ライフサイエンスコーパス: endothelium-dependent

1 in the radial artery, suggesting that it is endothelium-dependent.

2 nduced vasodilation in mesenteric vessels is endothelium-dependent.

3 To test this, we measured endothelium-dependent (acetylcholine) and -independent v

4 serotonin; both P = .03); relaxation to the endothelium-dependent agonist acetylcholine was attenuat

5 Under baseline conditions, responses to the endothelium-dependent agonist acetylcholine were not aff

6 d intradermal microdialysis perfusion of the endothelium-dependent agonist acetylcholine, alone and i

7 contraction amplifies vasodilatation to the endothelium-dependent agonist ACh, whereas there was no

8 Deletion of PTP1B improved both endothelium dependent and independent NO-mediated dilati

9 tionally, obese mice demonstrate an impaired endothelium dependent and independent vasodilation to ac

10 Endothelium-dependent and -independent arterial vasodila

11 Intravital microscopy was used to assess endothelium-dependent and -independent arteriolar vasodi

12 Resveratrol elicits endothelium-dependent and -independent dilation of retin

13 Endothelium-dependent and -independent flow-mediated vas

14 ears that the dilatation is mediated by both endothelium-dependent and -independent signalling pathwa

15 and SV graft vasomotor and flow responses to endothelium-dependent and -independent stimuli 5 years a

16 e, distal segments failed to respond to both endothelium-dependent and -independent stimuli.

17 al coronary segments that fail to respond to endothelium-dependent and -independent stimuli.

18 hy, higher white blood cell count, and lower endothelium-dependent and -independent vasodilation in t

19 ignificantly improved vascular reactivity to endothelium-dependent and -independent vasodilators as w

20 peroxynitrite (ONOO(-)) inhibitors improved endothelium-dependent and EDHF-mediated relaxations and

21 Forearm endothelium-dependent and endothelium-independent vasodi

22 gen species, endothelial cell apoptosis, and endothelium-dependent and endothelium-independent vasore

23 Endothelium-dependent and independent vasodilation was t

24 moglobin (Hb) may impact the transduction of endothelium-dependent and nitric oxide (NO)-mediated vas

25 Nebivolol but not metoprolol induced endothelium-dependent and nitric oxide-dependent relaxat

26 Flow-mediated, endothelium-dependent and nitroglycerin-induced, endothe

27 The activation of PAR-1 causes endothelium-dependent arterial vasodilation and the rele

28 The vasorelaxation properties seemed to be endothelium dependent, as well as nitric oxide (NO) and

29 al agents (either endothelium-independent or endothelium-dependent) at 1 year.

30 y during transfusion, followed by testing of endothelium-dependent blood flow with increasing doses o

31 erol supplementation significantly increased endothelium-dependent brachial artery flow-mediated dila

32 The primary outcome was change in endothelium-dependent brachial artery flow-mediated dila

33 of isolated arteries with palmitate impaired endothelium-dependent but not vascular smooth muscle fun

34 vasodilator responses to acetylcholine (ACh; endothelium dependent) but not sodium nitroprusside (SNP

35 stal segments: incremental atrial pacing for endothelium-dependent cases; and intracoronary nitroglyc

36 kidney function and hemodynamic responses to endothelium-dependent challenge were assessed in pigs af

37 en evaluated for suspected ischemia, and the endothelium-dependent component is linked with adverse o

38 d releases endothelin-1 (ET-1) and initiates endothelium-dependent constriction.

39 lial stimulation, and initiate ET-1-mediated endothelium-dependent constriction.

40 Thrombin did not cause endothelium-dependent contraction of young arteries.

41 Our findings show that endothelium-dependent control of swine coronary artery t

42 tment is effective in partial restoration of endothelium-dependent coronary flow.

