ライフサイエンスコーパス: enteral feeding

1 eks, and <30 days old prior to initiation of enteral feeding.

2 for renal protection, vasopressors, TPN, and enteral feeding.

3 s the delivery, digestion, and absorption of enteral feeding.

4 r treatment of diarrhea that develops during enteral feeding.

5 g, preterm infants during the first 12 wk of enteral feeding.

6 All patients were on enteral feeding.

7 pen-label trial of ketogenic versus standard enteral feeding.

8 also decreased in humans who are deprived of enteral feeding.

9 ght junction protein losses due to a lack of enteral feeding.

10 cluding medications, underlying illness, and enteral feeding.

11 eral feed tolerance thereby permitting early enteral feeding.

12 = 0.063), respectively, even at the start of enteral feeding.

13 n are lost with starvation and maintained by enteral feeding.

14 were trauma patients (83%), and 90% received enteral feeding.

15 mass and mucosal immunity when compared with enteral feeding.

16 Group Gf received nasogastric enteral feeding.

17 e to acute amino acid supplementation during enteral feeding.

18 ession, traumatic tissue injury, and lack of enteral feedings.

19 cal, as was the use of antibiotics, TPN, and enteral feedings.

20 ions were associated with the utilization of enteral feedings.

21 more cost-effective and timely initiation of enteral feedings.

22 and smell intervention was the time to full enteral feeding (150 ml per kilogram of body weight per

23 heelchair dependence (31% -> 57%), exclusive enteral feeding (22% -> 46%), and one-to-one assistance

24 o wheelchair dependence (31% 57%), exclusive enteral feeding (22% 46%), and one-to-one assistance for

25 "nothing by mouth" status (28%), started on enteral feeding (23%), or discharged from the intensive

26 cifications, 2) clinical/practical issues in enteral feeding, 3) gastrointestinal and surgical issues

27 calculated caloric requirements) or standard enteral feeding (70 to 100%) for up to 14 days while mai

28 failure-induced liver disease include early enteral feeding, a multidisciplinary approach to the man

29 s with acute lung injury, compared with full enteral feeding, a strategy of initial trophic enteral f

30 Enteral feeding affects MAdCAM-1 expression.

31 for the observed beneficial effects of early enteral feeding after major rectal surgery.

32 An IED is the preferred diet for early enteral feeding after severe blunt and penetrating traum

33 t of TPN support, can maintain themselves on enteral feedings after this intestinal rehabilitation pr

34 l period (C) of continuous high-carbohydrate enteral feeding alone, and (b) on the seventh day of ent

35 vival after transplant and both grew well on enteral feeding alone.

36 Lack of enteral feeding also induces mucosal effects.

37 and P = 0.002).A slow rate of progression of enteral feeding and a less favorable direct-breastfeedin

38 langiopancreatography acute pancreatitis and enteral feeding and antibiotics in severe acute pancreat

39 amping, stabilisation with intubation, early enteral feeding and caffeine administration.

40 curs in patients who develop diarrhea during enteral feeding and may be involved in its pathogenesis.

41 We showed directly a relation between enteral feeding and small-intestinal mucosal growth.

42 of oligopeptides as a source of nitrogen for enteral feeding and the use of oral route for delivery o

43 y acids (SCFAs) in patients starting 14-d of enteral feeding and to compare these changes between pat

44 age of 25 to 29 weeks, who were suitable for enteral feeding and who proved to be medically stable on

45 Early enteral feeding and, in particular, breast milk were cor

46 Infants without major anomalies who received enteral feedings and survived beyond postnatal day 7 wer

47 the major risk factors for NEC: prematurity, enteral feeding, and bacterial colonization.

48 sis, feeding intolerance, time to reach full enteral feeding, and duration of hospitalization.

49 ment of acute rejection, early postoperative enteral feeding, and ganciclovir prophylaxis.

