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1 -domain exoproteins found only in pathogenic enteric bacteria.
2 ixation in cyanobacteria and the survival of enteric bacteria.
3 changes in the growth rate for fast adapting enteric bacteria.
4 ersal regulator of virulence determinants in enteric bacteria.
5     A similar approach can be used for other enteric bacteria.
6 om a conserved master virulence regulator of enteric bacteria.
7 . thuringiensis-induced mortality depends on enteric bacteria.
8 in the host and an aberrant host response to enteric bacteria.
9 nt mucosal CD4+ T cell response to commensal enteric bacteria.
10 s a homodimeric DNA-binding protein found in enteric bacteria.
11 e extreme acid-resistance response common to enteric bacteria.
12  a crucial role in global gene regulation of enteric bacteria.
13 llow for spread of the pCoo plasmid to other enteric bacteria.
14 pid that is synthesized by all gram-negative enteric bacteria.
15  flagellar biosynthesis and swarming in many enteric bacteria.
16 t pathway that is typically used by LPS from enteric bacteria.
17 can be regulated by colonization with normal enteric bacteria.
18 hat resistance might be transferred to other enteric bacteria.
19 ntibodies, putatively in response to GalT(+) enteric bacteria.
20 s, the plague bacillus, from closely related enteric bacteria.
21 reside within a core regulon, shared by most enteric bacteria.
22 nd the generation of CD4 T cell responses to enteric bacteria.
23 ally by the adenylyltransferase (GlnE) as in enteric bacteria.
24 ation, and mechanisms of resistance to AP in enteric bacteria.
25  nrf operon promoter from various pathogenic enteric bacteria.
26 version of the genetic map relative to other enteric bacteria.
27 microorganisms, including some gram-negative enteric bacteria.
28 hism three orders of magnitude lower than in enteric bacteria.
29 probably of other physiological processes in enteric bacteria.
30 ipenem, which has broad-spectrum coverage of enteric bacteria.
31 s is significantly more complex than that of enteric bacteria.
32 erstanding of lipopolysaccharide function in enteric bacteria.
33 two-component Ntr regulatory system found in enteric bacteria.
34 ct from those that mediate oxygen control in enteric bacteria.
35 vation-induced cross-resistance to stress in enteric bacteria.
36 romosomally encoded antibiotic resistance in enteric bacteria.
37 t committed step in heme biosynthesis in the enteric bacteria.
38  of the well-characterized Che system of the enteric bacteria.
39 sequence similarity to the type I enzymes of enteric bacteria.
40 bout 80% identical and 90% similar to Fis in enteric bacteria.
41 ex regulatory pathway than that found in the enteric bacteria.
42 or as a dissimilatory metal ion reductase in enteric bacteria.
43 ocyte infiltration, which may be mediated by enteric bacteria.
44 ins (22 genera) of non-E. coli gram-negative enteric bacteria.
45 s by epithelial cells infected with invasive enteric bacteria.
46 ary-phase and osmotically inducible genes in enteric bacteria.
47  and acquisition of pathogenicity islands in enteric bacteria.
48 ritoneal cavity with gram-negative and other enteric bacteria.
49  and thus the regulator is not restricted to enteric bacteria.
50 ral regulator of peroxide stress response in enteric bacteria.
51 r resistance to oxidative and heat stress in enteric bacteria.
52 ation in a manner analogous to that found in enteric bacteria.
53 ays sequence homology to porin proteins from enteric bacteria.
54 CusR/CusS regulatory system present in other enteric bacteria.
55 lls required the presence of HBGA-expressing enteric bacteria.
56  and internalization of normally noninvasive enteric bacteria.
57 c resistance genes among clinically relevant enteric bacteria.
58 d to prevent systemic infection of mice with enteric bacteria.
59  the regulation of inflammatory responses to enteric bacteria.
60 effector that is widely conserved throughout enteric bacteria.
61 ed within dusB-fis operons of representative enteric bacteria.
62 les, along with discrimination against other enteric bacteria.
63 Escherichia coli, Salmonella spp., and other enteric bacteria.
64    Curli are functional amyloids produced by enteric bacteria.
65 roduce inflammatory cytokines in response to enteric bacteria.
