ライフサイエンスコーパス: endothelial dysfunction

1 (PAR1) to promote inflammatory responses and endothelial dysfunction.

2 red tube formation and migration, confirming endothelial dysfunction.

3 n in hyperhomocysteinemia (HHcy), leading to endothelial dysfunction.

4 tory APN levels exhibited pulmonary vascular endothelial dysfunction.

5 sease through monocyte activation-associated endothelial dysfunction.

6 s to investigate how exposure to As leads to endothelial dysfunction.

7 c arteries (aortic and carotid arteries) and endothelial dysfunction.

8 dding of the glycocalyx are early changes of endothelial dysfunction.

9 imetic drugs with protective effects against endothelial dysfunction.

10 ERK5 signalling may be useful to counteract endothelial dysfunction.

11 tic milieu characterized by blood stasis and endothelial dysfunction.

12 inducing oxidative stress, inflammation, and endothelial dysfunction.

13 ythematosus (SLE) is a known risk factor for endothelial dysfunction.

14 significantly increased vasoconstriction and endothelial dysfunction.

15 ostasis and prevent oxidative stress-induced endothelial dysfunction.

16 SAM develop endothelial dysfunction.

17 = 0.011), even in those patients with severe endothelial dysfunction.

18 angiotensin-converting enzyme expression and endothelial dysfunction.

19 e and in a small collective of patients with endothelial dysfunction.

20 or individuals with blindness due to corneal endothelial dysfunction.

21 he vasculature, p66Shc-induced ROS engenders endothelial dysfunction.

22 effect of GATA2 and NF-kappaB and consequent endothelial dysfunction.

23 on Willebrand factor (vWF) is a biomarker of endothelial dysfunction.

24 improved NO bioavailability, and ameliorated endothelial dysfunction.

25 ay a facilitating role, probably mediated by endothelial dysfunction.

26 from diabetic oxidative stress and vascular endothelial dysfunction.

27 filtration rate and showed evidence of renal endothelial dysfunction.

28 d to cell-based therapy for treating corneal endothelial dysfunction.

29 otects against systemic inflammation-induced endothelial dysfunction.

30 One of the first events in MVD is endothelial dysfunction.

31 F3 as an important protective factor against endothelial dysfunction.

32 (DMEK) for the surgical treatment of corneal endothelial dysfunction.

33 of pro-atherogenic genes and the subsequent endothelial dysfunction.

34 ammation and thrombogenicity associated with endothelial dysfunction.

35 ized (i.e., microvascular and macrovascular) endothelial dysfunction.

36 emic inflammation and coronary microvascular endothelial dysfunction.

37 ing fibrosis, immunologic abnormalities, and endothelial dysfunction.

38 nhealing wounds, and diabetes often involves endothelial dysfunction.

39 antigen levels as a marker for intrahepatic endothelial dysfunction.

40 ction of impaired renal microcirculation and endothelial dysfunction.

41 red to be a hallmark feature of pathological endothelial dysfunction.

42 ric oxide and prostanoid signalling underlie endothelial dysfunction.

43 rs to be particularly effective in reversing endothelial dysfunction.

44 scular microRNA-Sirtuin1 nexus that leads to endothelial dysfunction.

45 ofiles, and elevated biomarkers representing endothelial dysfunction.

46 modulate biological process associated with endothelial dysfunction.

47 egulation and as a potential therapeutic for endothelial dysfunction.

48 ctive platelets, hypercoagulable status, and endothelial dysfunction.

49 s in CHD patients, even in those with severe endothelial dysfunction.

50 ads to membrane disruption, ATP release, and endothelial dysfunction.

51 plasma Abeta42 correlated with diabetes and endothelial dysfunction.

52 .g. plasma [ATP]) as opposed to an intrinsic endothelial dysfunction.

53 wing transplantation and partially prevented endothelial dysfunction.

54 ine the real prevalence of microvascular and endothelial dysfunction.

55 of 20 analytes involved in angiogenesis and endothelial dysfunction.

56 ture cell therapy products destined to treat endothelial dysfunctions.