43 n the collateral dependent region, preserves endothelium-dependent coronary vessel function, and upre

44 This allows the combined assessment of endothelium-dependent (CPT) and endothelium-independent

45 osine hydroxylase-positive axon density) and endothelium-dependent dilatation (carbachol) of the MCA

46 Endothelium-dependent dilatation (ED) is abnormal in pat

47 aerobic exercise is associated with enhanced endothelium-dependent dilatation (EDD) in older humans,

48 Acetylcholine (ACh)-induced peak endothelium-dependent dilatation (EDD) was lower in isol

49 Endothelium-dependent dilatation (EDD), assessed by the

50 ximately 30% (P <0.05) reduction in arterial endothelium-dependent dilatation (EDD).

51 anism involved in the age-related decline in endothelium-dependent dilatation (EDD).

52 Spontaneous vasomotor tone, endothelium-dependent dilatation and adrenergic vasocons

53 Acetylcholine caused endothelium-dependent dilatation that was decreased in o

54 Heat therapy improved endothelium-dependent dilatation, arterial stiffness, in

55 y vascular endothelial dysfunction (impaired endothelium-dependent dilatation, EDD) and aortic stiffe

56 cular endothelial dysfunction (e.g. impaired endothelium-dependent dilatation, EDD).

57 protective endothelial functions, including endothelium-dependent dilatation.

58 ctase inhibitors (statins) improve cutaneous endothelium-dependent dilatation; however, whether stati

59 term high-fat diet feeding had no effect on endothelium dependent dilation.

60 Ex vivo carotid artery endothelium-dependent dilation (EDD) to increasing doses

61 al function, as indicated in part by reduced endothelium-dependent dilation (EDD).

62 e inhibitor (apocynin), or anti-TNF restored endothelium-dependent dilation in Lepr(db) mice.

63 bitor (apocynin), or anti-TNF-alpha restored endothelium-dependent dilation in the ZOF rats.

64 Endothelium-dependent dilation is mediated by the releas

65 ere no sex differences in either NO-mediated endothelium-dependent dilation or endothelium-independen

66 the control diet, the HM/LF diet diminished endothelium-dependent dilation to 10 micromol/L acetylch

67 ioglitazone treatment significantly improved endothelium-dependent dilation to bradykinin (P=0.01) wi

68 Endothelium-dependent dilation was blunted in MDD and me

69 Finally, endothelium-dependent dilation was lower (P < 0.01) in i

70 ion (n=8; 7 women; 19-37 years), NO-mediated endothelium-dependent dilation was preserved, but endoth

71 sion of these NAD(P)H subunits and abrogated endothelium-dependent dilation.

72 Vascular endothelial function (endothelium-dependent dilation; EDD), nitric oxide (NO)/

73 t sympathetic vasoconstriction competes with endothelium-dependent dilator activity to determine post

74 VC) to local intra-arterial infusion of ACh (endothelium-dependent dilator) during resting conditions

75 ure to high glucose has a concentration- and endothelium-dependent effect on the myogenic tone of rat

76 ear-activated Kir currents and inhibition of endothelium-dependent flow-induced vasodilatation (FIV)

77 and 30 normotensive control subjects during endothelium-dependent flow-mediated dilatation induced b

78 o measure changes in segmental lumen volume (endothelium-dependent function).

79 ) venous occlusion plethysmography to assess endothelium-dependent (% Hyper) and endothelium-independ

80 re sites of dynamic Ca(2+) events leading to endothelium dependent hyperpolarization (EDH)-mediated r

81 esenteric arteries the K(Ca)2.3 component of endothelium-dependent hyperpolarization (EDH) is lost fo

82 To further study the role of endothelium-dependent hyperpolarization (EDH), ACh trial

83 ions and limits the impact of the disease on endothelium-dependent hyperpolarization (EDH)-mediated v

84 ) channels serve as electrical amplifiers of endothelium-dependent hyperpolarization (EDH).