66 L-10, but not IL-12 p40, when activated with enteric bacteria.
67 ent a potential therapeutic intervention for enteric bacteria.
68 er range of exogenous fatty acids than other enteric bacteria.
69 s different from that of flgJ mutants in the enteric bacteria.
70 he cell by sensing the level of glutamine in enteric bacteria.
71 gest that this function may be restricted to enteric bacteria.
72  which c-di-GMP initiates this transition in enteric bacteria.
73  role associated with E. coli Lrp limited to enteric bacteria?
74 d antigen passages (GAPs), which translocate enteric bacteria across the intestinal epithelium.
75      Nitrogen regulatory protein C (NtrC) of enteric bacteria activates transcription of genes/operon
76 ing a global regulatory role evolved to help enteric bacteria adapt to their ecological niches and th
77                                           In enteric bacteria, adaptation to a number of different st
78                                           In enteric bacteria, adhesion to host cells is often promot
79 ndent acid resistance system, which protects enteric bacteria against the extreme acidity of the huma
80                                              Enteric bacteria also produce amyloids, termed curli, co
81  dga genes are largely restricted to certain enteric bacteria and a few species in the phylum Firmicu
82 dentify ZntB as a zinc efflux pathway in the enteric bacteria and assign a new function to the CorA f
83 ch helminths modulate the host's response to enteric bacteria and bacteria-mediated intestinal inflam
84 tosis but also to apoptosis induced by other enteric bacteria and by several toxic agents including r
85 p of genes involved in host-cell invasion by enteric bacteria and can be modeled to predict the amoun
86 able increases in levels of gastrointestinal enteric bacteria and Candida.
87 tory loops controlling expression of oxyR in enteric bacteria and characteristic of the LysR superfam
88  appendages are found on the surface of many enteric bacteria and enable the bacteria to bind to euka
89 t observation of direct interactions between enteric bacteria and enteric viruses, we observed a dire
90                     The interactions between enteric bacteria and genetic susceptibilities are major
91 es stable increases in gastrointestinal (GI) enteric bacteria and GI Candida levels with no introduct
92 protein H-NS that is conserved in a range of enteric bacteria and had no known function in Salmonella
93 oup IV polysaccharide capsules are common in enteric bacteria and have more recently been described i
94 is homologous to both CheA and CheY from the enteric bacteria and is therefore a novel CheA-CheY fusi
95    Different strategies are needed to reduce enteric bacteria and parasites at this critical young ag
96 step in the cysteine biosynthetic pathway in enteric bacteria and plants, substitution of the beta-ac
97 step in the cysteine biosynthetic pathway in enteric bacteria and plants, the replacement of the beta
98 step in the cysteine biosynthetic pathway in enteric bacteria and plants, the replacement of the beta
99  establish that gut epithelia actively sense enteric bacteria and play an essential role in maintaini
100 chical regulation by the sRNA SgrS, found in enteric bacteria and produced under conditions of metabo
101 , an abundant nucleoid-associated protein in enteric bacteria and related species, can recognize and
102 eruginosa that is different from that in the enteric bacteria and seems to be most similar to that of
103          By blocking the interaction between enteric bacteria and the innate immune system, CRX-526 m
104                Given the enormous numbers of enteric bacteria and the persistent threat of opportunis
105 m/methylammonium transport (Amt) proteins of enteric bacteria and their homologues, the methylammoniu
106 nclude mutually beneficial signaling between enteric bacteria and their mammalian hosts.
107                                              Enteric bacteria and their products play an important ro
108                Flagellin is secreted by many enteric bacteria and, upon reaching the basolateral memb
109        This study is aimed at determining if enteric bacteria and/or immune signals regulate their ph
110                                              Enteric bacteria and/or their products are necessary for
111 e of Escherichia coli, as well as many other enteric bacteria, and are involved in cell colonization
112  solfataricus, mutational data obtained from enteric bacteria, and the enzyme's mechanism of action.
