ライフサイエンスコーパス: energy balance

1 is and fat mass accumulation during positive energy balance.

2 that the hypothalamic transcriptome programs energy balance.

3 reeding states is often marked by a shift in energy balance.

4 thalamus (ARH) to other regions that control energy balance.

5 omponent of the hydrologic cycle and surface energy balance.

6 governing M1-M2 macrophage polarization and energy balance.

7 FXR function coordinately to integrate liver energy balance.

8 heir substantial contribution to the surface energy balance.

9 eals during situations of prolonged negative energy balance.

10 tity and quality are important for improving energy balance.

11 neurons in terms of the regulation of normal energy balance.

12 behavior and energy expenditure, to maintain energy balance.

13 reinforcement, depression-like behaviors, or energy balance.

14 effects were not explained by differences in energy balance.

15 s other metabolic pathways relevant to liver energy balance.

16 ish the DRN as an important node controlling energy balance.

17 re normal hypothalamic responses to negative energy balance.

18 uits to generate a central representation of energy balance.

19 se tissue stores excess lipids and maintains energy balance.

20 s in the alternative respiratory pathway for energy balance.

21 Ppargamma2 axis may contribute to whole-body energy balance.

22 CTH-induced adipose lipolysis and whole-body energy balance.

23 energy sensor that acts to sustain cellular energy balance.

24 hus influencing cloud properties and Earth's energy balance.

25 The lateral hypothalamus (LH) controls energy balance.

26 eutrality on a standard diet also had normal energy balance.

27 ly decreasing blood glucose without altering energy balance.

28 sis on Mito(VD), while minimizing changes in energy balance.

29 mediating leptin signaling and in regulating energy balance.

30 lved in sex- and diet-specific regulation of energy balance.

31 metabolic alterations indicative of impaired energy balance.

32 the quest of new genes/variants influencing energy balance.

33 y which each macronutrient may contribute to energy balance.

34 ptide (AMP) gene expression that compromised energy balance.

35 outflow, and endocrine function to maintain energy balance.

36 nal signals to regulate food consumption and energy balance.

37 es in maintaining glucose metabolism and net energy balance.

38 egulator of leptin signalling and organismal energy balance.

39 unctional role in modulating food intake and energy balance.

40 is no longer required for Pomc expression or energy balance.

41 ergy expenditure, thereby promoting negative energy balance.

42 ass than monotherapies by promoting negative energy balance.

43 ponse to altered environmental conditions or energy balance.

44 nesis, uncoupled respiration, and regulation energy balance.

45 to lack of standardization of participants' energy balance.

46 foraging intensity in response to changes in energy balance.

47 a determinant of thermogenesis and systemic energy balance.

48 thalamus (PVH) neurons are key regulators of energy balance.

49 ncentrating effect rather than from improved energy balance.

50 nd disease status, all potentially influence energy balance.

51 signaling pathway that regulates organismal energy balance.

52 ytohormones and by reducing sugar levels and energy balance.

53 regulate systemic insulin sensitivity and/or energy balance.

54 ptor plays a critical role in the control of energy balance.

55 anabolic metabolism coordinately to restore energy balance.

56 ng the modeling of AET and thus of water and energy balances.

57 s not only on homeostatic signals related to energy balance [1] but also on the presence of competing

58 continuous neurogenesis as a way to mediate energy balance [4-10].

