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1 al deficits and physiological changes to the enteric nervous system.
2 nd adaptive immunity, and dysfunction of the enteric nervous system.
3 HGF and MET are expressed in fetal and adult enteric nervous system.
4 n between the enteroendocrine system and the enteric nervous system.
5 e along with the drivers of their input, the enteric nervous system.
6       Little is known of how Tat affects the enteric nervous system.
7 ed during HIV infection are regulated by the enteric nervous system.
8 neuronal as well as glial progenitors in the enteric nervous system.
9  role of miRNAs in HFD-induced damage to the enteric nervous system.
10 motility disorders caused by diseases of the enteric nervous system.
11 eristalsis is significantly dependent on the enteric nervous system.
12 s show a significant increase in glia in the enteric nervous system.
13  (Hand2(+/-) mice) numbers of neurons in the enteric nervous system.
14  in contrast to the neurodegeneration in the enteric nervous system.
15 ural crest that colonize the gut to form the enteric nervous system.
16  role in the development and survival of the enteric nervous system.
17 eric neurons but also is latent in the human enteric nervous system.
18 es produced by enteroendocrine cells and the enteric nervous system.
19  neurite fasciculation necessary to form the enteric nervous system.
20 e most common congenital malformation of the enteric nervous system.
21 pin-releasing factor (CRF) in the guinea pig enteric nervous system.
22 nal development with a partial rescue of the enteric nervous system.
23 ory neurons (EP cells) within the developing enteric nervous system.
24 s of cells to the terminal bowel to form the enteric nervous system.
25 s a major inhibitory neurotransmitter in the enteric nervous system.
26  excitatory actions of CRF on neurons in the enteric nervous system.
27 n many inhibitory neurones of the vertebrate enteric nervous system.
28 ing factor (CRF) receptors in the guinea pig enteric nervous system.
29 noamine release in central neurons or in the enteric nervous system.
30 pes and glial cells present in the mammalian enteric nervous system.
31 ole of erbB2 in postnatal development of the enteric nervous system.
32 n myenteric and submucosal components of the enteric nervous system.
33 ing fast and slow synaptic excitation in the enteric nervous system.
34 ic and submucosal plexuses of the guinea pig enteric nervous system.
35 gically identified neurons in the guinea pig enteric nervous system.
36 ney organogenesis and the development of the enteric nervous system.
37 ng renal development and histogenesis of the enteric nervous system.
38 for neuronal replacement in disorders of the enteric nervous system.
39 iate into the neurons and glial cells of the enteric nervous system.
40 ty, suggesting that they may also affect the enteric nervous system.
41  neurotransmitter roles for NO and CO in the enteric nervous system.
42 l role in the development of the kidneys and enteric nervous system.
43 ecting the gut epithelium, immune system and enteric nervous system.
44 gulating the development and function of the enteric nervous system.
45 ng neurogenesis in the fully developed adult enteric nervous system.
46 odulating cholinergic neural reflexes in the enteric nervous system.
47 ant inhibitory motor neurotransmitter in the enteric nervous system.
48  inhibits cholinergic neural reflexes in the enteric nervous system.
49 nesyl-prelamin A are toxic to neurons of the enteric nervous system.
50 rs are localized to central, peripheral, and enteric nervous systems.
51 axonal number in the peripheral, central and enteric nervous systems.
52 hat which transports 5-HT in the central and enteric nervous systems.
53 eripheral, sympathetic, parasympathetic, and enteric nervous systems.
54 nce of cellular pathology in the central and enteric nervous systems.
55 differentiated neurons of the peripheral and enteric nervous systems.
56                                       In the enteric nervous system, 5-HITCA is detected without the
57 ow chest, and shortening of long bones), and enteric nervous system (aganglionosis).
