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1 ia, severe cerebral lesions, and necrotizing enterocolitis).
2 icular leukomalacia >grade 1, or necrotizing enterocolitis).
3  164 unaffected controls (no LOS/necrotizing enterocolitis).
4 intestinal pathologies including necrotizing enterocolitis.
5 criteria for Bell's stage 2 or 3 necrotising enterocolitis.
6 t typhoid fever, there are none that prevent enterocolitis.
7 erial burden during L. monocytogenes-induced enterocolitis.
8 ricular hemorrhage, and death or necrotizing enterocolitis.
9 ants who did and did not develop necrotizing enterocolitis.
10 mon to all infants who developed necrotizing enterocolitis.
11 e, norovirus, cytomegalovirus, and bacterial enterocolitis.
12 s that may lead to diseases like necrotizing enterocolitis.
13 is restricted to APCs, in the development of enterocolitis.
14 tract, or cerebrospinal fluid or necrotizing enterocolitis.
15 et inflammatory bowel disease with apoptotic enterocolitis.
16 oundly depleted in newborns with necrotizing enterocolitis.
17 es and T cells that lead to severe apoptotic enterocolitis.
18 ouse homologs of CXCL8 in the early phase of enterocolitis.
19 actice reduces the prevalence of necrotizing enterocolitis.
20 owards a diminished incidence of necrotizing enterocolitis.
21 she was diagnosed with allergic eosinophilic enterocolitis.
22 and host immune elements such as necrotizing enterocolitis.
23 regulation with life-threatening early-onset enterocolitis.
24 ntributes to the pathogenesis of necrotizing enterocolitis.
25 ch as the transfusion-associated necrotizing enterocolitis.
26 testinal atresias, volvulus, and necrotizing enterocolitis.
27  inflammatory bowel disease, and necrotizing enterocolitis.
28 cular leukomalacia; and death or necrotizing enterocolitis.
29 rious respiratory conditions and necrotizing enterocolitis.
30 en in developing intestine as in necrotizing enterocolitis.
31 enuated Salmonella-induced and noninfectious enterocolitis.
32 osal inflammation in a murine model of acute enterocolitis.
33 ich likely contributes to the development of enterocolitis.
34  weeks of age with intestinal distension and enterocolitis.
35  enterocytes during experimental necrotizing enterocolitis.
36  receptor B model of Hirschsprung-associated enterocolitis.
37  rate from the best quartile for necrotizing enterocolitis.
38 , rapid clinical improvement, and healing of enterocolitis.
39 of neonatal bacterial sepsis and necrotizing enterocolitis.
40 ifier, on the risk of developing necrotising enterocolitis.
41 ng-related antecedents of severe necrotising enterocolitis.
42 eriventricular leukomalacia, and necrotising enterocolitis.
43 se or colitis (6.3% and 2.7%), and bacterial enterocolitis (0.9% and 0%) (P = .03).
44 =0.045) and an increased rate of necrotizing enterocolitis (10.4% vs. 8.0%; relative risk, 1.31; 95%
45  to treat to prevent one case of necrotising enterocolitis 114, 95% CI 87 to 136).
46  CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).
47 auses of the abdominal pain were neutropenic enterocolitis (28%) and small bowel obstruction (12%); t
48 ncreases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23 [95% CI, 20
49 .3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and death or br
50  significantly less was death or necrotizing enterocolitis (73.5% with exposure to antenatal corticos
51 ary cohort, of whom 28 developed necrotising enterocolitis; 94 infants were used as controls.
52 ht predispose to or protect from necrotising enterocolitis, a severe illness linked to prematurity.
53 he survival of newborn mice with necrotizing enterocolitis, a severe pathology in preterm infants.
54 nchiolitis, arthralgia, ocular inflammation, enterocolitis, absence of autoantibodies, and mild immun
55 f 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of care and VLBW c
56  the population of patients with neutropenic enterocolitis admitted to an ICU and to investigate the
57 ifferential diagnosis in outbreaks of severe enterocolitis among puppies between 4 days and 21 weeks
58  protected mice from T-cell transfer-induced enterocolitis and death.