85 We propose that initiation of endothelium-dependent hyperpolarization is the underlyin

86 xation of vascular smooth muscle through the endothelium-dependent hyperpolarizing factor (EDHF) path

87 helium independent) and acetylcholine (ACh) (endothelium dependent) iontophoresis, flicker-light-indu

88 ascular superoxide and H2O2 production in an endothelium-dependent manner and elicited a concentratio

89 ferent physiologic or pharmacologic stimuli, endothelium-dependent (micro)vascular reactivity can be

90 he index of microvascular resistance; third, endothelium-dependent microvascular function assessment

91 In men, the HF F&V diet increased endothelium-dependent microvascular reactivity (P = 0.01

92 Endothelium-dependent microvascular relaxation response

93 The response to endothelium-dependent microvascular vasodilation was gre

94 Endothelium-dependent microvessel relaxation was moderat

95 d stable basal tone and the vasodilations to endothelium-dependent nitric oxide (NO)-mediated agonist

96 The authors examined whether CRP can affect endothelium-dependent nitric oxide (NO)-mediated dilatio

97 of CRP (7 microg/mL; 60 minutes) attenuated endothelium-dependent nitric oxide-mediated and prostacy

98 on the effects of aging and hypertension on endothelium-dependent nitric oxide-mediated vasodilation

99 We sought to determine whether endothelium-dependent NO regulates TG2 activity by S-nit

100 nificantly attenuated arteriolar dilation to endothelium-dependent NO-mediated agonists bradykinin an

101 CRP inhibits endothelium-dependent NO-mediated dilation in retinal ar

102 direct evidence for selective impairment of endothelium-dependent NO-mediated dilation of retinal ar

103 Endothelium-dependent NO-mediated dilation of retinal ar

104 nder normal conditions through constraint of endothelium-dependent NO-mediated vasodilatation in heal

105 The endothelium-dependent NO-mediated vasodilations to brady

106 By impairing endothelium-dependent NO-mediated vasoreactivity, CRP ca

107 Simvastatin elicits mainly an endothelium-dependent, NO-mediated dilation of retinal a

108 ial fluid, with emphasis on whether they are endothelium dependent or O(2) dependent.

109 CRP elicits endothelium-dependent oxidative stress and compromises n

110 olecular mechanisms underlying the pulmonary endothelium-dependent protective effects of APN.

111 his is the first study in humans to identify endothelium-dependent regulation of sympathetic vasocons

112 cellular signaling mechanism responsible for endothelium-dependent regulation of vascular smooth musc

113 ability of vascular rings to respond to the endothelium-dependent relaxant acetylcholine, both durin

114 Endothelium-dependent relaxation (EDR) is an initial key

115 ic tone was significantly potentiated, while endothelium-dependent relaxation (EDR) was impaired in s

116 38 prevented NADP(H) depletion and preserved endothelium-dependent relaxation and NO generation with

117 t to diabetes mellitus-induced impairment in endothelium-dependent relaxation and reendothelializatio

118 cids and GLP-1 were associated with improved endothelium-dependent relaxation compared with sham-oper

119 In addition, impaired acetylcholine-induced endothelium-dependent relaxation could be related to dec

120 2-CreNox2KO mice, along with preservation of endothelium-dependent relaxation during angiotensin II s

121 significantly improved acetylcholine-induced endothelium-dependent relaxation in AMPKalpha2(-/-) mice

122 ion of l-sepiapterin normalized the impaired endothelium-dependent relaxation in aortas isolated from

123 ease in STIM1 protein expression, attenuates endothelium-dependent relaxation in diabetic coronary ar

124 stress and SERCA oxidation and improved the endothelium-dependent relaxation in isolated mouse aorta

125 lose inspection reveals a specific effect on endothelium-dependent relaxation in mesenteric resistanc

126 We also observed impaired endothelium-dependent relaxation in resistant vessels fr

127 e control, exhibited striking improvement in endothelium-dependent relaxation in response to acetylch

128 Endothelium-dependent relaxation in response to shear st

129 Functionally, acetylcholine-induced, endothelium-dependent relaxation is impaired in T1DM mes

130 pid accumulation in the liver; and decreased endothelium-dependent relaxation of aorta.

