戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 with angiogenic capacity plus clear signs of endothelial to mesenchymal transition.
2 m endocardial endothelial cells via aberrant endothelial to mesenchymal transition.
3 ytes in vitro, and the ability to undergo an endothelial-to-mesenchymal transition.
4 ssion, revealed that REST silencing triggers endothelial-to-mesenchymal transition.
5 cence, pro-inflammatory gene expression, and endothelial-to-mesenchymal transition.
6 ed through pattern-recognition receptors and endothelial-to-mesenchymal transition.
7 ion and a synthetic repertoire suggestive of endothelial-to-mesenchymal transition.
8 , suggest that HYAL2 is important to inhibit endothelial-to-mesenchymal transition.
9 reases mitral valve area in association with endothelial-to-mesenchymal transition.
10 n-driven transcription program that leads to endothelial-to-mesenchymal transition.
11  gain of mesenchymal markers consistent with endothelial-to-mesenchymal transition.
12 genesis, enhanced vascular permeability, and endothelial-to-mesenchymal transition.
13 ndothelial cells suppressed induction of the endothelial-to-mesenchymal transition.
14 ich could lead to disturbed angiogenesis and endothelial-to-mesenchymal-transition.
15 umor vasculature that displayed hallmarks of endothelial-to-mesenchymal transition, a process of prev
16 expectedly, myelin debris engulfment induces endothelial-to-mesenchymal transition, a process that co
17 d L1CAM (L1 cell adhesion molecule), a known endothelial-to-mesenchymal transition activator, and its
18 hogenetic proteins 7 effectively ameliorates endothelial to mesenchymal transition and experimental E
19 vely active ALK2 in endothelial cells caused endothelial-to-mesenchymal transition and acquisition of
20 vation of TGF-beta signaling causes aberrant endothelial-to-mesenchymal transition and altered extrac
21 hearts and establish a possible link between endothelial-to-mesenchymal transition and cardiac fibros
22 D3 pathways, protecting the endothelium from endothelial-to-mesenchymal transition and consequent liv
23 s hypercontractility, via the attenuation of endothelial-to-mesenchymal transition and elastin breaks
24 y inactive HDAC3 expression rescues aberrant endothelial-to-mesenchymal transition and epigenetic sil
25 o the AVC of embryonic mouse hearts, undergo endothelial-to-mesenchymal transition and express marker
26 olarization, M2 macrophage infiltration, and endothelial-to-mesenchymal transition and improves endot
27   Moreover, Frs2alpha mutants have defective endothelial-to-mesenchymal transition and neural crest c
28              Hyaluronan (HA) is required for endothelial-to-mesenchymal transition and normal heart d
29  mice causes increased HA, which may promote endothelial-to-mesenchymal transition and proliferation
30 lls that functions to constitutively inhibit endothelial-to-mesenchymal transition and protect agains
31 ence of epithelial-to-mesenchymal transition/endothelial-to-mesenchymal transition and protein expres
32 mor-associated macrophages, is implicated in endothelial-to-mesenchymal transition and survival strat
33       In summary, our data point to aberrant endothelial to mesenchymal transition as a common denomi
34 sts is multifactorial, recent data implicate endothelial-to-mesenchymal transition as an important so
35 n the 2 endocardial cell monolayers, undergo endothelial-to-mesenchymal transition as marked by loss
36  to PAH revealed significant RV fibrosis and endothelial-to-mesenchymal transition, as well as elevat
37  subset of cardiac fibroblasts by undergoing endothelial-to-mesenchymal transition, but whether cardi
38 Particularly, the ubiquitous presentation of endothelial-to-mesenchymal transition CAF, which may int
39 ion in SSc skin from both myofibroblasts and endothelial-to-mesenchymal-transitioning cells (EndoMT).
