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1 or low frequency (LFS; 3 h at 10 Hz to mimic endurance training).
2 me of the adaptations to exercise induced by endurance training.
3 testing and underwent 20 weeks of supervised endurance training.
4 ere measured before and after 5 and 10 wk of endurance training.
5 instead, exhibits plasticity with long-term endurance training.
6 tation on changes in VO2max during prolonged endurance training.
7 cise and facilitates molecular adaptation to endurance training.
8 events the adaptive intrinsic bradycardia of endurance training.
9 tal muscle and enhanced adaptive response to endurance training.
10 gnificantly with age, sex, body surface, and endurance training.
11 egulate hallmark adaptations associated with endurance training.
12 strated physiology characteristic of extreme endurance training.
13 s sex, race, and age in response to 20 wk of endurance training.
14 ance and molecular adaptations to subsequent endurance training.
15 mia in an experimental large animal model of endurance training.
16 egulate hallmark adaptations associated with endurance training.
17 mitochondrial haplotype in the responses to endurance training.
18 ges in athletes who participate in long-term endurance training.
20 plementation neither enhances nor attenuates endurance training adaptations in young healthy men.
22 d palmitate and linoleate kinetics but after endurance training, all were realigned with those of dec
24 rehabilitation, which includes strength and endurance training and educational, nutritional, and psy
27 most mitochondrial adaptations to subsequent endurance training, but this effect may have been a resu
29 er." As a result, worldwide participation in endurance training, competitive long distance endurance
30 ty): seven participants performed 2 weeks of endurance training (cycling) and were tested pre-post in
31 he first 6 to 9 months after commencement of endurance training depending on the duration and intensi
32 rint interval training (SIT) and traditional endurance training elicit similar physiological adaptati
33 flavanone bioavailability.A 7-d cessation of endurance training enhanced, rather than reduced, the bi
35 iglyceride (IMTG) utilization is enhanced by endurance training (ET) and is linked to improved insuli
36 in peak oxygen uptake (V(O2peak)) following endurance training (ET) are primarily determined by cent
37 proposed as a time efficient alternative to endurance training (ET) for increasing skeletal muscle o
39 oxidation during exercise and the effect of endurance training, exercise intensity, and lipid supple
40 iovascular and haematological adaptations to endurance training facilitate greater maximal oxygen con
42 All group B patients underwent intensive endurance training for a median of 15 h/week (interquart
46 solute intensity, persons who have undergone endurance training have greater fat oxidation during exe
47 on including inspiratory muscle training and endurance training have shown a promising effect post-Fo
48 ty power training (HIPT), and high-intensity endurance training (HIET)] on the resting concentration
51 nvestigated transcriptomic changes caused by endurance training in mice deficient in plakophilin-2 (P
53 We hypothesized that prolonged and intensive endurance training in previously sedentary healthy young
55 garding the long-term effects of high-volume endurance training, including potential maladaptation.
56 e-type specific in the posterior muscles and endurance training increased its content in type I muscl
58 bility before and after exercise may augment endurance training-induced adaptations of human skeletal
59 idative and performance ability derived from endurance training-induced increases in muscle mitochond
63 le-component modalities, that is, continuous endurance training, interval training, and resistance tr
64 rcise interventions consisting of continuous endurance training, interval training, resistance traini
68 iated with immune dysfunction, but long-term endurance training may confer protective effects on immu
71 hanges are consistent with those elicited by endurance training of the limb muscles in normal subject
73 ated in both LRT and HRT rats that underwent endurance training on a treadmill compared with those th
75 y was, therefore, to determine the effect of endurance training on O2 saturation of Mb (SmbO2) and Pm
76 fatty acid (n-3 PUFA) supplementation during endurance training on tryptophan (Trp) metabolism and me
79 cardiovascular stiffening, whereas life-long endurance training preserves left ventricular (LV) compl
81 this study was to evaluate the effects of an endurance training program on microvasculature of skelet
82 sessed the age-dependent effects of a 4-week endurance training program on the catabolic-anabolic bal
83 ) followed a personalized moderate-intensity endurance training program, while the nontraining (n = 1
88 6J mice were compared with or without a 5 wk endurance training protocol at rest or after an acute ex
90 acidity and can be evaluated for monitoring endurance, training routines, or athletic performance.
91 consisted primarily of four 30- to 40-minute endurance training sessions (low-impact aerobics; brisk
94 ifferences in aerosol particle emission, but endurance-training subjects emitted significantly more a
95 pair the metabolic adaptations to short-term endurance training, suggesting that the ills of inactivi
96 ith type 2 diabetes show reversibility after endurance training through increased contributions of th
97 tissue (AT) lipolysis becomes elevated upon endurance training to cope with enhanced energy demands.
98 f a combination of a human PKP2 mutation and endurance training to trigger an ARVC-like phenotype.
100 biochemical responses to acute exercise and endurance training were investigated in female Fischer 3
101 In contrast, the right ventricle responds to endurance training with eccentric remodeling at all leve