ライフサイエンスコーパス: enteral

1 Patients were randomized 1:1 to enteral acetaminophen 1 g every 6 hours for 3 days (n =

2 e role of the mucus layer was tested via the enteral administration of a mucus surrogate.

3 the pediatric intensive care, we showed that enteral administration of acetaminophen results in less

4 Enteral administration of immune-modulating nutrients (e

5 In this model, enteral administration of immunogen trinitrobenzene sulf

6 Therefore, continuous enteral administration of lipid- and protein-rich nutrit

7 Enteral administration of lipid-rich nutrition was previ

8 t Th17 polarization could be reversed by the enteral administration of retinoic acid, which induced T

9 ease and gastrointestinal colonization after enteral administration of serotype-specific capsular ant

10 Enteral administration of talactoferrin reduced 28-day a

11 ing, negative pressure therapy, debridement, enteral and parenteral feeding, vitamin and mineral supp

12 surgery and was significantly higher in the enteral and parenteral groups than in the control group

13 Supplementation of these amino acids with enteral and parenteral nutrition before, during, and aft

14 Both enteral and parenteral solutions were prepared with comm

15 Hepatic GS contributes to both enteral and systemic ammonia detoxification.

16 d symptoms, and nutritional intake via oral, enteral, and parenteral routes to accurately assess the

17 in intestinal failure (IF) is adaptation and enteral autonomy (EA).

18 nt exhibited normal graft function with full enteral autonomy and without histological or endoscopic

19 ed with mortality and liver failure, whereas enteral autonomy correlates with small-bowel length.

20 intestinal adaptation after transplant, but enteral autonomy is attainable.

21 Enteral autonomy was associated with small bowel length

22 tation, IF-associated liver disease (IFALD), enteral autonomy, and sepsis.

23 bowel syndrome can aid in the achievement of enteral autonomy, but with a price of >$400,000 per y.

24 xpands absorptive surface areas and promotes enteral autonomy.

25 that leaves the food matrix, and crosses the enteral barrier to reach their cellular target.

26 an age of 6.1 mo; Q1-Q3, 1.8-20 mo) the mean enteral bioavailability was 72% (range, 11-91%).

27 fter head injury, the rats received a single enteral bolus of luminescent Escherichia coli Xen 14.

28 teral nutrition without supplementation, and enteral causes such as malabsorption.

29 dings in 90.0% of patients who received IV + enteral contrast and 90.4% of patients scanned with IV c

30 om a diverse group of hospitals suggest that enteral contrast can be eliminated in CT scans for suspe

31 Enteral contrast does not improve CT evaluation of appen

32 ed trials from tertiary centers suggest that enteral contrast does not improve diagnostic performance

33 s study of intravenous (IV)-only versus IV + enteral contrast in computed tomographic (CT) scans perf

34 Eliminating enteral contrast may improve efficiency, patient comfort

35 foration, odds ratio of concordance for IV + enteral contrast versus IV contrast alone was 0.95 (95%

36 ence in concordance between IV-only and IV + enteral contrast.

37 se with IV contrast only and 28.5% with IV + enteral contrast.

38 Enteral delivery of proteins altered the duodenal mucosa

39 We recently reported that enteral delivery of proteins markedly stimulated global

40 Enteral DHA supplementation at a dose of 60 mg per kilog

41 Eligible patients received a single enteral dose of 540,000 IU of vitamin D(3) or matched pl

42 r first enteral feeding to receive either an enteral emulsion providing DHA at a dose of 60 mg per ki

43 The mean delivery of enteral energy and protein was 36 +/- 35% and 37 +/- 38%

44 pathogenesis is incompletely understood, but enteral feed exposures are believed to affect risk.

45 calculated caloric requirements) or standard enteral feeding (70 to 100%) for up to 14 days while mai

46 for the observed beneficial effects of early enteral feeding after major rectal surgery.

47 and P = 0.002).A slow rate of progression of enteral feeding and a less favorable direct-breastfeedin

48 amping, stabilisation with intubation, early enteral feeding and caffeine administration.

49 Early enteral feeding and, in particular, breast milk were cor

50 start is associated with underweight, while enteral feeding can lead to obesity.

51 NALD who were unable to wean from PN to full enteral feeding developed cirrhosis and end-stage liver

52 s encountered during endoscopic placement of enteral feeding devices.