113 r CD4+ T cells react to Ags of the commensal enteric bacteria, and the latter can mediate colitis whe
114 he YcgR-FliG interaction is conserved in the enteric bacteria, and the N-terminal YcgR/PilZN domain o
115 hat the structurally related GlnB protein of enteric bacteria-apparently a paralogue of GlnK-cannot s
116                                    Commensal enteric bacteria are a required pathogenic factor in inf
117  work in several laboratories has shown that enteric bacteria are common inhabitants of the interior
118                                              Enteric bacteria are frequently found in aquatic environ
119           We have tested the hypothesis that enteric bacteria are necessary for formation of intestin
120          We conclude therefore that resident enteric bacteria are necessary for the development of sp
121        This study tested the hypothesis that enteric bacteria are necessary for the development of sp
122                   Multi-drug resistant (MDR) enteric bacteria are of increasing global concern.
123  the MgtA and MgtB Mg2+ transport systems of enteric bacteria are P-type ATPases by sequence homology
124               Virulence factors expressed by enteric bacteria are pivotal for pathogen colonization a
125                 Several proteins secreted by enteric bacteria are thought to contribute to virulence
126      These metabolic transitions, common for enteric bacteria, are often accompanied by multi-hour la
127 ells as a cellular target of noroviruses and enteric bacteria as a stimulatory factor for norovirus i
128 rogen metabolism and in the pathogenicity of enteric bacteria, available biochemical data are scarce.
129 t that glycogen storage may be widespread in enteric bacteria because it is necessary for maintaining
130 pression of outer membrane porin proteins in enteric bacteria, belongs to a large family of transcrip
131 other pathogenic E. coli strains and related enteric bacteria but differs in Salmonella enterica sero
132 ter crescentus or the peritrichous system of enteric bacteria but is pertinent to many Vibrio and Pse
133                   CheS has no counterpart in enteric bacteria but revealed distinct similarities to p
134 n is associated with dramatic alterations in enteric bacteria, but little is known about other microb
135 age chi is known to infect motile strains of enteric bacteria by adsorbing randomly along the length
136 MarR and MarA confer multidrug resistance in enteric bacteria by modulating efflux pump and porin exp
137 ed in the regulation of rRNA biosynthesis in enteric bacteria by modulating the efficiency of transcr
138  intestinal homeostasis to commensal luminal enteric bacteria by regulating NF-kappaB signaling in IE
139 t that dysregulated host immune responses to enteric bacteria can influence the development of extrai
140                               Populations of enteric bacteria can inhibit the NF-kappaB pathway by bl
141                                      Certain enteric bacteria can inhibit the NF-kappaB pathway by bl
142 cterial siderophore involved in virulence of enteric bacteria, can only be brought into the cell usin
143 modium infections increase the likelihood of enteric bacteria causing systemic infections.
144 ties to the major chemotaxis proteins of the enteric bacteria: CheA, CheY, CheW, CheR, CheB and Tar.
145                               In contrast to enteric bacteria, chemotaxis in Rhodobacter sphaeroides
146              In contrast to the situation in enteric bacteria, chemotaxis in Rhodobacter sphaeroides
147                                              Enteric bacteria circumvent the gastric acid barrier by
148 hogenic Escherichia coli strain UTI89 and by enteric bacteria Citrobacter koseri and Salmonella enter
149    The central nitrogen metabolic circuit in enteric bacteria consists of three enzymes: glutamine sy
150                   The PhoQ sensor domains of enteric bacteria contain an acidic cluster of residues (
151                                     As other enteric bacteria contain CpxA, this signal exploitation
152  the enteric pathogen salmonella, like other enteric bacteria, contains three putative membrane-assoc
153                                         Many enteric bacteria cope with phosphosugar accumulation and
154                Class I pauses, discovered in enteric bacteria, depend on interaction of a nascent RNA
155  DNA restriction and modification systems of enteric bacteria display several enzymatic activities du
156  we report that mycobacteria, in contrast to enteric bacteria, do not form higher-order structures (e
157 ar functional amyloids that are assembled by enteric bacteria during biofilm formation and host colon
158                Type III secretion systems of enteric bacteria enable translocation of effector protei
159                                       In the enteric bacteria Escherichia coli and Salmonella enteric
160 by the PhoP/PhoQ two-component system in the enteric bacteria Escherichia coli and Salmonella enteric
161            Application of this method to the enteric bacteria Escherichia coli and Salmonella enteric
162                                       In the enteric bacteria Escherichia coli and Salmonella enteric
163  three steps of siroheme biosynthesis in the enteric bacteria Escherichia coli and Salmonella enteric
164 esses containing commensal organisms, mostly enteric bacteria, even when reared under specific pathog
