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1 nsion, results in enlarged kidneys and often end stage kidney disease.
2 course of disease, preceding progression to end stage kidney disease.
3 th more frequent remission and less frequent end stage kidney disease.
4 tors are superior to other drugs in reducing end stage kidney disease.
5 ectories were associated with progression to end stage kidney disease.
6 die prematurely than they are to progress to end stage kidney disease.
7 podocyte injury and promotes progression to end-stage kidney disease.
8 ion in the form of AKI, progressive CKD, and end-stage kidney disease.
9 total mortality, nonvascular mortality, and end-stage kidney disease.
10 iovascular adverse events and progression to end-stage kidney disease.
11 protein requirements in patients with CKD or end-stage kidney disease.
12 y 3 wk after birth and progressed rapidly to end-stage kidney disease.
13 patients with atherosclerosis, diabetes, and end-stage kidney disease.
14 o prevent its development and progression to end-stage kidney disease.
15 nd, in steroid-resistant nephrotic syndrome, end-stage kidney disease.
16 causing early-onset nephrotic syndrome with end-stage kidney disease.
17 /min per 1.73 m(2) (stage 3 to 5), excluding end-stage kidney disease.
18 ey transplant for plasma cell dyscrasias and end-stage kidney disease.
19 mated glomerular filtration rate by >=40% or end-stage kidney disease.
20 ow-up of 6.5 years, 1,688 patients developed end-stage kidney disease.
21 pies/mL, and 55% of these patients developed end-stage kidney disease.
22 as the treatment of choice for children with end-stage kidney disease.
23 y ciliopathy and leading cause of hereditary end-stage kidney disease.
24 of morbidity and mortality in patients with end-stage kidney disease.
25 hly prevalent and can eventually progress to end-stage kidney disease.
26 kidney disease (DKD) is the leading cause of end-stage kidney disease.
27 th nephrotic syndrome that often progress to end-stage kidney disease.
28 iatric and adult patients, which can lead to end-stage kidney disease.
29 ffects on glomerular function or the risk of end-stage kidney disease.
30 independently associated with progression to end-stage kidney disease.
31 events and mortality as well as the risk for end-stage kidney disease.
32 ansplantation (KT) is the best treatment for end-stage kidney disease.
33 ion (KT) is the most effective treatment for end-stage kidney disease.
34 ) is the optimal treatment for children with end-stage kidney disease.
35 is the best health option for patients with end-stage kidney disease.
36 diverse and can result in the development of end-stage kidney disease.
37 ansplantation is the preferred treatment for end-stage kidney disease.
38 ogression of chronic kidney disease (CKD) to end-stage kidney disease.
39 disease and one of the most common causes of end-stage kidney disease.
40 decompensated HF, and acute kidney injury or end-stage kidney disease.
41 for type 1 diabetes mellitus and concurrent end-stage kidney disease.
42 despite having the highest global burden of end-stage kidney disease.
43 antation (KT) is the treatment of choice for end-stage kidney disease.
44 The condition introduces a high risk for end-stage kidney disease.
45 0 mL/min/1.73 m2, new-onset albuminuria, and end-stage kidney disease.
46 sistently associated with subsequent risk of end-stage kidney disease.
47 h type 2 diabetes at high risk of developing end-stage kidney disease.
48 ollow-up and information on the incidence of end-stage kidney disease.
49 of renal replacement therapy for those with end-stage kidney disease.
50 eight (LMW) proteinuria that can progress to end-stage kidney disease.
51 rimary outcomes were all-cause mortality and end-stage kidney disease.
52 lly irreversible process causing chronic and end-stage kidney disease.
53 , were the most effective strategies against end-stage kidney disease.
54 hanism to delay the onset and progression of end-stage kidney disease.
55 interstitial fibrosis underlies all forms of end-stage kidney disease.
56 as "myeloma kidney" that often progresses to end-stage kidney diseases.
61 the following: (1) all-cause mortality; (2) end-stage kidney disease; (3) a decline in estimated glo
62 ronic obstructive pulmonary disease, 3785 of end-stage kidney disease, 579 of cirrhosis, and 6271 of
63 less VVI pacemakers were more likely to have end-stage kidney disease (690 [12.0%] vs 226 [2.3%]; P <
64 tive risk of the renal-specific composite of end-stage kidney disease, a doubling of the creatinine l
66 ined 30% or more decline in eGFR or reaching end-stage kidney disease, accounting for a potentially r
67 dney disease and the fourth leading cause of end-stage kidney disease, accounting for over 50% of pre
68 sistently associated with subsequent risk of end-stage kidney disease across a range of cohorts, lend
69 its definition (Risk, Injury, Failure, Loss, End-stage kidney disease, Acute Kidney Injury Network, a
72 imary outcome of interest was development of end-stage kidney disease after a baseline period of 2 ye
73 (AHR, 2.23; 95% CI, 2.14-2.32), dialysis or end-stage kidney disease (AHR, 2.34; 95% CI, 2.24-2.44),
74 sts in Patients With Atrial Fibrillation and End-Stage Kidney Disease), an investigator-initiated PRO
75 a heightened risk of developing chronic and end-stage kidney disease, an association that is largely
77 ic uremic syndrome (aHUS) is a rare cause of end-stage kidney disease and associated with poor outcom
78 ing problem across the world and can lead to end-stage kidney disease and cardiovascular disease.