59                      The fear of necrotizing enterocolitis and feeding intolerance are major factors
60  sequencing of DNA from 1 infant with severe enterocolitis and her parents.
61 everity in mouse models of acute and chronic enterocolitis and improved, in synergy with glucocortico
62 icated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease.
63 ylobacter jejuni is a leading cause of human enterocolitis and is associated with postinfectious comp
64  breve BBG-001 for prevention of necrotising enterocolitis and late-onset sepis in very preterm infan
65            Probiotics may reduce necrotising enterocolitis and late-onset sepsis after preterm birth.
66 es (controls) and 8 infants with necrotizing enterocolitis and measured levels of S100A8 by immunoflu
67 species cause zoonotic infections, including enterocolitis and plague.
68 wborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality.
69     We enrolled 40 patients with early-onset enterocolitis and screened for mutations in IL10/IL10R u
70 e virus has been associated with necrotizing enterocolitis and severe and persistent diarrhea, as wel
71 psy findings showed features of eosinophilic enterocolitis and she was diagnosed with allergic eosino
72 or and in protection against immune mediated enterocolitis and these phenomena are significantly asso
73 ses a spectrum of human infections including enterocolitis and typhoid fever.
74 cal courses were consistent with necrotising enterocolitis and whose radiographs fulfilled criteria f
75 e PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanical ventilatio
76 sease, irritable bowel syndrome, necrotizing enterocolitis, and a variety of other disorders.
77 e development of auto-inflammatory diseases, enterocolitis, and cancer.
78  or suspected late-onset sepsis, necrotizing enterocolitis, and cerebral palsy.
79 ere intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease among infants le
80 syndrome, cataracts, sensorineural deafness, enterocolitis, and early lethality in two pedigrees: mal
81 ensive loss of intestinal villi, obstructive enterocolitis, and lethality within 10 days.
82 sia, retinopathy of prematurity, necrotizing enterocolitis, and periventricular leukomalacia.
83 mmatory diseases such as asthma, necrotizing enterocolitis, and sepsis.
84 oss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of prematurity.
85 emature infants with and without necrotizing enterocolitis, and successfully provided a total number
86 c meningitis, neutropenic fever, neutropenic enterocolitis, and transfussion-associated GVHD.
87 1%), and Hirschsprung's disease, necrotising enterocolitis, and volvulus neonatorum in 23 (45.1%) eac
88 g enterocolitis, the severity of necrotizing enterocolitis, and/or bacterial sepsis.
89  (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20), severe neu
90  Four other patients with steroid-refractory enterocolitis appeared to respond promptly to tumor necr
91 rom critically ill patients with neutropenic enterocolitis are scarce.
92  or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of prematurity, a
93                             Pseudomembranous enterocolitis associated with Clostridium difficile infe
94 butor to the enhanced severity of Salmonella enterocolitis associated with helminth coinfection.
95 actors, including development of necrotising enterocolitis, associated with gut bacterial populations
96  less than 1500 g and perforated necrotizing enterocolitis at 15 pediatric centers to undergo primary
97 ets for interventions to prevent necrotising enterocolitis, at least among infants born at less than
98                We determined the severity of enterocolitis based on disease activity index, histology
99 y, ELGAN/ELBW infants developing necrotizing enterocolitis before day 14 had higher expression of CCR
100 that could predispose infants to necrotising enterocolitis before we can develop new strategies for p
101        The primary outcomes were necrotising enterocolitis (Bell stage 2 or 3), blood culture positiv
102                     Infants with necrotizing enterocolitis born into midlevel hospitals (low-volume l
103 intracranial hemorrhage, sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, or death or i
104  morbidities: late-onset sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, periventricul
105 ssociated with a reduced risk of necrotizing enterocolitis but an increased risk of late-onset sepsis
106 roducts might reduce the risk of necrotising enterocolitis, but the absolute reduction is small.
107 roducts might reduce the risk of necrotising enterocolitis, but the absolute reduction is small.
