ライフサイエンスコーパス: endoplasmic reticulum

1 ein STIM1 upon Ca(2+) store depletion of the endoplasmic reticulum.

2 ed compared with WT, due to retention in the endoplasmic reticulum.

3 membrane proteins that are assembled at the endoplasmic reticulum.

4 activating protein (SCAP) and sterols in the endoplasmic reticulum.

5 tochondria, chloroplasts, lysosomes, and the endoplasmic reticulum.

6 onsistent with post-glycan processing in the endoplasmic reticulum.

7 of physiological Ca(2+) concentration in the endoplasmic reticulum.

8 P1 and MTTP are partially colocalized in the endoplasmic reticulum.

9 ks typically found in organelles such as the endoplasmic reticulum.

10 functions in nascent protein folding in the endoplasmic reticulum.

11 red specificity for protein targeting to the endoplasmic reticulum.

12 activities through these interactions in the endoplasmic reticulum.

13 hanks in part to the expert trimming done by endoplasmic reticulum aminopeptidases (ERAPs), the final

14 P alone, TurNP + EP treatment upregulated 66 endoplasmic reticulum and 193 mitochondrial proteins, en

15 Cl(-)/H(+) exchange and was retained in the endoplasmic reticulum and cis-Golgi, but not in early en

16 s studies of PQC degradation pathways in the endoplasmic reticulum and cytosol have led to the prevai

17 microscopy in EC cytoplasm (associated with endoplasmic reticulum and Golgi proteins).

18 e components including endosomes, lysosomes, endoplasmic reticulum and Golgi.

19 peptides in the oxidizing environment of the endoplasmic reticulum and is mediated by protein disulfi

20 a molecular chaperone protein located in the endoplasmic reticulum and plasma membranes and has been

21 free ribosomes, the appearance of elongated endoplasmic reticulum and strikingly increased number of

22 teasome cleavage, peptide transport into the endoplasmic reticulum and T-cell receptor repertoire, al

23 sociation of the SCAP-SREBP complex from the endoplasmic reticulum and the activation of SREBPs(1,2).

24 transgenic 16:0 desaturases targeted to the endoplasmic reticulum and the chloroplast to lower 16:0

25 chanisms to those regulating turnover of the endoplasmic reticulum and the clearance of protein aggre

26 apparatus and the close contacts between the endoplasmic reticulum and the plasma membrane, structure

27 spiration, volume, and interactions with the endoplasmic reticulum) and MSNs (i.e., dendritic complex

28 nobody-based approach to trap Wntless in the endoplasmic reticulum, and hence prevent all Wnt secreti

29 volving mitochondrial communication with the endoplasmic reticulum, and how retrograde signaling upre

30 ysosomal clearance of a model organelle, the endoplasmic reticulum, and of a model protein, polymerog

31 response, a cellular stress response of the endoplasmic reticulum, and removal of misfolded proteins

32 of cargo between the Golgi apparatus and the endoplasmic reticulum, as well as intra-Golgi transport.

33 Cx43 was found to be partly retained in the endoplasmic reticulum-associated cell compartments.

34 Here we report endoplasmic reticulum-associated degradation (ERAD) as a

35 , we report that protein quality control via endoplasmic reticulum-associated degradation (ERAD) gove

36 ked glycoproteins are selectively sorted for endoplasmic reticulum-associated degradation (ERAD) in r

37 l role in cellular homeostasis by regulating endoplasmic reticulum-associated degradation (ERAD), mit

38 In endoplasmic reticulum-associated protein degradation (ER

39 iptomics analysis revealed downregulation of endoplasmic reticulum-associated protein degradation pat

40 s including intranuclear vesicles containing endoplasmic reticulum-associated proteins, web-like adhe

41 AM enzyme viperin (virus-inhibitory protein, endoplasmic reticulum-associated, interferon-inducible)

42 Thus, the UPR, an endoplasmic-reticulum-associated response, quite unexpec

43 se, p97, a homolog of the human transitional endoplasmic reticulum ATPase (VCP/p97) protein.

44 (F/-)) triggers Golgi dispersal, dilation of endoplasmic reticulum, autophagy and exosome biogenesis

45 (HO-1) and the cytochromes P450 (P450s) are endoplasmic reticulum-bound enzymes that rely on the sam

46 tNAA50-EYFP localized to the cytosol and the endoplasmic reticulum but also to the nuclei.