57 in the fetal heart, and promoted peripheral endothelial dysfunction (70.9 +/- 5.6% AUC of normoxic c

58 RATIONALE: Endothelial dysfunction, a major predictor of late cardi

59 toid arthritis (RA) has been associated with endothelial dysfunction, a pathophysiological feature of

60 xidative stress, sympathetic activation, and endothelial dysfunction, all of which are critical media

61 injury, inflammation, oxidative stress, and endothelial dysfunction, all of which may perpetuate a n

62 ell and extracellular trap formation, induce endothelial dysfunction, alter the phenotypes of dendrit

63 (.)) production, but the direct link between endothelial dysfunction and aggravation of CHF is not di

64 In old mice, antibiotic treatment reversed endothelial dysfunction and arterial stiffening and atte

65 oxidative stress- and inflammation-mediated endothelial dysfunction and arterial stiffening, is the

66 ers of the arteries, predominantly caused by endothelial dysfunction and arterial stiffening.

67 e TRPV1 TRP box ameliorated diabetes-induced endothelial dysfunction and augmented vascular regenerat

68 This could promote the development of endothelial dysfunction and cardiovascular disease in SL

69 (MPs) have emerged as a surrogate marker of endothelial dysfunction and cardiovascular risk.

70 provide direct evidence of the link between endothelial dysfunction and CHF.

71 show that TNF-alpha, a known risk factor for endothelial dysfunction and CVD, induces H19 expression

72 evident after the occurrence of age-related endothelial dysfunction and diminished distensibility.

73 capacity in SCCOR participants, and between endothelial dysfunction and FEV1 or FEV1/FVC in HeartSCO

74 However, there was no association between endothelial dysfunction and FEV1, FEV1/FVC, low-attenuat

75 NOS(-/-) ) mice, which exhibit hypertension, endothelial dysfunction and FGR.

76 of endothelial PGC-1alpha sensitized mice to endothelial dysfunction and hypertension in response to

77 ecause of SOD2 hyperacetylation and promotes endothelial dysfunction and hypertension.

78 ension, and activation of TLR4 and -9 causes endothelial dysfunction and hypertension.

79 iR-204 in the aorta, and its absence rescues endothelial dysfunction and impaired blood pressure decl

80 1-knockout mice from type 1 diabetes-induced endothelial dysfunction and impaired vascular regenerati

81 e increased vascular permeability and edema, endothelial dysfunction and impaired vasomotion, microem

82 report that microRNA-204 (miR-204) promotes endothelial dysfunction and impairment in blood pressure

83 in gut microbiota linked with attenuation of endothelial dysfunction and increase in blood pressure i

84 Vascular endothelial dysfunction and increased arterial stiffness

85 Selective brain endothelial dysfunction and increased permeability of th

86 hree-variable model that included markers of endothelial dysfunction and inflammation accurately dete

87 CPB efficiently reduced cfDNA/NETs-mediated endothelial dysfunction and inflammation and might repre

88 Migraine is linked to endothelial dysfunction and is considered to be a system

89 could be one of the mechanisms of UA-induced endothelial dysfunction and kidney disease.

90 e stress-mediated arterial dysfunction (e.g. endothelial dysfunction and large elastic artery stiffen

91 esis that the vascular amylin deposits cause endothelial dysfunction and microvascular injury and are

92 In pulmonary arterial hypertension (PAH), endothelial dysfunction and obliterative vascular diseas

93 dium falciparum infection is associated with endothelial dysfunction and parasite sequestration.

94 ipid metabolism, PCSK9 levels associate with endothelial dysfunction and predict cardiovascular event

95 lar quercetin, have been shown to ameliorate endothelial dysfunction and reduce blood pressure (BP),

96 Endothelial dysfunction and reduced nitric oxide (NO) bi

97 Endothelial dysfunction and reduced nitric oxide (NO) si

98 in pregnancies associated with hypertension, endothelial dysfunction and reduced NO bioavailability.

99 eactivity through increased pulmonary artery endothelial dysfunction and remodeling, or enhanced gene

100 es and vascular oxidative stress, leading to endothelial dysfunction and subsequent development of va

101 PM2.5-induced endothelial dysfunction and systemic inflammation have b

102 identify a causal link between dietary salt, endothelial dysfunction and tau pathology, independent o

103 helial senescence is the main determinant of endothelial dysfunction and thus of age-related cardiova

104 d can improve cardiac recovery by minimizing endothelial dysfunction and tissue injury.

105 clusion of the pulmonary arterioles owing to endothelial dysfunction and uncontrolled proliferation o

106 tho deficiency lead to chronic inflammation, endothelial dysfunction and vascular calcifications.

