ライフサイエンスコーパス: enterochromaffin cell

1 icate goblet cells, whereas others implicate enterochromaffin cells).

2 viously unreported population that resembles enterochromaffin cells.

3 e adult stage in a subset of enteroendocrine/enterochromaffin cells.

4 by 5-hydroxytryptamine (5-HT) released from enterochromaffin cells.

5 g cells are identified as a subpopulation of enterochromaffin cells.

6 is both necessary and sufficient to generate enterochromaffin cells.

7 isthmus cells and uncover a role for Sox2 in enterochromaffin cells.

8 ses and an unexpected close association with enterochromaffin cells.

9 in neuroendocrine, including chromaffin and enterochromaffin, cells.

10 onstrated that 5-HT released from intestinal enterochromaffin cells activates 5-HT3 receptors on vaga

11 5-Hydroxytryptamine (5-HT) released from enterochromaffin cells activates secretory and peristalt

12 5-Hydroxytryptamine (5-HT) is released from enterochromaffin cells and activates neural reflex progr

13 e of 5-hydroxytryptamine (5-HT) from mucosal enterochromaffin cells and activation of 5-HT(3) recepto

14 e transcription factor Lmx1a is expressed in enterochromaffin cells and functions downstream of Nkx2.

15 Serotonin is also released from enterochromaffin cells and inflammatory/immune cells to

16 sis, consistent with localization of TGR5 to enterochromaffin cells and intrinsic primary afferent ne

17 med FFA2 and FFA3, are expressed in duodenal enterochromaffin cells and L cells, respectively.

18 of the cells expressed SOX9 (duct), SLC18A1 (enterochromaffin cells), and CDX2 (gut cells), respectiv

19 ivity was abundant in both enteric plexuses, enterochromaffin cells, and pancreatic ganglia.

20 release serotonin (5-hydroxytryptamine) from enterochromaffin cells, and stretch reflexes that determ

21 ceptor OR51E1 was notably expressed in ileal enterochromaffin cells, and the glucose-sensing sodium g

22 vious results showing guanylin expression in enterochromaffin cells appear to be a consequence of ant

23 These findings imply a role for enterochromaffin cells as "glucose sensors" during inges

24 Gut enterochromaffin cells can be converted to insulin-produ

25 We show that the number of mucosal enterochromaffin cells containing 5-HT changes with the

26 Paracrine signaling by enterochromaffin cells (EC), which release 5-HT, has not

27 of serotonin signaling in a newly developed enterochromaffin cell (ECC)-enriched gut organoid model.

28 re IECs that we identified to be a subset of enterochromaffin cells (ECC).

29 Serotonin production by enterochromaffin cells (ECs) is microbiota-dependent, bu

30 biota promote 5-HT biosynthesis from colonic enterochromaffin cells (ECs), which supply 5-HT to the m

31 tion to neurons, a subset of 5-HT-containing enterochromaffin cells expressed 5-HT1A immunoreactivity

32 le cells in the gut, including immune cells, enterochromaffin cells, gut microbiota and the derived l

33 Mucosal enterochromaffin cells have been postulated to be pressu

34 iven by ammoniagenesis in the gut, sensed by enterochromaffin cells in a TRPA1-dependent fashion and

35 onsistent with the hypothesis that 5-HT from enterochromaffin cells in response to mucosal stimuli in

36 neurons were activated by 5-HT release from enterochromaffin cells in the mucosa.

37 5-HT released from enterochromaffin cells initiates peristaltic, secretory,

38 Expression of uroguanylin in enterochromaffin cells is consistent with the hypothesis

39 5HT produced by enterochromaffin cells is critical in motility and secre

40 g kg-1) indicating that endogenous 5-HT from enterochromaffin cells is not essential for transduction

41 erotonin (5-HT), which abounds in intestinal enterochromaffin cells, is released in response to vario

42 e populations, and identify Lmx1a as a novel enterochromaffin cell marker that is also essential for

43 human carcinoid BON cells, an in vitro human enterochromaffin cell model, to understand the mechanism

44 athematical modeling, we show that, in human enterochromaffin cells, Myrip (1) inhibits a class of SG

45 imiting biosynthetic enzyme for serotonin in enterochromaffin cells of the duodenum.

46 the central nervous system (CNS) as well as enterochromaffin cells of the gastrointestinal tract.

47 a decrease in the synthesis of serotonin by enterochromaffin cells of the gut.

48 eptor FFAR2 was highest in ileal L-cells and enterochromaffin cells, olfactory receptor OR51E1 was no

49 Factors that control secretion of 5-HT from enterochromaffin cells or BON cells are important as par

50 , it is unclear whether they act directly on enterochromaffin cells or indirectly through an intermed

51 The enterochromaffin cell population was decreased in severe

52 phils, platelets, histaminergic neurons, and enterochromaffin cells produce varying amounts of histam

53 er RNA, encoding the rate-limiting enzyme in enterochromaffin cell serotonin (5-hydroxytryptamine [5H

54 Most RET(+) epithelial cells were either enterochromaffin cells that release serotonin or L-cells

55 mulated serotonin biosynthesis in intestinal enterochromaffin cells via a mechanism involving activat

56 kx2.2 mutant conditions, serotonin-producing enterochromaffin cells were the most severely reduced en

57 Human BON cells, derived from enterochromaffin cells, were treated with D-glucose, gal

58 release of endogenous 5-HT from the mucosal enterochromaffin cells, which acts on the 5-HT3 receptor