53 Initial trophic versus full enteral feeding for the first 6 days after randomization

54 e median net protein balance improved during enteral feeding from -8.6 to -5.8 mumol . kg body weight

55 ermiT (Permissive Underfeeding versus Target Enteral Feeding in Adult Critically Ill Patients) trial.

56 05), whereas PEG-CCK9 mimicked the impact of enteral feeding in fasted animals (p < 0.05).

57 In critical illness, delaying enteral feeding is associated with a reduction in small

58 Enteral feeding may ameliorate protein loss, but its eff

59 infants, 2) clinical and practical issues in enteral feeding of preterm infants, 3) gastrointestinal

60 ain-matched TLR4KO mice were provided either enteral feeding or TPN.

61 ICE 4: In patients with pancreatic necrosis, enteral feeding should be initiated early to decrease th

62 wer and intermediate rates of progression of enteral feeding strategies were associated with a higher

63 Early enteral feeding through a nasoenteric feeding tube is of

64 Enteral feeding to deliver a moderate amount of nonprote

65 and center) within 3 days after their first enteral feeding to receive either an enteral emulsion pr

66 The enteral feeding tube was withdrawn prematurely from 48.5

67 onic Health Evaluation II score, presence of enteral feeding tube, mechanical ventilation, and recent

68 s, neurologic impairment, and presence of an enteral feeding tube.

69 Enteral feeding tubes and parenteral nutrition should no

70 lization), major morbidities, and nutrition (enteral feeding type, macronutrient/energy intakes) with

71 r, as was the proportion of cumulative total enteral feeding volume provided as breast milk: median (

72 nal data have shown that slow advancement of enteral feeding volumes in preterm infants is associated

73 infants based on BW, EGA, day of life (DOL) enteral feeding was initiated and DOL of the first sampl

74 cifications, 2) clinical/practical issues in enteral feeding, 3) gastrointestinal and surgical issues

75 mends goal-directed hydration therapy, early enteral feeding, judicious use of endoscopic retrograde

76 ive underfeeding), as compared with standard enteral feeding, on 90-day mortality among critically il

77 DD], acidification of gastric content, early enteral feeding, prevention of microinhalation); circuit

78 tegies concerning the rate of progression of enteral feeding, the direct-breastfeeding policy, and th

79 on across neonatal networks, and investigate enteral feeding-related antecedents of severe necrotisin

80 also decreased in humans who are deprived of enteral feeding.

81 ght junction protein losses due to a lack of enteral feeding.

82 eks, and <30 days old prior to initiation of enteral feeding.

83 s the delivery, digestion, and absorption of enteral feeding.

84 lity evidence); no reduction in tolerance of enteral feeds (risk ratio, 0.94 [95% CI, 0.62-1.42]; p =

85 ld be considered only in cases where oral or enteral feeds are not feasible or tolerated.

86 ion with high-DHA compared with standard-DHA enteral feeds decreases the incidence and severity of pa

87 s) or standard-DHA (~0.3% total fatty acids) enteral feeds from 2-4 d of postnatal age until 40 wk po

88 Probiotics reduced the time to full enteral feeds in preterm neonates.

89 ta-analyses have shown early introduction of enteral feeds to be beneficial to hospital stay and pati

90 efits are seen in patients receiving minimal enteral feeds versus full caloric enteral nutrition.

91 s (n = 4527) estimated that the time to full enteral feeds was shorter in the probiotic group (mean d

92 ile, drugs (e.g. laxatives, antibiotics) and enteral feeds.

93 cohorts based on percentage of MOM and DM in enteral feeds.

94 tory pressures and timing of introduction of enteral feeds.

95 Protein-energy-enriched enteral formulas (PE-formulas) can improve nutrient inta

96 enriched in sodium nitrate--a precursor for enteral generation of nitrite and nitric oxide--and repl

97 ccurred significantly less frequently in the enteral group (1 patient) compared with parenteral suppl

98 roup and 409 of 1195 patients (34.2%) in the enteral group had died (relative risk in parenteral grou

99 erences between the parenteral group and the enteral group in the mean number of treated infectious c

100 e higher in the parenteral group than in the enteral group on postoperative day 1 (p = 0.027) and day

101 Mean length of stay in the enteral group was 13.4 +/- 2.2 days versus 16.7 +/- 2.3

102 1191 in the parenteral group and 1197 in the enteral group).