47 Protein secretion is initiated at the endoplasmic reticulum by the COPII coat, which self-asse

48 ted by direct physical interactions with the endoplasmic reticulum Ca(2+) sensor stromal interaction

49 n endolysosomal Ca(2+) uptake and filling of endoplasmic reticulum Ca(2+) stores, thereby regulating

50 channel IP(3)R and the activity of the sarco-endoplasmic reticulum Ca(2+)-ATPase (SERCA) pump during

51 differential sensitivity to the sarcoplasmic/endoplasmic reticulum Ca(2+)-ATPase pump inhibitors, and

52 SERCAs (sarco-endoplasmic reticulum Ca(2+)-ATPases) pump Ca(2+) into i

53 Sarcoplasmic/endoplasmic reticulum Ca2+ ATPase (SERCA) activity was r

54 , including the ER calcium pump sarcoplasmic/endoplasmic reticulum calcium ATPase (SERCA).

55 The results of our study indicate a role for endoplasmic reticulum calcium signaling via calreticulin

56 n, which in turn phosphorylates sarcoplasmic/endoplasmic reticulum calcium-ATPase 2a (SERCA2a) and ac

57 interaction leads to sequestration of HFE in endoplasmic reticulum, causing poorer surface expression

58 receptors are substantially localised to the endoplasmic reticulum, cellular sites of cytokinin perce

59 ally opens following Ca(2+) release from the endoplasmic reticulum, certain pathologic mutations rend

60 g proteins targeted to the cytosol, nucleus, endoplasmic reticulum, chloroplasts, mitochondria and pe

61 h in stretched axons was also accompanied by endoplasmic reticulum (ER) accumulation and, accordingly

62 Disruption of protein folding in the endoplasmic reticulum (ER) activates the unfolded protei

63 Endoplasmic reticulum (ER) acts as the largest intracell

64 bined action of signal peptide peptidase and endoplasmic reticulum (ER) aminopeptidase 1 (ERAP1) is r

65 myelinating Schwann cells, processed in the endoplasmic reticulum (ER) and delivered to myelin via t

66 y, membrane contact sites (MCSs) between the endoplasmic reticulum (ER) and endosomes have emerged as

67 organises membranes at the interface of the endoplasmic reticulum (ER) and ERGIC/Golgi (Raote et al.

68 nse (UPR) plays a central role in regulating endoplasmic reticulum (ER) and global cellular physiolog

69 protein is localized to the plasma membrane, endoplasmic reticulum (ER) and Golgi.

70 protein response (UPR) senses defects in the endoplasmic reticulum (ER) and orchestrates a complex pr

71 Tethered interactions between the endoplasmic reticulum (ER) and other membrane-bound orga

72 Polyomavirus SV40 traffics to the endoplasmic reticulum (ER) and penetrates a virus-induce

73 final seven steps occur in the lumen of the endoplasmic reticulum (ER) and require dolichylphosphate

74 compatibility complex (MHC) molecules in the endoplasmic reticulum (ER) and reroutes them to lysosome

75 4SS effectors localize to and/or disrupt the endoplasmic reticulum (ER) and secretory transport, but

76 ies are required for proSP-B export from the endoplasmic reticulum (ER) and sorting to LBs, the conve

77 which is involved in trafficking between the endoplasmic reticulum (ER) and the Golgi.

78 Both DENV and ZIKV rely on expansion of the endoplasmic reticulum (ER) and the induction of autophag

79 form vC) localize to the luminal face of the endoplasmic reticulum (ER) and to the cell surface, but

80 STING resides on the endoplasmic reticulum (ER) and traffics following stimul

81 Lysosomal enzymes are synthesized in the endoplasmic reticulum (ER) and transferred to the Golgi

82 f newly synthesized membrane proteins in the endoplasmic reticulum (ER) are assembled into multiprote

83 Misfolded proteins in the endoplasmic reticulum (ER) are degraded by ER-associated

84 In living cells, dynamics of the endoplasmic reticulum (ER) are driven by the cytoskeleto

85 Eeyarestatin 1 (ES1) is an inhibitor of endoplasmic reticulum (ER) associated protein degradatio

86 uclear membrane (INM) is continuous with the endoplasmic reticulum (ER) but harbors a distinctive pro

