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1 ial cell-wide response to copper toxicity in Enterobacteria.
2 , clinically and biotechnologically relevant enterobacteria.
3  kinase PhoQ and is widely distributed among enterobacteria.
4 uence and positioning is highly conserved in enterobacteria.
5 mbrane and elongating outside the cell as in enterobacteria.
6 n of the atlas that is depleted in high-risk enterobacteria.
7 PS) to elicit antibodies cross-reactive with enterobacteria.
8  of unknown function in Salmonella and other enterobacteria.
9  of the emergence of carbapenemase-producing enterobacteria.
10 he anaerobic fermentation of glucose by many enterobacteria.
11 e T4aP structure, function and biogenesis in enterobacteria.
12 he nature of the interaction is conserved in enterobacteria.
13 riptional regulation of purine metabolism in enterobacteria.
14 it that this regulation also occurs in other enterobacteria.
15 ion against ACA in tmRNAs was seen mostly in enterobacteria.
16 nated by Rho factor, an essential protein in enterobacteria.
17 stribution in pathogenic and commensal human enterobacteria.
18 as a c-di-GMP receptor affecting motility in enterobacteria.
19 olutionary divergence from animal-pathogenic enterobacteria.
20 ci, staphylococci, lactobacilli, yeasts, and enterobacteria.
21 on on the coding strand has been observed in enterobacteria.
22 xposed to either pathogenic or nonpathogenic enterobacteria.
23 linked arthritides follows an infection with enterobacteria.
24 , 4.5-log for yeasts and molds and 2-log for enterobacteria after 20 d of storage.
25             Diverse elements use ssrA; among enterobacteria alone, at least four different integrase
26 demonstrate an antagonistic correlation with enterobacteria and enterococci.
27  concatenation of the core set of TCSTs from enterobacteria and for individual TCST proteins from spe
28        The other group, mostly restricted to Enterobacteria and including Escherichia coli pheL, has
29 that occur in a conserved genomic context in Enterobacteria and is essential in the infection process
30 ible for intracellular formate generation in enterobacteria and other microbes, interacts specificall
31 cquired antibiotic resistance genes found in enterobacteria and pseudomonads are part of small mobile
32  glutamine amidotransferase-QueC homologs in Enterobacteria and Pseudomonas phage, and distant homolo
33 e determinants, its carriage in a variety of enterobacteria, and its presence in both nosocomial and
34 nserved across its entire length in numerous Enterobacteria, and mutational analysis revealed that tw
35           Total viable counts, pseudomonads, enterobacteria, and specific fish spoilers were reduced
36 otal anaerobes, aerobes, bifidobacteria, and enterobacteria, and to assay for beta-glucuronidase, nit
37                                Escherichia-, Enterobacteria-, and Enterococcus phages were over-repre
38                           Type 1 fimbriae of enterobacteria are heteropolymeric organelles of adhesio
39    The core bacteria include Pseudomonas and enterobacteria, both are shared in the sand flies in the
40 as found to be dominated by Lactobacilli and Enterobacteria, both typically facultative anaerobes.
41                       QseD is present in all enterobacteria but exists almost exclusively in O157:H7
42 jugated antibiotic that specifically targets enterobacteria by exploiting active import through oligo
43 ys show that processing of the model Ag from enterobacteria by mast cells is similar in efficiency to
44  close relatives all infect a broad range of enterobacteria by recognizing a plasmid-encoded conjugal
45                              Like Dam in the enterobacteria, CcrM plays a regulatory role in Caulobac
46               We tested a hypothesis that in enterobacteria CheV functions as an additional adaptor l
47 h whole genome sequence information from 250 enterobacteria clones to characterize the dissemination
48 ovora subsp. carotovora, like those of other enterobacteria, consists of flhD and flhC.
49   Comparative genomic analysis revealed that enterobacteria contain eight pairs of core TCSTs.
50 pportunistic infections with FimH-expressing enterobacteria could occur in a setting deprived of opso
51 Infections caused by carbapenemase-producing enterobacteria (CPE) are a major concern in clinical set
52 gmoidoscopy scores (SS) were associated with enterobacteria, desulfovibrios, type E Clostridium perfr
53 hey promote infection by the phytopathogenic enterobacteria Dickeya dadantii and Erwinia amylovora.
54                      Like the cells of other enterobacteria, E. coli cells acquire beta-lactam resist
55                                           In Enterobacteria, envelope stress caused by unfolded OMPs
56 ly (OMP) were consistently identified in the Enterobacteria Escherichia coli, Enterobacter cloacae, E
57                                              Enterobacteria, especially Escherichia coli, are abundan
58 Blochmannia pairs, plus Buchnera and related enterobacteria, estimates of sequence divergence at four
59  temperate double-stranded (ds) phages, like enterobacteria, exhibit significantly high relative abun
60                                   Pathogenic enterobacteria face various oxygen (O(2)) levels during
61          During flagellum assembly by motile enterobacteria, flagellar axial proteins destined for po
62 unctionally uncharacterized, suggesting that enterobacteria harbor a pool of evolutionarily young, de
63 r of carbapenemase-encoding plasmids between enterobacteria in the gut microbiota of individual patie
64  lactobacilli) and increase the abundance of enterobacteria including entropathogenic Escherichia col
65 essing occurs from a number of Gram-negative enterobacteria including Salmonella typhimurium and Esch
66 gen is an exopolysaccharide produced by many enterobacteria, including the majority of Escherichia co
67 a condition often associated with virulence, enterobacteria inhibit the ClpXP-dependent proteolysis o
68 toxins are PIN domain endonucleases that, in enterobacteria, inhibit translation by site-specific cle
69                                Speciation in enterobacteria involved horizontal gene transfer.
70 eir biogenesis in Escherichia coli and other enterobacteria involves two machineries: Isc and Suf.