103 n the parenteral group, as compared with the enteral group, in rates of hypoglycemia (44 patients [3.

104 arginine concentrations were measured in the enteral group, whereas a better clinical outcome was obs

105 Enteral IAP supplementation may represent a novel approa

106 nts with bleeding risk factors (1B); oral or enteral (if necessary) feedings, as tolerated, rather th

107 olled trials evaluating preoperative oral or enteral IMN in patients undergoing surgery for gastroint

108 Enteral IMN or CON was resumed postoperatively and conti

109 showed that lactoferrin given orally before enteral infection with pathogenic Escherichia coli reduc

110 om order and received, during 5 h, either an enteral infusion of maltodextrins alone (0.25 g . kg(-)(

111 ctional intestine to sustain themselves with enteral intake alone.

112 d-expiratory pressure (P < 0.01), and higher enteral intake in the first 7 days (P = 0.01).

113 Daily parenteral and enteral manganese intakes were calculated.

114 Our previous data showed that enteral nutrient deprivation or total parenteral nutriti

115 Likewise, enteral nutrient deprivation selectively affected the mo

116 Enteral nutrient deprivation via total parenteral nutrit

117 We used a model of enteral nutrient deprivation, or total parenteral nutrit

118 odel of total parenteral nutrition (TPN), or enteral nutrient deprivation, to study this interaction

119 ies, such as humans, undergo long periods of enteral nutrient deprivation.

120 tocols for the initiation and advancement of enteral nutrient intake had a lower prevalence of acquir

121 y bile salts upon gallbladder emptying after enteral nutrient stimulation.

122 strointestinal tract, which is influenced by enteral nutrients and microbial activity.

123 trition (TPN), which deprives the animals of enteral nutrients, displayed a significant decrease of I

124 ation) preoperatively into two groups: early enteral nutrition (early enteral nutrition, intervention

125 A nutritional intervention, exclusive enteral nutrition (EEN) can induce remission in patients

126 CD in 2010 and 2011 who commenced exclusive enteral nutrition (EEN) for 8 weeks were followed up for

127 Exclusive enteral nutrition (EEN) is recommended for children with

128 Exclusive enteral nutrition (EEN) is the only established dietary

129 iology of Crohn's Disease (CD) and exclusive enteral nutrition (EEN) is the primary induction treatme

130 dy was designed to investigate whether early enteral nutrition (EEN), as a bridge to a normal diet, c

131 Us) with relative contraindications to early enteral nutrition (EN) may benefit from parenteral nutri

132 Enteral nutrition (EN) was advanced and PN weaned as ind

133 lled trial (RCT) hypothesized that prolonged enteral nutrition (EN) with supplemental eicosapentanoic

134 Home enteral nutrition (HEN) has always been recognized as a

135 f research suggests that ongoing maintenance enteral nutrition (MEN) can be beneficial in maintaining

136 this study was to compare nasojejunal early enteral nutrition (NJEEN) with total parenteral nutritio

137 interleukin (IL)-6 levels were decreased by enteral nutrition (p < 0.05).

138 DED), a whole-food diet coupled with partial enteral nutrition (PEN), designed to reduce exposure to

139 ncluded rats with head injury fed a standard enteral nutrition (Sondalis HP, n = 10) and group 2 incl

140 on has a deleterious effect in comparison to enteral nutrition alone.

141 izing feeding practices improves delivery of enteral nutrition and decreases feeding complications.

142 Controlled studies of patients receiving enteral nutrition and observations made from patients on

143 might benefit from early intensive therapy, enteral nutrition and timely transfer to specialized cen

144 defecation was significantly shorter in the enteral nutrition arm than in the control arm (P = 0.04)

145 We compared the impact of administering enteral nutrition as either gastric feeding or jejunal f

146 tudy expands the immunomodulating effects of enteral nutrition as previously observed in rodents to m

147 When oral nutrition is not feasible, enteral nutrition by either nasogastric/duodenal or naso

148 average time from ICU admission to start of enteral nutrition compared to the control group (40.7-29

149 Early combined parenteral/enteral nutrition delayed recovery irrespective of sever

150 immunomodulatory potential of a custom-made enteral nutrition during systemic inflammation in man.