59 atients, were more likely to have a negative energy balance [45% compared with 32%, respectively; OR:

60 ion important for GLP-1R-mediated effects on energy balance, AAV-GLP-1R was injected into the NTS to

61 In pair-fed conditions during positive energy balance, activation of Agrp neurons improved meta

62 Energy balance affected maternal adiposity (recommended:

63 These mice also have altered energy balance, altered body composition, and a shift in

64 nough electrons to the adsorbed species; the energy balance and activation barriers for the individua

65 an a century that the brain controls overall energy balance and adiposity by regulating feeding behav

66 ceptor gene (MC4R), a key protein regulating energy balance and adiposity, have been related to obesi

67 biological basis for both AMPK regulation of energy balance and AgRP neuron state-dependent plasticit

68 Leptin regulates energy balance and also exhibits neurotrophic effects du

69 .5 mum, called PM2.5) can affect the surface energy balance and atmospheric heating rates and thus ma

70 ecessary to obtain its beneficial effects on energy balance and body weight, major contributors to it

71 of hepatic adropin expression appear to link energy balance and cellular stress to the intracellular

72 Energy balance and eating behavior were also normal, eve

73 ity (DIO) resistant and had a lower positive energy balance and energy intake, greater lipid fuel pre

74 ged as key players in the central control of energy balance and etiology of obesity.

75 glutamate cells may play a negative role in energy balance and food intake.

76 ein-coupled receptors that are essential for energy balance and food intake.

77 Epac1 plays an important role in regulating energy balance and glucose homeostasis by promoting lept

78 n plays a crucial role in how SH2B1 controls energy balance and glucose homeostasis.

79 ffectors of cAMP and play important roles in energy balance and glucose homeostasis.

80 d the roles for SH2B1alpha and SH2B1delta in energy balance and glucose metabolism, we generated mice

81 keletal muscle and play a role in regulating energy balance and glucose metabolism.

82 Altered energy balance and insulin resistance are important char

83 ain-derived hormone amylin promotes negative energy balance and is receiving increasing attention as

84 ol energy excretion that favorably influence energy balance and may help explain epidemiologic associ

85 growth, which is linked with alterations in energy balance and metabolic and gut microbiome profile.

86 tary lipids is a major determinant of animal energy balance and metabolic health.

87 Dietary lactalbumin and lactoferrin improved energy balance and metabolism, and decreased adiposity,

88 ts fractions lactalbumin and lactoferrin, on energy balance and metabolism.

89 key aspect of intracellular homeostasis and energy balance and plays a vital role in cell survival u

90 thways that mediate the relationship between energy balance and prognosis?

91 in the control of bodily functions, such as energy balance and reproduction.

92 The role of PVT in energy balance and reward control is however understudie

93 measure of propensity periods of a positive energy balance and should be examined in addition to mea

94 lically deficient with a significantly lower energy balance and significantly higher concentrations o

95 s), are reciprocally activated by changes in energy balance and that modulating their activity has op

96 n in light of the ambiguity of locomotion on energy balance and the different living conditions of th

97 We review the components of energy balance and the mechanisms acting to resist weigh

98 sions often integrate external cues, such as energy balance and the nutritional status of the organis

99 te critically to the long-term regulation of energy balance and the underlying physiological and beha

100 Leaf optical properties impact leaf energy balance and thus leaf temperature.

101 indicate that LDTg GLP-1R signaling controls energy balance and underscores the role of the LDTg in i

102 thalamus and hippocampus promoted a negative energy balance and weight loss.

103 n between partners A and B determines the SF energy balance and yields squared matrix elements |T*|(2

104 mass balance (in addition to the charge and energy balances) and provides new insight into the natur

105 effects of a LP diet on glucose homeostasis, energy balance, and body composition are unlikely to be

106 nvolved in the evolution of feeding ecology, energy balance, and body size in cetaceans.

107 Adipocytes play a central role in energy balance, and dysfunctional adipose tissue severel

108 lated peptide (NPY/AgRP)-expressing neurons, energy balance, and glucose homeostasis.

109 ng with other integrated signals that affect energy balance, and mediates the majority of hypothalami

110 region implicated in arousal, reproduction, energy balance, and memory processing.

111 er, its links to rhythms of nutrient intake, energy balance, and metabolic control remain poorly defi

112 mote meal irregularity, dysregulation of the energy balance, and poor metabolic health.