131 reduced ejection fraction, mitral E/A ratio, endothelium-dependent relaxation of coronary arteries, t

132 emporal profile of Ca(2+) dynamics underlies endothelium-dependent relaxation of swine coronary arter

133 re no acute changes in BP or the NO-mediated endothelium-dependent relaxation of the brachial artery

134 the Ca(2+) concentration in the ER, and (3) endothelium-dependent relaxation that was attenuated in

135 Endothelium-dependent relaxation was enhanced in male CF

136 Vasodilator prostanoid contribution to endothelium-dependent relaxation was reduced in lobar ar

137 ared to the wild type, acetylcholine-induced endothelium-dependent relaxation was significantly impai

138 heterozygout mice (db/m) mice and effects on endothelium-dependent relaxation, insulin sensitivity, a

139 ha release, and improved coronary arteriolar endothelium-dependent relaxation.

140 arteries and the role played by caveolae in endothelium-dependent relaxation.

141 ae structure and integrity are essential for endothelium-dependent relaxation.

142 hereas, in pregnant rats, PVAT-media reduced endothelium-dependent relaxation.

143 ed with decreased NO production and impaired endothelium-dependent relaxation.

144 ich restored NO bioavailability and improved endothelium-dependent relaxations and HDL endothelium-pr

145 Impaired endothelium-dependent relaxations in renal arteries, car

146 mice exhibited an accelerated impairment of endothelium-dependent relaxations in response to in vitr

147 Hcy impaired endothelium-dependent relaxations of rat aortae and led

148 ived hyperpolarizing factor (EDHF)-mediated, endothelium-dependent relaxations of small mesenteric ar

149 helium-independent and prostacyclin-mediated endothelium-dependent relaxations were not changed.

150 gh glucose in vitro induced an impairment of endothelium-dependent relaxations, which was prevented b

151 ction, which is characterized by an impaired endothelium-dependent response to vasodilators and hyper

152 the alteration in neurovascular coupling and endothelium-dependent responses in somatosensory cortex

153 Endothelium-dependent responses to acetylcholine in pres

154 a-arterial infusion of ACh or ATP to augment endothelium-dependent signalling during exercise attenua

155 dy, we tested the hypothesis that increasing endothelium-dependent signalling during exercise in olde

156 In this study, increasing endothelium-dependent signalling during exercise signifi

157 However, augmentation of endothelium-dependent signalling via infusion of ACh or

158 exercise + infusion of ACh or ATP to augment endothelium-dependent signalling.

159 monstrate that, given a sufficient stimulus, endothelium-dependent sympatholysis remains intact in ol

160 ver, H(2)O(2) has never been shown to be the endothelium-dependent transferrable hyperpolarization fa

161 peraldosteronism is associated with impaired endothelium-dependent vascular reactivity owing to incre

162 contributes, at least partially, to impaired endothelium-dependent vascular relaxation and hypertensi

163 oxLDL-induced impairment of endothelium-dependent vascular relaxation of vascular ri

164 riments from mice showed that L5 compromised endothelium-dependent vascular relaxation through a nitr

165 ild and moderate HHcy aggravated HG-impaired endothelium-dependent vascular relaxation to acetylcholi

166 xposure of WKY rat aortas to IL-17F impaired endothelium-dependent vascular relaxation, whereas IL-17

167 hat this mechanism is important in impairing endothelium-dependent vascular relaxation.

168 mental role in regulating endothelial NO and endothelium-dependent vascular tone by deacetylating eNO

169 lationship between the two in the context of endothelium-dependent vascular tone is unknown.