151 High-protein enteral nutrition enriched with immune-modulating nutrie

152 and no clear disadvantage of providing early enteral nutrition following elective gastrointestinal su

153 ally ventilated patients expected to receive enteral nutrition for >/=2 d were randomly assigned to r

154 The group suggested exclusive enteral nutrition for induction therapy and biologic tum

155 ilated for more than 72 hours and to require enteral nutrition for more than 72 hours were randomized

156 stomy (PEG) is an effective and safe mode of enteral nutrition for patients needing chronic enteric n

157 ity: 44.4% of patients died in the intensive enteral nutrition group (95% confidence interval [CI], 3

158 In the intensive enteral nutrition group, enteral nutrition was given via

159 Also, low-dose or trophic enteral nutrition has similar benefits with less gastroi

160 l, the Early Parenteral Nutrition Completing Enteral Nutrition in Adult Critically Ill Patients (EPaN

161 trial (Early Parenteral Nutrition Completing Enteral Nutrition in Adult Critically Ill Patients [EPaN

162 Current guidelines recommend enteral nutrition in critically ill adults; however, poo

163 sed the evidence on effects of probiotics on enteral nutrition in preterm neonates.

164 the effects of probiotic supplementation on enteral nutrition in preterm neonates.

165 r strains used specifically for facilitating enteral nutrition in this population.

166 enteral nutrition to supplement insufficient enteral nutrition increases morbidity in the intensive c

167 The optimization of enteral nutrition is a priority in preterm neonates worl

168 Early enteral nutrition is associated with less anastomotic le

169 Providing effective enteral nutrition is important during critical illness.

170 hic lateral sclerosis (ALS), the question of enteral nutrition is increasingly raised in NIV users AL

171 Early enteral nutrition is safe and associated with significan

172 ovide preliminary evidence that hypercaloric enteral nutrition is safe and tolerable in patients with

173 Using a mouse TPN model, removing enteral nutrition leads to decreased crypt proliferation

174 s adverse events considered to be related to enteral nutrition occurred in 5 patients.

175 ohn's disease patients starting therapy with enteral nutrition or anti-TNFalpha antibodies and reveal

176 n additional 48 hours and who were receiving enteral nutrition or expected to start imminently.

177 rwent intravenous cannulation and were given enteral nutrition or TPN for 7 days.

178 The amount of enteral nutrition patients with acute lung injury need i

179 luded rats with head injury fed the standard enteral nutrition plus arginine (4 g/kg/d, n = 11).

180 :1) to groups that received either intensive enteral nutrition plus methylprednisolone or conventiona

181 Early enteral nutrition reduced postoperative ileus, anastomot

182 Continuous administration of enteral nutrition resulted in a rapid anti-inflammatory

183 xplores management strategies for delivering enteral nutrition safely and effectively to this high-ri

184 The groups received similar volumes of enteral nutrition solution [1221 mL/d (95% CI: 1120, 132

185 he substitution of a 1.0- with a 1.5-kcal/mL enteral nutrition solution administered at the same rate

186 receive 1.5 (n = 57) or 1.0 (n = 55) kcal/mL enteral nutrition solution at a rate of 1 mL/kg ideal bo

187 ne whether the substitution of a 1.5-kcal/mL enteral nutrition solution for a 1.0-kcal/mL solution re

188 PURPOSE OF REVIEW: Enteral nutrition support is often required in patients

189 combination of corticosteroid and intensive enteral nutrition therapy is more effective than cortico

190 Management of enteral nutrition varies widely both pre and postoperati

191 nteral nutrition, 354 (36%) of whom received enteral nutrition via the postpyloric route.

192 proportion of targeted energy delivered from enteral nutrition was 72% for the early nasojejunal nutr

193 In both groups, enteral nutrition was attempted early and intravenous mi

194 Intolerance to enteral nutrition was based only on regurgitation and vo

195 nutrition in critically ill patients in whom enteral nutrition was contraindicated did not significan

196 gal anti-inflammatory reflex with lipid-rich enteral nutrition was demonstrated to prevent tissue dam

197 ith corticosteroids, we found that intensive enteral nutrition was difficult to implement and did not

198 In the intensive enteral nutrition group, enteral nutrition was given via feeding tube for 14 days

199 f prescribed protein and energy delivered by enteral nutrition was greater in the intervention sites

200 d to stay in ICU for at least 3 days in whom enteral nutrition was indicated.