87 RNA translation to CK2alpha translation with endoplasmic reticulum (ER) Ca(2+) release versus cytopla

88 -permeable TRP channels, but the function of endoplasmic reticulum (ER) Ca(2+) stores in this importa

89 However, depletion of endoplasmic reticulum (ER) Ca(2+) stores reduced capsaic

90 eceptor stimulation induces depletion of the endoplasmic reticulum (ER) Ca(2+) stores, which is sense

91 The type 2a sarco/endoplasmic reticulum (ER) Ca(2+)-ATPase (SERCA2a) plays

92 of calcium influx at the plasma membrane by endoplasmic reticulum (ER) calcium stores, a process com

93 Accumulation of unfolded proteins in the endoplasmic reticulum (ER) causes ER stress and activate

94 Sigma-1 receptor (S1R) is an endoplasmic reticulum (ER) chaperone that not only regul

95 SURF4 resulted in accumulation of EPO in the endoplasmic reticulum (ER) compartment and that SURF4 an

96 we show that the disruption of mitochondria-endoplasmic reticulum (ER) contact sites (MERCs) phenoco

97 igomerization, which constricts membranes at endoplasmic reticulum (ER) contact sites.

98 7B interaction with HSP70, thus accelerating endoplasmic reticulum (ER) degradation of the mutant pro

99 Protein maturation in the endoplasmic reticulum (ER) depends on a fine balance bet

100 NGO1 is a transmembrane protein localised at endoplasmic reticulum (ER) exit sites, where it binds bu

101 While the effect of ROS on mitochondria and endoplasmic reticulum (ER) has been well documented, its

102 The endoplasmic reticulum (ER) has recently emerged as a pro

103 often exhibit augmented capacity to maintain endoplasmic reticulum (ER) homeostasis under adverse con

104 The endoplasmic reticulum (ER) immunoglobulin binding protei

105 cell surface and retain MR1 molecules in the endoplasmic reticulum (ER) in an immature form.

106 lly is concentrated in a subapical region of endoplasmic reticulum (ER) in cholangiocytes, but both i

107 In vivo imaging reveals instability of endoplasmic reticulum (ER) in mouse AD models and genome

108 Here, we report that DRP1 translocates to endoplasmic reticulum (ER) in response to beta-adrenergi

109 The endoplasmic reticulum (ER) is a fundamental organelle in

110 Lipid droplet (LD) formation from the endoplasmic reticulum (ER) is accompanied by the targeti

111 Membrane protein biogenesis in the endoplasmic reticulum (ER) is complex and failure-prone.

112 ion of unfolded or misfolded proteins at the endoplasmic reticulum (ER) is emerging as a possible dri

113 The endoplasmic reticulum (ER) is the entry point to the sec

114 The endoplasmic reticulum (ER) is the main site of protein s

115 The endoplasmic reticulum (ER) is the reservoir for calcium

116 of the intrinsic activation of the PKR-like endoplasmic reticulum (ER) kinase (PERK) in the immunoin

117 fically phosphorylated protein kinase R-like endoplasmic reticulum (ER) kinase (PERK-P) signaling-a p

118 Endoplasmic reticulum (ER) macroautophagy (hereafter cal

119 In yeast, the endoplasmic reticulum (ER) makes extensive intermembrane

120 P) cation channel expressed primarily on the endoplasmic reticulum (ER) membrane and primary cilia of

121 The endoplasmic reticulum (ER) membrane complex (EMC) cooper

122 Interestingly, NRF1 is synthesized as an endoplasmic reticulum (ER) membrane protein and when cel

123 se proteins into the membrane depends on the endoplasmic reticulum (ER) membrane protein complex (EMC

124 The endoplasmic reticulum (ER) membrane protein complex (EMC

125 Ubiquitin ligases (E3s) embedded in the endoplasmic reticulum (ER) membrane regulate essential c

126 mian virus 40 (SV40) must penetrate the host endoplasmic reticulum (ER) membrane to enter the cytosol

127 inserts tail-anchored (TA) proteins into the endoplasmic reticulum (ER) membrane with an insertase (y

128 eased lipid packing order when comparing the endoplasmic reticulum (ER) membrane, plasma membrane, an