170 Nor-NOHA markedly increased endothelium-dependent vasodilatation (up to 2-fold) in p

171 Higher endothelium-dependent vasodilatation by ACh or leptin wa

172 ilure Assessment score, leukocyte count, and endothelium-dependent vasodilatation conferred an increa

173 ctional hyperaemia are reduced with age, and endothelium-dependent vasodilatation declines with age i

174 ance (HOMA-IR), was associated with impaired endothelium-dependent vasodilatation in FH- (p < 0.03, a

175 related increases in oxidative stress impair endothelium-dependent vasodilatation in humans, leading

176 termine whether arginase activity diminishes endothelium-dependent vasodilatation in skeletal muscle

177 rginine completely inhibited the increase in endothelium-dependent vasodilatation induced by nor-NOHA

178 e results demonstrate that the impairment of endothelium-dependent vasodilatation induced by old age

179 We hypothesized that impaired endothelium-dependent vasodilatation is a predictor of m

180 In vivo bedside assessment of endothelium-dependent vasodilatation is an independent p

181 potassium (Kir) channels, but their role in endothelium-dependent vasodilatation is not clear.

182 Endothelium-dependent vasodilatation is reduced with adv

183 sine triphosphate release and stimulation of endothelium-dependent vasodilatation may explain impairm

184 Ageing reduces endothelium-dependent vasodilatation through an endothel

185 An endothelium-dependent vasodilatation value of 0.5% or le

186 hat contracting muscle selectively amplifies endothelium-dependent vasodilatation via activation of K

187 Endothelium-dependent vasodilatation was calculated as t

188 Endothelium-dependent vasodilatation was impaired in cor

189 Endothelium-dependent vasodilatation was impaired in hig

190 annel function was not altered, although the endothelium-dependent vasodilatation was severely impair

191 In logistic regression analysis, endothelium-dependent vasodilatation was the only predic

192 adrenergic-induced vasoconstriction, reduced endothelium-dependent vasodilatation, and enhanced hypox

193 ostatic conditions demonstrated a decline in endothelium-dependent vasodilatation, but restored the f

194 probably due (in part) to an improvement in endothelium-dependent vasodilatation.

195 airment of coronary blood flow responses and endothelium-dependent vasodilatation.

196 nnels, thereby causing hyperpolarization and endothelium-dependent vasodilatation.

197 Endothelium-dependent vasodilatations in response to mus

198 It induces endothelium dependent vasodilation, but the precise rece

199 The extract possesses the ability to induce endothelium dependent vasodilation, which is dependent o

200 tolerance and brachial artery flow-mediated, endothelium-dependent vasodilation (EDV) were assessed i

201 Almonds, compared with control, increased endothelium-dependent vasodilation (mean difference 4.1%

202 The change in endothelium-dependent vasodilation achieved with fasudil

203 IRT1 in the endothelium of arteries inhibits endothelium-dependent vasodilation and decreases bioavai

204 active hyperemic index (RHI), which measures endothelium-dependent vasodilation and is a surrogate ma

205 Here we show that SIRT1 promotes endothelium-dependent vasodilation by targeting endothel

206 ng-severe systolic hypertension and impaired endothelium-dependent vasodilation due to uncoupled NO s

207 MA levels and reduce nitric oxide levels and endothelium-dependent vasodilation in a murine model and

208 Fasudil increased endothelium-dependent vasodilation in CAD subjects from

209 pressure in healthy individuals and improves endothelium-dependent vasodilation in obese and overweig

210 Indeed, both flow-mediated dilation and endothelium-dependent vasodilation in response to acetyl

211 Impaired endothelium-dependent vasodilation is a hallmark of obes

212 determine whether putative modifications in endothelium-dependent vasodilation of the principal nutr

213 glutathionylation is increased with impaired endothelium-dependent vasodilation that is restored by t

214 One mechanistic hypothesis involves impaired endothelium-dependent vasodilation through reactive oxyg

215 histocompatibility complex class I inhibited endothelium-dependent vasodilation to acetylcholine.

216 de for a possible coupling mechanism linking endothelium-dependent vasodilation to bone remodeling.

217 mCMV-ND mice exhibited impaired endothelium-dependent vasodilation versus mock-ND at 9 a

218 PNA endothelium-dependent vasodilation was assessed in vitro

219 Coronary endothelium-dependent vasodilation was examined by infus

220 on was associated with larger improvement in endothelium-dependent vasodilation) (r=-0.48; P=0.01).