108 ased the histopathologic severity of chronic enterocolitis by repairing crypt epithelium and simultan
109             18 infants developed necrotising enterocolitis (cases) and 26 were controls.
110 infants who subsequently develop necrotising enterocolitis (cases) and those who do not (controls).
111 nt complement-mediated killing compared with enterocolitis-causing strains of S Typhimurium.
112  (9%) in the probiotic group had necrotising enterocolitis compared with 66 (10%) in the placebo grou
113 n infants who went on to develop necrotising enterocolitis compared with controls.
114 fy the burden of severe or fatal necrotising enterocolitis confirmed by laparotomy, leading to death,
115           In mice with S typhimurium-induced enterocolitis, CoPP reduced the numbers of live S typhim
116 development was most impaired by necrotizing enterocolitis (d = -0.40; P < .001) and meningitis (d =
117 development was most impaired by necrotizing enterocolitis (d = -0.66; P < .001).
118                                  Necrotizing enterocolitis, defined as Bell stage 2 or greater by pre
119                                              Enterocolitis, defined by grade 3/4 clinical presentatio
120 the national incidence of severe necrotising enterocolitis, describe variation across neonatal networ
121 FN signaling during L. monocytogenes-induced enterocolitis did not recapitulate STING deficiency.
122 eration performed for perforated necrotizing enterocolitis does not influence survival or other clini
123 mall-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface areas and promo
124 for more than one decade with chronic active enterocolitis, fistula disease as well as previous oral
125                         Food protein-induced enterocolitis (FPIES) is a non-IgE cell- mediated food a
126 g diagnoses: intestinal atresia, necrotizing enterocolitis, gastroschisis, and midgut volvulus.
127 tropenia, and pulmonary embolism [n=2 each]; enterocolitis, general physical health deterioration and
128                      Hirschsprung-associated enterocolitis (HAEC) is a significant and life-threateni
129 eatments for biliary atresia and necrotising enterocolitis have been achieved through specialisation,
130  such as reduction in sepsis and necrotizing enterocolitis, have been reported for premature infants
131 ne mediated toxicities including dermatitis, enterocolitis, hypophysitis, uveitis, hepatitis, and nep
132 onic intestinal inflammation and necrotizing enterocolitis in animal models.
133  spontaneous colitis in mice and early onset enterocolitis in humans.
134 hogen that causes meningitis and necrotizing enterocolitis in infants.
135 lammatory bowel diseases (IBD) in humans and enterocolitis in mice.
136                Here we show that necrotizing enterocolitis in neonate mice is accompanied by elevatio
137 sence of IL-10 signaling causes inflammatory enterocolitis in neonates.
138  may be associated with neonatal necrotizing enterocolitis in premature infants.
139 ally may reduce the incidence of necrotizing enterocolitis in preterm infants.
140 y that inhibited Slamf1 reduced the level of enterocolitis in Rag(-/-) mice.
141 the drug of choice to treat pseudomembranous enterocolitis in the gastrointestinal tract.
142 onalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infants, and hepa
143 specially Negativicutes) precede necrotising enterocolitis in very low birthweight infants.
144 nd find that specific fatty acids exacerbate enterocolitis in zebrafish.
145  more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary dysplasia, se
146  events were reported in five patients (6%): enterocolitis, infusion-related reaction, aminotransfera