40             We showed that Adora2a-dependent endothelial-to-mesenchymal transition contributed to the
41                                              Endothelial-to-mesenchymal transition, detected as alpha
42 ag1, similar to the loss of Notch1, disrupts endothelial-to-mesenchymal transition during endocardial
43  patients in inducing tubular senescence and endothelial to mesenchymal transition (EndMT) and analyz
44 asing number of studies have also implicated endothelial to mesenchymal transition (EndMT) as a poten
45                                              Endothelial to mesenchymal transition (EndMT) is a leadi
46                                              Endothelial to mesenchymal transition (EndMT) is a proce
47                                              Endothelial to mesenchymal transition (EndMT) is a proce
48                                         Such endothelial to mesenchymal transition (EndMT) is associa
49                                              Endothelial to mesenchymal transition (EndMT) plays a ma
50                                              Endothelial to mesenchymal transition (EndMT) was origin
51 ere, we investigate the effects of leptin on endothelial to mesenchymal transition (EndMT), which imp
52 icant number of endothelial cells resembling endothelial to mesenchymal transition (EndMT), which is
53     Here, we investigated the role played by endothelial-to-mesenchymal transition (EndMT) and its ke
54 ism by which M1-polarized macrophages induce endothelial-to-mesenchymal transition (EndMT) and promot
55        Ablation of ERG expression results in endothelial-to-mesenchymal transition (EndMT) and sponta
56 orithm (SingleR), we showed that DS promoted endothelial-to-mesenchymal transition (EndMT) by inducin
57                                              Endothelial-to-mesenchymal transition (EndMT) has been i
58                                              Endothelial-to-mesenchymal transition (EndMT) is a cellu
59                                              Endothelial-to-mesenchymal transition (EndMT) is a cruci
60                                              Endothelial-to-mesenchymal transition (EndMT) is crucial
61                                              Endothelial-to-mesenchymal transition (EndMT) is now wid
62                                              Endothelial-to-mesenchymal transition (EndMT) occurs dur
63  on published literature on inflammation and endothelial-to-mesenchymal transition (EndMT) pathway ge
64 (PVEC) from patients with PVT identified the endothelial-to-mesenchymal transition (EndMT) pathway up
65 al cells into mesenchymal cells featuring an endothelial-to-mesenchymal transition (EndMT) phenotype.
66 ovisa et al. demonstrate the contribution of endothelial-to-mesenchymal transition (EndMT) to kidney
67                                              Endothelial-to-mesenchymal transition (EndMT) was assess
68 ociation between cardiovascular diseases and endothelial-to-mesenchymal transition (EndMT), a cellula
69 lar changes are attributed to disruptions in endothelial-to-mesenchymal transition (EndMT), a key pro
70 e source of myofibroblasts in fibrosis is an endothelial-to-mesenchymal transition (EndMT), a process
71                               Disruptions in endothelial-to-mesenchymal transition (EndMT), a process
72 tivation of TGF-beta signaling, induction of endothelial-to-mesenchymal transition (EndMT), and cardi
73 ied the diagnostic value of three markers of endothelial-to-mesenchymal transition (EndMT), fascin1,
74 e a mesenchymal phenotype by a process named endothelial-to-mesenchymal transition (EndMT), which aff
75 ted signaling in endothelial cells (ECs) and endothelial-to-mesenchymal transition (EndMT).
76 elial cells (ECs) including pro-inflammatory endothelial-to-mesenchymal transition (EndMT).
77 hanical forces are frequently accompanied by endothelial-to-mesenchymal transition (EndMT).
78 e in TGFbeta signaling leading to widespread endothelial-to-mesenchymal transition (EndMT).
79 fferentiation of EC through a process termed endothelial-to-mesenchymal transition (EndMT).
80  which is suggestive of the process known as endothelial-to-mesenchymal transition (EndMT).
81 ts during kidney fibrosis via the process of endothelial-to-mesenchymal transition (EndMT).
