ライフサイエンスコーパス: encephalomyelitis

1 tened sensitivity to experimental autoimmune encephalomyelitis.

2 A viruses that are important causes of viral encephalomyelitis.

3 from degeneration in experimental autoimmune encephalomyelitis.

4 glycoprotein-induced experimental autoimmune encephalomyelitis.

5 om 13 marmosets with experimental autoimmune encephalomyelitis.

6 thogenic function in experimental autoimmune encephalomyelitis.

7 ut, also ameliorated experimental autoimmune encephalomyelitis.

8 lowed during induced experimental autoimmune encephalomyelitis.

9 dent protection from experimental autoimmune encephalomyelitis.

10 system of mice with experimental autoimmune encephalomyelitis.

11 viral infection and experimental autoimmune encephalomyelitis.

12 to protect mice from experimental autoimmune encephalomyelitis.

13 ady state and during experimental autoimmune encephalomyelitis.

14 peutic reduction in, experimental autoimmune encephalomyelitis.

15 ngham virus-dependent progressive multifocal encephalomyelitis.

16 esponses are seen in experimental autoimmune encephalomyelitis.

17 reduced severity of experimental autoimmune encephalomyelitis.

18 nd its animal model, experimental autoimmune encephalomyelitis.

19 vivo development of experimental autoimmune encephalomyelitis.

20 system of mice with experimental autoimmune encephalomyelitis.

21 s system (CNS) inflammation, including viral encephalomyelitis.

22 sease: from multiple sclerosis to autoimmune encephalomyelitis.

23 suppression of ongoing experimental allergic encephalomyelitis.

24 ofound resistance to experimental autoimmune encephalomyelitis.

25 e T cells and modulate experimental allergic encephalomyelitis.

26 ltiple sclerosis and experimental autoimmune encephalomyelitis.

27 induced arthritis or experimental autoimmune encephalomyelitis.

28 chronic or relapsing experimental autoimmune encephalomyelitis.

29 ive phases of murine experimental autoimmune encephalomyelitis.

30 s the MS mouse model experimental autoimmune encephalomyelitis.

31 ed susceptibility to experimental autoimmune encephalomyelitis.

32 arameters missing in experimental autoimmune encephalomyelitis.

33 A viruses that are important causes of viral encephalomyelitis.

34 neuroinflammation in experimental autoimmune encephalomyelitis.

35 in the spleen during experimental autoimmune encephalomyelitis.

36 297 prevented murine experimental autoimmune encephalomyelitis (a model of human multiple sclerosis)

37 less susceptible to experimental autoimmune encephalomyelitis (a model of MS) and is accompanied by

38 t the progression of experimental autoimmune encephalomyelitis, a model for multiple sclerosis, even

39 ins protection against experimental allergic encephalomyelitis, a mouse model for human multiple scle

40 less-severe signs of experimental autoimmune encephalomyelitis, a murine model of MS, thus implicatin

41 ltiple sclerosis (MS) and acute disseminated encephalomyelitis (ADEM) have been difficult to study an

42 h encephalitis other than acute disseminated encephalomyelitis (ADEM) recruited from 40 secondary and

43 reported in children with acute disseminated encephalomyelitis (ADEM).

44 vous system diseases like acute disseminated encephalomyelitis (ADEM).

45 in mice paralysed by experimental autoimmune encephalomyelitis ameliorates symptoms.

46 suppression of ongoing experimental allergic encephalomyelitis (an MS animal model), and the disease

47 and protects mice from experimental allergic encephalomyelitis, an animal model of multiple sclerosis

48 s the development of experimental autoimmune encephalomyelitis, an autoimmune inflammation in the CNS

49 Here, using experimental autoimmune encephalomyelitis, an established mouse MS model, we rep

50 cells (FRCs), during experimental autoimmune encephalomyelitis and colitis.

51 mi of mice at different stages of autoimmune encephalomyelitis and healthy controls.

52 ression in mice with experimental autoimmune encephalomyelitis and K/BxN serum transfer-induced RA.

53 te to multiple sclerosis (MS) and autoimmune encephalomyelitis and likely play a role in traumatic br

54 o diseases caused by RNA viruses, alphavirus encephalomyelitis and measles, and was enriched with sev

55 e severe symptoms of experimental autoimmune encephalomyelitis and reduced proportions of IL-10(+) Tr

56 of actively induced experimental autoimmune encephalomyelitis and salt-sensitive hypertension by mod

57 disorders, including experimental autoimmune encephalomyelitis and sepsis in mice.