221 diabetic mice exhibited a marked decrease in endothelium-dependent vasodilation, a modest decrease in

222 s an important determinant of eNOS coupling, endothelium-dependent vasodilation, and superoxide produ

223 ysfunction, we measured endothelial markers, endothelium-dependent vasodilation, arteriolar glycocaly

224 placebo, allopurinol significantly improved endothelium-dependent vasodilation, by both forearm veno

225 LRs on endothelial cells leading to impaired endothelium-dependent vasodilation, increased vascular t

226 (*-) generation, is associated with impaired endothelium-dependent vasodilation.

227 pothesis that Ca(2+) influx via TRPA1 causes endothelium-dependent vasodilation.

228 n, including Ca(2+) signaling, integrity and endothelium-dependent vasodilation.

229 rate for nitric oxide synthesis, and impairs endothelium-dependent vasodilation.

230 ange in augmentation index in response to an endothelium-dependent vasodilator (salbutamol).

231 nses to intra-arterial administration of the endothelium-dependent vasodilator acetylcholine (P=0.03)

232 Coronary blood flow in response to the endothelium-dependent vasodilator acetylcholine as well

233 d by neural activity, hypercapnia, or by the endothelium-dependent vasodilator acetylcholine.

234 n but did not ameliorate the response to the endothelium-dependent vasodilator acetylcholine.

235 alpha(1) -agonist) during (i) infusion of an endothelium-dependent vasodilator alone (Protocol 1: ACh

236 /min), SLIGKV (160 to 800 nmol/min), and the endothelium-dependent vasodilator bradykinin (100 to 100

237 Similar to Ercc1(d/-) mice, age-related endothelium-dependent vasodilator dysfunction in Xpd(TTD

238 response to norepinephrine, and an impaired endothelium-dependent vasodilator response to acetylchol

239 on and no impairments in vasoconstrictor and endothelium-dependent vasodilator responses, associated

240 Elevation of IP(3) by the endothelium-dependent vasodilator, acetylcholine, increa

241 absolute forearm blood-flow responses to the endothelium-dependent vasodilator, acetylcholine, increa

242 cid (LPA) has been recognized recently as an endothelium-dependent vasodilator, but several lines of

243 ) were calculated during (1) infusion of the endothelium-dependent vasodilators acetylcholine (ACh) a

244 ion responses of precontracted arterioles to endothelium-dependent vasodilators adenosine 5'-diphosph

245 blood flow elicited by neural activity or by endothelium-dependent vasodilators in WT mice but not in

246 IK(Ca)/SK(Ca)), NS309 (10(-5) M), and to the endothelium-dependent vasodilators, substance P (10(-8)

247 Endothelium-dependent vasodilators, such as acetylcholin

248 itric oxide bioavailability at rest and with endothelium-dependent vasodilators.

249 t K(IR) channels function as 'amplifiers' of endothelium-dependent vasodilators.

250 endothelial intracellular [Ca(2+)] regulate endothelium-dependent vasodilatory pathways, the molecul

251 In young adults, increasing endothelium-dependent vasodilatory signalling during mil

252 first in humans to demonstrate that specific endothelium-dependent vasodilatory signalling is amplifi

253 etal muscle contractions selectively amplify endothelium-dependent vasodilatory signalling via activa

254 We tested the hypothesis that stimulating endothelium-dependent vasodilatory signalling will enhan

255 1.0 [0.9-1.0], P<0.001), and displayed less endothelium-dependent vasomotion (% change segmental lum

256 Per2, a circadian gene, is known to regulate endothelium-dependent vasomotion.

257 X-1 contributes to adverse effects of CRP on endothelium-dependent vasomotor function in resistance a

258 Endothelium-dependent vasomotor function, as measured by

259 identify p53 as an important determinant of endothelium-dependent vasomotor function.