82                                              Endothelial to mesenchymal transition (EndoMT) is essent
83                                    Recently, endothelial to mesenchymal transition (EndoMT), a newly
84 beta1 stimulated higher pSmad2 signaling and endothelial to mesenchymal transition (EndoMT)-related g
85 y2 transcription factor in radiation-induced endothelial-to-mesenchymal transition (EndoMT) and its i
86 bility of TGFB-related genes associated with endothelial-to-mesenchymal transition (EndoMT) and parti
87                                              Endothelial-to-mesenchymal transition (EndoMT) and shedd
88 ith COUP-TFII knockdown also readily undergo endothelial-to-mesenchymal transition (EndoMT) and subse
89                                    Recently, endothelial-to-mesenchymal transition (EndoMT) and the r
90 this study, we determined how epsins enhance endothelial-to-mesenchymal transition (EndoMT) in athero
91 orming growth factor beta (TGF-beta) induces endothelial-to-mesenchymal transition (EndoMT) in murine
92                                          The endothelial-to-mesenchymal transition (EndoMT) is a cruc
93 retrieved from the lung showed expression of endothelial-to-mesenchymal transition (EndoMT) markers.
94                                              Endothelial-to-mesenchymal transition (EndoMT) may be a
95 immune encephalomyelitis (EAE), we show that endothelial-to-mesenchymal transition (EndoMT) occurs in
96                                              Endothelial-to-mesenchymal transition (EndoMT) plays an
97 hlight interesting parallels between EHT and endothelial-to-mesenchymal transition (EndoMT), which oc
98 tribute to cardiac development by undergoing endothelial-to-mesenchymal transition (EndoMT).
99 etion of TbetaRII in the endothelium reduced endothelial-to-mesenchymal transition (EndoMT).
100 ontribute to fibrosis through the process of endothelial-to-mesenchymal transition (EndoMT).
101 s characterized by endothelial inflammation, endothelial-to-mesenchymal transition, endothelial-to-im
102 ooth muscle (SM)-like mesenchymal phenotype (endothelial-to-mesenchymal transition), explaining both
103 ated in epithelial-to-mesenchymal transition/endothelial-to-mesenchymal transition) expression in per
104                                              Endothelial-to-mesenchymal transition has been described
105 c endothelial cells were more susceptible to endothelial-to-mesenchymal transition in response to tra
106 ndothelial dysfunction in the kidney, and of endothelial-to-mesenchymal transition in streptozotocin-
107                 Positivity for markers of an endothelial-to-mesenchymal transition indicates possible
108  serum reference range (100-800 nM) reverted endothelial-to-mesenchymal transition, inhibited cellula
109 f nitric oxide, endothelial cell injury, the endothelial-to-mesenchymal transition, insulin resistanc
110 of endothelial specificity in the process of endothelial-to-mesenchymal transition involved in renal
111    We further demonstrate that such aberrant endothelial to mesenchymal transition involving endocard
112 ced epithelial-to-mesenchymal transition and endothelial-to-mesenchymal transition, lessened mitochon
113 ell adhesion molecule 1 (CD31) and increased endothelial-to-mesenchymal transition markers, alphaSM a
114 c endocardial defects contribute to abnormal endothelial-to-mesenchymal transition, NOTCH signaling,
115 n and not derived from bone marrow lineages, endothelial-to-mesenchymal transition, or blood.
116  differentiation from bone marrow (35%), the endothelial-to-mesenchymal transition program (10%) and
117 nished nitric oxide production, and fostered endothelial to mesenchymal transition, providing potenti
118 e-specific LRP1 expression repression by the endothelial-to-mesenchymal transition regulator SNAIL.
119                                       Excess endothelial-to-mesenchymal transition, resulting in incr
120 markers and transcription factors regulating endothelial-to-mesenchymal transition, similar to TGFbet
121       Furthermore, Poly(I:C) and LPS induced endothelial to mesenchymal transition that was reversed
122 ated that this signaling pathway promotes an endothelial-to-mesenchymal transition that supplies cell
123 thin the atrioventricular canal undergoes an endothelial-to-mesenchymal transition to give rise to mi
124 olarization, M2 macrophage infiltration, and endothelial-to-mesenchymal transition were greatly suppr

 
Page Top