82 omerulosclerosis (FSGS) is a common cause of end-stage kidney disease and frequently recurs after kid
83 It occurs predominantly in patients with end-stage kidney disease and has high mortality, elusive
84 lication of type 2 diabetes that can lead to end-stage kidney disease and is associated with high car
85 ar calcification is common in the setting of end-stage kidney disease and is associated with increase
87 review current data on the global burden of end-stage kidney disease and the distribution of major r
89 versies Conference on CAD and CKD (including end-stage kidney disease and transplant recipients) seek
90 E criteria (Risk, Injury, Failure, Loss, and End-stage kidney disease) and assessed using logistic re
91 admissions to hospital for heart failure and end stage kidney disease, and possibly cardiovascular de
92 o dialysis, 7% had progressed to dialysis or end-stage kidney disease, and 7% had been readmitted for
93 ion or died of kidney disease, 335 developed end-stage kidney disease, and 943 had acute kidney injur
94 ), and non-vascular and all-cause mortality, end-stage kidney disease, and adverse events, as well as
97 CVD, stroke, coronary artery disease (CAD), end-stage kidney disease, and mortality using Cox propor
99 Diabetic nephropathy is a major cause of end-stage kidney disease, and overactivity of the endoca
100 diagnosis, 5-20% of patients with LN develop end-stage kidney disease, and the multiple comorbidities
101 enrolled in Medicare Advantage, did not have end-stage kidney disease, and were not institutionalized
103 factors associated with early progression to end-stage kidney disease assessed by Cox proportional mu
104 significantly associated with lower risk for end-stage kidney disease at week 52 (RR: 0.96: 0.95-0.97
105 SGS), HIV-associated nephropathy (HIVAN) and end-stage kidney disease attributed to hypertension (H-E
106 The growing number of elderly patients with end-stage kidney disease awaiting transplantation has re
107 ant morbidity and mortality to patients with end-stage kidney disease but are resistant to therapy.
108 the optimal treatment for most patients with end-stage kidney disease but organ shortage is a major c
109 y transplantation is the optimal therapy for end-stage kidney disease but requires lifelong immunosup
111 transplant (LDKT) is the ideal treatment for end-stage kidney disease, but racial disparities in LDKT
113 increased risk for post-donation mortality, end-stage kidney disease, cardiovascular disease, non-pr
116 5 groups of EHMs: chronic kidney disease and end-stage kidney disease (CKD and ESKD), type 2 diabetes
119 lar morbidity and mortality in patients with end-stage kidney disease could be partially caused by ex
120 defined by Risk, Injury, Failure, Loss, and End-stage kidney disease criteria and new need for renal
121 njury, Failure, Loss of kidney function, and End-stage kidney disease criteria for acute kidney injur
123 rom baseline; first eGFR <15 mL/min/1.73 m2; end-stage kidney disease; death from kidney causes).
124 nt of ammonia in the breath of patients with end-stage kidney disease demonstrated its significant re
125 sease (DKD) remains the most common cause of end-stage kidney disease despite multifactorial interven
126 =15 mL/min/1.73 m2, dialysis, transplant, or end-stage kidney disease diagnosis), and at least 30%, 4
128 study two variants in APOL1 associated with end-stage kidney disease discovered by admixture mapping
130 erum creatinine (sustained for >=30 days) or end-stage kidney disease (eGFR <15 mL/min per 1.73 m(2)
131 nors relative to the number of patients with End Stage Kidney Disease (ESKD) has led to prolonged wai
133 timated glomerular filtration rate (eGFR) or end stage kidney disease (ESKD) using clinical risk fact
134 with low or low-moderate risk of developing end stage kidney disease (ESKD), and a weak recommendati
135 follow-up and included all-cause mortality, end stage kidney disease (ESKD), serious infections, dis
136 omerulosclerosis (FSGS) is a common cause of end-stage kidney disease (ESKD) across the lifespan.
137 the other hand, are at the increased risk of end-stage kidney disease (ESKD) after donation compared
140 stry to assess adults (aged >=18 years) with end-stage kidney disease (ESKD) and adult KT candidates.