50 Nursing care procedures, antacids, enteral feeding, and prophylactic antibiotics were admin

51 arenteral nutrition (PN), 41% were receiving enteral feeding, and the remaining 18% had already achie

52 y in the medical intensive care unit, use of enteral feeding, and use of sucralfate.

53 icant improvement in glucose control, use of enteral feeding, antibiotic use, adult respiratory distr

54 r upper GI surgery for malignancy with early enteral feeding are limited.

55 gth of hospital stay and early initiation of enteral feedings as compared with bedside self-migrating

56 Targeted enteral feeding before liver transplant improves HGS, an

57 For infants who need long-term enteral feeding but are unable to maintain sufficient or

58 espiratory tract immunity was preserved with enteral feeding but not with intravenous feeding.

59 start is associated with underweight, while enteral feeding can lead to obesity.

60 These data indicate that enteral feeding can prolong survival and decrease renal

61 scopists play a key role in the placement of enteral feeding catheters.

62 ictive dose, and may offset the benefit from enteral feeding, causing iatrogenic stresses to the syst

63 effective in reducing the time to reach full enteral feeding compared with placebo.

64 Thus, we tested the hypothesis that enteral feeding could prevent renal ischemic injury usin

65 ot the leaching from plastic tubing used for enteral feeding, could expose premature neonates to a cu

66 2021, the median (IQR) time to achieve full enteral feeding decreased from 18 (14-28) days to 14 (10

67 NALD who were unable to wean from PN to full enteral feeding developed cirrhosis and end-stage liver

68 s encountered during endoscopic placement of enteral feeding devices.

69 problems encountered during the placement of enteral feeding devices.

70 At full enteral feeding, donor milk is estimated to provide 1.3

71 These observations underscore the value of enteral feeding during health and disease.

72 The most recent data suggest that early enteral feeding, even when patients are receiving vasopr

73 The primary outcome was time to achieve full enteral feeding (FEF) defined as an enteral intake of 15

74 The primary outcome was the time to full enteral feeding (FEF), defined as an enteral intake of 1

75 Initial trophic versus full enteral feeding for the first 6 days after randomization

76 randomized to receive either trophic or full enteral feeding for the first 6 days.

77 teral feeding, a strategy of initial trophic enteral feeding for up to 6 days did not improve ventila

78 The patients then received enteral feedings for 2 hrs, after which the gastric intr

79 e median net protein balance improved during enteral feeding from -8.6 to -5.8 mumol . kg body weight

80 ts, best practice guidelines for withholding enteral feeding from intubated patients before scheduled

81 the length of time necessary for withholding enteral feeding from intubated patients before scheduled

82 rograms for the length of time they withhold enteral feeding from intubated patients before seven sch

83 Enteral feeding has been strongly recommended in severe

84 Enteral feeding has largely replaced the parenteral rout

85 est that early initiation and advancement of enteral feeding have the potential to reduce the risk of

86 udies suggest that early achievement of full enteral feeding improves clinical outcomes among preterm

87 ermiT (Permissive Underfeeding versus Target Enteral Feeding in Adult Critically Ill Patients) trial.

88 ceiving cimetidine) were mixed with 60 mL of enteral feeding in an airtight container; the PCO2 of th

89 05), whereas PEG-CCK9 mimicked the impact of enteral feeding in fasted animals (p < 0.05).

90 Enteral feeding in malnourished patients may result in r

91 , duration of parenteral nutrition and early enteral feeding in neonates with congenital duodenal obs

92 Time to achieve full enteral feeding in the open pyloromyotomy group was (med

93 thogenesis of diarrhea in patients receiving enteral feeding includes colonic water secretion, antibi

94 ing oesophagus may require oesophagectomy or enteral feeding into the stomach.

95 Enteral feeding intolerance (EFI) is a frequent problem

96 d with rats infused with diet A after 3 d of enteral feeding irrespective of endotoxin co-infusion.