107 ndings, we observed elevations in markers of endothelial dysfunction and vascular damage in the serum

108 ted with maternal ancestry may contribute to endothelial dysfunction and vascular disease.

109 w approach to treat diseases associated with endothelial dysfunction and vascular hyperpermeability.

110 Medin induces endothelial dysfunction and vascular inflammation, yet d

111 Endothelial dysfunction and vascular leak, pathogenic ha

112 se life-threatening disease characterized by endothelial dysfunction and vascular leakage.

113 eed, by fashioning an underlying template of endothelial dysfunction and vulnerability, the robust in

114 thout SDB, which seems to be associated with endothelial dysfunction and, in part, increased MSNA res

115 m that describes overlapping "hyperhemolytic-endothelial dysfunction" and "high hemoglobin-hypervisco

116 apy can also cause myocardial damage, induce endothelial dysfunction, and alter cardiac conduction.

117 metabolism, homocysteine, oxidative stress, endothelial dysfunction, and cardiovascular and potentia

118 on in renal vascular resistance, reversal of endothelial dysfunction, and increased activation of the

119 orrelated with decreased NO bioavailability, endothelial dysfunction, and increased blood pressure.

120 is typical in people with diabetes, reflects endothelial dysfunction, and increases the risk of end-o

121 lic dysfunction, pulmonary vascular disease, endothelial dysfunction, and peripheral abnormalities.

122 modeling, reversal of cardiac and peripheral endothelial dysfunction, and recovery of deranged vascul

123 intercellular adhesion molecule-1 (ICAM-1), endothelial dysfunction, and smooth muscle cell (SMC) pr

124 lar ER stress and ER stress-induced vascular endothelial dysfunction, and that miR-204 promotes vascu

125 have been associated with disease severity, endothelial dysfunction, and vasodilation.

126 Oxidative/nitrosative stress and endothelial dysfunction are hypothesized to be central t

127 er, the GPCR signaling pathways that promote endothelial dysfunction are incompletely understood.

128 As disturbed angiogenesis and endothelial dysfunction are strongly implicated in T2D a

129 n, high VWF:Ag levels, probably representing endothelial dysfunction, are associated with prognosis i

130 In old mice, antibiotics reversed endothelial dysfunction [area-under-the-curve carotid ar

131 WD feeding resulted in aortic stiffness and endothelial dysfunction as determined in vivo by pulse w

132 of evidence that point to both platelet and endothelial dysfunction as essential components of COVID

133 d develop significantly different degrees of endothelial dysfunction as measured by syndecan-1.

134 Endothelial dysfunction, as demonstrated by FMD reductio

135 D (SAM-WD) and SAM on regular diet displayed endothelial dysfunction, as evidenced by impaired acetyl

136 racellular BiP/GRP78 as a novel regulator of endothelial dysfunction associated with ALI.

137 t may have a role in preventing the vascular endothelial dysfunction associated with As exposure.

138 remains about how diet affects the vascular endothelial dysfunction associated with disordered insul

139 of therapeutic benefit in the prevention of endothelial dysfunction associated with preeclampsia.

140 Conversely, laminar flow protects against endothelial dysfunction, at least in the initial phases

141 that global deletion of collectrin leads to endothelial dysfunction, augmented salt sensitivity, and

142 culating levels of systemic inflammation and endothelial dysfunction biomarkers in patients with LI.

143 Concentrations of inflammatory and endothelial dysfunction biomarkers were determined at cl

144 increased risk of myocardial infarction and endothelial dysfunction, but a link between endothelial

145 Radiation causes endothelial dysfunction, but no study has investigated r

146 ane, from MetS patients were shown to induce endothelial dysfunction, but their role in early stage o

147 Shear stress antagonises endothelial dysfunction by increasing nitric oxide forma

148 In conclusion, shear stress counteracts endothelial dysfunction by suppressing the pro-inflammat

149 04 (miR-204) promotes vascular ER stress and endothelial dysfunction by targeting the Sirtuin1 (Sirt1

150 Prevention of endothelial dysfunction, by endothelial PTP1B deficiency

151 Endothelial dysfunction caused by the combined action of

152 al translocation, inflammatory response, and endothelial dysfunction), coagulopathy, and angiogenesis