141 Diabetic kidney disease (DKD) can lead to end-stage kidney disease (ESKD) and mortality; however,
142 ary care perspective emphasizing the risk of end-stage kidney disease (ESKD) and need for renal repla
143 to recovery occurred in 4% of patients with end-stage kidney disease (ESKD) and was more common amon
144 k for type 2 diabetic (T2D) and non-diabetic end-stage kidney disease (ESKD) appear strong in African
148 nerenone significantly reduced the hazard of end-stage kidney disease (ESKD) by 20% versus placebo (0
151 y associated with risk of the development of end-stage kidney disease (ESKD) during 10-year follow-up
152 hine learning (ML) in predicting the risk of end-stage kidney disease (ESKD) from patients with chron
153 er, long-term outcomes such as mortality and end-stage kidney disease (ESKD) have been rarely reporte
154 sociation with CKD risk factors and incident end-stage kidney disease (ESKD) in 4,843 participants of
157 It is estimated that >50% of patients with end-stage kidney disease (ESKD) in low-resource countrie
158 y, major adverse cardiac events (MACEs), and end-stage kidney disease (ESKD) in patients with dialysi
159 intraglomerular hemodynamic dysfunction with end-stage kidney disease (ESKD) in people with type 2 di
161 year following the index hospitalization and end-stage kidney disease (ESKD) in the 10 years followin
162 change using transplant rates normalized to end-stage kidney disease (ESKD) incidence have not been
163 ranging from stage 1 to stage 5 CKD, whereas end-stage kidney disease (ESKD) is defined as permanent
165 ents with diabetes and advanced-stage CKD or end-stage kidney disease (ESKD) is not well understood.
169 in in-hospital treatment and outcomes among end-stage kidney disease (ESKD) patients receiving perit
171 estigated risks of mortality, heart failure, end-stage kidney disease (ESKD), and atherosclerotic car
172 with maintenance dialysis for patients with end-stage kidney disease (ESKD), but it is limited to ap
173 s follows: first-degree relative(s) with CKD/end-stage kidney disease (ESKD), early-onset CKD, focal
174 , depression, chronic kidney disease without end-stage kidney disease (ESKD), ESKD, liver disease, ca
175 to decline in glomerular filtration rate and end-stage kidney disease (ESKD), has been replaced by a
176 nances health care for most US patients with end-stage kidney disease (ESKD), regardless of age.
177 on of proteinuric kidney diseases leading to end-stage kidney disease (ESKD), requiring renal replace
185 condary outcomes included the development of end-stage kidney disease (ESKD); a composite of a decrea
186 a has been suggested to increase the risk of end-stage kidney disease (ESKD); however, most studies w
187 The increased burden of chronic kidney and end-stage kidney diseases (ESKD) in populations of Afric
188 d the composite clinical endpoint of treated end-stage kidney disease, estimated glomerular filtratio
189 han 1% absolute reduction in 10-year risk of end-stage kidney disease, even at early stages of chroni
193 e burden of chronic kidney disease (CKD) and end-stage kidney disease falls disproportionately on Bla
194 nd few or no liver cysts; 8 subjects reached end-stage kidney disease from 62 to 91 years of age.
195 -for-service Medicare receiving dialysis for end-stage kidney disease from January 2017 to November 2
197 sclerosis (FSGS) and hypertension-attributed end-stage kidney disease (H-ESKD) are associated with tw
201 nsplant waitlisting within the first year of end-stage kidney disease have remained unchanged over th
202 r renal failure to an accepted treatment for end-stage kidney disease, heart disease, liver disease,
203 % CI, 0.43-0.84), and acute kidney injury or end-stage kidney disease (HR, 0.73; 95% CI, 0.57-0.92).
204 any biochemical disturbances associated with end-stage kidney disease improve in the first year after
206 on between change in albuminuria and risk of end-stage kidney disease in a large individual participa
209 sease, is the most frequent genetic cause of end-stage kidney disease in children and young adults.
