ライフサイエンスコーパス: end-diastole

1 the mitral valve leaflets at end systole and end diastole.

2 d measurement of balloon luminal pressure at end diastole.

3 normalized ventricular elastance at arterial end diastole.

4 stimated normalized ventricular elastance at end diastole.

5 umes and sphericity index at end-systole and end-diastole.

6 =.005), indicating a less circular shape at end-diastole.

7 and mass were calculated at end-systole and end-diastole.

8 c magnetic resonance scans of 91 subjects at end-diastole.

9 myocardial wall thickness increased in both end diastole (11.5 +/- 2.7 to 13.7 +/- 2.4 mm, p = 0.03)

10 t significantly lower in the Physio group at end diastole (8.4+/-3.8, 6.7+/-2.3, and 3.4+/-0.6 mm, re

11 in Physio (23+/-11%, 24+/-7%, and 12+/-2% at end diastole and 42+/-17%, 37+/-17%, and 21+/-10% at end

12 difference in capillary blood volume between end diastole and end systole at baseline.

13 PM were also projected onto the MA plane at end diastole and end systole to assess PM dynamics.

14 ented contours, and a correspondence between end diastole and end systole was computed with a novel a

15 with triggering performed separately at both end diastole and end systole.

16 reased left ventricular internal diameter at end-diastole and decreased fractional shortening.

17 annulus and LV base-apex length increased at end-diastole and end-systole (all +1 mm, P<0.05).

18 MR severity, LV volumes at end-diastole and end-systole, and LA volumes were measur

19 Reduction of LV volumes at end-diastole and LA volumes, but not LV volumes at end-s

20 hree-dimensional RV models were segmented at end-diastole and processed using statistical shape model

21 in short axis and length) were calculated in end-diastole at stress and rest.

22 In end diastole, both the ostial and distal intramural sect

23 Baseline LV sphericity at end diastole (ED) (r = 0.13, p = 0.6) did not correlate

24 and three-dimensional marker coordinates at end diastole (ED) and end systole (ES) were computed.

25 rtrophy (interventricular septal diameter at end-diastole >1.5 cm and diastolic dysfunction).

26 even excessive, LV trabeculation measured in end-diastole in asymptomatic population-representative i

27 The first was over the left ventricle at the end-diastole including the aortic valve plane area, and

28 dels were constructed from the MRI images at end-diastole, isovolumic systole, peak-systole and end-s

29 t was first determined from their respective end-diastole LV geometries segmented from the cardiac ma

30 ase in left ventricular internal diameter at end-diastole (LVIDd; 95% CI, -0.92 to -0.67; P = 2.3 x 1

31 gree of structural (LV internal dimension at end-diastole [LVIDd]) and functional (LV ejection fracti

32 in early diastole, and minimum MA area near end-diastole; maximum area reduction was 12+/-1% (P< or

33 ngle-fixed region of interest (ROI) drawn at end-diastole, often underestimates the left ventricular

34 od at end-systole (r = 0.91, p < 0.0001) and end-diastole (r = 0.86, p < 0.0001).

35 calculated using volumetric measurements at end diastole ([right atrial+atrialized right ventricular

36 We hypothesized that (1) in end diastole, the intramural segment is narrower, more e

37 s the percent change in the cavity area from end diastole to end systole (fractional area change [FAC

38 nd functional MR, reduction in LV volumes at end-diastole was associated with degree of residual MR a

39 f LVNC (noncompacted/compacted ratio >2.3 in end-diastole) was confirmed in all patients.

40 mage series reconstructed at end systole and end diastole were used to measure LV-LAS.

41 e closure time was defined as the time after end diastole when the distance between leaflet edge mark

42 ntricular volume ratio (LRVR) was defined at end diastole, whereas the left-to-right atrial volume ra