58 t accumulation of 5-HITCA in the central and enteric nervous systems, along with data showing the deg
59  appear to be an intrinsic capability of the enteric nervous system and are not related to slow waves
60 inergic receptor subtype is expressed in the enteric nervous system and at intestinal neuromuscular j
61 the autonomic nervous systems, including the enteric nervous system and central autonomic network.
62 ion between these genes is restricted to the enteric nervous system and does not affect renal, coat c
63 pathway of adrenergic differentiation in the enteric nervous system and have defined a transient requ
64 ides a selective mechanism through which the enteric nervous system and innate immune system integrat
65 lionosis, is a developmental disorder of the enteric nervous system and is the most common cause of i
66 e involves an enterotoxin, activation of the enteric nervous system and malabsorption, suggesting tha
67 d carbon monoxide - that are produced by the enteric nervous system and share common molecular target
68 s displayed pathological changes in both the enteric nervous system and smooth muscle.
69                                     Both the enteric nervous system and the central nervous system ca
70 singly evident that interactions between the enteric nervous system and the immune system play an imp
71 des convey nutrient-regulated signals to the enteric nervous system and to distal organs, acting as c
72 he possible common origin of sympathetic and enteric nervous systems and provides new hope that we ma
73 e sensory, sympathetic, parasympathetic, and enteric nervous systems and the kidneys, as well as for
74  inhibitory nitrergic neural inputs from the enteric nervous system, and (iv) stretch receptors that
75  the immaturity of their intestinal barrier, enteric nervous system, and immune response to pathogens
76 unctions, the functional architecture of the enteric nervous system, and immune responses in the gut
77 ions between the central nervous system, the enteric nervous system, and the gastrointestinal tract.
78  the interactions among the vagus nerve, the enteric nervous system, and the intestinal epithelium ma
79 he genetic control of the development of the enteric nervous system, and the potential role of the in
80 st-derived components of the sympathetic and enteric nervous systems, and cardiac fibroblasts.
81 iation of progenitor cells in the developing enteric nervous system are controlled by molecules such
82 tent of the sacral crest contribution to the enteric nervous system are not well established in roden
83  neuronal subtypes during development of the enteric nervous system are poorly understood despite its
84 es a viral enterotoxin and activation of the enteric nervous system, as well as malabsorption, sugges
85 on sequence variants in genes related to the enteric nervous system, as well as with monogenic and ch
86 nding and isolating early progenitors of the enteric nervous system based on their ability to form di
87 ardenburg-Shah syndrome combines the reduced enteric nervous system characteristic of Hirschsprung's
88 Vagal neural crest-derived precursors of the enteric nervous system colonize the bowel by descending
89 is sequence also modulates expression in the enteric nervous system consistent with its proposed role
90                                          The enteric nervous system consists of two ganglionated neur
91 ition to ionotropic glutamate receptors, the enteric nervous system contains functional group I metab
92                         We conclude that the enteric nervous system contains intrinsic dopaminergic n
93                                          The enteric nervous system contains intrinsic primary affere
94                                          The enteric nervous system controls a variety of gastrointes
95                  The myenteric plexus of the enteric nervous system controls the movement of smooth m
96          The combination of pigmentation and enteric nervous system defects makes colourless mutation
97 ibution of strain background to variation in enteric nervous system deficits.
98 y recently to discuss the latest research on enteric nervous system development at a meeting organise
99 ver been implicated in stem cell function or enteric nervous system development.
100  developing neurons before the completion of enteric nervous system development.
101 uch that its loss leads to severe defects in enteric nervous system development.