97 In critical illness, delaying enteral feeding is associated with a reduction in small

98 Enteral feeding is challenging in preterm infants becaus

99 hy and impaired mucosal transport occur when enteral feeding is not provided, residual transport can

100 ting a higher limit for glucose control when enteral feeding is principally used.

101 olerance (ie, achieving and maintaining full enteral feedings) is a significant problem in preterm in

102 mends goal-directed hydration therapy, early enteral feeding, judicious use of endoscopic retrograde

103 Enteral feeding may ameliorate protein loss, but its eff

104 distribution, and clearance, and concomitant enteral feeding may decrease fluoroquinolone bioavailabi

105 dback, the rate of intestinal transit during enteral feeding may depend on a balance between the acce

106 a direct reaction between gastric fluid and enteral feedings may generate CO2, 30-mL aliquots of gas

107 In preterm infants, both parenteral and enteral feeding methods are modeled on term breast milk.

108 The continuous intragastric in vivo enteral feeding model in the rat developed by Tsukamoto

109 alues were similar to levels achieved in the enteral feeding model.

110 to 4 weeks via intragastric feeding using an enteral feeding model.

111 In such instances, enteral feeding must be temporarily discontinued or seve

112 ndard parenteral nutrition (n = 7) or normal enteral feeding (n = 8).

113 he consequences of frequent interruptions of enteral feeding need to be weighed against the possible

114 Preoperative enteral feeding (odds ratio = 0.68; 0.52-0.9) and open s

115 We compared the median hours of withholding enteral feeding of intubated patients according to train

116 infants, 2) clinical and practical issues in enteral feeding of preterm infants, 3) gastrointestinal

117 further information defining the effects of enteral feeding on mucosal immunity.

118 here are clinical trials showing benefits of enteral feeding on outcome of acute pancreatitis as well

119 ive underfeeding), as compared with standard enteral feeding, on 90-day mortality among critically il

120 e directives should be addressed long before enteral feeding or assistive ventilatory support might b

121 s no evidence to support specific methods of enteral feeding or increased frequency of ventilator cir

122 rtant clinical outcomes of parenteral versus enteral feeding or intravenous fluids in patients with t

123 did not show any effects on the time to full enteral feeding or on body composition at 4 months of co

124 ain-matched TLR4KO mice were provided either enteral feeding or TPN.

125 Compared with enteral feeding, parenteral nutrition was associated wit

126 s at 28 days, patient-days receiving oral or enteral feeding, patient-days under light sedation, and

127 feeding alone, and (b) on the seventh day of enteral feeding plus exogenous insulin (200 pmol/h = 28

128 DD], acidification of gastric content, early enteral feeding, prevention of microinhalation); circuit

129 ethanol diet or high-fat control diet via an enteral-feeding protocol for 3 weeks.

130 Enteral feeding provides nutrients for patients who requ

131 teworthy studies on endoscopic approaches to enteral feeding published from January 2005 to the prese

132 Lower enteral feeding rates and changes in weight, as well as

133 Enteral feeding reduced the death rate and organ permeab

134 ) or intermittent (6x/day, intervention arm) enteral feeding regimen.

135 on across neonatal networks, and investigate enteral feeding-related antecedents of severe necrotisin

136 The increased intraluminal CO2 following enteral feeding results in a spuriously low gastric intr

137 ICE 4: In patients with pancreatic necrosis, enteral feeding should be initiated early to decrease th

138 In experiment 1, enteral feeding significantly reduced the death rate com

139 Enteral feeding significantly reduces the incidence of p

140 outcomes were the median daily increment of enteral feeding, signs of feeding intolerance, effective

141 range of disorders from abscess drainage to enteral feeding solutions to treating hydronephrosis.

142 cal microbiota or SCFAs were observed during enteral feeding, stark alterations occurred within indiv

143 Enteral feeding stimulates the secretion of hydrogen ion

144 wer and intermediate rates of progression of enteral feeding strategies were associated with a higher

145 is, 87% of surviving patients do not require enteral feeding support.