147     In mixed models, the time-by-necrotising-enterocolitis interaction was positively associated with
148    Owing to the rarity of severe necrotising enterocolitis, international collaborations are needed f
149                       Perforated necrotizing enterocolitis is a major cause of morbidity and mortalit
150                                  Necrotising enterocolitis is a neonatal gastrointestinal inflammator
151  We have reported this case because allergic enterocolitis is becoming a topic of concern.
152        Given that a Mendelian form of severe enterocolitis is due to up-regulation of the interleukin
153                                  Necrotizing enterocolitis is the leading cause of death from gastroi
154 acterium breve BBG-001 to reduce necrotising enterocolitis, late-onset sepsis, and death in preterm i
155 suggested, as TLR4 activation in necrotizing enterocolitis led to reduced proliferation and increased
156                   Infections and necrotizing enterocolitis, major causes of mortality and morbidity i
157 fficile colitis, viral colitis, inflammatory enterocolitis, mesenteric ischemia, radiation-induced ga
158  composite of late-onset sepsis, necrotising enterocolitis (modified Bell stage >/=2), or death in in
159  bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches have failed t
160 ecome immune deficient and develop apoptotic enterocolitis, multiple intestinal atresia, and recurren
161 ), intestinal atresia (n=5), and necrotizing enterocolitis (n=4).
162 uded all-cause mortality, severe necrotizing enterocolitis (NEC) (Bell stage II or more), and culture
163                                  Necrotizing enterocolitis (NEC) affects up to 10% of premature infan
164 wn to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic candidates
165 iota alterations associated with necrotizing enterocolitis (NEC) and feeding intolerance among preter
166  (RBC) transfusion and anemia to necrotizing enterocolitis (NEC) are conflicting.
167 een associated with outbreaks of necrotizing enterocolitis (NEC) as well as infant sepsis and meningi
168                                  Necrotizing enterocolitis (NEC) continues to be a major cause of mor
169                                  Necrotizing enterocolitis (NEC) develops in response to elevated TLR
170                                  Necrotizing enterocolitis (NEC) has long remained a significant caus
171 ation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wall rupture in
172                                  Necrotizing enterocolitis (NEC) is a common and often fatal inflamma
173                                  Necrotizing enterocolitis (NEC) is a devastating disease affecting p
174                                  Necrotizing enterocolitis (NEC) is a devastating disease of prematur
175                                  Necrotizing enterocolitis (NEC) is a devastating disease of prematur
176                                  Necrotizing enterocolitis (NEC) is a devastating gastrointestinal di
177                                  Necrotizing enterocolitis (NEC) is a devastating gastrointestinal em
178                                  Necrotizing enterocolitis (NEC) is a devastating inflammatory bowel
179                                  Necrotizing enterocolitis (NEC) is a devastating intestinal disease
180                                  Necrotizing enterocolitis (NEC) is a disease of neonatal preterm inf
181                                  Neutropenic enterocolitis (NEC) is a life-threatening disease with s
182                                  Necrotizing enterocolitis (NEC) is a major cause of morbidity and mo
183                                  Necrotizing enterocolitis (NEC) is a major cause of neonatal morbidi
184                                  Necrotizing enterocolitis (NEC) is a severe disease of the gastroint
185                                  Necrotizing enterocolitis (NEC) is a severe disease that affects the
186                                  Necrotizing enterocolitis (NEC) is an idiopathic, inflammatory bowel
187                                  Necrotizing enterocolitis (NEC) is an inflammatory bowel necrosis of
188                                  Necrotizing enterocolitis (NEC) is an inflammatory disease of the in
189                                  Necrotizing enterocolitis (NEC) is an inflammatory intestinal disord
190                                  Necrotizing enterocolitis (NEC) is characterized by interferon-gamma
191                   Development of necrotising enterocolitis (NEC) is considered to be dependent on the
192                                  Necrotizing enterocolitis (NEC) is one of the most serious disorders
193                                  Necrotizing enterocolitis (NEC) is the leading cause of death from g
194                                  Necrotizing enterocolitis (NEC) is the leading cause of death from g
195                                  Necrotizing enterocolitis (NEC) is the leading cause of gastrointest
196                                  Necrotizing enterocolitis (NEC) is the most common and serious gastr
197                                  Necrotizing enterocolitis (NEC) is the most common gastrointestinal
198                                  Necrotizing enterocolitis (NEC) is the most common gastrointestinal
199                                  Necrotizing enterocolitis (NEC) is the most common gastrointestinal
200 nd is involved in development of necrotizing enterocolitis (NEC) of the immature intestine.
