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1  and sterol transporter that facilitates the enterohepatic and renal-hepatic circulation of bile acid
2 F15)/cholesterol-7alpha-hydroxylase (Cyp7a1) enterohepatic axis and eventually provide protection aga
3 BP effects on the biochemical changes in the enterohepatic axis caused by a high-fat diet (HFD) remai
4 stinal acidification confirming an important enterohepatic axis of metabolite-microbiome interaction
5  improve the lipid or glucose alterations in enterohepatic axis of rats fed HFD.
6 pid, glucose, or inflammatory changes in the enterohepatic axis of rats fed HFD.
7 e acid (BA) transporters are involved in the enterohepatic BA circulation between the liver and gut,
8                              Interruption of enterohepatic BA cycling after partial external biliary
9 nt mice maintained free of the Gram-negative enterohepatic bacteria Helicobacter spp. for up to 9 mon
10            We investigated the effect of the enterohepatic bacterial pathogen Helicobacter hepaticus
11                            Disruption of the enterohepatic bile acid circulation during biliary tract
12  farnesoid X receptor dramatically increases enterohepatic bile acid levels and jet-lag-induced HCC,
13 rovide an important contributing role in the enterohepatic bile acid metabolism and cholesterol homeo
14                                Replacing the enterohepatic bile acid pool with DCA restored FXR mRNA
15 through pyruvate dehydrogenase and elevating enterohepatic bile acid recirculation are promising new
16 s of increased mitochondrial respiration and enterohepatic bile acid recirculation due to improvement
17 t exporter protein levels, thereby promoting enterohepatic bile acid recirculation, leading to activa
18 ent, bile acid transport inhibitor, prevents enterohepatic bile acid recirculation.
19 clinically important transporter involved in enterohepatic bile acid recycling with currently no high
20 FXR is the sensor of physiological levels of enterohepatic bile acids, the end products of cholestero
21                                        Thus, enterohepatic circulated NA is preferentially used in th
22 tabolites into bile as well as a slowdown of enterohepatic circulation (bile acid recirculation) of b
23 findings triggered us to study the liver and enterohepatic circulation (EHC) following intra-amniotic
24 to 57% +/- 5% of paired controls with intact enterohepatic circulation (P < 0.0001).
25 nal selective FXR reactivation normalized BA enterohepatic circulation along with up-regulation of in
26           (11)C-cholylsarcosine underwent an enterohepatic circulation and reappeared in liver tissue
27 -uptake of bile acids, thus interrupting the enterohepatic circulation and reducing the total bile ac
28 urnal variation has been demonstrated in the enterohepatic circulation and the gut microbiota, existi
29 n hepatocytes and maintained in vivo through enterohepatic circulation between the liver and small in
30 acid (BA) synthesis and transport within the enterohepatic circulation has revealed potential targets
31 from enterocytes of the small intestine into enterohepatic circulation in response to bile-induced FX
32          Cholate feeding to rats with intact enterohepatic circulation increased mdr2 transcriptional
33 ects of bile acids on tissues outside of the enterohepatic circulation may be of major pathophysiolog
34                                    Thus, the enterohepatic circulation of all conjugated bile acids w
35 We analyzed expressions of factors mediating enterohepatic circulation of BA using ileal and colonic
36 r, they suggest a potential role for altered enterohepatic circulation of BAs in improving insulin se
37 tic BA uptake machinery maintains a (slower) enterohepatic circulation of BAs, although it is occasio
38  in the distal ileum plays a key role in the enterohepatic circulation of BAs.
39 defects in gallbladder emptying that disrupt enterohepatic circulation of BAs.
40 ver bile acid compositions via the disturbed enterohepatic circulation of bile acids and the disturba
41 n of bile acids, a rate-limiting step in the enterohepatic circulation of bile acids and transactivat
42                                          The enterohepatic circulation of bile acids is maintained by
43                          Interruption of the enterohepatic circulation of bile acids leads to increas
44 l diarrhea, steatorrhea, interruption of the enterohepatic circulation of bile acids, and reduced pla
45 hASBT, SLC10A2) plays a critical role in the enterohepatic circulation of bile acids, as well as in c
46 y pump, a critical component involved in the enterohepatic circulation of bile acids.