165               Homologues of the amtB gene of enteric bacteria exist in all three domains of life.
166 binding sites at the nir promoter in related enteric bacteria fix the level of nir operon expression
167                                       In the enteric bacteria, flgJ null mutants fail to produce the
168  and is an essential enzyme in gram-negative enteric bacteria for maintenance of bacterial viability.
169 different larger systems of proteins used by enteric bacteria for molecular recognition and signaling
170 protects Escherichia coli and possibly other enteric bacteria from exposure to the strong acid enviro
171  the distribution of antimicrobial-resistant enteric bacteria from three ethnic groups in Tanzania.
172 ila, 20 vertebrate, 17 Staphylococcus and 20 enteric bacteria genomes, EvoPrinterHD employs a modifie
173 s were colonized with specific-pathogen-free enteric bacteria grown overnight either in anaerobic or
174  Germfree transgenic rats reconstituted with enteric bacteria grown under anaerobic conditions had mo
175 ction of crotonobetaine) from L-carnitine by enteric bacteria has been demonstrated in rats and human
176                            Quorum sensing in enteric bacteria has been elusive for a long time.
177  The nitrogen regulatory protein (NtrC) from enteric bacteria has been the model for this family of a
178    Although the oligomerization of H-NS from enteric bacteria has been the subject of intense investi
179          We describe how genetic analysis of enteric bacteria has contributed to these issues.
180 nces reveal that chromosome heterogeneity in enteric bacteria has resulted from the acquisition and d
181             Dysregulated T cell responses to enteric bacteria have been implicated as a common mechan
182 pathways of infection utilized by pathogenic enteric bacteria have important implications for their c
183 rotaviruses, Sapporo-like caliciviruses, and enteric bacteria (i.e., Salmonella, Clostridium, and Shi
184 ses to a subset of commensal (nonpathogenic) enteric bacteria in genetically predisposed individuals.
185  with choledochocholedochostomy (CDC) and by enteric bacteria in patients with choledochojejunostomy
186  function of reducing horizontal transfer of enteric bacteria in poultry.
187                                Chemotaxis of enteric bacteria in spatial gradients toward a source of
188 of undesirable immune responses to foods and enteric bacteria in the gastrointestinal tract.
189               This is in contrast to certain enteric bacteria in which the absence of a terminal deco
190 ral regulator of peroxide stress response in enteric bacteria) in Mycobacterium leprae, this gene is
191 (ECA), a surface glycolipid ubiquitous among enteric bacteria, in S. Typhimurium pathogenesis.
192                                         Some enteric bacteria including Salmonella have evolved the p
193  in many pathogenic strains of Gram-negative enteric bacteria, including E. coli, Salmonella spp., an
194  greater numbers of each of seven strains of enteric bacteria, including Listeria monocytogenes (two
195  for metabolizing 1,2-propanediol in certain enteric bacteria, including Salmonella.
196 n is also conserved in several Gram-negative enteric bacteria, indicating that this Mn(2+)-responsive
197                        Normal, nonpathogenic enteric bacteria induce and perpetuate chronic intestina
198               This regulatory network in all enteric bacteria involves genetic, allosteric, and physi
199                Type III secretion (T3S) from enteric bacteria is a co-ordinated process with a hierar
200      An early process in the pathogenesis of enteric bacteria is colonization of the intestinal epith
201                     The RpoS sigma factor of enteric bacteria is either required for or augments the
202               An aberrant T cell response to enteric bacteria is important in inflammatory bowel dise
203 xpression of histidine biosynthetic genes in enteric bacteria is regulated by an attenuation mechanis
204                     The RpoS sigma factor of enteric bacteria is required for the increased expressio
205                  The host immune response to enteric bacteria is tightly compartmentalized to the muc
206 ichia coli cytoplasm, but how nitrate enters enteric bacteria is unknown.
