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1 f DNA replication in the absence of mitosis (endomitosis).
2 ough the stochastic occurrence of premeiotic endomitosis.
3  through the ectopic induction of premeiotic endomitosis.
4 s arise from mononucleated Hodgkin cells via endomitosis.
5 es are platelet precursor cells that undergo endomitosis.
6 hly expressed in young MKs, in parallel with endomitosis.
7  division but continued to accumulate DNA by endomitosis.
8  for understanding the molecular basis of MK endomitosis.
9  from polyploid megakaryocytes which undergo endomitosis.
10 er a phase of DNA synthesis, thus leading to endomitosis.
11 totic cell cycle while not detectable during endomitosis.
12 oiesis, but is dispensable for megakaryocyte endomitosis.
13 ls undergoing a mitotic cell cycle or during endomitosis.
14  within a single nucleus, a process known as endomitosis.
15 is currently unknown how human cells undergo endomitosis.
16 nviable polyploid (>=8 N) cells arising from endomitosis.
17 e how human hepatocytes initiate and execute endomitosis.
18 Gp1balpha also participates in megakaryocyte endomitosis, a form of controlled and precise whole-geno
19  in many animal tissues, where they arise by endomitosis, a non-canonical cell cycle in which cells e
20 loidization in megakaryocytes is achieved by endomitosis, a specialized cell cycle in which DNA repli
21 an colon cancer derived cell line results in endomitosis and a single multilobulated nucleus with chr
22 feration to yield diploid cells, followed by endomitosis and acquisition of polyploidy.
23  that the pathways leading to megakaryocytic endomitosis and c-Myc-induced tetraploidy are mechanisti
24 teins should be informative in understanding endomitosis and cell cycle control.
25 etarded tapetum development, delayed tapetum endomitosis and cell wall degeneration, resulting in enl
26 ant proportion of these cells do not undergo endomitosis and express markedly lower levels of mRNA of
27  at high levels in megakaryocytes undergoing endomitosis and is markedly upregulated following exposu
28 cape senescence via restitutional multipolar endomitosis and other noncanonical modes of cell divisio
29 cus plays an important role in megakaryocyte endomitosis and terminal maturation.
30 in mice, suggesting that RUNX1B can regulate endomitosis and thrombopoiesis.
31 ow an important role for MAPK in TPO-induced endomitosis and underscore the value of primary cells wh
32 at Polo-like kinase 1 (Plk1) is required for endomitosis, and ablation of the Plk1 gene in megakaryoc
33 ine stimulating megakaryocyte proliferation, endomitosis, and platelet production.
34                      The factors controlling endomitosis are accessible to analysis in our megakaryoc
35 ry evaluated; the molecular underpinnings of endomitosis are being increasingly understood; the intra
36 ell fusion of unrelated Hodgkin cells nor to endomitosis, but rather is mediated by re-fusion of daug
37 ntrol polyploidization and the transition to endomitosis by impeding cell cycling and promoting apopt
38                   We aimed to define whether endomitosis consists of a continuous phase of DNA synthe
39         Thus, CDKN1B-mediated suppression of endomitosis contributes to the initiation of a reversibl
40 We conclude that mitosis is abrogated during endomitosis due to the absence of cdk1 and the failure t
41 aryocytes (MKs) undergo successive rounds of endomitosis during differentiation, resulting in polyplo
42                                              Endomitosis (EnM) in megakaryocytes (MKs) is characteriz
43 cted during the mitotic cell cycle or during endomitosis; however, cyclin B1-dependent Cdc2 kinase ac
44  integrin-binding protein 1 (CIB1) regulates endomitosis in Dami cells.
45 egulates Tpo-mediated cell proliferation and endomitosis in hematopoietic cell lines and primary hema
46 , we have elucidated how hepatocytes undergo endomitosis in human Hep-Orgs, providing new insights in
47 ctor is distinct from TPO because it induces endomitosis in IL-3-generated megakaryocytes in vitro, w
48 leukins, and SDF1a, that can induce in vitro endomitosis in immature human megakaryocytes in the pres
49 roviding new insights into the mechanisms of endomitosis in mammals.
50 iological role of miR-1300 as a regulator of endomitosis in megakaryocyte differentiation where block
51           The role of cyclin D3 in promoting endomitosis in other lineages programmed to abrogate mit
52  kinase pathway is important for TPO-induced endomitosis in primary megakaryocytes (MKs).
53 on of c-myc oncogene on megakaryopoiesis and endomitosis in vivo, using transgenic mice carrying c-my
54  involves a specialized cell division called endomitosis, in which repeated nuclear divisions occur w
55                                              Endomitosis is a unique form of cell cycle used by megak
56                                              Endomitosis is a unique megakaryocyte (MK) differentiati
57 study indicates that Fanca expression during endomitosis is crucial for normal megakaryopoiesis and p
58  the mosquito host, an atypical rapid closed endomitosis is observed.
59 ow; this process is aborted in megakaryocyte endomitosis, leading to polyploidy.
60                        We find that cells in endomitosis M phase have normal mitotic timings, but los
61 nesis have been shown to be inhibited during endomitosis M phase in rodents, but it is currently unkn
62 lls, the extent to which it is necessary for endomitosis of megakaryocytes remains controversial.
63 oles of 2 nonmuscle myosin IIs (NMIIs) on MK endomitosis: only NMII-B (MYH10), but not NMII-A (MYH9),
64 teins that may eventually lead to changes in endomitosis process.
65 tensions, and reduced phosphorylation of the endomitosis regulators LIM domain kinase 1, cofilin, and
66  expression of both GEF-H1 and ECT2 prevents endomitosis, resulting in proliferation of 2N Mks.
67 telet lineage display a striking increase in endomitosis, similar to changes seen following Mpl ligan
68 ficiency does not alter megakaryopoiesis and endomitosis, the final step of megakaryocyte maturation
69 with TGCs endocycling rather than undergoing endomitosis, they have low expression of M-phase genes.
70 opmental Cell, Gao et al. trace the basis of endomitosis to sequential downregulation of guanine nucl
71 K contractile ring and implicated in mitosis/endomitosis transition.
72  identified miR-1300 as a novel regulator of endomitosis with translatable potential for therapeutic