201 regulate enterocyte apoptosis in necrotizing enterocolitis (NEC) remain incompletely understood, alth
202           The pathophysiology of necrotizing enterocolitis (NEC) remains poorly understood.We assesse
203 sequence of intestinal injury in necrotizing enterocolitis (NEC) remains unknown.
204 ave proposed using outcomes from necrotising enterocolitis (NEC) surgery for revalidation of neonatal
205 gen associated with the cases of necrotizing enterocolitis (NEC) that result from formula contaminati
206 mmatory cascade, is activated in necrotizing enterocolitis (NEC), a devastating condition of intestin
207 sease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gastrointestinal
208  of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affecting prematur
209 ture neonates are predisposed to necrotizing enterocolitis (NEC), an idiopathic, inflammatory bowel n
210 antibiotic resistance, fungemia, necrotizing enterocolitis (NEC), and mortality.
211 , otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death syndrome (S
212 s associated with lower rates of necrotizing enterocolitis (NEC), but an understanding of mechanism i
213 ead use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time-consuming m
214 nal diseases, including neonatal necrotizing enterocolitis (NEC), the leading cause of death from gas
215                                  Necrotizing enterocolitis (NEC), the leading cause of gastrointestin
216 HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading and intract
217 h can lead to the development of necrotizing enterocolitis (NEC)--a devastating inflammatory disease
218 egnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during infancy.
219 (BAs) may mediate development of necrotizing enterocolitis (NEC).
220 y associated with development of necrotizing enterocolitis (NEC).
221 racterized by ISC loss including necrotizing enterocolitis (NEC).
222  in prophylaxis and treatment of necrotizing enterocolitis (NEC).
223 order to reduce the incidence of necrotizing enterocolitis (NEC).
224 is, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.95; medical o
225 ated in the mutant animals, with less severe enterocolitis observed in vivo and reduced macrophage TN
226 Late-onset invasive infection or necrotizing enterocolitis occurred in 32% of infants (19 of 60) in g
227                                  Necrotizing enterocolitis occurred in 70 of 1394 infants (5.0%) in t
228                                  Necrotizing enterocolitis occurred less frequently in the liberal ox
229                                  Necrotizing enterocolitis occurred more frequently in infants on res
230                                  Neutropenic enterocolitis occurs in about 5.3% of patients hospitali
231 age of 32 weeks developed severe necrotising enterocolitis, of whom 222 (48.1%) died.
232 1 (0.4%) babies developed severe necrotising enterocolitis, of whom 247 (46.5%) died (139 after lapar
233 78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventricular hemor
234 y bypass, as well as in neonatal necrotizing enterocolitis or persistent ductus arteriosus.
235 y of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perforation were
236 vere retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 weeks of post
237 nfection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first 60 days of
238 ntricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinopathy of prem
239      Interestingly, we find that necrotizing enterocolitis patients also exhibit decreased expression
240 10/IL-10R-deficient patients had intractable enterocolitis, perianal disease, and fistula formation.
241 unreported syndrome featuring neonatal-onset enterocolitis, periodic fever, and fatal or near-fatal e
242 diographic evidence of extensive necrotizing enterocolitis (pneumatosis intestinalis), gestational ag
243 he adjusted network incidence of necrotising enterocolitis ranged from 2.51% (95% CI 1.13-3.60) to 3.
244 s system injury decreased, while necrotizing enterocolitis-related deaths increased.
245                     Infants with necrotizing enterocolitis represent a high-risk subgroup of the very
246 s restrictive thresholds groups, necrotizing enterocolitis requiring surgical intervention occurred i
247        Cases presented with severe apoptotic enterocolitis resembling acute intestinal graft-versus-h
248 compared with 11% and 2% in patients without enterocolitis, respectively (P = .0065 for MM and P = .0
249 ic periventricular leukomalacia, necrotizing enterocolitis, retinopathy of prematurity stage 3-5, or
250 h or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, and severe in
251  neonatal morbidities, including necrotizing enterocolitis, retinopathy of prematurity, bronchopulmon
252 ome, and significance of the immune-mediated enterocolitis seen with ipilimumab is presented.