58 f the Coronaviridae family) results in acute encephalomyelitis and viral persistence associated with

59 ica spectrum disorder and acute disseminated encephalomyelitis) and from non-demyelinating disorders

60 ediated encephalitis, 18% acute disseminated encephalomyelitis, and 6% anti-N-methyl-d-aspartate rece

61 m disorders and relapsing acute disseminated encephalomyelitis, and characterizing cohorts for antibo

62 multiple sclerosis, experimental autoimmune encephalomyelitis, and show that gene inactivation of co

63 ogic function in the experimental autoimmune encephalomyelitis animal model of multiple sclerosis was

64 pathogenesis and the experimental autoimmune encephalomyelitis animal model.

65 -induced glomerulonephritis and experimental encephalomyelitis are attenuated in ICER/CREM-deficient

66 and its animal model experimental autoimmune encephalomyelitis, are neuroinflammatory diseases driven

67 signs of disease in experimental autoimmune encephalomyelitis, as well as maintain normoglycemia in

68 IL4I1 into mice with experimental autoimmune encephalomyelitis at disease onset significantly reverse

69 with MOG to promote experimental autoimmune encephalomyelitis because NFM-deficient synonymous with

70 ion and induction of experimental autoimmune encephalomyelitis but normal thymocyte development.

71 ic test for chronic fatigue syndrome/Myalgic Encephalomyelitis (CFS/ME).

72 Remarkably, in experimental autoimmune encephalomyelitis, cholesterol supplementation does not

73 he Multi-Site Clinical Assessment of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (MCAM), we re

74 Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) invo

75 Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a

76 Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a

77 Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is a

78 Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a

79 Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a

80 if any, biological test to diagnose myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).

81 nefit from B-lymphocyte depletion in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).

82 ing phenotype: those with acute disseminated encephalomyelitis demonstrated abnormal trajectories of

83 were protected from experimental autoimmune encephalomyelitis, demonstrating that this pathway is cr

84 In an experimental autoimmune encephalomyelitis disease model of neurodegeneration in

85 endent manner during experimental autoimmune encephalomyelitis (EAE) and drain both CSF and cells tha

86 ufficient to trigger experimental autoimmune encephalomyelitis (EAE) and facial allodynia in immunize

87 neurodegeneration in experimental autoimmune encephalomyelitis (EAE) and in in vitro studies regardin

88 m, for inhibition of experimental autoimmune encephalomyelitis (EAE) and induction of tolerance.

89 progeny can suppress experimental autoimmune encephalomyelitis (EAE) and pancreatic beta cell autorea

90 an important role in experimental autoimmune encephalomyelitis (EAE) and perhaps MS.

91 the pathogenesis of experimental autoimmune encephalomyelitis (EAE) and, ostensibly, in multiple scl

92 CNS inflammation in experimental autoimmune encephalomyelitis (EAE) and, potentially, MS.

93 CNS inflammation in experimental autoimmune encephalomyelitis (EAE) and, potentially, multiple scler

94 ltiple sclerosis and experimental autoimmune encephalomyelitis (EAE) are inflammatory diseases of the

95 o the development of experimental autoimmune encephalomyelitis (EAE) as a result of an increase of pr

96 ts preclinical model experimental autoimmune encephalomyelitis (EAE) by single-cell RNA sequencing in

97 evelop a more severe experimental autoimmune encephalomyelitis (EAE) course compared to wildtype mice

98 sponses and enhanced experimental autoimmune encephalomyelitis (EAE) development.

99 in vitro and during experimental autoimmune encephalomyelitis (EAE) development.

100 ces inflammation and experimental autoimmune encephalomyelitis (EAE) disease scores via the ligand-ac

101 pment of spontaneous experimental autoimmune encephalomyelitis (EAE) during adolescence and early you

102 (NPCs) in mice with experimental autoimmune encephalomyelitis (EAE) impairs the accumulation of infl

103 17 cells that induce experimental autoimmune encephalomyelitis (EAE) in animals.

104 Using experimental autoimmune encephalomyelitis (EAE) in C57BL/6 mice, a well-studied

105 sclerosis in man or experimental autoimmune encephalomyelitis (EAE) in mice [poly(Y,E,A,K)(n), known

106 cells and exacerbate experimental autoimmune encephalomyelitis (EAE) in mice.