102                                Later, during enteric nervous system differentiation, Meis3-depleted e
103                          Most neurons in the enteric nervous system discharge in response to distorti
104                             In contrast, the enteric nervous system displays little or no loss of cho
105 losion of interest in the development of the enteric nervous system driven by the need to understand
106 ation of immunity, changes that occur in the enteric nervous system during inflammation, the fundamen
107 nism by measuring colonization in hosts with enteric nervous system dysfunction due to a mutation in
108 cells (NCC) that ultimately give rise to the enteric nervous system (ENS) along the entire length of
109 Enteric glia are important components of the enteric nervous system (ENS) and also form an extensive
110                   These ganglia comprise the enteric nervous system (ENS) and are derived from migrat
111 uding the gastrointestinal tract to form the enteric nervous system (ENS) and hematopoietic organs (b
112      We developed a technique to image human enteric nervous system (ENS) and other intramural cells
113  the hypotheses that OT is endogenous to the enteric nervous system (ENS) and that OTR signaling may
114 ous system (CNS), there is evidence that the enteric nervous system (ENS) and the peripheral nervous
115     The majority of neurones and glia of the enteric nervous system (ENS) are derived from the vagal
116                                          The enteric nervous system (ENS) arises from the coordinated
117 t signaling is critical for formation of the enteric nervous system (ENS) because Ret activation prom
118 derived progenitors (ENPs) that generate the enteric nervous system (ENS) can lead to aganglionosis i
119 (HSCR) is a severe congenital anomaly of the enteric nervous system (ENS) characterized by functional
120                                          The enteric nervous system (ENS) comprises a complex neurona
121                                          The enteric nervous system (ENS) consists of neurons and gli
122                                          The enteric nervous system (ENS) controls the gastrointestin
123                                          The enteric nervous system (ENS) coordinates diverse functio
124                                          The enteric nervous system (ENS) coordinates essential intes
125  (5-HT) regulates central nervous system and enteric nervous system (ENS) development and long-term f
126 irschsprung disease is a serious disorder of enteric nervous system (ENS) development caused by the f
127                                              Enteric nervous system (ENS) development is governed by
128                                              Enteric nervous system (ENS) development is relevant to
129                                       Normal enteric nervous system (ENS) development relies on numer
130                                              Enteric nervous system (ENS) development requires comple
131 disease (HSCR), a multifactorial disorder of enteric nervous system (ENS) development, is associated
132 rify the role of Ret signaling components in enteric nervous system (ENS) development, we evaluated E
133 ital disorder, arising from abnormalities in enteric nervous system (ENS) development.
134                                          The enteric nervous system (ENS) develops from neural crest
135                                          The enteric nervous system (ENS) develops from neural crest
136                                          The enteric nervous system (ENS) exists in close proximity t
137                                          The enteric nervous system (ENS) forms from migrating neural
138                                          The enteric nervous system (ENS) forms from the neural crest
139 l gut function relies on the activity of the enteric nervous system (ENS) found within the wall of th
140                         The formation of the enteric nervous system (ENS) from neural crest-derived c
141  and A30P lines show robust abnormalities in enteric nervous system (ENS) function and synuclein-immu
142                          Although the mature enteric nervous system (ENS) has been shown to retain st
143  sodium channel (VGSC) alpha subunits in the enteric nervous system (ENS) has not been described.
144 lation of two main human tau isoforms in the enteric nervous system (ENS) in CD but not in UC.
145 ssessed the effects of TLR2 signaling on the enteric nervous system (ENS) in mice.
146 root ganglia to the autonomic ganglia of the enteric nervous system (ENS) in the colon.
147 stinal obstruction due to the absence of the enteric nervous system (ENS) in the distal bowel and is
148 se (HSCR) is characterized by absence of the enteric nervous system (ENS) in the distal bowel.
149                  During the formation of the enteric nervous system (ENS) in the moth Manduca, approx
150 iets, the consumers' gut microbiota, and the enteric nervous system (ENS) interact to regulate gut mo
151 uring the migration of the precursors of the enteric nervous system (ENS) into the colon.