207 stimulation, from Escherichia coli and other enteric bacteria, is an interwined alpha-helical homodim
208 t of redox-cycling compounds, whereas in the enteric bacteria it defends the cell against the same ag
209 fis operon structures were identified in the enteric bacteria Klebsiella pneumoniae, Serratia marcesc
210 higella, and Yersinia, three other genera of enteric bacteria known to cause apoptosis.
211               The only regulatory protein in enteric bacteria known to serve exclusively as an NO-res
212 type; second, K. aerogenes and several other enteric bacteria lack a gene homologous to gltF; and thi
213 der region of the his biosynthetic operon of enteric bacteria like Escherichia coli.
214 e and utilize pseudocobalamin contrasts with enteric bacteria like Salmonella.
215                     Eradication of commensal enteric bacteria mitigates intestinal inflammation and I
216      To successfully colonize the human gut, enteric bacteria must activate acid resistance systems t
217                                     Invasive enteric bacteria must pass through the intestinal epithe
218                  To reach the mammalian gut, enteric bacteria must pass through the stomach.
219 ed as a single organism (n=57), with another enteric bacteria (n=23), or with coagulase negative stap
220 y 1 hour of incubation with pure cultures of enteric bacteria, namely, Salmonella typhimurium (two st
221 ellar components, heterologous expression in enteric bacteria of the putative switch basal body genes
222 ntestinal and extraintestinal infections and enteric bacteria on digestive motor function continues t
223 tion in intestinal epithelia and the role of enteric bacteria on VDR expression.
224                             Translocation of enteric bacteria or their components (or both) has been
225 mpetitive advantage this system provides for enteric bacteria, particularly those that inhabit an ana
226 ce of different exposure routes by measuring enteric bacteria (pathogenic Escherichia coli) and virus
227 e investigated the relative roles of various enteric bacteria populations in the induction and perpet
228                                              Enteric bacteria possess multiple fimbriae, many of whic
229                                   In the non-enteric bacteria Pseudomonas aeruginosa and Streptomyces
230 rminants may play a role in the evolution of enteric bacteria quite apart from, and perhaps with prec
231           The mutS-rpoS intergenic region of enteric bacteria ranges in size from 88 bp in Yersinia t
232 ic challenges in the gastrointestinal tract, enteric bacteria rapidly accumulate salts of glutamate a
233                 Secretory IgA, which targets enteric bacteria, regulates the number, composition, and
234              In Escherichia coli and related enteric bacteria, repair of base-base mismatches is perf
235 lactamase-1 (NDM-1) has been found to confer enteric bacteria resistance to nearly all beta-lactams,
236 cuits that exist between the closely related enteric bacteria Salmonella and E. coli.
237 hanism responsible for the divergence of the enteric bacteria Salmonella enterica and Escherichia col
238                  Small intestines exposed to enteric bacteria secreted zonulin.
239 there is increasing evidence to suggest that enteric bacteria sense and respond to the host NE stress
240                      The deamidase OspI from enteric bacteria Shigella flexneri deamidates a glutamin
241       By contrast, Lrp-related proteins from enteric bacteria show more than 97% amino acid identity.
242           The sequence preceding ORF1 in the enteric bacteria showed a very strong similarity to the
243        The chemotaxis signal protein CheY of enteric bacteria shuttles between transmembrane methyl-a
244 r treatment, however, has limited effects on enteric bacteria so we tested the hypothesis that excret
245 otein component of curli fibers assembled by enteric bacteria such as Escherichia coli and Salmonella
246 i are functional amyloid fibers assembled by enteric bacteria such as Escherichia coli and Salmonella
247          The former is typically observed in enteric bacteria such as Escherichia coli and Salmonella
248                                              Enteric bacteria such as Escherichia coli must tolerate
249                                              Enteric bacteria such as Escherichia coli utilize variou
250 ntestinal tract is vital for the survival of enteric bacteria such as Escherichia coli.