113 tin-4 receptor (MC4R) regulates food intake, energy balance, and somatic growth in both mammals and t

114 m, snack episodes may contribute to positive energy balance, and the risk may be higher in women.

115 suggest that JNK activity promotes positive energy balance, and the therapeutic intervention inhibit

116 Rather various components of energy balance are dynamically interrelated and weight l

117 Impaired glucose homeostasis and energy balance are integral to the pathophysiology of di

118 lecular mechanisms of SF-1 in the control of energy balance are largely unknown.

119 y temperature (BT), blood pressure (BP), and energy balance are poorly understood.

120 adipocytes may be a novel means to modulate energy balance as a treatment of obesity and related cli

121 TrkB receptor is critical for the control of energy balance, as mutations in its gene (NTRK2) lead to

122 implicated in reproduction, homeostasis, and energy balance, as well as neuromodulation of social beh

123 e transfer of heat in the reservoir, and the energy balance at the surface.

124 s may have an influential role on subsequent energy balance behaviors and weight.

125 The adipokine leptin mediates energy balance between adipose tissue and brain.

126 ral rotations, which, in turn, determine the energy balance between Ni(3+) /Fe(4+) and Ni(4+) /Fe(3+)

127 Surprisingly, we reveal a fine energy balance between the core cohesive energy and the

128 ether this is associated with alterations in energy balance, blood metabolomics and fecal microbiota

129 her delineate the neuro-endocrine control of energy balance/body mass and demonstrates that this mole

130 t thermoneutrality (24 degrees C) and during energy balance both at thermoneutrality (24 degrees C) a

131 ence that this inflammation not only impairs energy balance but also contributes to obesity-associate

132 ) are important regulators of metabolism and energy balance, but excess BAs cause cholestatic liver i

133 on, has emerged as an important regulator of energy balance, but the underlying cellular and molecula

134 e occur in response to positive and negative energy balance, but the underlying mechanism remains unc

135 tinal cannabinoid that controls appetite and energy balance by engagement of the enteric nervous syst

136 n system both play vital roles in regulating energy balance by modulating energy intake and utilizati

137 Energy balance calculation were used to assess the physi

138 Our results demonstrate that an unclosed energy balance caused the largest uncertainties, in part

139 ting energy expenditure (REE), the estimated energy balance, clinical and biological markers of cache

140 ripheral appetite hormones during a negative energy balance.CONCLUSIONThese results indicate that blu

141 eveloping molecular probes to study negative energy balance conditions and unidentified functions of

142 theories to address how demography and mass-energy balance constrain allocation of biomass to surviv

143 of lipid utilization in response to negative energy balance contributes to subsequent weight maintena

144 of adipose tissue plasticity under positive energy balance, contributing to adipose tissue metabolic

145 as an ARC gliopeptide playing a key role in energy balance control and exerting strong anorectic eff

146 like peptide-1 (GLP-1) system is critical to energy balance control and is a target for obesity pharm

147 reviously unknown role for CNS astrocytes in energy balance control by GLP-1 signaling.

148 t astrocytes within the NTS are relevant for energy balance control by GLP-1 signaling.

149 derlying mechanisms of NRs in the context of energy balance control may facilitate the identification

150 e role of endogenous NTS GLP-1R signaling in energy balance control.

151 y and potentially preclinically relevant for energy balance control.

152 rius (NTS), a hindbrain nucleus critical for energy balance control.

153 role for NTS astrocytic GLP-1R signaling in energy balance control.

154 receptor substrate 4 (IRS4(PVH)) involved in energy balance control.

155 signalling has a key role in food intake and energy-balance control and is often impaired in obese in

156 zing tubular cell mitochondrial function and energy balance could be an important preventative strate

157 in increases of precipitation and changes in energy balance, creating significant uncertainty for cli

158 ion, mammary tumorigenesis, and survival are energy balance dependent in association with epigenetic

159 BMI: 31.4 +/- 0.5) consumed a 3-wk baseline energy-balance diet with 0.8 g protein . kg baseline bod

160 es impact glucose homeostasis and whole-body energy balance during development of obesity, weight los

161 ster regulator of metabolism, which restores energy balance during metabolic stress both at the cellu

162 tatory projections mediate cisplatin-induced energy balance dysregulation.