253 also decreases the prevalence of necrotizing enterocolitis, sepsis, and intraventricular hemorrhage (
254 antly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease, intraventri
255 hage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dysplasia, or sev
256       No differences in rates of necrotizing enterocolitis, severe bronchopulmonary dysplasia, or sev
257 regular colon cleanouts during treatment for enterocolitis (slow responders).
258 f prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perforation, and n
259 maturity (stage 3 or higher), or necrotizing enterocolitis (stages 2-3).
260      The estimated numbers of admissions for enterocolitis suggest an increasing trend from 466 to 40
261                         Food protein-induced enterocolitis syndrome (FPIES) is a form of non-IgE-medi
262                         Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo
263                         Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo
264                         Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo
265                         Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated gas
266                         Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated gas
267 oderate to severe acute food protein-induced enterocolitis syndrome (FPIES) typically consists of int
268  and pathophysiology of food protein-induced enterocolitis syndrome (FPIES), an under-recognized and
269 d allergies and include food protein-induced enterocolitis syndrome (FPIES), food protein-induced all
270 l food allergy known as food protein-induced enterocolitis syndrome (FPIES), with several recent publ
271 n facing a patient with food protein-induced enterocolitis syndrome avoiding delayed diagnosis with u
272                 Mainly, food protein-induced enterocolitis syndrome diagnosis is based on clinical hi
273                         Food protein-induced enterocolitis syndrome is still a mysterious disease, pa
274 ns which are similar to food protein-induced enterocolitis syndrome reactions.
275 nique entities, such as food protein-induced enterocolitis syndrome with acute presentation.
276 , pollen food syndrome, food-protein-induced enterocolitis syndrome, food-induced proctocolitis, eosi
277 cations of prematurity including necrotizing enterocolitis, systemic infections and bronchopulmonary
278 ssociated with susceptibility to spontaneous enterocolitis that is microbiome dependent.
279 The frequencies of air leaks and necrotizing enterocolitis, the duration of respiratory support, and
280  bacteria may reduce the rate of necrotizing enterocolitis, the severity of necrotizing enterocolitis
281 nfants, 3586 stools, 46 cases of necrotising enterocolitis), there were increased proportions of Gamm
282 erleukin (IL)10-null mice are susceptible to enterocolitis, they maintained the same body weight as t
283 range of disease manifestations ranging from enterocolitis to typhoid fever.
284 y (the UK Neonatal Collaborative Necrotising Enterocolitis [UKNC-NEC] Study) of babies born in Englan
285 ture infants are associated with necrotizing enterocolitis, underscoring the critical role of host mu
286 sms against intestinal pathogens, Salmonella enterocolitis (using Salmonella enterica serovar Typhimu
287 its absence, mice develop aggravated chronic enterocolitis via an imbalance of colitogenic Th1 cells
288                                      Chronic enterocolitis was induced by the transfer of wild-type o
289 tricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born in hospitals
290                The occurrence of necrotizing enterocolitis was significantly higher in the control gr
291 ition of IL18 has been shown to reverse this enterocolitis, we undertook a Mendelian randomization st
292 us parents with severe exfoliative apoptotic enterocolitis; we also detected TTC7A mutations in 2 unr
293 ective tumor response rates in patients with enterocolitis were 36% for MM and 35% for RCC, compared
294 , just 28.6% of the infants with necrotizing enterocolitis were born into high-level, high-volume hos
295                       If vaccines preventing enterocolitis were to be developed, they would likely pr
296 ase reaches 20% in patients with neutropenic enterocolitis when enteritis is considered.
297 pidemic of poultry-associated, self-limiting enterocolitis, whereas in sub-Saharan Africa it is a maj
298                Chorioamnionitis, necrotizing enterocolitis, white matter injury on cranial ultrasound
299 a 5-month-old female infant who had allergic enterocolitis with protein-losing enteropathy and had lo
300 sed in ICU patients admitted for neutropenic enterocolitis with radiologically assessed enteritis.

 
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