146 tegies concerning the rate of progression of enteral feeding, the direct-breastfeeding policy, and th

147 a model unvalidated for non-clear fluids as enteral feeding, the scanning protocol was not clearly d

148 hed preterm infants commonly receive minimal enteral feedings, the aim being to enhance intestinal fu

149 Early enteral feeding through a nasoenteric feeding tube is of

150 h protein supplements are routinely added to enteral feeding to correct protein malnutrition, little

151 Enteral feeding to deliver a moderate amount of nonprote

152 and center) within 3 days after their first enteral feeding to receive either an enteral emulsion pr

153 This report describes a novel technique of enteral feeding tube placement, using external magnetic

154 The enteral feeding tube was withdrawn prematurely from 48.5

155 onic Health Evaluation II score, presence of enteral feeding tube, mechanical ventilation, and recent

156 s, neurologic impairment, and presence of an enteral feeding tube.

157 Enteral feeding tubes and parenteral nutrition should no

158 Enteral feeding tubes are often used in this situation,

159 e enabled endoscopists to successfully place enteral feeding tubes in patients who previously require

160 ch as radiographs, fluoroscopic placement of enteral feeding tubes, and insertion of vena cava filter

161 determined in those patients receiving total enteral feeding (two-thirds polymeric/one-third elementa

162 lization), major morbidities, and nutrition (enteral feeding type, macronutrient/energy intakes) with

163 with no DHA) from within the first 3 days of enteral feeding until 36 weeks' postmenstrual age or dis

164 uch as cholestasis might be reduced by early enteral feedings, ursodeoxycholic acid, and cholecystoki

165 r, as was the proportion of cumulative total enteral feeding volume provided as breast milk: median (

166 nal data have shown that slow advancement of enteral feeding volumes in preterm infants is associated

167 ch additional 1-week delay in achieving full enteral feeding was 16% higher (adjusted relative risk [

168 infants based on BW, EGA, day of life (DOL) enteral feeding was initiated and DOL of the first sampl

169 ith a nasogastric tonometer in situ, in whom enteral feeding was initiated.

170 of the gastric fluid before and after adding enteral feeding was measured tonometrically.

171 Moreover, enteral feeding was significantly earlier in the JFT tha

172 The time to full enteral feeding was similar among the infants who were e

173 Delays in achieving full enteral feeding were also associated with a higher risk

174 postnatal day 7, delays in establishing full enteral feeding were associated with a higher risk of la

175 enty patients starting exclusive nasogastric enteral feeding were monitored for 14 d.

176 ormula, which was fed from the age when full enteral feedings were tolerated through expected term, o

177 Other important mechanisms include lack of enteral feeding, which leads to reduced gut hormone secr

178 ths, she had undergone Stamm gastrostomy for enteral feeding with a Pezzer catheter.

179 Short-term enteral feeding with an eicosapentaenoic acid-enriched o

180 Early enteral feeding with an IEF has been associated with imp

181 Early enteral feeding with an IEF was not beneficial and shoul

182 was to determine whether early postoperative enteral feeding with an immune-enhancing formula (IEF) d

183 Additionally, patients received enteral feeding with an immunonutrition formula (experim

184 present study determined whether short-term enteral feeding with diets enriched with either eicosape

185 retrospectively testing the hypothesis that enteral feeding with EPA+GLA could reduce alveolar-capil

186 be rapidly modified by continuous short-term enteral feeding with EPA- and GLA-enriched diets irrespe

187 Early enteral feeding with FOSL-HN was safe and well tolerated

188 he patient's ICU stay in which they required enteral feeding, with a maximum of 90 days.

189 and hepatic 125I albumin leak compared with enteral feeding without increasing pulmonary myeloperoxi

190 ith functioning grafts are currently on full enteral feeding without need for any intravenous supplem