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1 patients, was a complex mass or masses in an enlarged spleen.
2 CT as multiple hypoattenuating masses in an enlarged spleen.
3 He had no peripheral lymphadenopathy and no enlarged spleen.
4 ent had no peripheral lymphadenopathy and no enlarged spleen.
5 fe; 60% of children younger than 5 years had enlarged spleens.
6 rved that MIM-deficient mice often developed enlarged spleens.
7 tive risk 0.65 [14-50], p=0.0023), 26% fewer enlarged spleens (46/79 [58%] vs 67/90 [74%], p=0.0045),
8 these animals reveal that they have greatly enlarged spleens, altered thymic histology, and lymphocy
9 usly develop autoimmune phenotypes including enlarged spleen and lung inflammation infiltrated with I
10 oly(Y,F,A,K)n have been established from the enlarged spleen and lymph nodes that result from copolym
13 At 2 y of age the mice showed significantly enlarged spleens and an increase in the CD5(+) B-cell po
14 jection (HDI) of c-Myc into mice resulted in enlarged spleens and lethal HCC associated with an incre
16 e (37.7 degrees C or above), nailbed pallor, enlarged spleen, and being seen at one of the clinics ra
17 terations of hemopoietic tissues, such as an enlarged spleen due to lymphoid hyperplasia, extramedull
18 pes include shortened long bones, a markedly enlarged spleen, elevated neutrophil counts, an enlarged
21 , LLC, and CT26 tumor-bearing mice displayed enlarged spleen (splenomegaly), and exercise training re
22 review found no evidence of infection and an enlarged spleen that showed active germinal centers.
23 cKO (conditional knockout) mice exhibited an enlarged spleen with disrupted spleen architecture and l
25 At this stage, mice showed significantly enlarged spleens with abnormal B cell-derived white pulp
26 acking AMPKalpha2, and the mice had markedly enlarged spleens with dramatically increased proportions