129 (ZIKV), rely heavily on the availability of endoplasmic reticulum (ER) membranes throughout their li

130 ctivity localizes to the plasma membrane and endoplasmic reticulum (ER) of cells, whereas TRPV3 resid

131 Upon ligand stimulation, the endoplasmic reticulum (ER) protein STING translocates to

132 ng MS-based proteomics, we elucidate how the endoplasmic reticulum (ER) proteostasis network differen

133 ction between mutant calreticulin (CALR) and endoplasmic reticulum (ER) resident protein 57 (ERp57) a

134 Sterol O-acyltransferase 1 (SOAT1) is an endoplasmic reticulum (ER) resident, multi-transmembrane

135 The metazoan endoplasmic reticulum (ER) serves both as a hub for matu

136 Overexpression of GRP78 and PDI following endoplasmic reticulum (ER) stress and activation of the

137 s misfolding and polymerization, which cause endoplasmic reticulum (ER) stress and liver disease thro

138 nstrated that AKT1 protein deficiency caused endoplasmic reticulum (ER) stress and potentiated beta c

139 ion of unfolded immunoglobulins, which cause endoplasmic reticulum (ER) stress and sensitivity to pro

140 rgeting ability and its capability to induce endoplasmic reticulum (ER) stress by reactive oxygen spe

141 unfolded protein response (UPR) to mitigate endoplasmic reticulum (ER) stress caused by cellular onc

142 Endoplasmic reticulum (ER) stress in AEC has been observ

143 gene and protein expression showed beta-cell endoplasmic reticulum (ER) stress in both sexes.

144 Persistent endoplasmic reticulum (ER) stress in neurons is associat

145 We assessed the role of endoplasmic reticulum (ER) stress in the cross-talk betw

146 cilitating mucilage production by mitigating endoplasmic reticulum (ER) stress in the developing appr

147 AS, and CA-BRAF down-regulated expression of endoplasmic reticulum (ER) stress proteins, and reduced

148 Additionally, Rebaudioside A improved endoplasmic reticulum (ER) stress related gene expressio

149 l activation of innate immune sensing or the endoplasmic reticulum (ER) stress response contributes t

150 FVIII expression activates the endoplasmic reticulum (ER) stress response, causes oxida

151 ded proteins in cells exposed to As leads to endoplasmic reticulum (ER) stress response, which, if no

152 e-specific phospholipase C2 functions in the endoplasmic reticulum (ER) stress response.

153 n to the activation of genes involved in the endoplasmic reticulum (ER) stress response.

154 ng sites for XBP1, a transcription factor of endoplasmic reticulum (ER) stress response.

155 th inflammation resolution and triggering an endoplasmic reticulum (ER) stress response.

156 ges IRE1alpha signaling in the absence of an endoplasmic reticulum (ER) stress signature, leading to

157 increased lipid peroxidation byproducts and endoplasmic reticulum (ER) stress, (2) decreased protect

158 eptibility to retinal degeneration caused by endoplasmic reticulum (ER) stress, and developmental def

159 rly signs of stress in different conditions: endoplasmic reticulum (ER) stress, calcium overload, oxi

160 ated by several stress conditions, including endoplasmic reticulum (ER) stress, executed by protein k

161 lies and inhibits ERK activity, resulting in endoplasmic reticulum (ER) stress, the unfolded protein

162 Hepatic endoplasmic reticulum (ER) stress, whether triggered by

163 ice lacking functional cone CNG channel show endoplasmic reticulum (ER) stress-associated cone degene

164 igenetic state (H3K9me3), and the content of endoplasmic reticulum (ER) stress-associated transcripts

165 Here, we report that the chronic endoplasmic reticulum (ER) stress-induced ATF4-CHOP-GADD

166 sitic infections, activated Rho GTPases, and endoplasmic reticulum (ER) stress.

167 n the accumulation of misfolded proteins and endoplasmic reticulum (ER) stress.

168 ing T-cell activation as well as thermal and endoplasmic reticulum (ER) stress.

169 adverse environmental conditions can trigger endoplasmic reticulum (ER) stress.

170 2 protease (S2P) sequentially in response to endoplasmic reticulum (ER) stress.

