ライフサイエンスコーパス: endodontic

1 d promising approach for future regenerative endodontics.

2 nd improving dental materials, especially in endodontics.

3 scaffolds and/or stem cells in regenerative endodontics.

4 mately one-third of the RAG-2 mice developed endodontic abscesses, while no immunocompetent controls

5 tically assessing the recent advancements in endodontic AI research for clinical applications, includ

6 in clinical practice and requires effective endodontic and regenerative periodontal therapy.

7 membrane in association with teeth that had endodontic and/or periodontic involvement, and a nearly

8 the impact of technical advances for modern endodontics and endodontic microsurgery on the long-term

9 The outcomes of traditional periodontal, endodontic, and prosthodontic treatment approaches are c

10 ernary ammonium methacrylates (QAMs) against endodontic bacteria and their biofilms.

11 This allows for effective eradication of endodontic biofilms via on-site free radical generation

12 e for detecting vital bacteria within mature endodontic biofilms, with an improved sensitivity over c

13 has been a steady increase in AI studies in endodontics but limited clinical application.

14 As there was a periapical radiolucency, an endodontic consultation was obtained.

15 ions and challenges imposed by the nature of endodontic data sets, AI transparency and generalization

16 o attenuate inflammation and bone erosion in endodontic disease and other inflammation-related osteol

17 tooth-factors, age, diagnosis of caries and endodontic disease and percentage of bone loss at baseli

18 ur results demonstrated that ODN can inhibit endodontic disease development, bone erosion, and immune

19 knockdown in periapical tissues can inhibit endodontic disease development, bone resorption, and inf

20 ooth brushing, initial bone loss, caries and endodontic disease in predicting tooth survival in a pri

21 Endodontic disease is characterized by inflammation and

22 We generated endodontic disease mice through bacterial infection as a

23 Dental caries may lead to endodontic disease, where the bacterial infection progre

24 bone resorption by 80% in the mouse model of endodontic disease.

25 improve the health of those who suffer from endodontic disease.

26 ts regarding systemic health consequences of endodontic disease.

27 ing RvE1 as a novel therapeutic for treating endodontic diseases.

28 faecalis and can potentially be applied for endodontic disinfection.

29 ion (BM), in preventing major complications (endodontic/extractions/pain)-a patient-centred outcome-d

30 tion was coronal tooth fracture, followed by endodontic failure such as root fracture.

31 acent to teeth with apical lesions; previous endodontic failures, retained root fragments, etc.

32 ts with apical periodontitis, an intractable endodontic infection affecting half of the global adult

33 Most common etiology of apical RPI is endodontic infection from neighboring teeth, which was d

34 tic lesion (EL) is a common manifestation of endodontic infection where Porphyromonas endodontalis is

35 Bacterial DNA typical for endodontic infection, mainly oral viridans streptococci,

36 so on a cross-sectional tooth slice model of endodontic infection.

37 and can be used to identify a wider range of endodontic-infection-related bacteria including the pres

38 results can help explain the persistence of endodontic infections and demonstrate a new virulence me

39 Dental caries, endodontic infections and periodontal diseases are bacte

40 protects the host from the dissemination of endodontic infections and that RAG-2 mice are more susce

41 sults indicate that bacterial communities in endodontic infections are more diverse than previously d

42 was to explore the microbial communities of endodontic infections at their apical portion by 16S rRN

43 The polymicrobial etiology of endodontic infections has long been established.

44 Bacterial diversity in endodontic infections has not been sufficiently studied.

45 re cultivable; hence, bacterial diversity in endodontic infections is underestimated.

46 fect of TLR4 deficiency on host responses to endodontic infections is unknown.

47 ed a more diverse microflora associated with endodontic infections than that revealed by cultural met

48 is study investigated bacterial diversity in endodontic infections through taxonomic classification b

49 s from 15 de novo and 26 refractory cases of endodontic infections were assessed.

50 iques can detect the presence of bacteria in endodontic infections when culture techniques yield a ne

51 has been identified in peri-implantitis, in endodontic infections, and in patients with localized ag

52 Endodontic infections, in which oral bacteria access the

53 tive in reducing or eliminating endotoxin in endodontic infections-single or multiple-session treatme

54 bic polymicrobial consortium responsible for endodontic infections.

55 quencings were performed on 7 specimens from endodontic infections.

56 A hybridization to examine the microbiota of endodontic infections.

57 y-six samples were collected from teeth with endodontic infections.

58 et of the oral microflora is responsible for endodontic infections.

59 Endodontic instruments were incorporated into 168 root c

60 Separated endodontics instruments and high-density obturating mate

61 Endodontics is the dental specialty foremost concerned w

62 The goal of regenerative endodontics is to reinstate normal pulp function in necr

63 An endodontic lesion (EL) is a common manifestation of endo

64 infections, such as periodontal diseases or endodontic lesion formation.

65 radiographically as a common periodontal or endodontic lesion.

66 Periodontitis associated with endodontic lesions are among the most daunting diagnosti

67 Chronic periodontic-endodontic lesions are not uncommon in clinical practice

68 the diagnosis and management of periodontal-endodontic lesions is also presented.

69 dity in chronic otitis media, periodontitis, endodontic lesions, and loosening of orthopedic implants

70 healing process of periapical tissues after endodontic micro-surgery procedure.

