to core features of autoimmune encephalitis and/or animal models of NMDAR antibody-medi
79 ability to reactivate to cause blindness and encephalitis, and high prevalence in human populations.
80 ovarian teratoma, prior herpes simplex virus encephalitis, and isolated psychiatric presentations (tw
81 ngitis, 31 (72%) children showed evidence of encephalitis, and ten (23%) children met our case defini
82 atric patients hospitalized with meningitis, encephalitis, and/or myelitis showed 92% sensitivity and
83 s, and 6% anti-N-methyl-d-aspartate receptor encephalitis; and 17% (95% CI, 13%-21%) had an unknown c
84 in adults and children with ADEM, seizures, encephalitis, anti-aquaporin-4-antibody (AQP4-Ab)-serone
85 usually ovarian teratoma, and herpes simplex encephalitis are known triggers of NMDAR autoimmunity.
89 databases (across multiple cancer types) of encephalitis associated with checkpoint inhibitor regime
90 (<=14 years of age) admitted with suspected encephalitis at 5 major pediatric hospitals nationally b
91 ive adult patients diagnosed with anti-NMDAR encephalitis at the French National Reference Centre, ad
95 9/23 with autoantibodies were diagnosed with encephalitis, by contrast to 0/196 without autoantibodie
99 Although there was a 20% reduction in NiV encephalitis cases treated with the drug, the difference
101 CSF antibodies from patients with anti-NMDAR encephalitis cause reversible psychotic-like features ac
103 from 0.32 to 0.009) and admissions for mumps encephalitis decreased by 98% (from 0.60 to 0.01) after
107 Then, we focus on several antibody-mediated encephalitis disorders that associate with seizures and
108 sible for an unprecedented outbreak of human encephalitis during 1975-1976 in Sao Paulo State, Brazil
110 d pathologic scores in experimental allergic encephalitis (EAE) and in the cuprizone model of demyeli
111 rotective effects on experimental autoimmune encephalitis (EAE) responses but the detailed roles of S
112 iple sclerosis (MS), experimental autoimmune encephalitis (EAE), we evaluated the role of gut microbi
114 we show that during La Crosse Virus-induced encephalitis, egress of iMOs was surprisingly independen
119 k-transmitted pathogen that may cause severe encephalitis; experimentally, it can be transmitted with
120 (CSF) using the BioFire FilmArray meningitis/encephalitis (FA-M/E) panel permits rapid, simultaneous
121 nalysis of hospital admission statistics for encephalitis for individuals aged 0-19 years using natio
122 nosis of infectious causes of meningitis and encephalitis from cerebrospinal fluid (CSF) in a license
124 ecades, the diagnosis rate for patients with encephalitis has remained poor despite advances in patho
126 Sleep disorders in people with autoimmune encephalitis have received little attention, probably ov
132 ctious, para-infectious, and post-infectious encephalitis, hypercoagulable states leading to stroke,
133 SF obtained from patients with meningitis or encephalitis improved diagnosis of neurologic infections
141 e virus (WNV) is an important cause of viral encephalitis in birds and animals, including humans.