47 otransporter superfamily and function in the enterohepatic circulation of bile acids.
48 and intestine and controls the synthesis and enterohepatic circulation of bile acids.
49 e acid transporter (ASBT) is crucial for the enterohepatic circulation of bile acids.
50 ns (ILBPs) are involved in the transport and enterohepatic circulation of bile acids.
51  acid amidation, a critical component of the enterohepatic circulation of bile acids.
52  bile acid cotransporter participates in the enterohepatic circulation of bile acids.
53 oduct is likely to play an essential role in enterohepatic circulation of bile acids; further charact
54  transporter (ASBT, SLC10A2) facilitates the enterohepatic circulation of bile salts and plays a key
55                                          The enterohepatic circulation of bile salts is an important
56 nse of the transport process involved in the enterohepatic circulation of bile salts to obstructive c
57 ux, and are each essential for the efficient enterohepatic circulation of bile salts.
58 g activity of FGF19 in organs engaged in the enterohepatic circulation of bile salts.
59 roteins and is thought to play a role in the enterohepatic circulation of bile salts.
60  function in the transcellular transport and enterohepatic circulation of bile salts.
61               We propose the existence of an enterohepatic circulation of lymphocytes, whereby some m
62 fecal excretion, suggests the possibility of enterohepatic circulation of this drug.
63 gation of bile acids entering liver from the enterohepatic circulation rather than in de novo bile ac
64 oea (BAD) can occur due to disruption to the enterohepatic circulation such as following cholecystect
65 ofluids, and several tissues involved in the enterohepatic circulation were measured and compared to
66 transformation enzyme activities, changes in enterohepatic circulation, altered bioavailability of en
67 terocyte, along with the contribution of the enterohepatic circulation, are considered.
68 results suggest that systemic alterations in enterohepatic circulation, as well as host and microbiot
69 ials, and the related feedback mechanisms in enterohepatic circulation, have been considered targets
70                    MCBAs fed to mice entered enterohepatic circulation, in which liver and gallbladde
71  bile salts, a critical determinant of their enterohepatic circulation, is mediated primarily by the
72  However, the lead compound 1a suffered from enterohepatic circulation, preventing further developmen
73                                   Within the enterohepatic circulation, reabsorbed bile acids act as
74 a measurement (one patient) is suggestive of enterohepatic circulation.
75 ll intestine and only in animals with intact enterohepatic circulation.
76 e quantity of bile salts fluxing through the enterohepatic circulation.
77 lysis and limited access of L -Tyr-CA to the enterohepatic circulation.
78 l absorption, and rat-human extrapolation of enterohepatic circulation.
79 olate cotransporting polypeptide, within the enterohepatic circulation.
80 ossing biological membranes, and clearing by enterohepatic circulation.
81 ed rats and mice, and in mice with an intact enterohepatic circulation.
82 al CFTR(inh)-172 accumulation facilitated by enterohepatic circulation.
83 ted charcoal, to interrupt enterovascular or enterohepatic circulations, offers benefit compared with
84 xiliary transporters are able to sustain the enterohepatic cycle in its absence.
85 d X receptor (FXR) plays a major role in the enterohepatic cycling of bile acids, but the impact of n
86  salts in the circulation suggested residual enterohepatic cycling of bile salts.
87 ults are consistent with the hypothesis that enterohepatic cycling of bilirubin occurs with bile salt
88 ats), indices of bile salt malabsorption and enterohepatic cycling of bilirubin were measured, includ
89            Because ileectomy in rats induces enterohepatic cycling of bilirubin, the hypothesis that
90 orption, dietary UDCA and cholesterol induce enterohepatic cycling of bilirubin.
91 o test the hypothesis that ileectomy induces enterohepatic cycling of bilirubin.
92 xtrahepatic exposure and underwent extensive enterohepatic cycling.
93 us-associated tumors in woodchucks or causes enterohepatic disease in cats.
94                Clostridium piliforme induces enterohepatic disease in many domestic and laboratory an
95 liforme infection (Tyzzer's disease) induces enterohepatic disease in many domestic and laboratory an
96 ation of other Helicobacter spp. involved in enterohepatic disease.