260 Thus, the relationship between coronary endothelium-dependent vasomotor reactivity and atheroma

261 icate that endothelial Rac1 is essential for endothelium-dependent vasomotor response and ischemia-in

262 5-MTHF improved NO-mediated endothelium-dependent vasomotor responses and reduced va

263 However, the role of SIRT1 in regulating endothelium-dependent vasomotor tone is not known.

264 can partially restore the normal response to endothelium-dependent vasorelaxants and myocardial perfu

265 ditions, Sirt1(endo-/-) mice showed impaired endothelium-dependent vasorelaxation and angiogenesis, a

266 ary arteries, sCD40L significantly decreased endothelium-dependent vasorelaxation and eNOS mRNA level

267 temic antagonism of miR-204 rescues impaired endothelium-dependent vasorelaxation and vascular Sirt1,

268 Improvement of endothelium-dependent vasorelaxation by antibiotics is l

269 s in vitro, confirming that ceramide impairs endothelium-dependent vasorelaxation in a tissue-autonom

270 nificantly improved myocardial perfusion and endothelium-dependent vasorelaxation in chronically isch

271 her also improved vascular H(4)B content and endothelium-dependent vasorelaxation in diabetes.

272 vailable vascular nitric oxide, and improves endothelium-dependent vasorelaxation in mouse aortas.

273 o reduce systemic blood pressure and improve endothelium-dependent vasorelaxation in patients with at

274 ) and the 3-plex significantly also impaired endothelium-dependent vasorelaxation in response to brad

275 Endothelium-dependent vasorelaxation in response to brad

276 RAAS inhibition normalized MAP and improved endothelium-dependent vasorelaxation in sGCalpha1-defici

277 release, at least partly contributes to the endothelium-dependent vasorelaxation induced by the KATP

278 onses in resistance arteries and facilitates endothelium-dependent vasorelaxation only when CO(2)/HCO

279 Collectrin knockout mice display impaired endothelium-dependent vasorelaxation that is associated

280 downregulated endothelial Cav1 and impaired endothelium-dependent vasorelaxation that was rescued by

281 endothelial dysfunction measured by blunted endothelium-dependent vasorelaxation to acetylcholine, w

282 endothelial dysfunction measured by blunted endothelium-dependent vasorelaxation to acetylcholine, w

283 Maximal endothelium-dependent vasorelaxation to BK (bradykinin;

284 Endothelium-dependent vasorelaxation was impaired in you

285 malization, blunted ROS production, restored endothelium-dependent vasorelaxation, and attenuated apo

286 arrow endothelial progenitor cells, improved endothelium-dependent vasorelaxation, and markedly delay

287 elial nitric oxide synthase (eNOS), impaired endothelium-dependent vasorelaxation, and mild hypertens

288 othelial ER stress, associated impairment of endothelium-dependent vasorelaxation, and preserves endo

289 tigates angiotensin II-induced impairment of endothelium-dependent vasorelaxation, decrease in bioava

290 dothelial dysfunction, signalled by impaired endothelium-dependent vasorelaxation, is an early marker

291 ss of type I IFN receptor signaling improved endothelium-dependent vasorelaxation, lipoprotein parame

292 administration markedly attenuated impaired endothelium-dependent vasorelaxation, SERCA oxidation, E

293 tion, and rescue decline of aortic Sirt1 and endothelium-dependent vasorelaxation, triggered by high-

294 cg1(-/-) mice exhibited a marked decrease in endothelium-dependent vasorelaxation, while Abca1(-/-) m

295 ent increase in blood pressure, and impaired endothelium-dependent vasorelaxation.

296 e, insulin-mediated glucose utilization, and endothelium-dependent vasorelaxation.

297 al membrane potential and play a key role in endothelium-dependent vasorelaxation.

298 es in the regulation of vasoconstriction and endothelium-dependent vasorelaxation.

299 R-204 expression, reduced Cav1, and impaired endothelium-dependent vasorelaxation.

300 chanical stress, and its knock-down inhibits endothelium-dependent vasorelaxation.