107 are desensitized to experimental autoimmune encephalomyelitis (EAE) induction, a model that is frequ

108 Following experimental autoimmune encephalomyelitis (EAE) induction, Treg-specific Il27ra(

109 Experimental autoimmune encephalomyelitis (EAE) is a mouse disease model with br

110 n the MS mouse model experimental autoimmune encephalomyelitis (EAE) is largely confined to induction

111 and its mouse model experimental autoimmune encephalomyelitis (EAE) is temporarily suppressed by pre

112 Experimental autoimmune encephalomyelitis (EAE) is the most common model of mult

113 relapsing-remitting experimental autoimmune encephalomyelitis (EAE) mice.

114 e first employed the experimental autoimmune encephalomyelitis (EAE) model and observed pronounced im

115 eak disease from the experimental autoimmune encephalomyelitis (EAE) model of MS ameliorates disease

116 Using the experimental autoimmune encephalomyelitis (EAE) model of MS, we determined that

117 Using the experimental autoimmune encephalomyelitis (EAE) model of MS, which recapitulates

118 patients and in the experimental autoimmune encephalomyelitis (EAE) model of MS.

119 riptome in ON in the experimental autoimmune encephalomyelitis (EAE) model of MS.

120 s development of the experimental autoimmune encephalomyelitis (EAE) model of multiple sclerosis (MS)

121 inflammation in the experimental autoimmune encephalomyelitis (EAE) model of multiple sclerosis (MS)

122 velop disease in the experimental autoimmune encephalomyelitis (EAE) model of multiple sclerosis.

123 nd in vivo using the experimental autoimmune encephalomyelitis (EAE) model of Th17 cell-driven pathol

124 Using the experimental autoimmune encephalomyelitis (EAE) model, we were able to study not

125 als in vivo using an experimental autoimmune encephalomyelitis (EAE) model.

126 in MS using a mouse experimental autoimmune encephalomyelitis (EAE) model.

127 sease in colitis and experimental autoimmune encephalomyelitis (EAE) models.

128 lerosis (MS) and its experimental autoimmune encephalomyelitis (EAE) models.

129 firmed in vivo in an experimental autoimmune encephalomyelitis (EAE) mouse model, which demonstrated

130 the CNS of mice with experimental autoimmune encephalomyelitis (EAE) originate in the gut and produce

131 and its animal model experimental autoimmune encephalomyelitis (EAE) remain undefined.

132 rotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) using AhR knockout mice.

133 n the development of experimental autoimmune encephalomyelitis (EAE) was explored.

134 ltration by B cells, experimental autoimmune encephalomyelitis (EAE) was induced in transgenic mice t

135 Disease severity of experimental autoimmune encephalomyelitis (EAE) was significantly exacerbated in

136 m in which mice with experimental autoimmune encephalomyelitis (EAE) were administered a sublethal do

137 his using a model of experimental autoimmune encephalomyelitis (EAE) with hippocampal degeneration in

138 the priming phase of experimental autoimmune encephalomyelitis (EAE), a CD4(+) T cell-driven mouse mo

139 Experimental autoimmune encephalomyelitis (EAE), a model for multiple sclerosis,

140 tation protects from experimental autoimmune encephalomyelitis (EAE), a model of MS.

141 7 cell generation in experimental autoimmune encephalomyelitis (EAE), a mouse model for human multipl

142 t the development of experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple scler

143 r Bhlhe40 to mediate experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple scler

144 In female mice with experimental autoimmune encephalomyelitis (EAE), a murine model of MS, adoptive

145 arthritis but also in experimental allergic encephalomyelitis (EAE), a murine model of multiple scle

146 d the development of experimental autoimmune encephalomyelitis (EAE), a preclinical model of MS, in p

147 ervous system during experimental autoimmune encephalomyelitis (EAE), a widely studied animal model o

148 UV light suppresses experimental autoimmune encephalomyelitis (EAE), a widely used animal model of M

149 me that occur during experimental autoimmune encephalomyelitis (EAE), an animal model for MS.

150 eveloped more severe experimental autoimmune encephalomyelitis (EAE), an animal model of human multip

151 n serum of mice with experimental autoimmune encephalomyelitis (EAE), an animal model of MS, as compa

152 oratory to attenuate experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

153 ord leptomeninges in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

154 o reduce severity of experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

155 disease severity in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

156 the pathogenesis of experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

157 ective properties in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple scl

158 ultiple sclerosis or experimental autoimmune encephalomyelitis (EAE), an established mouse model mimi

159 anscriptome occur in experimental autoimmune encephalomyelitis (EAE), an MS model.

