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1 ve role for Clostridiales while negative for Enterobacteriales.
2 were Desulfovibrionales, Pseudomonadales and Enterobacteriales.
3 ement of the Cra regulator characteristic of Enterobacteriales.
4            Eighty-five isolates of the order Enterobacteriales (12 mcr positive) were tested by CBDE
5  or TTSS-secreted proteins, are required for enterobacterial aggregative multicellular behavior.
6 developed genetics of E. coli by integrating enterobacterial ampRC genes into the E. coli chromosome.
7 (IL-1beta) level; the relative abundances of Enterobacteriales and Enterobacteriaceae and the interfe
8 relations between the relative abundances of Enterobacteriales and Enterobacteriaceae and the sCD14 l
9  relative abundances of Gammaproteobacteria, Enterobacteriales, and Enterobacteriaceae and the interl
10 in-resistant E.faecium, carbapenem-resistant Enterobacteriales, and extended-spectrum beta-lactamase-
11                                              Enterobacterial animal pathogens exhibit aggregative mul
12 igh homology to previously reported ViI-like enterobacterial bacteriophage genomes.
13 sly constructed an assay system for studying enterobacterial beta-lactam resistance mutations using t
14 rial amyloids, are an important component of enterobacterial biofilms.
15  responses in the gut can generate transient enterobacterial blooms in which conjugative transfer occ
16 tics to inhibit enteric pathogens and reduce enterobacterial blooms.
17 em is a phosphorelay responsible for sensing enterobacterial cell envelope stresses.
18 propose a model for LdcI function inside the enterobacterial cell, providing a structural and mechani
19 e, we assess the impact of a time-restricted enterobacterial challenge to long-term ILC3 activation i
20             The mechanism of sugar uptake by enterobacterial channels, such as Escherichia coli LamB
21 igosaccharides preferred by LamB and related enterobacterial channels.
22                                  We targeted enterobacterial coliphages because they are better than
23 tification of a water-soluble cyclic form of enterobacterial common antigen (ECA(CYC)) from Escherich
24                      Phosphoglyceride-linked enterobacterial common antigen (ECA(PG)) is a cell surfa
25 embly of the phosphoglyceride-linked form of enterobacterial common antigen (ECA(PG)) occurs by a mec
26 biosynthesis of two surface polysaccharides: enterobacterial common antigen (ECA) and a high-molecula
27                 The polysaccharide chains of enterobacterial common antigen (ECA) are comprised of th
28 tools revealed that rffH, a gene involved in enterobacterial common antigen (ECA) biosynthesis, is pa
29                                              Enterobacterial common antigen (ECA) is expressed by Gra
30 ese loci is responsible for synthesis of the enterobacterial common antigen (ECA), a glycolipid situa
31 otransferase involved in the biosynthesis of enterobacterial common antigen (ECA), a non-essential ou
32        Here, we characterize the role of the enterobacterial common antigen (ECA), a surface glycolip
33 ogues of genes required for the synthesis of enterobacterial common antigen (ECA), suggesting that H.
34 cteriaceae express a polysaccharide known as enterobacterial common antigen (ECA), which is an attrac
35 cose and its derivatives, used to synthesize enterobacterial common antigen (ECA).
36 accharide repeat unit in the biosynthesis of enterobacterial common antigen (ECA).
37 d A and complete LOS core (galU), as well as enterobacterial common antigen (wecB and wecC), is impor
38 acid, lipopolysaccharide, peptidoglycan, and enterobacterial common antigen biosynthesis in Proteobac
39 en with the use of MAB-T88 in the bacteremic enterobacterial common antigen group (p <.05).
40 tive sepsis or in those patients with proven enterobacterial common antigen infections.
41 in wzyE, encoding an enzyme that polymerizes enterobacterial common antigen, a surface polysaccharide
42 ibits growth in bile only in the presence of enterobacterial common antigen, an outer-membrane glycol
43 occurrence of a water-soluble cyclic form of enterobacterial common antigen, ECA(CYC), purified from
44 n the biosynthesis of lipopolysaccharide and enterobacterial common antigen.
45 n insertion in wecE was unable to synthesize enterobacterial common antigen.
46 luding group I capsule, group II capsule and enterobacterial common antigen; (iii) genes involved in
47 he type 5 capsule biosynthetic locus restore enterobacterial common-antigen expression to Escherichia
48 -1 fimbrial subunit, FimH, was the necessary enterobacterial component for mast-cell activation and n
49 egion differs substantially from the typical enterobacterial cores.
50 uences occurs at the attachment site of each enterobacterial element, apparently serving as a transcr
51 ance of Proteobacteria, Gammaproteobacteria, Enterobacteriales, Enterobacteriaceae, Erysipelotrichi,
52 a that differences in structural features in enterobacterial FlhD are responsible for differential su
53 n DNA condensation and is a key regulator of enterobacterial gene expression in response to changes i
54 B porin was transferred into three different enterobacterial genera.
55         Mauve has been applied to align nine enterobacterial genomes and to determine global rearrang
56 ion as well as those of additional published enterobacterial genomes is underway and will be publicly
57     The EnteriX suite currently includes >15 enterobacterial genomes, generates views centered on fou
58 er 300 vertically inherited prophages within enterobacterial genomes.
59  A backbone, usually present in highly toxic enterobacterial Gram-negative lipid A.
60 haperones, facilitating incorporation of the enterobacterial hook-associated axial proteins (HAPs) Fl
61             Highest mortality was found with Enterobacteriales in non-Fournier NF.
62  of Desulfovibrionales, Pseudomonadales, and Enterobacteriales in produced water globally.