163 populations that control discrete aspects of energy balance (e.g. oxytocin (OXT), neuronal nitric oxi

164 nowledge, no study has evaluated the role of energy balance (EB) in modulating metabolic adaptation.

165 ient (RQ) in a whole-room calorimeter during energy balance (EBL) (50% carbohydrate, 30% fat) and the

166 ot significantly increased when adjusted for energy balance (EEDLWDeltaRQ = 139 +/- 89 kcal/d; P = 0.

167 Hunger, driven by negative energy balance, elicits the search for and consumption o

168 Models of energy balance employed in public health [1-3] should be

169 main, particularly regarding the role of the energy balance entering pregnancy.We investigated how pr

170 nutritional risk index, daily energy intake, energy balance (equal to daily energy intakes minus the

171 ysiological adaptations on both sides of the energy balance equation that try to bring body weight ba

172 s and experimental measurements based on the energy balance equations to measure the [Formula: see te

173 om the Australian Health Survey 2011-12, and energy balance equations.

174 rain, vegetation and ground properties using energy balance equations.

175 ggestions for how best to design and conduct energy balance experiments in rodents.

176 te-median eminence complex (Arc-ME) controls energy balance, fertility and growth through molecularly

177 ering first-principles to calculate mass and energy balances for each process.

178 e risks by affecting circadian regulation of energy balance, glucose metabolism, and postprandial inf

179 ion appears to disrupt the delicately poised energy balance governing thin filament regulation.

180 ored the effects of a high-fat diet (HFD) on energy balance, gut microbiota, and risk factors of meta

181 apping neuroendocrine mechanisms controlling energy balance has highlighted combination therapies as

182 othalamic neuronal populations that regulate energy balance have been identified.

183 ssential for the physiological regulation of energy balance; however, its role in glucose homeostasis

184 erlapping neural substrates in this critical energy balance hub.

185 that serotonin is a critical mediator of the energy balance impact of GLP-1 receptor (GLP-1R) activat

186 nd given the effect of clouds on the Earth's energy balance, improved prediction of aerosol-cloud-cli

187 nutritional variations to maintain a proper energy balance in cells.

188 ning therapeutic opportunities for restoring energy balance in depressed patients.

189 s between breakfast habits and components of energy balance in free-living obese humans.

190 d that NCS have no impact on weight gain and energy balance in high fat diet induced obesity.

191 estioning the role of leptin as regulator of energy balance in humans.

192 T-knockout mice, we found that CRT regulates energy balance in IECs and thereby epithelial integrity

193 ll functions, it was first found to maintain energy balance in liver cells.

194 indicates the triazole ureas may affect the energy balance in mice through multiple molecular target

195 t necessary for the maintenance of long-term energy balance in normal eating conditions.

196 with the regulation of metabolic health and energy balance in obese animals, and suggest that specif

197 ose-dependent insulin secretion and improves energy balance in patients with type 2 diabetes mellitus

198 s that astrocytic GLP-1R signaling regulates energy balance in rats.

199 hanism for fine-tuning energy expenditure to energy balance in real time.

200 hat contributes to homeostatic regulation of energy balance in response to metabolic stress.

201 heat transport, hydrodynamic evolution, and energy balance in systems ranging from astrophysical obj

202 cid synthase activity could underlie altered energy balance in these mice.