171 tes the delivery of MHC-I molecules from the endoplasmic reticulum (ER) to phagosomes, and increases

172 a critical role in endocytic trafficking and endoplasmic reticulum (ER) to plasma membrane (PM) trans

173 t C88 and inhibited its trafficking from the endoplasmic reticulum (ER) to the Golgi complex.

174 Proteins that clog the endoplasmic reticulum (ER) translocon prevent the moveme

175 Here, we report the discovery of a family of endoplasmic reticulum (ER) transmembrane proteins that a

176 astin GTPase involved in homotypic fusion of endoplasmic reticulum (ER) tubules in the formation of t

177 of proteins stabilize the high curvature of endoplasmic reticulum (ER) tubules.

178 anslational targeting and insertion into the endoplasmic reticulum (ER) via the Guided-Entry of TA pr

179 st, it accounts for only 5% of lipids in the endoplasmic reticulum (ER)(1).

180 ins are inserted, modified and folded at the endoplasmic reticulum (ER)(2).

181 central hubs in the chaperone network of the endoplasmic reticulum (ER), acting as gatekeepers to the

182 s vesicles distinct from hydrogenosomes, the endoplasmic reticulum (ER), and Golgi complex.

183 lly focus on the links between mitochondria, endoplasmic reticulum (ER), and lysosomes in HSC metabol

184 Myelin proteins, which are produced in the endoplasmic reticulum (ER), are essential and necessary

185 s the unfolded protein response (UPR) in the endoplasmic reticulum (ER), are two mechanisms that enab

186 otein response (UPR) sensor IRE1alpha in the endoplasmic reticulum (ER), but not other UPR sensors, s

187 (PD) and use cell endomembranes, mostly the endoplasmic reticulum (ER), for delivery of viral genome

188 k TMD oligomers locate preferentially to the endoplasmic reticulum (ER), heterooligomerization betwee

189 in 4 (FATP4), a transmembrane protein in the endoplasmic reticulum (ER), is a recently identified neg

190 cts and observed outward co-movement of MTs, endoplasmic reticulum (ER), mitochondria, acidic organel

191 In the endoplasmic reticulum (ER), secretory proteins are packa

192 pairs and families within the context of the endoplasmic reticulum (ER), the main cellular hub of lip

193 ge of the signal peptide upon entry into the endoplasmic reticulum (ER), the peptide precursors are p

194 argeting the immature V(o) subcomplex in the endoplasmic reticulum (ER), thus providing insight into

195 The cholesterol moves to the endoplasmic reticulum (ER), where it inhibits production

196 d level, the excess cholesterol moves to the endoplasmic reticulum (ER), where it regulates the SREBP

197 e most cellular cholesterol resides, and the endoplasmic reticulum (ER), where the protein machinery

198 nuclear envelope (NE) is continuous with the endoplasmic reticulum (ER), yet the NE carries out many

199 l., 2015) revealed that UBIAD1 also inhibits endoplasmic reticulum (ER)-associated degradation (ERAD)

200 clones carry mutations in genes involved in Endoplasmic Reticulum (ER)-based lipid homeostasis and a

201 ation, and protein processing/sorting in the Endoplasmic Reticulum (ER)-Golgi in a temporal order con

202 lasts showed OsPIP1;3 mislocalization in the endoplasmic reticulum (ER)-like neighborhood, whereas co

203 ate that reconstitution of VRCs on GUVs with endoplasmic reticulum (ER)-like phospholipid composition

204 SEIPIN proteins are localized to endoplasmic reticulum (ER)-lipid droplet (LD) junctions

205 ive 20 (SCAR20)-associated protein Snx14, an endoplasmic reticulum (ER)-lipid droplet (LD) tethering

206 lasmic Ca(2+) concentration ([Ca(2+)](i)) by endoplasmic reticulum (ER)-localized inositol 1,4,5-tris

207 icity, 2) Hsp70, 3) Hsp90, 4) proteasome, 5) endoplasmic reticulum (ER)-mediated folding inhibition,

208 elated protein (Drp1) severs mitochondria at endoplasmic reticulum (ER)-mitochondria contact sites, w

209 interaction molecules STIM1 and STIM2 within endoplasmic reticulum (ER)-plasma membrane (PM) contact

210 MYLK-L augmented SOCE by increasing endoplasmic reticulum (ER)-plasma membrane (PM) junction

211 calreticulin (TcCalr) is a multifunctional, endoplasmic reticulum (ER)-resident chaperone that, tran

212 that Rab18, by directly interacting with the endoplasmic reticulum (ER)-resident protein kinectin-1,

213 The endoplasmic reticulum (ER)-resident protein TANGO1 assem

214 (Ribo-seq) we find specific dysregulation of endoplasmic reticulum (ER)-targeted mRNA translation in

215 Notably, PSEN1 loss of function impedes Endoplasmic Reticulum (ER)-to-lysosome delivery of ClC-7

216 al lipid storage organelles assembled at the endoplasmic reticulum (ER).