201 arly initiation of parenteral nutrition when enteral nutrition was insufficient (early parenteral nut

202 Enteral nutrition was subsequently interrupted on averag

203 Enteral nutrition was systematically offered, although P

204 critically ill patients suitable to receive enteral nutrition were compared with 12 healthy subjects

205 Enteral nutrition with gastric or jejunal feeding in hea

206 l ventilation for more than 2 days and given enteral nutrition within 36 hours after intubation at 9

207 than 24 hours, of which 74,771 were ordered enteral nutrition within the first 48 hours.

208 A total of 985 subjects received enteral nutrition, 354 (36%) of whom received enteral nu

209 odulating nutrients vs standard high-protein enteral nutrition, initiated within 48 hours of ICU admi

210 o two groups: early enteral nutrition (early enteral nutrition, intervention) by nasojejunal tube (n

211 7 trials that compared parenteral nutrition, enteral nutrition, or nutritional supplements to no nutr

212 djunctive nutritional support (parenteral or enteral nutrition, or nutritional supplements) to patien

213 Calorie delivery from enteral nutrition, parenteral nutrition, propofol, and d

214 versy exists on the use of immune-modulating enteral nutrition, reflected by lack of consensus in gui

215 omponents were protective ventilation, early enteral nutrition, standardization of antibiotherapy for

216 Current evidence suggests that enteral nutrition, started as soon as possible after acu

217 g mechanical ventilation and receiving early enteral nutrition, the absence of gastric volume monitor

218 ASN, the American Society for Parenteral and Enteral Nutrition, the Academy of Nutrition and Dietetic

219 Among patients ordered early enteral nutrition, the risk of mortality in the body mas

220 However, among those ordered early enteral nutrition, the survival disadvantage for body ma

221 Given that birth marks the first exposure to enteral nutrition, we investigated how nutrient-regulate

222 s is a viable long-term treatment option for enteral nutrition, with complication rates similar to th

223 itically ill patients anticipated to receive enteral nutrition.

224 died critically ill patients receiving early enteral nutrition.

225 initial hypocaloric-hyponitrogenous dose of enteral nutrition.

226 ng minimal enteral feeds versus full caloric enteral nutrition.

227 with amyotrophic lateral sclerosis receiving enteral nutrition.

228 malabsorptive diarrhea upon ingestion of any enteral nutrition.

229 t common myths and misconceptions related to enteral nutrition.

230 and who were already receiving percutaneous enteral nutrition.

231 Animals were fasted or received lipid-rich enteral nutrition.

232 pneumonia (VAP) in patients receiving early enteral nutrition.

233 are frequently placed in patients to provide enteral nutrition.

234 d among a subgroup of patients ordered early enteral nutrition.

235 dense (1.5 kcal/ml) or routine (1.0 kcal/ml) enteral nutrition.Measurements and Main Results: Partici

236 o differences in need for postoperative (par)enteral nutritional support, other complications, hospit

237 gan failure, new-onset systemic dysfunction, enteral or pancreatic-cutaneous fistula, bleeding and pe

238 ssigned to the endoscopic approach developed enteral or pancreatic-cutaneous fistulae compared with 2

239 ntrolled trial in adult inpatients receiving enteral or parenteral nutrition (or both) who required s

240 Enteral or parenteral nutrition before, during, and afte

241 eous insulin therapy in inpatients receiving enteral or parenteral nutrition or both.

242 d controlled trials (RCTs) have investigated enteral or parenteral nutritional support, and evidence-

243 ve known roles in carbohydrate digestion and enteral or renal glucose transport, suggesting that gene

244 onia-detoxifying capacity through either the enteral or the intravenous route is approximately 160 mu

245 formance indicators were early initiation of enteral (oral or tube feeds) or parenteral nutrition; av

246 t anaphylaxis, were subjected to a high-dose enteral OVA desensitization protocol (OIT).

247 nutritional guidelines recommend the use of enteral over parenteral nutrition in patients undergoing

248 Cumulative days with diarrhea (P = 0.27), enteral (P = 0.42) or intravenous fluids (P = 0.19), oth

249 afting (CABG) were randomly assigned between enteral, parenteral, or no nutrition (control) from 2 d

250 ics (70.3%), prolonged drainage (67.8%), and enteral/parenteral nutrition (54.7%).