160 pressing MOG-induced experimental autoimmune encephalomyelitis (EAE), and they downregulated chronic

161 ltiple sclerosis and experimental autoimmune encephalomyelitis (EAE), but how it impacts neuroinflamm

162 y used animal model, experimental autoimmune encephalomyelitis (EAE), does not present this aspect of

163 nd its animal model, experimental autoimmune encephalomyelitis (EAE), expansion of pathogenic, myelin

164 emyelination through experimental autoimmune encephalomyelitis (EAE), have provided valuable insights

165 xacerbated course of experimental autoimmune encephalomyelitis (EAE), highlighting a role for this nu

166 onding animal model, experimental autoimmune encephalomyelitis (EAE), is widely used to understand di

167 s TH17 cell-mediated experimental autoimmune encephalomyelitis (EAE), it also disrupts thymocyte deve

168 its mouse model, the experimental autoimmune encephalomyelitis (EAE), miRNA dysregulation has been ma

169 ve days to mice with experimental autoimmune encephalomyelitis (EAE), PLG-H particles had significant

170 ing the animal model experimental autoimmune encephalomyelitis (EAE), substantial evidence from patie

171 t animals undergoing experimental autoimmune encephalomyelitis (EAE), the animal model of MS, resulte

172 ltiple sclerosis and experimental autoimmune encephalomyelitis (EAE), the C-C chemokine receptor 6 (C

173 nd its animal model, experimental autoimmune encephalomyelitis (EAE), the mechanisms of how these cel

174 utically ameliorates experimental autoimmune encephalomyelitis (EAE), the mouse model of MS.

175 ry from the signs of experimental autoimmune encephalomyelitis (EAE), the mouse model of multiple scl

176 murine model of MS, experimental autoimmune encephalomyelitis (EAE), to evaluate the hypothesis that

177 oimmune model of MS, experimental autoimmune encephalomyelitis (EAE), together with a genetically div

178 animal model of MS, experimental autoimmune encephalomyelitis (EAE), we show here that injection of

179 11 or 511, in murine experimental autoimmune encephalomyelitis (EAE), we show here that loss of endot

180 nd its animal model, experimental autoimmune encephalomyelitis (EAE), we used intravital microscopy,

181 d autoimmune disease experimental autoimmune encephalomyelitis (EAE).

182 brain lesions during experimental autoimmune encephalomyelitis (EAE).

183 rotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE).

184 ffector CD4 cells in experimental autoimmune encephalomyelitis (EAE).

185 ord injury (SCI) and experimental autoimmune encephalomyelitis (EAE).

186 e neuroinflammation, experimental autoimmune encephalomyelitis (EAE).

187 odels of colitis and experimental autoimmune encephalomyelitis (EAE).

188 n (MOG)35-55-induced experimental autoimmune encephalomyelitis (EAE).

189 ting activity on the Experimental Autoimmune Encephalomyelitis (EAE).

190 d of its mouse model experimental autoimmune encephalomyelitis (EAE).

191 multiple sclerosis, experimental autoimmune encephalomyelitis (EAE).

192 of C57BL/6J mice to experimental autoimmune encephalomyelitis (EAE).

193 and its animal model experimental autoimmune encephalomyelitis (EAE).

194 ferred resistance to experimental autoimmune encephalomyelitis (EAE).

195 he manifestations of experimental autoimmune encephalomyelitis (EAE).

196 spinal cord, during experimental autoimmune encephalomyelitis (EAE).

197 idely used MS model, experimental autoimmune encephalomyelitis (EAE).

198 for the treatment of experimental autoimmune encephalomyelitis (EAE).

199 and the mouse model experimental autoimmune encephalomyelitis (EAE).

200 nd its animal model, experimental autoimmune encephalomyelitis (EAE).

201 in a murine model of experimental autoimmune encephalomyelitis (EAE).

202 responses, including experimental autoimmune encephalomyelitis (EAE).

203 tenuated severity of experimental autoimmune encephalomyelitis (EAE).

204 nd its animal model, experimental autoimmune encephalomyelitis (EAE).

205 nd its animal model, experimental autoimmune encephalomyelitis (EAE).

206 and demyelination in experimental autoimmune encephalomyelitis (EAE).

207 own-regulated during experimental autoimmune encephalomyelitis (EAE).