217 produced in peroxisomes and degraded in the endoplasmic reticulum (ER).

218 poorly secreted and localized largely to the endoplasmic reticulum (ER).

219 that have failed to properly assemble in the endoplasmic reticulum (ER).

220 interaction molecules (STIM) located in the endoplasmic reticulum (ER).

221 t storage organelles that originate from the endoplasmic reticulum (ER).

222 n the late endosomes/lysosomes (LEL) and the endoplasmic reticulum (ER).

223 nown to be essential for its assembly in the endoplasmic reticulum (ER).

224 triphosphate-dependent Ca2+ release from the endoplasmic reticulum (ER).

225 oad of unfolded or misfolded proteins in the endoplasmic reticulum (ER).

226 asia that jeopardize proteostasis within the endoplasmic reticulum (ER).

227 ates the stability of RNAs translated at the endoplasmic reticulum (ER).

228 esterol synthesis enriched this lipid in the endoplasmic reticulum (ER).

229 are integral membrane enzymes located in the endoplasmic reticulum (ER).

230 pathogenic JUNV, causes GPC retention in the endoplasmic reticulum (ER).

231 of ethylene receptors in the membrane of the endoplasmic reticulum (ER); a protein kinase, called con

232 ERAD substrate that became aggregated in the endoplasmic reticulum following ERAD deficiency.

233 nsulin secretion, transcription, metabolism, endoplasmic reticulum function, and the stress response.

234 unction and highlight that mitochondrial and endoplasmic reticulum functions are intertwined through

235 living cells independently of the classical endoplasmic reticulum-Golgi exocytic route.

236 In lipid bilayers that mimic the endoplasmic reticulum-Golgi intermediate compartment (ER

237 g to ITPR1 controls calcium release from the endoplasmic reticulum in macrophages and monocytes.

238 haperone protein generally restricted to the endoplasmic reticulum in normal tissues, but which is ex

239 binding domain, cause ATP7B retention in the endoplasmic reticulum, inhibit Cu-transport, and lower A

240 erexpression facilitated the accumulation of endoplasmic reticulum, integrins and Rab11 endosomes in

241 The endoplasmic reticulum is a cellular hub of lipid metabol

242 acid (C18:1) to linoleic acid (C18:2) in the endoplasmic reticulum is critical to the accumulation of

243 Yet, ablation of protein kinase R-like endoplasmic reticulum kinase (PERK) also ameliorated the

244 ER-stress conditions, protein kinase R-like endoplasmic reticulum kinase (PERK) phosphorylates eukar

245 ctivation of the protein kinase R (PKR)-like endoplasmic reticulum kinase (PERK), a key component of

246 R) stress, executed by protein kinase R-like endoplasmic reticulum kinase (PERK).

247 the SG process using a protein kinase R-like endoplasmic reticulum kinase inhibitor (GSK2606414) or t

248 erminal kinase and the protein kinase R-like endoplasmic reticulum kinase.

249 the distal axon, whilst removing Protrudin's endoplasmic reticulum localization, kinesin-binding or p

250 r P4Hs, a third P4H-the poorly characterized endoplasmic reticulum-localized transmembrane prolyl 4-h

251 henols with specific biological alterations (endoplasmic reticulum, lysosomes, and NFkB) caused by th

252 synthesis, lipid droplet formation, nuclear/endoplasmic reticulum membrane morphology, vacuole fusio

253 IRE1 spans the endoplasmic reticulum membrane, comprising a sensory lum

254 ng, recruits the other COPII proteins to the endoplasmic reticulum membrane.

255 equired for its translocation to the nuclear/endoplasmic reticulum membrane.