153 PG involvement may provide early evidence of endothelial dysfunction, consistent in BA women with the

154 KEY POINTS: Ageing-induced endothelial dysfunction contributes to organ dysfunction

155 Since vascular and endothelial dysfunction develop in patients with coarcta

156 Endothelial dysfunction does not mediate the association

157 of adult age, the R6/2 mouse developed overt endothelial dysfunction due to an inability to increase

158 Maternal endothelial dysfunction due to circulating factors of fe

159 ve populations may have in common underlying endothelial dysfunction due to genetic or environmental

160 These data support the concept that endothelial dysfunction due to high circulating sFLT1 ma

161 From 66 patients with irreversible corneal endothelial dysfunction dues to Fuchs' dystrophy who enr

162 Endothelial dysfunction (ED) is a parameter of early ACD

163 Endothelial dysfunction (ED) is involved in the developm

164 IR is responsible for Endothelial Dysfunction (ED) through the impairment of e

165 Few studies have assessed endothelial dysfunction (ED), an early marker of subclin

166 the subjects respectively; and microvascular endothelial dysfunction (endothelial coronary flow reser

167 -standing anterior uveitis increases risk of endothelial dysfunction, especially in the setting of in

168 inally, chronic high-salt ingestion produces endothelial dysfunction, even in salt-resistant subjects

169 ciation between FEV1 and atherosclerosis for endothelial dysfunction had no impact.

170 However, the role of CIRP in causing endothelial dysfunction has not been investigated.

171 ) in cancer therapy, adverse effects such as endothelial dysfunction have to be considered.

172 ations and correction of learning/cognition, endothelial dysfunction, hemostasis, bone mineralization

173 Systemic inflammation, endothelial dysfunction, hepatic insulin resistance, oxi

174 events in PAD patients is likely related to endothelial dysfunction, highlighting the necessity for

175 lucose metabolism, inflammation, adipokines, endothelial dysfunction, IGF axis, and iron store plus a

176 ascular diseases, driven largely by vascular endothelial dysfunction (impaired endothelium-dependent

177 retains eNOS substrate activity and reverses endothelial dysfunction: implications for the COX-2/ADMA

178 m uric acid levels with allopurinol improves endothelial dysfunction in 80 participants >/=18 years o

179 pressing endothelial autophagy and promoting endothelial dysfunction in a chronic pressure overload m

180 during the day is pathognomonic for corneal endothelial dysfunction in advanced Fuchs endothelial co

181 We hypothesized that androgen drives endothelial dysfunction in AE-PCOS women and oestradiol

182 shear adaptation is an essential part of the endothelial dysfunction in all forms of PAH and tested w

183 es CHF and further suggest a causal role for endothelial dysfunction in CHF development.

184 Hyperuricemia may contribute to endothelial dysfunction in CKD.

185 ther NFAT plays a role in the development of endothelial dysfunction in diabetes.

186 c avenue for mitigating post shock pulmonary endothelial dysfunction in hemorrhage shock.

187 ents and lipid oxidation products may induce endothelial dysfunction in HIV infection that could be p

188 Androgens drive endothelial dysfunction in lean and obese AE-PCOS.

189 annabinoid and autonomic nervous systems and endothelial dysfunction in mediating the complex interpl

190 inflammatory cascade potentially leading to endothelial dysfunction in OSA.

191 Chronic heart failure (CHF) induces endothelial dysfunction in part because of decreased nit

192 disease (PD) is known to be associated with endothelial dysfunction in patients with coronary artery

193 oscillatory shear stress further exacerbates endothelial dysfunction in patients with moderate-severe

194 ive against oscillatory shear stress-induced endothelial dysfunction in patients with moderate-severe

195 ide intracellular channel 4) is a feature of endothelial dysfunction in pulmonary arterial hypertensi

196 udy were to determine how IFN-alpha promotes endothelial dysfunction in SLE, focusing on its regulati

197 ghting DECT-PBV as a biomarker of reversible endothelial dysfunction in smokers with CAE.

198 Microvascular and endothelial dysfunction in the nonculprit artery territo

199 PC4 provides a Ca(2+) source associated with endothelial dysfunction in the pathophysiology of PAH.

200 evel of certain macronutrients contribute to endothelial dysfunction in vascular diabetes complicatio

201 that cigarette smoke extract (CSE) triggers endothelial dysfunction in vitro by initiating oxidative

202 esults illustrate the importance of studying endothelial dysfunction in vitro over prolonged periods.