152                                          The enteric nervous system (ENS) is a complex network consti
153                                          The enteric nervous system (ENS) is a major division of the
154 tanding the neurochemical composition of the enteric nervous system (ENS) is critical for elucidating
155                                          The enteric nervous system (ENS) is derived from vagal and s
156                                          The enteric nervous system (ENS) is derived from vagal and s
157 h the idea that phenotypic expression in the enteric nervous system (ENS) is determined, in part, by
158                                          The enteric nervous system (ENS) is essential for digestive
159                                          The enteric nervous system (ENS) is essential for normal gas
160                                          The enteric nervous system (ENS) is formed from vagal and sa
161                                          The enteric nervous system (ENS) is mainly derived from vaga
162                                          The enteric nervous system (ENS) is organized into neural ci
163 ronal development in vitro; nevertheless, an enteric nervous system (ENS) is present in mice lacking
164                                          The enteric nervous system (ENS) is sometimes called the "se
165                                          The enteric nervous system (ENS) is the largest branch of th
166                                          The enteric nervous system (ENS) is the largest component of
167 S: Neural stem and progenitor cells from the enteric nervous system (ENS) might serve as a source of
168 en have aggregated alpha-synuclein (aSyn) in enteric nervous system (ENS) neurons, which may be assoc
169 es in resident and inflammatory cells in the enteric nervous system (ENS) of macaques during the acut
170                                          The enteric nervous system (ENS) of the gastrointestinal tra
171                             In the embryonic enteric nervous system (ENS) of the moth Manduca sexta,
172                  During the formation of the enteric nervous system (ENS) of the moth Manduca sexta,
173 ocate calcium (Ca2+) channel proteins in the enteric nervous system (ENS) of the rat and guinea pig.
174                                 By using the enteric nervous system (ENS) of the tobacco hornworm Man
175 ce and identity of neural progenitors in the enteric nervous system (ENS) of vertebrates is a matter
176 rinsic denervation but play unknown roles in enteric nervous system (ENS) physiology.
177  complex equilibrium, here, we show that the enteric nervous system (ENS) plays an essential and non-
178 ant oligogenic birth defect that occurs when enteric nervous system (ENS) precursors fail to colonize
179                                              Enteric nervous system (ENS) precursors migrate extensiv
180                                              Enteric nervous system (ENS) precursors undergo a comple
181                                          The enteric nervous system (ENS) predominantly originates fr
182 tion of the bowel, results from a failure of enteric nervous system (ENS) progenitors to migrate, pro
183                                          The enteric nervous system (ENS) provides the intrinsic neur
184                       BACKGROUND & AIMS: The enteric nervous system (ENS) regulates gastrointestinal
185                                          The enteric nervous system (ENS) regulates numerous gastroin
186                                          The enteric nervous system (ENS) represents a vast network o
187                                          The enteric nervous system (ENS) senses and reacts to the dy
188             Advances in understanding of the enteric nervous system (ENS) support the brain-in-the-gu
189  Crohn's disease (CD) and the ability of the enteric nervous system (ENS) to produce PGD2 in inflamma
190               Disordered neurobiology of the enteric nervous system (ENS) underlies a broad assortmen
191 cation and differentiation in the developing enteric nervous system (ENS) was tested.
192  a balanced microbial community and that the enteric nervous system (ENS), a chief regulator of physi
193  array of functions, the bowel relies on the enteric nervous system (ENS), an intricate network of mo
194 estinal obstruction, striking defects in the enteric nervous system (ENS), and abnormal intestinal mo
195 rons are the major excitatory neurons of the enteric nervous system (ENS), and include intrinsic sens
196  function is controlled by its own intrinsic enteric nervous system (ENS), but it is additionally reg
197  of axonal tract configuration in the mature enteric nervous system (ENS), but profound abnormalities
198 ty alterations maybe due to an effect on the enteric nervous system (ENS), but the underlying mechani
199 ointestinal tract, and more specifically the enteric nervous system (ENS), in search of an early biom
200                                The zebrafish enteric nervous system (ENS), like those of all other ve
201 e leads to degeneration and autophagy in the enteric nervous system (ENS), particularly in the submuc
202 the neural crest, impacts development of the enteric nervous system (ENS), possibly by regulating the
203                                          The enteric nervous system (ENS), the intrinsic innervation
204 ium signaling plays an essential role in the enteric nervous system (ENS), the role of CaMKII in neur
205  pathway in the development of the mammalian enteric nervous system (ENS), very little is known regar
206 ointestinal physiology are controlled by the enteric nervous system (ENS), which is composed of neuro
207 long the gut to promote the formation of the enteric nervous system (ENS).