63 NF-alpha) to recruit neutrophils to sites of enterobacterial infection.
64                          Chlamydial and many enterobacterial infections can trigger reactive arthriti
65 idely distributed among Salmonella and other enterobacterial isolates from agricultural sources and h
66 onses to Pseudomonas and Xanthomonas but not enterobacterial lipid A or lipopolysaccharide preparatio
67  glucosamine disaccharide characteristic for enterobacterial lipid A was replaced by a 2,3-diamino-2,
68  --> 6)-glucosamine disaccharide, typical of enterobacterial lipid A.
69        Exposure of mononuclear phagocytes to enterobacterial LPS induces a state of transient hypores
70                                              Enterobacterial LPS is recognized by the TLR4 signaling
71  activity was established: IL-6 induction by enterobacterial LPS was inhibited by cylindrically shape
72 demonstrate that in contrast to protein-free enterobacterial LPS, a similarly purified preparation of
73 ta support the conclusion that TLR4 mediates enterobacterial LPS-induced HIV transcription via IL-1 s
74 surface receptor and molecular mechanisms of enterobacterial LPS-induced HIV transcription.
75 to differ structurally and functionally from enterobacterial LPS.
76 mune subsets typically depleted by canonical enterobacterial LPSs.
77                                              Enterobacterial MlaA proteins form stable complexes with
78 he genes are designated mntH because the two enterobacterial NRAMPs encode H+-stimulated, highly sele
79 4 signaling complex, whereas LPS of some non-enterobacterial organisms is capable of signaling indepe
80 ococci declined (P = 0.041), whereas that of Enterobacteriales (P = 0.005), particularly Escherichia
81  in contrast to the 2 position seen with the enterobacterial PagP.
82 cterize a DyP-containing encapsulin from the enterobacterial pathogen Klebsiella pneumoniae.
83 d TCSTs in Erwinia amylovora, a severe plant enterobacterial pathogen, at genome-wide level.
84 ugment and curate annotations for genomes of enterobacterial pathogens and for additional genome sequ
85                                  Most of the enterobacterial pathogens encode at least one T3SS, a ma
86                                      In some enterobacterial pathogens, but not in Escherichia coli,
87 chment protein of Escherichia coli and other enterobacterial pathogens.
88 prevalence of resistance among opportunistic enterobacterial pathogens.
89                                          The enterobacterial phytopathogen Erwinia amylovora causes f
90                         The ardA gene of the enterobacterial plasmid CollbP-9 acts to alleviate restr
91 which is closely related to the genes in the enterobacterial plasmid R64.
92                   The multiple activities of enterobacterial RecBCD helicase-nuclease are coordinated
93 atory ability of this procedure with that of enterobacterial repeat intergenic consensus (ERIC2) PCR,
94 wo Polymerase Chain Reaction (PCR) analyses: Enterobacterial Repetitive Intergenic Consensus (ERIC) a
95 ined by polymerase chain reaction (PCR) with enterobacterial repetitive intergenic consensus (ERIC) p
96  phylogenetic groups was further assessed by enterobacterial repetitive intergenic consensus (ERIC) t
97        Clonal relatedness was assessed using enterobacterial repetitive intergenic consensus (ERIC)-P
98  larvae, which largely confirms the previous enterobacterial repetitive intergenic consensus (ERIC)-p
99 ection of Escherichia coli isolates typed by enterobacterial repetitive intergenic consensus (ERIC)-P
100  by pulsed-field gel electrophoresis (PFGE), enterobacterial repetitive intergenic consensus (ERIC)-P
101 tis were compared by DNA fingerprinting with enterobacterial repetitive intergenic consensus primers.
102 al and clinical isolates were genotyped with enterobacterial repetitive intergenic consensus sequence
103 lsed-field gel electrophoresis (PFGE) and/or enterobacterial repetitive intergenic consensus sequence
104 ss discriminating than MLST, ribotyping, and enterobacterial repetitive intergenic consensus sequence
105                Genetic profiling was done by enterobacterial repetitive intergenic consensus sequence
106 isruptor whose omission helped stabilize the Enterobacteriales root.
107     The incidence of ciprofloxacin-resistant Enterobacteriales showed a significant increasing trend
108 t grows under iron-limiting conditions using enterobacterial siderophores.
109 ance plasmid pOXA-48 can use a wide range of enterobacterial species as hosts, but it is usually asso
110  to modulate the tissue tropism of different enterobacterial species represents a novel function for
111 ison of rsmB sequences from several of these enterobacterial species revealed a highly conserved 34-m
112   The phylogenetic relationships of multiple enterobacterial species were reconstructed based on 16S
113 assettes into arbitrary genomic loci of four enterobacterial species with an efficiency of 50-100%.
114 xed and user-supplied sequences from related enterobacterial species, anchored on a reference genome.
115 a collection of 17 SPI-7 related ICEs within enterobacterial species, of which six are novel.
116 enomic analysis of TCSTs in 53 genomes of 16 enterobacterial species.
117 n of fliA and motility varies depending upon enterobacterial species.
118 gation dynamics in a collection of wild-type enterobacterial strains isolated from hospitalized patie
119 ges' host range across a panel of pathogenic enterobacterial strains.
120  important step in the evolution of virulent enterobacterial strains.
121 he local extracellular environment to ensure enterobacterial survival at low pH.
122 a and their phages, and (3) dCTP/dUTPases in enterobacterial T4-like phages.
123  distinct binding specificities of different enterobacterial type 1 fimbriae cannot be ascribed solel
124  in the scale (from 1 to 2 target operons in Enterobacteriales up to 20 operons in Aeromonadales) and
125 d extended-spectrum beta-lactamase-producing Enterobacteriales were recently observed.
126 Instability was observed for the root of the Enterobacteriales, with nearly equal subsets of the prot

 
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