256 thesizing machinery associates with modified endoplasmic reticulum membranes that are transformed int

257 ile acid recirculation due to improvement of endoplasmic reticulum-mitochondria calcium homeostasis w

258 (sixfold higher protein abundance) and novel endoplasmic reticulum morphology.

259 els, and/or internal release from the smooth endoplasmic reticulum) near the postsynaptic density to

260 HL14 is in close proximity to the BCR in the endoplasmic reticulum of MCD cell line models and promot

261 monooxygenase associated externally with the endoplasmic reticulum of plant cells.

262 Endoplasmic reticulum omega-oxidation, a minor fatty aci

263 s implicate Ste24 as a key factor in several endoplasmic reticulum processes, including the unfolded

264 PMP22 under CMT1A conditions overwhelms the endoplasmic reticulum quality control system, leading to

265 The sarco-endoplasmic reticulum (SR/ER) plays an important role in

266 1) and TRP vanilloid-3 (TRPV3) in regulating endoplasmic reticulum stress (ERS) and cytotoxicity in h

267 ons promote protein misfolding, resulting in endoplasmic reticulum stress and activation of the unfol

268 response in COVID-19 infection by modulating endoplasmic reticulum stress and stimulating the resolut

269 vated GCN2 kinase, whereas mitochondrial and endoplasmic reticulum stress did not.

270 Cells undergoing endoplasmic reticulum stress express spliced X-box bindi

271 Induction of endoplasmic reticulum stress in knockout macrophages inc

272 he NF-kappaB-inducible nitric oxide synthase-endoplasmic reticulum stress pathway.

273 ion causes tissue damage, which triggers the endoplasmic reticulum stress response and subsequent eic

274 itochondrial respiration, as a result of the endoplasmic reticulum stress response induced by high pr

275 The unfolded protein response (UPR), an endoplasmic reticulum stress response pathway, has been

276 effect due to the latter's ability to induce endoplasmic reticulum stress response.

277 e incidence of inflammation and induction of endoplasmic reticulum stress responses during an extende

278 ndidate genes at the beta-cell level and the endoplasmic reticulum stress signalling that contributes

279 HFpEF-specific downregulated genes engaged endoplasmic reticulum stress, autophagy, and angiogenesi

280 esterol (FC) in macrophages, which can cause endoplasmic reticulum stress, cholesterol crystal format

281 ly response genes, oxidative, heat shock and endoplasmic reticulum stress, DNA damage responses, indu

282 ted on PCs demonstrated that LCDD LC induces endoplasmic reticulum stress, likely accounting for the

283 ipotoxicity with impaired membrane function, endoplasmic reticulum stress, mitochondrial dysfunction,

284 lular stress, including oxidative stress and endoplasmic reticulum stress, secondary to increased dem

285 CN results from mutant ELANE, which triggers endoplasmic reticulum stress, UPR, and apoptosis.

286 ular insults, such as nutrient deficiency or endoplasmic reticulum stress.

287 d T-helper cell type 17 (Th17) signaling and endoplasmic reticulum stress.

288 , ubiquitous Ca(2+)-buffering protein in the endoplasmic reticulum that controls transcriptional acti

289 ses the accumulation of PILS proteins at the endoplasmic reticulum, thereby increasing nuclear abunda

290 MHC I) proteins, from their synthesis in the endoplasmic reticulum to folding and trafficking via the

291 mary mediators of vesicular traffic from the endoplasmic reticulum to the Golgi apparatus.

292 peptide and not requiring transport from the endoplasmic reticulum to the Golgi apparatus.

293 s defective folding and trafficking from the endoplasmic reticulum to the LE/Ly compartments.

294 ed ADHCAI and suggested a role for FAM83H in endoplasmic reticulum-to-Golgi vesicle trafficking and p

295 Here, we show circadian clock regulation of endoplasmic reticulum-to-plasma membrane procollagen tra

296 such as the unfolded protein response of the endoplasmic reticulum (UPR(ER)), become defunct with age

297 membranes of other organelles, including the endoplasmic reticulum, via N-terminal glycine myristoyla

298 Phosphorylated PCK1 translocates to the endoplasmic reticulum, where it uses GTP as a phosphate

299 ation through sequestration of beta2M inside endoplasmic reticulum, which contributes toward inhibiti

300 es displayed substantial distribution in the endoplasmic reticulum with partial colocalization in mit