71 The endodontic microbiota was far more complex than previous

72 echnical advances for modern endodontics and endodontic microsurgery on the long-term prognosis of to

73 the relationship between incident lesions of endodontic origin and time to CHD diagnosis.

74 incident radiographically evident lesions of endodontic origin were related to development of coronar

75 ose < or = 40 years old, incident lesions of endodontic origin were significantly associated with tim

76 to periodontal disease as well as lesions of endodontic origin.

77 s commonly associated with these infections (endodontic pathogens [EP]), i.e., Fusobacterium nucleatu

78 d and infected with a mixture of four common endodontic pathogens, and bone destruction was determine

79 We conclude that the endodontic pathogens, particularly P. intermedia, can ef

80 ns, including the detection and diagnosis of endodontic pathologies such as periapical lesions, fract

81 hogen that has recently been associated with endodontic pathology.

82 studies that investigated the maternal true endodontic-periodontal lesion (EPL) in offspring are sca

83 Treatment of concomitant endodontic-periodontal lesions remains a challenge in cl

84 promoting periodontal healing in concomitant endodontic-periodontal lesions.

85 viable modality in the treatment of chronic endodontic/periodontal lesions.

86 us cell carcinoma that presented as a benign endodontic-periodontic lesion with a 7-mm periodontal po

87 Zirconia endodontic posts (n = 23) were used to function as impla

88 Regenerative endodontic procedures (REPs) are a treatment alternative

89 Regenerative endodontic procedures are stem cell-based treatments for

90 The translation of regenerative endodontic procedures into treating mature teeth depends

91 gative effect on the outcome of regenerative endodontic procedures.

92 teeth with apical rarefactions had received endodontic procedures.

93 prosthetic (risk difference [RD], 0.21) and endodontic (RD, 0.11) treatment was higher among histori

94 More recently, cell-based approaches in endodontic regeneration based on pulpal mesenchymal stem

95 pain (n = 210, 78.95%), and had restorative/endodontic-related clinical findings (n = 55, 20.68%).

96 uators (periodontist, periodontics resident, endodontics resident, dental student) assessed the photo

97 ment (IET), non-surgical (NET), and surgical endodontic retreatments (SET).

98 t common bacteria isolated from recalcitrant endodontic (root canal) infections.

99 Species commonly detected in endodontic samples included P. tannerae, Prevotella oris

100 al pathogen Porphyromonas gingivalis and the endodontic species Enterococcus faecalis were grown to e

101 depth, PI, and BOP), and background factors (endodontic status, smoking status, and surgeon's experie

102 ations on diagnosis, treatment planning, and endodontic success.

103 e lesions did not affect the success rate of endodontic surgeries.

104 n had been present for 42 years since having endodontic surgery at teeth #8 and #9 after a traumatic

105 regeneration techniques in combination with endodontic surgery using an induced perio-endo defect mo

106 l in the healing of periapical lesions after endodontic surgery.

107 illing materials are commonly used following endodontic surgical procedures; however, their effect on

108 old for use within regenerative (reparative) endodontic techniques.

109 h conditions were assessed, including decay, endodontic therapy (root canal therapy [RCT]), and/or ex

110 estore the lesion, and facilitate subsequent endodontic therapy and restoration.

111 Endodontic therapy often faces the challenge of complete

112 e present findings demonstrate that adequate endodontic therapy performed >/=6 months before surgical

113 f the tooth with nerve fiber ablation (i.e., endodontic therapy), generally alleviates pain and allow

114 canal system and are therefore promising for endodontic therapy.

115 ical and difficult concerns for regenerative endodontics therapy (RET).

116 lation, performed to evaluate the quality of endodontic treatment (ET) and the condition of the peria

117 The impact of the endodontic treatment (ET) of the tooth on the healing po

118 oth implants (SI) to teeth receiving initial endodontic treatment (IET), non-surgical (NET), and surg

119 OR, 1.52), restorative treatment (OR, 1.35), endodontic treatment (OR, 1.63), and more than 10 oral c

120 care (PDC), PDC without an extraction and/or endodontic treatment (PDC without Ext/Endo), PDC with an

121 , with minimum 4 weeks between completion of endodontic treatment and actual implant placement.

122 pair treatments include fillings for caries, endodontic treatment for pulp necrosis, and dental impla

123 A major cause of apical periodontitis after endodontic treatment is the bacterial infiltration which

124 ant and adjacent tooth and/or with time from endodontic treatment of adjacent tooth to implant placem

125 ded in this study (N = 20) were referred for endodontic treatment of mature teeth with apical lesions

126 During surgical endodontic treatment, periapical lesion tissue was colle

127 d differentiation potential for personalized endodontic treatment.

128 natomical variations that can complicate the endodontic treatment.

129 properties can increase the success rate of endodontic treatment.

130 special attention and careful assessment for endodontic treatment.

131 for dentin/pulp tissue engineering in future endodontic treatment.

132 for dentin/pulp tissue engineering in future endodontic treatment.

133 n of human dental pulp stem cells (DPSCs) in endodontic treatment.

134 4 chronic apical abscess) underwent surgical endodontic treatment.

135 t term but the survival and success rates in endodontic treatments dropped more rapidly than single t

136 of this study, single tooth implants and the endodontic treatments yielded predictable survival and s

137 with the survival and success of implant and endodontic treatments.

138 e, AI will significantly affect the everyday endodontic workflow, education, and continuous learning.