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1 ene-specific activation complex known as an "enhanceosome".
2 YGO2, are constitutive components of the Wnt enhanceosome.
3 iated chromatin-modifying enzymes to form an enhanceosome.
4 ation and appears to recycle to maintain the enhanceosome.
5 ively to adjacent sites and form a bacterial enhanceosome.
6 at these factors may function together as an enhanceosome.
7 ay a role in the transcriptionally active mu enhanceosome.
8 ription factors in formation of the IFN-beta enhanceosome.
9 endogenous IFN-beta enhancers as part of the enhanceosome.
10 ctivation of transcription from the IFN beta enhanceosome.
11 P) form a stable core complex within the Wnt enhanceosome.
12 two fatty-acid-bound TGA3 dimers to form an enhanceosome.
13 TGA transcription factor dimers, forming an enhanceosome.
14 more distally involving the canonical CIITA enhanceosome.
15 n of beta-catenin, a co-activator of IFNbeta enhanceosome.
16 assembles a nucleoprotein complex termed MHC enhanceosome.
17 promoting the formation of a Foxp3-specific enhanceosome.
18 ater moved to the promoter to form the c-Rel enhanceosome.
19 main components of the human interferon-beta enhanceosome.
20 1 complex is part of the trichome initiation enhanceosome.
21 s the key DNA-binding component of the MHCII enhanceosome.
22 of the mammalian interferon-beta (IFN-beta) enhanceosome.
23 cific multiprotein complex, termed the MHCII enhanceosome.
24 oid X receptor from a distal RARE to form an enhanceosome.
25 as the recruitment of other proteins to the enhanceosome.
26 otein complexes, leading to the formation of enhanceosomes.
27 or complexes forming secondary structures or enhanceosomes.
28 he regulated assembly and disassembly of the enhanceosome, a higher-order nucleoprotein complex forme
30 cently emerged as the core module of the Wnt enhanceosome, a multiprotein complex that primes develop
32 HMGI(Y) by CBP at lysine-65 destabilizes the enhanceosome, acetylation of HMGI(Y) by PCAF/GCN5 at lys
33 romoters or activators, the natural IFN-beta enhanceosome activates transcription by causing a dramat
39 is is the first description of an RA-induced enhanceosome and demonstrates that GABP and p300 are ess
42 tion of CIITA through a disruption of MHC-II enhanceosome and relevant coactivator-transcription fact
43 I gene expression is enhanced by the T cell enhanceosome and results from a direct interaction of th
44 t least in part, to interactions between the enhanceosome and the transcriptional coactivator CREB, c
45 ed by the dynamic interplay between specific enhanceosomes and specific local chromatin structure.
47 s that regulate IFNB enhancer activity (the "enhanceosome") and highlight opportunities for deeper un
49 eg cell development is controlled by a c-Rel enhanceosome, and strategies targeting Rel-NF-kappaB can
50 IITA, the assembly of CIITA, NF-YB, and RFX5 enhanceosome, and the extent of H3 acetylation at the MH
51 Wnt-dependent docking of beta-catenin to the enhanceosome apparently causes a rearrangement that appo
52 ining half to assemble a complete picture of enhanceosome architecture in the vicinity of the DNA.
58 IFN-beta gene activation via multifactorial enhanceosome assembly is potentiated in LPS-stimulated c
60 n accessible IFN-beta core promoter prior to enhanceosome assembly results in major changes in the ge
61 t a highly dynamic yet intrinsically ordered enhanceosome assembly to maintain the finely balanced tr
62 sential architectural component required for enhanceosome assembly, at distinct lysine residues, caus
63 Here, we demonstrate that the first step in enhanceosome assembly, i.e. HMG I(Y)-dependent recruitme
64 e HMG-I/Y protein, proposed as orchestrating enhanceosome assembly, interacts specifically with the P
71 cruitment of the CBP-PolII holoenzyme by the enhanceosome, because its depletion from the extract dec
72 r that participates in the formation of this enhanceosome, binding specifically to the positive regul
73 pts the association of CIITA with the MHC-II enhanceosome by binding to the components of the RFX com
74 tor interacts with the components of the EGF-enhanceosome (co-activators: glucocorticoid-receptor-int
75 r may involve the binding of a multi-protein enhanceosome complex at the CRX triplet and the PCE-1-li
76 sequent alteration of the composition of the enhanceosome complex binding to IFNA promoters in vivo,
77 s, these results suggest the existence of an enhanceosome complex comprised of p300 and multiple semi
78 es on the dynamic assembly of a multiprotein enhanceosome complex that is initiated by the activation
79 d histone acetyltransferase Ep300 to form an enhanceosome complex that promoted Gata2 expression.
83 p300, and RNA polymerase II (Pol II) to form enhanceosome complexes that contain HIF1alpha, STAT3, CB
84 recruiting coactivators CBP and p300 to form enhanceosome complexes that contain HIF2alpha, USF2, CBP
86 is driven by oncogenic transcription factor enhanceosomes comprising chromatin and epigenetic regula
87 rect interactions with a preassembled MHC-II enhanceosome consisting of cyclic AMP response element-b
88 ation of class I transcription by the T cell enhanceosome consisting of Runx1, CBFbeta, and LEF1.
