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1  that blood and lymphatic vessels within the endomysial and perimysial spaces undergo significant cha
2               Mild inflammatory exudates, in endomysial and perivascular spaces, consisted of lymphoc
3      Group 2: the sensitivities of Simtomax, endomysial and tissue-transglutaminase antibodies were c
4    BACKGROUND & AIMS: Tests to measure serum endomysial antibodies (EMA) and antibodies to tissue tra
5 cted and apparently asymptomatic adults with endomysial antibodies (EmA) benefit from a gluten-free d
6 psy; in 5 countries, HLA, and in one country endomysial antibodies (EMA) were not available.
7 awn for later evaluations of serum IgA-class endomysial antibodies (EMA).
8 noglobulins, IgA/IgG anti-gliadin (AGA), and endomysial antibodies (EMA).
9  serum IgA antigliadin, IgG antigliadin, and endomysial antibodies (EMA).
10                                              Endomysial antibodies and HLA typing were routinely avai
11                                              Endomysial antibodies indicate celiac sprue in asymptoma
12                                          IgA endomysial antibodies tests were associated with high sp
13 th IBD had a slight decrease in risk of anti-endomysial antibodies vs controls (RR 0.70; 95% CI 0.18-
14 of IgA, IgA antitissue transglutaminase, and endomysial antibodies were measured, and a duodenal biop
15 rology (IgA anti-transglutaminase 2 and anti-endomysial antibodies) and villous atrophy on small-inte
16 d for IgA anti-tTG, and if positive, for IgA endomysial antibodies.
17 d on analysis of tissue transglutaminase and endomysial antibodies.
18 issue transglutaminase and, if abnormal, for endomysial antibodies.
19  tests for anti-TG2, tested positive for the endomysial antibody (anti-EMA), had total serum levels o
20 sis of tissue transglutaminase (tTg) levels; endomysial antibody (EMA) levels were analyzed in sample
21 sue transglutaminase antibody (tTG) and anti-endomysial antibody (EMA).
22  tTG) and, if positive, testing for IgA anti-endomysial antibody (IgA EMA) as a confirmatory test.
23  IgA against deamidated gliadin peptide, and endomysial antibody (IgA).
24 positive TG2-IgA is combined with a positive endomysial antibody in a second blood sample, the positi
25 lutaminase or deamidated gliadin peptide, or endomysial antibody, should be considered to have celiac
26 and, if titer results were above 2 U/mL, for endomysial antibody.
27 al concepts of bag filling within the muscle endomysial "bag", which is limited by the "bag" size unl
28 and deposition of complement causes lysis of endomysial capillaries and muscle ischaemia.
29 delta T cells are cytotoxic against adherent endomysial cardiac cells, mostly macrophages, but not ag
30 re evaluated by measuring serum CK activity, endomysial CD45 immunoreactive inflammation area, endomy
31 with muscle fiber invasion by leukocytes and endomysial collagen deposition, was associated with elev
32 ysial CD45 immunoreactive inflammation area, endomysial collagen III deposition, and hind limb grip s
33 owed prominent FGF23 immunoreactivity in the endomysial connective tissue and along the muscle membra
34 emistry, FRZB was predominantly localized to endomysial connective tissue and to a lesser extent musc
35 a fibrogenic cytokine immunolocalized in the endomysial connective tissue of pretreatment DM muscles,
36 ents of complement were present diffusely in endomysial connective tissue.
37 often with a prominent B cell component, and endomysial dendritic cells.
38                      Although studies on the endomysial expression of cytokines and cell adhesion mol
39                                     However, endomysial fibrosis (distance between myocytes within bu
40 01), myocyte diameter (r = 0.97, p < 0.001), endomysial fibrosis (r = 0.98, p < 0.001) and capillary
41 a severe muscular dystrophy in DKO mice with endomysial fibrosis and ectopic calcification.
42                                       Atrial endomysial fibrosis content was lower and myocyte diamet
43 ontinuity of the epicardial layer because of endomysial fibrosis may reduce its synchronizing effect,
44 confirmed the potential impact of epicardial endomysial fibrosis on AF complexity.
45 cence, obvious foci of severe capillary loss/endomysial fibrosis, and muscle infarcts predicted chron
46 and endomysial fibrosis, spatial patterns of endomysial fibrosis, fibroblast density, capillary densi
47 myofiber size, centrally located nuclei, and endomysial fibrosis, in the absence of intracytoplasmic
48 ification of cardiomyocyte size, overall and endomysial fibrosis, spatial patterns of endomysial fibr
49 uclei, elevated creatine kinase activity and endomysial fibrosis.
50 muscle fibers, increased central nuclei, and endomysial fibrosis.
51 , central nuclei, rimmed fibers, and/or mild endomysial fibrosis.
52 (TG2-IgA), IgA testing, and less frequently, endomysial IgA testing.
53 iac disease was confirmed using an assay for endomysial IgA.
54 found that CD8+ T lymphocytes persist in the endomysial infiltrates in all biopsies during a 19-22 mo
55 ve the strength of some patients, and reduce endomysial inflammation and stressor molecules.
56  with mRNA of ICOS, ICOS-L and the degree of endomysial inflammation as assessed in coded haematoxyli
57 at least two members from each family showed endomysial inflammation, red-rimmed vacuoles, intracellu
58 n expression was compared with the degree of endomysial inflammation.
59   The HIV gag antigen was present on several endomysial macrophages but not within the muscle fibers.
60 omyositis specimens contained C5b-9 reactive endomysial microvessels but none of these or other vesse
61  particle antibodies; immune myopathies with endomysial pathology (IM-EP), illustrated by brachio-cer
62  model predicts that the blood volume in the endomysial space increases 24% and decreases 22% for a 2
63 t only peripheral blood lymphocytes but also endomysial T cells and alters the natural course of the
64 n of certain Vb families was noted among the endomysial T cells with evidence of in situ expansion.
65                    The clonal restriction of endomysial T cells, compared with peripheral blood, was
66 r understanding of TCR gene rearrangement in endomysial T cells, regulation of MHC expression, activi
67 istent, clonally expanded and antigen-driven endomysial T cells, the disease is resistant to immunoth
68 ysed the T-cell receptor (TCR) gene usage by endomysial T lymphocytes in three sequential muscle biop
69  putative antibody may not be limited to the endomysial vessels but may also involve the blood vessel