251 hanism exhibited by many of the well-studied enteric bacteria, such as enteroinvasive Escherichia col
252 zation have been extensively studied in some enteric bacteria, such as Klebsiella pneumoniae; however
253  genomes in comparison with those of related enteric bacteria suggest that extensive changes includin
254                                              Enteric bacteria synthesize glutamate by the combined ac
255 A and for outer membrane proteins from other enteric bacteria than either an isogenic DeltarfaH deriv
256  part because of an increasing prevalence of enteric bacteria that are resistant to 3(rd)-generation
257 epithelial cells infected with gram-negative enteric bacteria that can bypass TLR activation.
258 A binding regulator with orthologues in many enteric bacteria that exhibits classical regulator activ
259 nents of the outer membrane of gram-negative enteric bacteria that function as potent stimulators of
260 IS) is a 22 kDa homodimeric protein found in enteric bacteria that is involved in the stimulation of
261 nal epithelium may recruit E. coli and other enteric bacteria that possess a Tsr-like receptor to pre
262                    RpoS, the sigma factor of enteric bacteria that responds to stress and stationary
263  h postwithdrawal, morphine-treated mice had enteric bacteria that were detected in the Peyer's patch
264 ented internalization of selected strains of enteric bacteria that were preferentially adherent on th
265                                           In enteric bacteria, the cellular response to oxidative str
266       In contrast to the rfaF (waaF) gene of enteric bacteria, the H. ducreyi waaF gene was not locat
267                                           In enteric bacteria, the key player of carbon catabolite re
268                                           In enteric bacteria, the kinase LsrK catalyzes the phosphor
269                                           In enteric bacteria, the transcription factor sigma(E) main
270 ellular pH or synthesizing ATP in many other enteric bacteria; therefore, we used degenerate primers
271              Since cyanobacteria predate the enteric bacteria, this procapsid-mediated assembly pathw
272    We found that Paneth cells directly sense enteric bacteria through cell-autonomous MyD88-dependent
273 xtends the family affiliations identified in enteric bacteria to a wide range of other genera.
274 ity, flagellum expression, and resistance of enteric bacteria to acetic acid and bile salts.
275 ecting organisms away from staphylococci and enteric bacteria to Aspergillus species, although staphy
276 iderophore enterobactin (Ent) is produced by enteric bacteria to mediate iron uptake.
277 obactin (Ent) is produced by many species of enteric bacteria to mediate iron uptake.
278                    The ability of species of enteric bacteria to recognize and colonize unique niches
279                                              Enteric bacteria tumble, swim slowly, and are then paral
280                                              Enteric bacteria use a limited array of macromolecular s
281                                         Most enteric bacteria use intestinal brushborder glycoconjuga
282 nces in our understanding of how pathogenic, enteric bacteria use quorum sensing to regulate several
283                    Recent data indicate that enteric bacteria use several quorum-sensing mechanisms i
284 doubling time and the glutamine pool size in enteric bacteria was also seen in phylogenetically dista
285 V infection and the family-level taxonomy of enteric bacteria was detected.
286 een glutamine pool size and doubling time in enteric bacteria was far less obvious in B. subtilis.
287              The stimulatory material in the enteric bacteria was trypsin sensitive and restricted by
288 ver the 2-week period; however, detection of enteric bacteria was variable if specimens were not refr
289 a nucleoid-associated DNA-binding protein of enteric bacteria, was discovered 35 years ago and subseq
290  termination at the intrinsic terminators of enteric bacteria, we observed, by using single-molecule
291 e genomes from prophages embedded in diverse enteric bacteria, we produced gokushoviruses in an exper
292 appears to be limited to motile, chemotactic enteric bacteria, we propose that CheAs may play an impo
293 leotide sequences of envZ genes from several enteric bacteria were determined.
294 phore associated with increased virulence of enteric bacteria, were mapped within a pathogenicity isl
295 share homology with chemotaxis proteins from enteric bacteria, which are encoded in the frzA-F putati
296  this constraint is not shared by many other enteric bacteria, which can use either cysteine or methi
297            We show further that like H-NS in enteric bacteria, which functions as a transcriptional s
298  limit proliferation for ceftiofur resistant enteric bacteria while preserving the ability to use thi
299                             Dairy herds with enteric bacteria with known low tetracycline susceptibil
300              Genome-wide comparisons between enteric bacteria yield large sets of conserved putative

 
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