142 vere hand, foot, and mouth disease and viral encephalitis in children across the Asia-Pacific region,
145 s, caused by rabies virus (RABV), is a fatal encephalitis in humans and other mammals, which continue
146 ult in acute respiratory syndrome and severe encephalitis in humans, leading to 40 to 100% mortality
147 (VEEV), which elicits flu-like symptoms and encephalitis in humans, with an estimated 14% of cases r
156 imate of the prevalence of autoimmune KLHL11 encephalitis in Olmsted County, Minnesota, was 2.79 case
157 s that causes near-annual outbreaks of fatal encephalitis in South Asia-one of the most populous regi
160 mplex virus (HSV) is the main cause of viral encephalitis in the Western world, and the type I interf
161 inoculation with MuPyV, we found that MuPyV encephalitis in wild-type mice causes an encephalopathy,
162 0 per month to 5.6 per month, admissions for encephalitis increased from 0.4 per month to 1.4 per mon
165 strocytes undergo apoptosis.IMPORTANCE Viral encephalitis is a significant cause of worldwide morbidi
166 Anti-N-methyl-d-aspartate receptor (NMDAR) encephalitis is an immune-mediated disease characterized
169 Anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis is the most common autoimmune encephalitis
171 s develops in NMDA receptor (NMDAR) antibody encephalitis, it usually has an acute or subacute onset,
172 ugh several different flaviviruses may cause encephalitis, Japanese encephalitis virus is the most si
176 nya (CHIKV), Yellow fever (YFV) and Japanese encephalitis (JEV) virus in different geographical regio
177 ed from dengue (DENV), Zika (ZIKV), Japanese encephalitis (JEV), West Nile (WNV), and yellow fever (Y
179 of neuroinflammatory responses during viral encephalitis may facilitate the development of new treat
180 and performance of the FilmArray meningitis/encephalitis (ME) panel has received limited investigati
181 QR}, 0.1-6.9]) compared with immune-mediated encephalitis (median age, 7.6 years [IQR, 4.6-12.4]).
184 appears broader as cases of encephalopathy, encephalitis, meningitis, myelitis, and seizures have al
186 d symptoms, but severe complications such as encephalitis, meningitis, or orchitis can also occur.
187 2009 in South Africa, measles inclusion body encephalitis (MIBE) was identified in several HIV-infect
189 nician awareness of Balamuthia as a cause of encephalitis might lead to earlier diagnosis and initiat
190 more susceptible to experimental autoimmune encephalitis, mirroring the susceptibility to autoimmune
192 nflammatory CNS syndromes (n = 12) including encephalitis (n = 2, para- or post-infectious), acute di
194 rists, the psychopathology of NMDAR-antibody encephalitis needs to be clearly defined to encourage ac
195 icella zoster, and enteroviruses) meningitis/encephalitis, neuroborreliosis, autoimmune neuroinflamma
196 ed whether IL-1 signaling contributes to the encephalitis observed in mouse adenovirus type 1 (MAV-1)
200 ents with adult onset epilepsy and suspected encephalitis of unresolved etiology and equivalent resul
201 al fluid (CSF) from patients with anti-NMDAR encephalitis or controls, and the effects on clusters of
203 ildren with demyelinating syndromes and with encephalitis other than acute disseminated encephalomyel
204 these 116 patients included ADEM (46 [68%]), encephalitis other than ADEM (22 [19%]), optic neuritis
205 yelinating syndromes (cohort A) and 296 with encephalitis other than ADEM (cohort B) were recruited.
206 hlighted because of their prominent roles in encephalitis, other infectious pathogens are also consid
207 s warranted in subjects with cytomegalovirus encephalitis, owing to the low penetration of antivirals
209 urthermore, in mouse experimental autoimmune encephalitis, p73-deficient mice have increased IFNgamma
210 ily available using the FilmArray Meningitis/Encephalitis panel (FA-ME; BioFire Diagnostics, Salt Lak
212 inal fluid (CSF) of patients with anti-NMDAR encephalitis (preabsorbed or not with GluN1) and a human
214 rus or Ebola, Chikungunya, or eastern equine encephalitis pseudoviruses was inhibited with greater ef
215 The distinctive aspect of NMDAR-antibody encephalitis psychopathology is complexity; core aspects
218 ) encephalitis is the most common autoimmune encephalitis related to autoantibody-mediated synaptic d
219 measles and rubella, yellow fever, Japanese encephalitis, rotavirus, and invasive bacterial diseases