89 such as TCR genes, are regulated by a T cell enhanceosome consisting of RUNX1, CBFbeta, LEF1, and Aly
90 virus infection requires the assembly of an enhanceosome, consisting of the transcriptional activato
91 virus infection requires the assembly of an enhanceosome, consisting of the transcriptional activato
93 beta (IFN-beta) gene requires assembly of an enhanceosome containing ATF-2/c-Jun, IRF-3/IRF-7, and NF
94 beta (IFN-beta) gene requires assembly of an enhanceosome containing the transcription factors ATF-2/
97 co-repressor, and also to the Chip/LDB1-SSDP enhanceosome core complex via an evolutionary conserved
98 the activation domains in the context of the enhanceosome decreases both recruitment of CBP and trans
99 iments showed that the formation of a stable enhanceosome-dependent preinitiation complex require coo
100 presence of a domain in IRF-2 that prevents enhanceosome-dependent recruitment of the CBP-Pol II hol
103 STAT3-dependent, glucocorticoid-supplemented enhanceosome for the alpha2-macroglobulin (alpha2-M) gen
104 uirement of BCL9/9l, constituents of the Wnt-enhanceosome, for intestinal transformation following lo
105 binding to the cyclin D1 promoter led to an enhanceosome formation and facilitated cyclin D1 express
107 A dominant negative p300 construct disrupts enhanceosome formation and reduces the RA responsiveness
108 t emerged from studying how HMGB1 stimulates enhanceosome formation by the Epstein-Barr viral activat
110 itectural transcription factors facilitating enhanceosome formation on a variety of mammalian promote
111 exhibited a weaker binding to STAT3, and the enhanceosome formation on the socs3 promoter was inhibit
113 and an alternative model of IFNgamma-driven enhanceosome formation that may allow for other adaptors
114 noise originate from the complexity of IFNB1 enhanceosome formation, which leads to a range up to man
116 n factors, is often critical for stabilizing enhanceosomes formed from trans-acting proteins separate
118 omotypic clusters, heterotypic clusters, and enhanceosomes, from real TF binding motifs from diverse
120 ur results demonstrate a key role of the Wnt enhanceosome in beta-catenin-dependent intestinal tumour
123 coactivators (p160 members and CBP) form an enhanceosome in the enhancer region of the hSP-B gene.
124 (H2BNTac) as a defining feature of oncogenic enhanceosomes in androgen receptor (AR)-positive prostat
127 thin an additional 30 min of the established enhanceosome indicates that renewal of STAT3 and GR bind
128 veral transcriptional components of the Ucp1 enhanceosome interact synergistically to achieve large d
130 emonstrate that once assembled, the IFN-beta enhanceosome is an unusually stable nucleoprotein struct
133 show that the stereospecific assembly of the enhanceosome is critical for the efficient recruitment o
135 ce that recruitment of the holoenzyme by the enhanceosome is due, at least in part, to interactions b
136 cells by M. tuberculosis a unique TNF-alpha enhanceosome is formed, and it is distinct from the TNF-
139 that the inducer-specific assembly of unique enhanceosomes is a general mechanism by which a single g
140 factors (TFs) at the composite DNA element (enhanceosome), is central for amplification of weak acti
141 complex, however, stimulates assembly of the enhanceosome itself such that the entire reaction can oc
142 onstrate NF-kappa B-dependent assembly of an enhanceosome-like complex on the promoter region of bfl-
143 olled during early mouse embryogenesis by an enhanceosome-like control region, termed the early enhan
145 00 assembles at the IL-2 promoter to form an enhanceosome-like signal transduction target that is cen
147 nowledge, this is the first demonstration of enhanceosome-mediated regulation of a cell death inhibit
148 WNT targets such as AXIN2 and BMP4, and WNT enhanceosome members including TCF4, LEF1 and BCL9 were
150 ected alveolar epithelial cells supports the enhanceosome model for RANTES gene transcription, which
152 r interaction with DNA that conforms to the 'enhanceosome' model, and furthermore identify associatio
156 Run-on transcription shows a lag after full enhanceosome occupation that can be largely but not comp
159 between STAT3 and retinoid nuclear receptor enhanceosome proteins in pulmonary epithelial cells.
164 ate that this synergy, in the context of the enhanceosome, requires a new protein-protein interaction
165 anscription factors are key components of an enhanceosome responsible for activating SCL transcriptio
166 As a consequence, IRF-2 incorporation into enhanceosomes restricts the number of IFN-beta promoters
168 we characterize NSD2 as an essential AR neo-enhanceosome subunit that enables its oncogenic activity
170 t for the assembly of an ER-stress-inducible enhanceosome that activates CHOP gene expression in resp
172 ting that DEK is part of a tissue-restricted enhanceosome that contains BMP4-dependent and -independe
173 ormed, and it is distinct from the TNF-alpha enhanceosome that forms in T cells stimulated by antigen
174 refore function as part of a hypoxia-induced enhanceosome that helps to promote transcription of COX-
175 jun, are well-characterized components of an enhanceosome that mediates virus induction of the human
177 ate the competing actions of Stat5-assembled enhanceosomes that promote Th1- and Treg-cell developmen
178 hypothesize that a multiprotein complex--an enhanceosome--that includes GABP, other transcription fa
179 Here we report that within the IFN-beta enhanceosome the ATF-2-c-jun heterodimer binds in a spec
180 on in B cells is known to involve the B cell enhanceosome, the molecular basis for high constitutive
181 require cooperative interactions between the enhanceosome; the general transcription factors TFID, TF
183 host immune system by disrupting the MHC-II enhanceosome through binding with RFX transcription fact
184 To examine the relative prevalence of the 'enhanceosome' versus the 'TF collective' model of combin
185 cruitment of the CBP/p300 coactivator to the enhanceosome, via a new activating surface assembled fro
187 gher-order transcription enhancer complexes (enhanceosomes), which is dependent upon inducer-specific
188 LRC5 participates in an MHC class I-specific enhanceosome, which assembles on the conserved W/S-X-Y c
189 of a higher order nucleoprotein complex, the enhanceosome, which consists of the transcriptional acti
190 virus infection requires the assembly of an enhanceosome, which instructs a recruitment program of c
191 aB-interacting factors in the putative CXCL1 enhanceosome will provide key information in developing