71  can bloom in the inflamed gut; expansion of enterobacteria is a hallmark of microbial imbalance know
72      It appears that cellulose production in enterobacteria is controlled by a two-tiered c-di-GMP-de
73              Respiratory enzyme synthesis in enterobacteria is controlled in response to electron acc
74 understanding how the virulence phenotype in enterobacteria is expressed and regulated.
75 he phage shock protein (Psp) system found in enterobacteria is induced in response to impaired inner
76 ssembly of CoA in Escherichia coli and other enterobacteria is well understood, except for the events
77  Since the alpha gal epitope is found on gut enterobacteria, it has been hypothesized that anti-gal a
78  operate on the chromosomal fis genes of the enterobacteria Klebsiella pneumoniae, Serratia marcescen
79                                           In enterobacteria like Salmonella, biogenesis of cell surfa
80 imited contribution to the noncore genome of enterobacteria, likely due to the limitations on the siz
81 s have elucidated aspects of Lpt function in enterobacteria, little is known about how this system op
82 ersification of Cu-resistance systems within enterobacteria, little research has focused on how this
83 a dominance was inversely related to that of Enterobacteria (mainly Thorsellia and Serratia).
84 relatively conserved, and suggest that these enterobacteria may have maintained their ancient core TC
85       Escherichelin production by colonizing enterobacteria may help human hosts resist opportunistic
86                             Contrasting with Enterobacteria, members of the dominant genus Bacteroide
87                                   Pathogenic enterobacteria need to survive the extreme acidity of th
88 d chemical analysis in four plant-associated enterobacteria of the Serratia and Dickeya genera.
89 ved in c-di-GMP synthesis and degradation in enterobacteria, only a handful of c-di-GMP receptors/eff
90                 In this setting, E. coli and Enterobacteria orbit around the dispersed particles.
91 hese OMP peptides observed are unique to the Enterobacteria order.
92 NAs, was recently detected in genomic DNA of Enterobacteria phage 9g and was proposed to protect phag
93                                              Enterobacteria phage P1 expresses two types of tail fibr
94 ate Tetrahymena thermophila, and the viruses Enterobacteria phage Rb49 and Bacteriophage Felix 01.
95 ovel sister clade to the Microvirus genus of Enterobacteria phages.
96                              Some strains of enterobacteria possess a mrkD1C allele that is associate
97                           Many gram-negative enterobacteria produce surface-associated fimbriae that
98 tructure analysis of the S10 leaders of five enterobacteria (Salmonella typhimurium, Citrobacter freu
99 iocontrol and phytopathogenic strains of the enterobacteria, Serratia and Dickeya.
100 a to cefiderocol killing, pyoverdine and the enterobacteria siderophore enterobactin displaced iron f
101 nt high levels of proteobacteria, especially enterobacteria species including E. coli, observed in cl
102 artii and E. rhapontici, as well as in other enterobacteria such as Escherichia coli, Salmonella ente
103                                           In enterobacteria such as Escherichia coli, the general str
104                                     All four enterobacteria tested produce tau and gamma homologs.
105 ere up to 20-fold less efficient in clearing enterobacteria than control WBB6F1 +/+ (+/+) mice or mas
106                Shigella species are invasive enterobacteria that cause dysentery, a severe form of di
107                              Salmonellae are enterobacteria that have the unique ability to change th
108 m plays an essential role in the response of enterobacteria to the environment of their mammalian hos
109                                           In enterobacteria, under non-stress conditions, PspA as a l
110 or the rapid classification of Gram-negative Enterobacteria using on-slide solubilization and trypsin
111 a-Proteobacteria, for example, in genomes of Enterobacteria, Vibrio, and Halomonas species, and in ty
112 of S. typhimurium LT2 genes in eight related enterobacteria was determined using previously completed
113 ells have been shown to phagocytose and kill enterobacteria, we wished to determine whether they coul
114                            Total aerobes and enterobacteria were less affected by diet and neosugar.
115 cated in the repression state of AmpR in the enterobacteria, were also shown to play a structural rol
116  sadA; orthologous operons are only found in enterobacteria, whereas other TAAs are not typically ass
117 hE belongs to the flhBAE flagellar operon in Enterobacteria, whose first two members function in Type
118 on genomic comparisons between Eca and other enterobacteria, with particular emphasis on the differen
119 tained for its role in growth homeostasis of Enterobacteria within the gut environment, thereby allow

 
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