220 with detectable autoantibodies, but without encephalitis, showed excellent long-term outcomes (modif
223 this disease, whereas in anti-NMDA receptor encephalitis, sleep disorders vary according to the dise
224 cal, mild, or attenuated forms of autoimmune encephalitis, some authors feel that that these cases ar
225 Antibodies from patients with anti-NMDAR encephalitis specifically alter the function of NMDARs i
228 Our results indicate that patients with VZV encephalitis suffer from cognitive impairment long time
229 es of autoantibody-positive patients without encephalitis suggests immunotherapy administration shoul
233 Southern Sweden is endemic for tick-borne encephalitis (TBE), with Stockholm County as one of the
235 tis, but only RSA59 (PP) produced widespread encephalitis that extended deeply into the brain parench
236 fficiently different from typical autoimmune encephalitis to establish a new category of so-called au
239 la elimination and the expansion of Japanese encephalitis vaccination in countries where it is endemi
240 cine and live attenuated SA 14-14-2 Japanese encephalitis vaccine (LJEV) are recommended for simultan
242 such as typhoid conjugate (TCV) and Japanese encephalitis vaccines are risk based and only used in co
243 uctural proteins (nsPs) of Venezuelan equine encephalitis (VEE) replicon that promoted subgenome expr
249 ne encephalitis virus (VEEV), eastern equine encephalitis virus (EEEV), and western equine encephalit
251 ns against neurotropic flaviviruses.Japanese encephalitis virus (JEV) is a Flavivirus responsible for
255 three flaviviruses, DENV, WNV, and Japanese encephalitis virus (JEV), using a high-content immunoflu
256 rus (DENV; nine isolates analyzed), Japanese encephalitis virus (JEV; one isolate analyzed) and Zika
261 inally, mice infected with Venezuelan equine encephalitis virus (VEEV) were successfully treated with
262 e infection with wild-type Venezuelan equine encephalitis virus (VEEV), a highly myeloid-cell-tropic
265 NCE RNA viruses, including Venezuelan equine encephalitis virus (VEEV), have high mutation rates that
266 ection with the alphavirus Venezuelan equine encephalitis virus (VEEV), which causes flu-like symptom
267 currently unavailable for Venezuelan equine encephalitis virus (VEEV), which elicits flu-like sympto
268 ncephalitis virus (EEEV), and western equine encephalitis virus (WEEV) are arthropod-borne positive-s
269 tron microscopy structure of mature Japanese encephalitis virus at near-atomic resolution, which reve
270 sed plasmid VRC5288 (Zika virus and Japanese encephalitis virus chimera), and the VRC 320, done in on
271 including West Nile virus and Eastern equine encephalitis virus has been detected in wild Atlantic bo
273 laviviruses may cause encephalitis, Japanese encephalitis virus is the most significant, being respon
274 with the corresponding region from Japanese encephalitis virus NS1 to create chimeric DJ NS1 protein
275 es with the corresponding region of Japanese encephalitis virus NS1 to generate a chimeric DJ NS1 pro
278 , Jamestown Canyon virus, and eastern equine encephalitis virus, as well as the tick-borne Powassan v
280 of neurovirulence and stability in Japanese encephalitis virus, opening up new avenues for therapeut
281 with epidemics, required RelA, and Japanese encephalitis virus, which produced relatively minor cyto
285 uding Dengue, Zika, West Nile and Tick-borne encephalitis viruses, activate the unfolded protein resp
289 Using a mouse model of West Nile virus (WNV) encephalitis, we show that RIPK3 restricts WNV pathogene
290 lt (aged >=17 years) patients with confirmed encephalitis were enrolled, and 194 (57%) had unknown et
293 nia is N-methyl-D-aspartate receptor (NMDAR) encephalitis, which can account for the full spectrum of
295 rus clearance and neurotoxicity during viral encephalitis with a focus on the cytokines essential for
296 escription of the morphology of human BoDV-1 encephalitis, with histopathological verification of ima
297 death were explained by active granulomatous encephalitis, with widespread involvement of other organ
298 OT) recipients from a common donor developed encephalitis within 1 week of transplantation, prompting
300 terologous challenges of West Nile, Japanese encephalitis, Zika, and dengue viruses did not affect re