203 veral manifestations of cerebral microvessel endothelial dysfunction including blood-brain barrier dy

204 oxidative stress in mediating microvascular endothelial dysfunction, including potential modulatory

205 xcretion, and vascular inflammation leads to endothelial dysfunction, increased vascular resistance,

206 elet activation, oxidation and inflammation, endothelial dysfunction, increased vascular stiffness, c

207 ), which was associated with protection from endothelial dysfunction induced by Ang II.

208 e most apparent for biomarkers of adiposity, endothelial dysfunction, inflammatory cell recruitment,

209 on, attenuated oxidative stress, ameliorated endothelial dysfunction, inhibited inflammation, and sup

210 ngs presented herein suggest that As-induced endothelial dysfunction involves the hyperactivation of

211 Endothelial dysfunction is a central pathomechanism in d

212 Endothelial dysfunction is a characteristic of many vasc

213 Endothelial dysfunction is a characteristic of systemic

214 Endothelial dysfunction is a crucial step in atheroscler

215 ammation, hemostasis, and vascular tone, and endothelial dysfunction is a hallmark of atherosclerosis

216 Endothelial dysfunction is a hallmark of preeclampsia, a

217 Endothelial dysfunction is a hallmark of tissue injury a

218 lular activation and inflammation leading to endothelial dysfunction is associated with cardiovascula

219 Endothelial dysfunction is characterised by aberrant red

220 Endothelial dysfunction is evident in major depressive d

221 Endothelial dysfunction is induced by inflammatory media

222 direct evidence of vascular inflammation and endothelial dysfunction is lacking.

223 al charcoal-heated hookah smoking, the acute endothelial dysfunction is masked by high levels of carb

224 Endothelial dysfunction is of interest in relation to sm

225 e remained unaffected, thus the mechanism of endothelial dysfunction is poorly defined.

226 n the development of renal disease, in which endothelial dysfunction is regarded as the key mechanism

227 Endothelial dysfunction is the common molecular basis of

228 Corneal endothelial dysfunction is the leading indication for co

229 Endothelial dysfunction is widely implicated in cardiova

230 In this review, we focus on what endothelial dysfunction is, what causes it, how it leads

231 Endothelin-1, a marker of endothelial dysfunction, is a potent vasoconstrictor rel

232 blood flow in driving pathologies, including endothelial dysfunction, ischemia, and stroke.

233 iparum malaria and have been associated with endothelial dysfunction (L-arginine), metabolic acidosis

234 thelial cells (ECs), one of the hallmarks of endothelial dysfunction leading to cardiovascular disord

235 Thus, endothelial dysfunction leads to a wide variety of disea

236 Endothelial dysfunction links thrombotic microangiopathy

237 Pulmonary vascular disease, pulmonary endothelial dysfunction, liver fibrosis, renal disease,

238 xic pregnancy in young offspring accelerated endothelial dysfunction (maximal arterial relaxation to

239 Vascular endothelial dysfunction may play an important role in th

240 Endothelial dysfunction may pose a particularly signific

241 Improvement of endothelial dysfunction may reduce colorectal cancer ris

242 Endothelial dysfunction may underlie this; however, the

243 METHODS AND hIRECO demonstrated significant endothelial dysfunction measured by blunted endothelium-

244 hIRECO demonstrated significant endothelial dysfunction measured by blunted endothelium-

245 Portal venous pressure and intrahepatic endothelial dysfunction might account for the selective

246 l for targeting novel pathways implicated in endothelial dysfunction, mitochondrial oxidative stress,

247 adipokines, and biomarkers of inflammation, endothelial dysfunction, myocyte injury and stress, and

248 Endothelial dysfunction/NO bioavailability is associated

249 (ER) stress has been implicated in vascular endothelial dysfunction of obesity, diabetes, and hypert

250 According to this concept, endothelial dysfunction of the renal microcirculation ca

251 migraine and coronary heart disease such as endothelial dysfunction or vasospasm should be discussed

252 hat miR-214(-/-) mice were protected against endothelial dysfunction, oxidative stress, and increased

253 epletion blunted Ang II-induced SBP rise and endothelial dysfunction (P<0.05), compared with isotype

254 In addition, cerebral endothelial dysfunction (particularly at the level of th

255 Endothelial dysfunction plays a pivotal role in the path

256 utic strategy to limit the development of an endothelial dysfunction post-ACS.