208 mice from developing experimental autoimmune encephalomyelitis (EAE).

209 s the progression of experimental autoimmune encephalomyelitis (EAE).

210 cing in animals with experimental autoimmune encephalomyelitis (EAE).

211 cal and histological experimental autoimmune encephalomyelitis (EAE); and identified CMKLR1 small mol

212 and its animal model experimental autoimmune encephalomyelitis (EAE); however, the role of NF-kappaB

213 ack myelin sheath in experimental autoimmune encephalomyelitis (EAE, an animal model for multiple scl

214 tion in an MS model (experimental autoimmune encephalomyelitis [EAE]) is partly due to central nervou

215 multiple sclerosis [experimental autoimmune encephalomyelitis, (EAE)] and a stabilized version of mo

216 otein A-1 vectors in experimental autoimmune encephalomyelitis, even at low doses devoid of hematolog

217 ated inflammation in experimental autoimmune encephalomyelitis has been extensively studied in an eff

218 ne processes involved in recovery from viral encephalomyelitis have been identified.

219 immune diseases like experimental autoimmune encephalomyelitis; however, its role in the pathogenesis

220 ctive local Ab within the CNS following JHMV encephalomyelitis.IMPORTANCE CD19 activation is known to

221 ly in the CNS during experimental autoimmune encephalomyelitis in a competitive setting than their wi

222 tic acid ameliorated experimental autoimmune encephalomyelitis in a therapeutic mouse model by regula

223 d the development of experimental autoimmune encephalomyelitis in adulthood in male, but not female,

224 of the recipients to experimental autoimmune encephalomyelitis in an IL-1 receptor-dependent manner.

225 is virus (SINV) can infect neurons and cause encephalomyelitis in mice.

226 ition of GSK3 limits experimental autoimmune encephalomyelitis in mice.

227 ) is an alphavirus that causes age-dependent encephalomyelitis in mice.

228 chronic progressive experimental autoimmune encephalomyelitis in nonobese diabetic mice, an experime

229 atitis, enteritis, respiratory diseases, and encephalomyelitis in the central nervous system (CNS).

230 nduced arthritis and experimental autoimmune encephalomyelitis, indicating that NKG2D is an important

231 promoted consistent experimental autoimmune encephalomyelitis induction, unlike mice challenged with

232 Experimental autoimmune encephalomyelitis is a CD4(+) T cell-mediated demyelinat

233 Experimental autoimmune encephalomyelitis is a model for multiple sclerosis.

234 ntigen-specific Bmem in the CNS during viral encephalomyelitis is largely undefined.

235 Myalgic Encephalomyelitis (ME), also known as Chronic Fatigue Sy

236 ic collection of data on measures of myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS).

237 phalopathy, encephalitis, acute disseminated encephalomyelitis, meningitis, ischemic and hemorrhagic

238 clinically-relevant experimental autoimmune encephalomyelitis mice model.

239 administered in the experimental autoimmune encephalomyelitis model of MS.

240 Similarly, in experimental autoimmune encephalomyelitis model of multiple sclerosis, TAGAP def

241 n a nonhuman primate experimental autoimmune encephalomyelitis model that an EBV-related lymphocrypto

242 severity in a mouse experimental autoimmune encephalomyelitis model, demonstrating the viability of

243 Using a neurotropic mouse hepatitis virus encephalomyelitis model, this study demonstrated an esse

244 on of T cells in the experimental autoimmune encephalomyelitis model.

245 plasma (pEVs) in an experimental autoimmune encephalomyelitis mouse model of central nervous system

246 ed in neurons in the experimental autoimmune encephalomyelitis mouse model of CNS inflammation and in

247 he late phase in the experimental autoimmune encephalomyelitis mouse model of multiple sclerosis.

248 a murine model of AD, experimental allergic encephalomyelitis murine model of demyelination and in p

249 ara- or post-infectious), acute disseminated encephalomyelitis (n = 9), with haemorrhage in five, nec

250 autoimmune disease, experimental autoimmune encephalomyelitis, often mediated by Th17 cells.