203 these brain NRs regulate multiple aspects of energy balance, including feeding, energy expenditure an

204 n: What are the comparative contributions of energy balance, including weight, dietary patterns, and

205 An energy balance indicates that flavins must be recycled f

206 animal models, taking into consideration the energy balance involving both the growth of tumor and th

207 When energy balance is altered by aerobic exercise, starvatio

208 r, the data indicate that a natural shift in energy balance is associated with changes in NPY and OX

209 r understanding of the central regulation of energy balance is continually being refined as new detai

210 ers within the neuronal circuits that govern energy balance is discussed, offering evidence of a bidi

211 ion, yet the role of astrocytes in mediating energy balance is largely unstudied.

212 ropeptides interact in the brain to regulate energy balance is not known.

213 l Kinase (JNK) in the control of feeding and energy balance is not well understood.

214 f hypothalamic neuropeptides and hormones in energy balance is paramount in the search for approaches

215 , whether they interact in order to regulate energy balance is poorly understood.

216 al care, objectively assessed information on energy balance is urgently needed.

217 neurons in the regulation of body weight and energy balance is well appreciated, little is known abou

218 The positive energy balance linked with obesity induces a variety of

219 Despite this well-known shift in energy balance, little work has explored seasonal differ

220 Despite similar fat mass and energy balance, M(IL10) mice were protected from aging-a

221 ship between sugar and health is affected by energy balance, macronutrient substitutions, and diet an

222 re found to have been in a moderate negative energy balance (mean +/- SD RDA: 209 +/- 213 kcal/d; 2RD

223 subsequent defects in maintaining whole-body energy balance might be early events that contribute to

224 A simple energy balance model is applied to obtain the maximum ho

225 Here, I use an advanced energy balance model to show that such an HZ would be co

226 zone water balance model and another from an energy balance model, to partition annual ET into green

227 This analysis supports the results from the energy balance model.

228 measurement height and canopy surface; (iv) energy balance non-closure; (v) uncertainties in net eco

229 By taking into account the energy balance of all processes during fault movement, w

230 e study corresponding to an overall negative energy balance of approximately 300 kcal/d.

231 d significantly affect the movements and the energy balance of polar bears (Ursus maritimus) which fo

232 likely due to an essential role of COX6A2 in energy balance of PV(+) interneurons, underscored by a d

233 The energy balance of the Earth is controlled by the shortwa

234 However, the direct impact of dust on the energy balance of the Earth system remains poorly quanti

235 tmospheric circulation controls the mass and energy balance of the Greenland ice sheet through its im

236 Further, net energy balance of the NiA(mox)MDC was the highest (NiA(m

237 A careful examination of the energy balance of the system surprisingly revealed that

238 number and mortality of offspring; and mass-energy balance on allocation of energy to growth and rep

239 ontaining 100% (mean +/- SD: 9.3 +/- 1.3 MJ; energy balance) or 25% [2.3 +/- 0.3 MJ; energy restricti

240 Maintaining energy balance over a wide range of temperatures is crit

241 thermogenesis; and how to sustain a negative energy balance over many years of treatment.

242 ctic action of central TTR in the control of energy balance, providing a potential novel target for t

243 Examination of the surface energy balance (radiative and turbulent fluxes) reveals

244 eby an exercise- or dietary-induced negative energy balance reduces human subcutaneous white adipose

245 accompanied by a transient induction of the energy balance regulating hormone FGF21 (fibroblast grow

246 the expression of vGlut2 may play a role in energy balance regulation, genetic deletion of vGlut2 in

247 ight loss may have functional importance for energy balance regulation, with greater losses of FFM po

248 (GLP-1) and serotonin play critical roles in energy balance regulation.

249 lease amino acid transmitters that can alter energy balance regulation.

250 lease amino acid transmitters that can alter energy balance regulation.

251 ystem serves as a prototype for hierarchical energy balance regulation.

252 depletion in VMH SF1 neurons did not affect energy balance regulation.

253 lar glutamate, but their direct influence on energy balance-relevant behaviors is largely understudie

254 erscores the role of the LDTg in integrating energy balance-relevant signals to modulate feeding.