251 therapy and a need for opioid analgesic and enteral/parenteral nutrition, with an effect on patient

252 Enteral progesterone administration increased plasma lev

253 Unexpectedly, the glucoregulatory actions of enteral progesterone did not require classical incretin

254 iable practices at the bedside might enhance enteral protein delivery in the PICU with a potential fo

255 tian in the PICU were associated with higher enteral protein delivery.

256 ved during early hypocaloric-hyponitrogenous enteral protein feeding in these patients.

257 lished a method of quantifying the effect of enteral protein feeding on whole-body protein turnover a

258 In relation to mean enteral protein intake <20%, intake >/=60% of the prescr

259 The adequacy of enteral protein intake was significantly associated with

260 This might point to a ceiling effect for enteral protein intake with respect to its influence on

261 ine effects on growth of different levels of enteral protein supplementation in predominantly human m

262 We aimed to assess the influence of an enteral protein supply on the duodenal mucosal proteome

263 In the carbohydrate fed state, enteral proteins but not glutamine increased duodenal pr

264 RDS were randomly assigned to receive either enteral rosuvastatin or placebo in a double-blind manner

265 y 100% of recommended calorie intake via the enteral route during critical illness compared with a le

266 uman DAF did not facilitate infection by the enteral route either in immunocompetent animals or in an

267 he enhanced protein-energy provision via the enteral route feeding protocol is safe and results in mo

268 he enhanced protein-energy provision via the enteral route feeding protocol was associated with a dec

269 acebo three times a day orally or by another enteral route for 28 days or until ICU discharge.

270 intraperitoneal injection, infection by the enteral route is very inefficient.

271 criptions received by study patients via the enteral route over the first 12 days in the ICU.

272 be fed through either the parenteral or the enteral route to a delivery route, with nutritional supp

273 ibed caloric intake (+/- SE) received by the enteral route was 64% +/- 2 in the active group and 65%

274 nteral route is superior to that through the enteral route.

275 re susceptible to echovirus infection by the enteral route.

276 trointestinal tract through hematogenous and enteral routes.

277 ARDS within the previous 48 hours to receive enteral simvastatin at a dose of 80 mg or placebo once d

278 ppropriate parenteral nutrition was added to enteral sources.

279 s also being pioneered by studies looking at enteral sphincter augmentation and regeneration.

280 ically ill patients by using intravenous and enteral stable-isotope Phe tracers.

281 lation and treatment with levodopa-carbidopa enteral suspension can help individuals with medication-

282 ents such as therapy with levodopa-carbidopa enteral suspension or deep brain stimulation.

283 Dietary intervention studies have shown that enteral therapy, with defined formula diets, helps child

284 n the LBD that was successfully treated with enteral treatment using a calcium chloride infusion.

285 nutritional supplements (ONS; 11 RCTs), and enteral tube feeding (1 RCT)] with a control comparison

286 March 2016 we randomly allocated 59 women to enteral tube feeding and 57 women to standard care.

287 dverse effects (34%).In women with HG, early enteral tube feeding does not improve birth weight or se

288 0 wk of gestation were randomly allocated to enteral tube feeding for >/=7 d in addition to standard

289 /- SD birth weight was 3160 +/- 770 g in the enteral tube feeding group compared with 3200 +/- 680 g

290 We hypothesized that in women with HG, early enteral tube feeding in addition to standard care improv

291 was associated with increased dependence on enteral tube feeding or total parenteral nutrition [odds

292 Enteral tube feeding was discontinued within 7 d of plac

293 Of the women allocated to enteral tube feeding, 28 (47%) were treated according to

294 equently, she was fed an elemental diet with enteral tube feeding, and her condition gradually improv

295 eaned from parenteral nutrition to exclusive enteral tube feeding.

296 utrition was slowly tapered while increasing enteral tube feeding.

297 oric) or 50% of that value (hypocaloric) via enteral tube feeds or parenteral nutrition, with an equa

298 critically ill patients should be started on enteral tube feeds within 48 h of intubation whenever po

299 es morbidity through dermal injury, impaired enteral uptake and subsequent fluid imbalance.

300 Early administration of high-dose enteral vitamin D(3) did not provide an advantage over p