251 reported previously in patients with myalgic encephalomyelitis or chronic fatigue syndrome (ME/CFS),

252 T cells controls initiation of opticospinal encephalomyelitis (OSE), a murine model of multiple scle

253 The high incidence of acute disseminated encephalomyelitis, particularly with haemorrhagic change

254 isplay an attenuated experimental autoimmune encephalomyelitis phenotype accompanied by decreased CNS

255 similar ameliorated experimental autoimmune encephalomyelitis phenotype as Sephin1-treated mice, and

256 as did patients with non-acute disseminated encephalomyelitis presentations associated with lesions

257 tant to induction of experimental autoimmune encephalomyelitis, presumably by dampening the excessive

258 elination as well as experimental autoimmune encephalomyelitis, principal animal models of multiple s

259 A supplementation in experimental autoimmune encephalomyelitis reduced the severity of disease throug

260 relapsing-remitting experimental autoimmune encephalomyelitis (RREAE).

261 that TSSP increases experimental autoimmune encephalomyelitis severity by limiting central tolerance

262 The effect on experimental autoimmune encephalomyelitis severity was MHC class II allele depen

263 ntiation and reduced experimental autoimmune encephalomyelitis severity.

264 ally infects neurons in rodents to induce an encephalomyelitis similar to the human disease.

265 ression in experimental models of autoimmune encephalomyelitis-, SOD1(G93A) and rotenone models, mimi

266 eveloped spontaneous experimental autoimmune encephalomyelitis (spEAE), which was associated with sev

267 ic treatment reduced experimental autoimmune encephalomyelitis symptoms and was accompanied by an inc

268 s (55.9%, p = 0.013), and acute disseminated encephalomyelitis syndrome was more frequent in children

269 and in TH17-induced experimental autoimmune encephalomyelitis (TH17-EAE).

270 re more susceptible to experimental allergic encephalomyelitis than mice sufficient for the HR (13R(+

271 In experimental autoimmune encephalomyelitis, the animal model for MS, myelin-react

272 adult marmosets with experimental autoimmune encephalomyelitis, vessel-wall fibrosis was detected ear

273 mptive ancestor-and porcine hemagglutinating encephalomyelitis virus (PHEV).

274 Intracerebral Theiler's murine encephalomyelitis virus (TMEV) infection in mice induces

275 Theiler's murine encephalomyelitis virus (TMEV) infection induces a well-

276 Theiler's murine encephalomyelitis virus (TMEV) infection of the CNS is c

277 Infection by Theiler's murine encephalomyelitis virus (TMEV) is a model for neurologic

278 persistently infected with Theiler's murine encephalomyelitis virus (TMEV) undergo apoptosis, result

279 lecules to brain atrophy in Theiler's murine encephalomyelitis virus (TMEV)-infected transgenic FVB m

280 analyzed PRF in the related Theiler's murine encephalomyelitis virus (TMEV).

281 peaks from murine parvovirus, norovirus, and encephalomyelitis virus samples, obtained in our improve

282 with the Daniel's strain of Theiler's murine encephalomyelitis virus were treated with the FDA-approv

283 es following infection with Theiler's Murine Encephalomyelitis Virus, a neurotropic virus.

284 roteoglycan accumulation in Theiler's murine encephalomyelitis virus-induced demyelinating disease.

285 of multiple sclerosis, the Theiler's murine encephalomyelitis virus-induced demyelinating disease.

286 sions in the spinal cord of Theiler's murine encephalomyelitis virus-infected mice.

287 sions in the spinal cord of Theiler's murine encephalomyelitis virus-infected mice.

288 cal and histological experimental autoimmune encephalomyelitis was observed in approximately 29% of i

289 multiple sclerosis, experimental autoimmune encephalomyelitis, we demonstrate that in vivo administr

290 multiple sclerosis, experimental autoimmune encephalomyelitis, we demonstrated that Sephin1 delayed

291 In autoimmune encephalomyelitis, we find that most expanded CD4(+) T c

292 rom a mouse model of experimental autoimmune encephalomyelitis, we observed that Ag potency modulates

293 key brain induced by experimental autoimmune encephalomyelitis, which is the most-studied animal mode

294 verity of subsequent experimental autoimmune encephalomyelitis, which was associated with an expansio

295 lting in less severe experimental autoimmune encephalomyelitis, which was associated with decreased i

296 virus A59 strain [MHV-A59]) developed severe encephalomyelitis with hind-limb paralysis and succumbed

297 developed fulminant experimental autoimmune encephalomyelitis with massive CNS infiltration of activ

298 ied IgGs from four patients with progressive encephalomyelitis with rigidity and myoclonus or stiff p

299 tial proportion of patients with progressive encephalomyelitis with rigidity and myoclonus, and less

300 are a significant and growing cause of viral encephalomyelitis worldwide.