255 t that inhibits this circuit during negative energy balance remains controversial.

256 effect of whole grains on the regulation of energy balance remains controversial.We aimed to determi

257 CNS drive annual changes in body weight and energy balance remains uncharacterized.

258 Our findings can help predict energy balance responses to stress and nutritional state

259 Components of energy balance (resting metabolic rate, physical activit

260 These cows experience a negative energy balance, resulting in a distinct blood metabolism

261 ack of chlorophyll exerts growth control via energy balance sensing, which is upstream of the known g

262 commonly used to treat cancer despite severe energy balance side effects.

263 , has been demonstrated to have an impact on energy balance, since it affects both feeding and energy

264 wiring to synaptic function and plasticity, energy balance, social behaviors, emotions, and cognitio

265 ssion is positively correlated with positive energy balance, suggesting a potential central role for

266 s provide a mechanism to attain the positive energy balance that sustains gestation, dysregulation of

267 e in physical activity leading to a negative energy balance, the dense built environment, pervasive f

268 ctor that directs the coordinated control of energy balance, thermogenesis and glucose homoeostasis.

269 mus (PVH) plays a pivotal role in regulating energy balance, though circuit mechanisms remain obscure

270 P2Y(6)R) plays a crucial role in controlling energy balance through central mechanisms.

271 inalis (BNST) may be critical for monitoring energy balance through changes in synaptic strength.

272 c potential in mediating amylin's effects on energy balance through gamma-aminobutyric acid receptor

273 f the hypothalamic BBSome for the control of energy balance through regulation of trafficking of impo

274 c requirements of bone formation with global energy balance through the regulation of insulin product

275 s in adults is integral to the regulation of energy balance, tissue/stem cell homeostasis, and diseas

276 ering thermogenesis can be used to influence energy balance to prevent or even treat obesity.

277 als integrate external stimuli with internal energy balance to regulate major developmental and repro

278 lut2 in POMC neurons were unable to maintain energy balance to the same extent as control mice when f

279 ve to the other SH2B1 isoforms likely shifts energy balance toward a lean phenotype via a primarily l

280 of adipose tissue in response to a positive energy balance underlies obesity and occurs through both

281 kes an appreciable contribution to protein's energy balance, up to 2 kcal/mol.

282 hat regulate host cholesterol metabolism and energy balance via several nuclear receptors and/or G-pr

283 SH2B1 has been suggested to impact energy balance via the modulation of leptin action.

284 Energy balance was achieved in both groups through varia

285 ing-induced increases in body temperature on energy balance, we compared rats fed chronically by eith

286 t and theoretical analysis of the organismal energy balance, we further show that the mass allometry

287 weight loss was observed in matched negative-energy-balance (weight loss) diets (P = 0.02) and in neu

288 weight loss) diets (P = 0.02) and in neutral-energy-balance (weight-maintaining) diets (P = 0.03), an

289 This behavior is consistent with an energy balance, where some of the energy of the injected

290 e role for IRS4(PVH) neurons in PVH-mediated energy balance which raises the possibility of developin

291 ing leptin concentrations reflect a negative energy balance, which augments sympathetic nervous syste

292 eptin is an adipokine involved in regulating energy balance, which has been identified as a potential

293 ions may, however, be confounded by negative energy balance, which may obscure the results.

294 Adipose tissue is fundamental to energy balance, which underpins fitness and survival.

295 s capable of safely achieving a net negative energy balance while avoiding unwanted cardiovascular si

296 ts provides a potential means of integrating energy balance with energy expenditure.

297 by integrating internal metabolic signals of energy balance with external environmental cues such as

298 vestigated the role of hypothalamic GRP78 on energy balance, with particular interest in thermogenesi

299 s of patients for whom purposefully altering energy balance would be deleterious to prognosis?

300 hat neurons in brain regions associated with energy balance would possess neuroanatomical connections