210 Review describes the incidence and causes of end-stage kidney disease in children on long-term dialys
212 e in albuminuria as a surrogate endpoint for end-stage kidney disease in clinical trials of progressi
214 epresents the most frequent genetic cause of end-stage kidney disease in the first three decades of l
215 igher levels of urinary PEDF associated with end-stage kidney disease in the Seattle Kidney Study, wi
216 disease is the leading cause of chronic and end-stage kidney disease in the United States and worldw
220 universal transplantation as the therapy for end-stage kidney disease include the economic limitation
224 to kidney transplantation for patients with end-stage kidney disease is a national clinical and poli
226 guide clinicians in developing comprehensive end-stage kidney disease Life-Plans with hemodialysis ac
229 isease, stage 4 or 5 chronic kidney disease, end-stage kidney disease, metastatic cancer, and no bloo
232 The primary composite outcome was incident end-stage kidney disease or a 40% decline in estimated g
234 and incidence of myocardial infarctions, and end-stage kidney disease or kidney failure (moderate to
235 ed period of 8 months to 4 years, subsequent end-stage kidney disease or mortality follow-up data, an
236 lar filtration rate (eGFR) decline, incident end-stage kidney disease, or all-cause mortality over 2
237 idney failure (confirmed 40% eGFR reduction, end-stage kidney disease, or all-cause mortality), and s
238 [or increase in creatinine], progression to end-stage kidney disease, or death attributable to kidne
239 a sustained decline in eGFR of 50% or more, end-stage kidney disease, or death from a kidney disease
240 at least 1 glucose-lowering medication, had end-stage kidney disease, or had stage 5 chronic kidney
242 only results in higher costs but also worse end stage kidney disease outcomes for an already underse
243 eterogeneity and exponential accumulation of end-stage kidney diseases over time prevent long-term pr
245 and to determine the prognostic factors for end-stage kidney disease patients admitted to the ICU.
246 n among 34 857 incident, adult (18-79 years) end-stage kidney disease patients from 690 dialysis faci
248 (PBUTs) cause various deleterious effects in end-stage kidney disease patients, because their removal
249 cs, clinical outcomes in SOT recipients, and end-stage kidney disease patients, transplant activity d
251 n patients with type 1 diabetes mellitus and end-stage kidney disease prevents worsening of diabetic
252 f these RNases were evident in patients with end stage kidney disease prior to PD initiation, and ome
253 heart failure, lower-extremity complication, end-stage kidney disease, proliferative retinopathy, tre
254 In individuals with type 1 diabetes and end-stage kidney disease, PT was associated with an impr
255 research using hard renal endpoints such as end stage kidney disease rather than surrogate markers m
257 aortic valve calcification in patients with end-stage kidney disease receiving hemodialysis in addit
259 and serious complication in individuals with end-stage kidney disease receiving peritoneal dialysis.
262 he past few decades, rates of progression to end-stage kidney disease remain high with no beneficial
263 outcomes were kidney events (a composite of end-stage kidney disease, renal death, development of an
264 ine estimated glomerular filtration rate and end stage kidney disease requiring renal replacement the
267 as meeting Risk, Injury, Failure, Loss, and End-stage kidney disease (RIFLE) Injury or Failure crite
268 here was no detectable effect on the risk of end-stage kidney disease (RR: 0.85; 95% CI: 0.49 to 1.49
269 001) but was not associated with the risk of end-stage kidney disease (SHR, 0.96 [95% CI, 0.62-1.50];
270 failures and adverse kidney outcomes such as end-stage kidney disease, significantly reduced kidney f
271 roved substantially in the past decade, with end-stage kidney disease still developing in 5-30% of pa
272 ency, once established, tends to progress to end-stage kidney disease, suggesting some common mechani
273 s of kidney transplantation as a therapy for end-stage kidney disease that surpasses dialysis treatme
275 lerability may be a problem in patients with end-stage kidney disease.The impact of HCV infection on
276 n of modifiable risk factors for chronic and end-stage kidney disease to low- and middle-income count
278 es among 346 828 Medicare beneficiaries with end-stage kidney disease treated with hemodialysis who u
280 ve veterans with type 2 diabetes and without end-stage kidney disease treated with metformin who star
281 rt study with 127 AA and white patients with end-stage kidney disease undergoing evaluation for KT (D
283 entage change in albuminuria with subsequent end-stage kidney disease using Cox regression in each co
284 group; the risk of the composite of death or end-stage kidney disease was 16.9% and 19.6%, respective
285 to 0.81; P<0.001), and the relative risk of end-stage kidney disease was lower by 32% (hazard ratio,
287 Patients with a faster progression toward end-stage kidney disease were at higher risk of developi
289 E criteria (risk, injury, failure, loss, and end-stage kidney disease) were employed in identifying c
291 85%, and 69% of patients remained free from end-stage kidney disease, which compares favorably to a
292 of renal significance, face a high burden of end-stage kidney disease, which limits survival and qual
293 timated glomerular filtration rate (eGFR) or end-stage kidney disease, whichever occurred first].
294 ta System Registry to identify patients with end-stage kidney disease who initiated dialysis between
295 tudy in individuals with type 1 diabetes and end-stage kidney disease who underwent PT between 1999 a
296 ommend follow-up and treatment.Patients with end-stage kidney disease without cirrhosis and selected
300 antation remains the preferred treatment for end-stage kidney disease, yet donor shortages limit its