208 tivity is integral to the development of the enteric nervous system (ENS).
209 plored for the treatment of disorders of the enteric nervous system (ENS).
210 r neurogenesis occurs in the adult mammalian enteric nervous system (ENS).
211 in (EC) cells and a smaller 5-HT pool in the enteric nervous system (ENS).
212  their cell bodies are not components of the enteric nervous system (ENS).
213 anized network of ganglia that comprises the enteric nervous system (ENS).
214 icrobes, marketed as probiotics, affects the enteric nervous system (ENS).
215 he entire gastrointestinal tract to form the enteric nervous system (ENS).
216  neural crest cells (ENCCs) to establish the enteric nervous system (ENS).
217 nt roles in the development of the mammalian enteric nervous system (ENS).
218 od flow are controlled and integrated by the enteric nervous system (ENS).
219 e rise to the neurons and glial cells of the enteric nervous system (ENS).
220 s determined by integrative functions of the enteric nervous system (ENS).
221  the neurobiology of P2X(7) receptors in the enteric nervous system (ENS).
222 glia that span its entire length, called the enteric nervous system (ENS).
223 ression of Sema3a, Sema3c, and Sema3d in the enteric nervous system (ENS).
224 ral control of gut functions mediated by the enteric nervous system (ENS).
225 n essential mode of neurotransmission in the enteric nervous system (ENS).
226 ortant in the functional neurobiology of the enteric nervous system (ENS); nevertheless, details for
227 natide reduces food intake and activates the enteric nervous system (ENS; myenteric and submucosal pl
228 ord primary motor neurons (PMN), kidney, and enteric nervous systems (ENS) and have identified areas
229 , have been identified and several mammalian enteric nervous systems express CB1 receptors and produc
230 kinase RET, which is essential for mammalian enteric nervous system formation.
231 effect of this Gaucher mutation on motor and enteric nervous system function in these transgenic anim
232 otential of neural crest stem cells from the enteric nervous system (gut NCSCs) in vivo to evaluate t
233                                          The enteric nervous system has been studied thus far as an i
234                                          The enteric nervous system has many neuronal subtypes that c
235  (EGCs), the major cellular component of the enteric nervous system, have long been considered mere s
236                       These defects included enteric nervous system hypoplasia, slow GI transit, dimi
237 l neural crest contributed precursors to the enteric nervous system in a regionalised manner.
238 and provide a paradigm for understanding the enteric nervous system in health and disease.
239 pts migration of NCCs and development of the enteric nervous system in mice.
240 nteractions can influence development of the enteric nervous system in mouse models and suggests that
241 emonstrating the intimate involvement of the enteric nervous system in mucosal immunity.
242           Here we examine development of the enteric nervous system in the basal jawless vertebrate t
243  sacral neural crest cells contribute to the enteric nervous system in the hindgut.
244 iving cholinergic excitatory inputs from the enteric nervous system in the murine fundus.
245  studies to describe the regeneration of the enteric nervous system in the sea cucumber Holothuria gl
246 stal ileum, confined to follicles and/or the enteric nervous system, in almost all animals.
247 isceral afferents (sensory) pathways and the enteric nervous system, including the interstitial cells
248  progenitor function in the dentate gyrus or enteric nervous system, indicating regional differences
249                                          The enteric nervous system integrates secretion and motility
250 ory detection in these processes, disordered enteric nervous system integration in diarrhea and const
251 es reveals that, in the absence of Pax3, the enteric nervous system is ablated from its inception.
