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1 decrease complication rates associated with endoscopic therapy.
2 S) have lower rebleeding rates compared with endoscopic therapy.
3 are limited because most have already failed endoscopic therapy.
4 cent hemorrhage (SRH) and decreased need for endoscopic therapy.
5 sult in transfusion-dependent anemia despite endoscopic therapy.
6 every 28 days or standard of care, including endoscopic therapy.
7 % of these examinations, enabling subsequent endoscopic therapy.
8 in relief among people who do not respond to endoscopic therapy.
9 residual colorectal neoplasia after previous endoscopic therapy.
10 e resolution after no more than 12 months of endoscopic therapy.
11 g medical conditions which require immediate endoscopic therapy.
12 ients with T1b disease may also benefit from endoscopic therapy.
13 Forrest grade Ia to IIb ulcers were offered endoscopic therapy.
14 and intravenous proton pump inhibitor after endoscopic therapy.
15 indispensable component following successful endoscopic therapy.
16 in the vast majority of patients undergoing endoscopic therapy.
17 CLE, and the technique may be used to guide endoscopic therapy.
18 omy are recurrent and refractory to standard endoscopic therapy.
19 to a shift in treatment algorithms favoring endoscopic therapy.
20 r stigmata of recent hemorrhage and need for endoscopic therapy.
21 ation-induced telangiectasias is amenable to endoscopic therapy.
22 an emphasis on novel imaging techniques and endoscopic therapies.
23 going resection: esophagectomy, 98.6 months; endoscopic therapy, 77.7 months; chemoradiation, 17.3 mo
26 hree or larger than 15 mm) received standard endoscopic therapies and UDCA + CBD stenting (group B) a
30 Rebleeding occurs in 20% of patients after endoscopic therapy, and so can we provide better outcome
32 the efficacy and safety of pharmacologic or endoscopic therapies as primary prophylaxis or that prev
34 ne GEP-NET subtypes that are appropriate for endoscopic therapy as well as to understand long-term ou
35 ng patients with achalasia continue to offer endoscopic therapies before recommending operative myoto
36 g Heller myotomy for achalasia, 154 received endoscopic therapy before being referred for surgery (10
37 article is to describe the role of Barrett's endoscopic therapy (BET) in patients with Barrett's esop
38 ion of esophageal neoplasia enables curative endoscopic therapy, but the current diagnostic standard
39 cation, magnetic sphincter augmentation, and endoscopic therapies can benefit patients with well-char
40 to medical treatment with beta-blockers and endoscopic therapy can be managed by variceal decompress
41 ett esophagus and dysplasia or early cancer, endoscopic therapy consisting of resection and ablation
42 to current guidelines, and use of drugs and endoscopic therapy (D+E) or p-TIPS was based on individu
44 We assessed trends in treatment; compared endoscopic therapy, esophagectomy, chemoradiation, and n
45 There has been an increase in interest in endoscopic therapy (ET) for intramucosal (T1a) or submuc
46 g stigmata of recent hemorrhage and need for endoscopic therapy, even when tested in an external pati
47 orporeal shockwave lithotripsy combined with endoscopic therapy failed to benefit patients with calci
50 Research and clinical experience with the endoscopic therapies for Barrett's esophagus continue to
51 garding the durability and role of different endoscopic therapies for dysplastic Barrett's oesophagus
56 m toxin injection is the most common initial endoscopic therapy for achalasia, most likely due to its
59 ts with HCC and liver cirrhosis who received endoscopic therapy for EV bleeding between 2017 and 2022
60 ween treatment groups and that the choice of endoscopic therapy for EVH must still rely on clinical g
61 ial recommendations of the AGA Institute on "Endoscopic Therapy for Gastroesophageal Reflux Disease."
62 anding of the indications and limitations of endoscopic therapy for HGD and IMC permits a tailored ap
63 py for HGD is preferred to surveillance, (6) endoscopic therapy for HGD is preferred to surgery, (7)
64 opy is necessary for accurate diagnosis, (5) endoscopic therapy for HGD is preferred to surveillance,
65 gical intervention should be considered over endoscopic therapy for long-term treatment of patients w
66 dose intravenous proton pump inhibitor after endoscopic therapy for peptic ulcer bleeding has been re
73 performed of the initial 40 patients who had endoscopic therapy from 2001 to 2010 at 1 center by 1 ph
76 g occurred in 5 of 61 (8.2%) patients in the endoscopic therapy group, compared with 21 of 85 (24.7%)
77 up period, none of the patients who received endoscopic therapy had a pseudocyst recurrence, compared
84 ents in the treatment of these sequelae, new endoscopic therapies have emerged to treat gastroesophag
88 ctice advice statements regarding the use of endoscopic therapies in treating patients with non-varic
89 ntion (median, 178 days) revealed successful endoscopic therapy in 43 (81%) and persistence of WOPN i
90 es transfusion requirements and the need for endoscopic therapy in patients with angiodysplasia-relat
91 re recommended, and clips are suggested, for endoscopic therapy in patients with high-risk stigmata.
104 diagnosed, treated, or palliated with timely endoscopic therapy, it is appropriate to consider endosc
107 tered during the past 2 decades during which endoscopic therapy, liver transplantation, and TIPS have
108 ve of this study was to assess the impact of endoscopic therapy, liver transplantation, and transjugu
112 phagogastroduodenoscopy (EGD) or their first endoscopic therapy of early neoplastic BE, from April 20
114 hy in the evaluation of biliary disease, and endoscopic therapy of postoperative liver transplantatio
118 patients with GERD to treatment with either endoscopic therapy or surgery according to the size of h
119 ancer and low risk of lymph node metastasis, endoscopic therapy or surgery alone is potentially curat
120 using EPCS mainly as salvage for failure of endoscopic therapy or TIPS is not supported by the defin
123 h high-risk stigmata who have had successful endoscopic therapy receive high-dose proton-pump inhibit
128 fication in responders and non-responders to endoscopic therapy showed that non-responders had signif
131 We analyzed data from a multicenter study of endoscopic therapy to identify factors associated with p
134 ccurrence of recurrent EV bleeding requiring endoscopic therapy was investigated as the outcome.
137 risk bleeding peptic ulcers after successful endoscopic therapy were randomly assigned as oral lansop
138 overed at an early stage can be treated with endoscopic therapy, whereas advanced cancers are primari
139 the future, the less invasive alternative of endoscopic therapy will need to be balanced against the
140 group B) and controls only received standard endoscopic therapies with only CBD stenting (group A).
141 achieving hemostasis for patients who failed endoscopic therapy with epinephrine injection, clip, or