257 le atrophy, which is common in patients with endothelial dysfunction related pathologies.

258 ved circulating PAR2 agonist and mediator of endothelial dysfunction-related microvascular diabetes c

259 beta synthase (CBS) gene are known to cause endothelial dysfunction responsible for cardiovascular a

260 seudophakic eyes of 54 patients with corneal endothelial dysfunction resulting from Fuchs endothelial

261 haracterized by chronic hemolytic anemia and endothelial dysfunction, resulting in a constant state o

262 the recognition of a crucial role played by endothelial dysfunction secondarily igniting cardiovascu

263 Instead, airflow limitation and endothelial dysfunction seem to be unrelated and mutuall

264 Patients with diabetes develop endothelial dysfunction shortly after diabetes onset tha

265 biota promotes atherosclerosis, and vascular endothelial dysfunction, signalled by impaired endotheli

266 We demonstrate that IFN-alpha promotes an endothelial dysfunction signature in HUVECs that is char

267 in angiotensin-converting enzyme expression, endothelial dysfunction, smooth muscle contractility, an

268 hk1(-/-) mice were characterized by enhanced endothelial dysfunction, suggesting a local protective r

269 re both associated with oxidative stress and endothelial dysfunction, suggesting common mechanistic o

270 atory response (IP-10, IL-8, IL-6, and OPG), endothelial dysfunction (sVCAM-1 and sICAM-1), and coagu

271 netically predisposed and is associated with endothelial dysfunction that is induced by oxidative str

272 ular amylin deposition as a trigger of brain endothelial dysfunction that is modulated by plasma apol

273 Hyperglycaemia is implicated in driving endothelial dysfunction that might underpin the link bet

274 Oxidative stress and inflammation promote endothelial dysfunction thereby hampering cerebral perfu

275 o-ANP processing, and defects in Corin cause endothelial dysfunction through MAPK and eNOS signaling

276 use endothelial damage and contribute to the endothelial dysfunction typical for PE.

277 mechanisms by which exposure to As leads to endothelial dysfunction using a mouse model and cultured

278 hat Sirt3 depletion in hypertension promotes endothelial dysfunction, vascular hypertrophy, vascular

279 (Sirt3 overexpression), which protects from endothelial dysfunction, vascular oxidative stress, and

280 High levels of sFlt-1 and sEng result in endothelial dysfunction, vasoconstriction, and immune dy

281 The detrimental role of miR-19b in inducing endothelial dysfunction was confirmed in vivo.

282 nk between declining adropin and age-related endothelial dysfunction was documented by a progressivel

283 CHF-induced endothelial dysfunction was less marked in endoPTP1B(-/-

284 Endothelial dysfunction was manifested by decreased leve

285 f e-cigarette users, increased ROS linked to endothelial dysfunction was observed, as indicated by im

286 s had a nonpathological study: macrovascular endothelial dysfunction was present in 60% of the patien

287 Our measure of cardiovascular dysfunction (endothelial dysfunction) was most profound in lean women

288 A total of 216 patients with endothelial dysfunction were screened, and 50 eyes of 38

289 d to inflammation (inflammatory response and endothelial dysfunction) were related to the severity of

290 xide signalling contributes significantly to endothelial dysfunction, whereas in resistance arteriole

291 diabetic macro- and microvascular disease is endothelial dysfunction, which appears well before any c

292 now considered a cornerstone in I/R-related endothelial dysfunction, which further impairs local mic

293 iodontal disease is associated with systemic endothelial dysfunction, which has been implicated in pr

294 Endothelial dysfunction, which is caused by endothelial

295 Patients with structural CMD have endothelial dysfunction, which leads to diminished peak

296 red e-liquids or e-cigarette use exacerbates endothelial dysfunction, which often precedes cardiovasc

297 Loss of TM and eNOS causes endothelial dysfunction, which results in suppressed gen

298 IFN-gamma ex vivo caused significant endothelial dysfunction, which was reduced by superoxide

299 , 6, and 12 months in patients with isolated endothelial dysfunction with similar complication rates.

300 sights into the molecular mechanisms linking endothelial dysfunction with the pathogenesis of Alzheim