252 flecting the current interest in the way the enteric nervous system is altered in disease and the sec
253 flecting the current interest in the way the enteric nervous system is altered in disease.
254                       The development of the enteric nervous system is dependent upon the actions of
255 ibution of sacral crest-derived cells to the enteric nervous system is not affected by cocolonization
256 the requirement for erbB2 in maintaining the enteric nervous system is not cell autonomous, but rathe
257                                          The enteric nervous system is plastic and continues to under
258 tor, free fatty acid receptor (FFA)3, to the enteric nervous system is unknown.
259           Adenosine receptors (ADORs) in the enteric nervous system may be of importance in the contr
260         Thus, differential engagement of the enteric nervous system may partake in bifurcating pro- o
261          Abnormalities in development of the enteric nervous system might therefore contribute to the
262 eural crest cells fated to contribute to the enteric nervous system migrate ventrally away from the n
263                                              Enteric nervous system neuropathy causes a wide range of
264                                      BBS, an enteric nervous system neuropeptide, reverses PN-induced
265                                       In the enteric nervous system, neurotransmitters initiate chang
266 major changes in the smooth muscle layers or enteric nervous system occurred in the absence of these
267  processes of enteric glial cells within the enteric nervous system of CWD-infected Tg(CerPrP-E) mice
268 ade an important contribution to the ancient enteric nervous system of early jawless vertebrates, a r
269                                          The enteric nervous system of jawed vertebrates arises prima
270 ons of both brain stem emetic nuclei and the enteric nervous system of the gut; and 4) whether select
271                            In the developing enteric nervous system of the moth Manduca sexta, an ide
272        This study provides evidence that the enteric nervous system of this echinoderm regenerates af
273 r and neuromodulator in both the central and enteric nervous systems of mammals.
274  in responses to endogenous ligands from the enteric nervous system or dietary sources.
275 be selectively ablated by ganciclovir in the enteric nervous system, or in the injured forebrain or s
276                 We provide evidence that the enteric nervous system organizes mixing movements to gen
277 l neural crest to interrupt the migration of enteric nervous system precursor cells and thus create a
278 e expressed very early in the development of enteric nervous system precursors, and are already prese
279 und that control of differentiation of mouse enteric nervous system progenitor cells by EDN3 requires
280                                              Enteric nervous system progenitor cells may therefore po
281 cial role in the maintenance of multilineage enteric nervous system progenitors.
282 l derived neurotrophic factor (GDNF) induces enteric nervous system regeneration in mouse models of H
283                                          The enteric nervous system regulates these events, and Mulle
284                         Motor neurons in the enteric nervous system release ATP as an inhibitory neur
285            Nitric oxide (NO) produced by the enteric nervous system represents an important regulator
286  recent advances in our understanding of the enteric nervous system, sensory physiology underlying pa
287                           In the central and enteric nervous systems, SERT is located in serotonergic
288      We describe neurochemical coding of the enteric nervous system, specifically the myenteric plexu
289 ating GDNF and ET-3 in the patterning of the enteric nervous system, suggest that specific pairing of
290 rity of neurons and glia that constitute the enteric nervous system, the intrinsic innervation of the
291 tite and energy balance by engagement of the enteric nervous system through cannabinoid receptors.
292                In 5-HT-incubated central and enteric nervous system tissue samples and differentiated
293 terface by demonstrating the capacity of the enteric nervous system to influence the microbiota.
294                                          The enteric nervous system was not affected.
295                           Development of the enteric nervous system was studied in Sufu(f/f), Gli3(De
296 year's issue on developmental aspects of the enteric nervous system, we have focused on several key t
297 nal progenitors capable of reconstituting an enteric nervous system when transplanted into a normal d
298             It is mediated by neurons of the enteric nervous system, which form an integrated circuit
299 fore alter innervation and morphology of the enteric nervous system, which may contribute to colonic
300 hesized that Lewy pathology initiates in the enteric nervous system years prior to debut of clinical

 
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