97 a bona fide novel therapeutic agent to treat enterohepatic disorders such as cholestasis, NASH, and i
98 nductance regulator (CFTR) deficiency on the enterohepatic disposition of bile acids (BAs).
99 or PXR (pregnane X receptor), a regulator of enterohepatic drug metabolism and clearance, results in
100 ental Cell, Ji et al. (2019) now describe an enterohepatic feedback loop that balances tissue size an
101 nthesis is controlled, in part, by a complex enterohepatic feedback regulatory mechanism.
102 dary modification to FXR agonists, enhancing enterohepatic feedback via FGF19.
103                    We found that circulating enterohepatic FGF15 stimulates hepatic receptor FGFR4 to
104  However, the activating ligand (DCA) in the enterohepatic flux is necessary for FXR-mediated transcr
105                 We propose that the enhanced enterohepatic flux of bile acids during HF-LC consumptio
106 icobacter gastritis, we investigated whether enterohepatic Helicobacter bilis modulates Helicobacter
107 bacter cinaedi is the most commonly reported enterohepatic helicobacter in humans, there are no repor
108                   Helicobacter hepaticus, an enterohepatic helicobacter in mice, is known to cause he
109 es of known virulence factors found in other enterohepatic helicobacter species (EHS) and H. pylori T
110                 To investigate how different enterohepatic Helicobacter species (EHS) influence Helic
111 ency (IBD), and non-Helicobacter pylori-like enterohepatic Helicobacter species (IBD).
112   Discrimination of this organism from other enterohepatic Helicobacter species and Campylobacter spe
113                                              Enterohepatic Helicobacter species are associated with s
114                 Helicobacter hepaticus is an enterohepatic Helicobacter species that induces lower bo
115 eptible to colitis induced by the pathogenic enterohepatic Helicobacter species, H. hepaticus.
116 sis of hepatitis and enterocolitis caused by enterohepatic Helicobacter species.
117  Cdt with other enteric pathogens, including enterohepatic Helicobacter species.
118 sults suggest a possible association between enterohepatic Helicobacter spp and cholesterol cholelith
119 7L mice were infected with several different enterohepatic Helicobacter spp or left uninfected and fe
120                       Helicobacter bilis, an enterohepatic helicobacter, is associated with chronic h
121                 Consistent with other murine enterohepatic helicobacters, WT(Hc) did not cause typhlo
122 ver to the gut as one branch of a postulated enterohepatic lymphocyte circulation.
123 g cells can induce gut tropism supporting an enterohepatic lymphocyte circulation.
124 ly susceptible to colitis induced by another enterohepatic microaerobe, Helicobacter hepaticus, which
125                                              Enterohepatic nuclear receptors including farnesoid X re
126  Helicobacter hepaticus, a widespread murine enterohepatic pathogen.
127 bt expression, fecal bile acid excretion, or enterohepatic pool size that might explain the phenotype
128                                              Enterohepatic recirculation (EHR) of parent drug or reve
129 following biliary excretion is well-known as enterohepatic recirculation (EHR).
130                                This leads to enterohepatic recirculation and an increase of toxic sec
131 amage, breakdown of intercellular integrity, enterohepatic recirculation and neutrophil activation by
132                                              Enterohepatic recirculation most probably contributes to
133 hought to be critical for the maintenance of enterohepatic recirculation of bile acids and hepatocyte
134                                     Possible enterohepatic recirculation of flavan-3-ols is discussed
135          The gut microbiome also impacts the enterohepatic recirculation of mycophenolate, resulting
136 amage, breakdown of intercellular integrity, enterohepatic recirculation, and neutrophil activation b
137                   Through a process known as enterohepatic recirculation, more than 90% of secreted b
138 dependent bile acid transporters involved in enterohepatic recirculation, the Na(+)-taurocholate co-t
139  from the intestinal lumen to preserve their enterohepatic recirculation.
140  stores, since progesterone does not undergo enterohepatic recirculation.
141 tudies suggested that the compound undergoes enterohepatic recirculation.
142 4%, elimination half-life of 4 hours, and an enterohepatic recirculation.
143 P5 in reconjugation of bile acids during the enterohepatic recirculation.
144 y orally administered phenolic drugs undergo enterohepatic recycling (EHR), presumably mediated by th
145  or drug interactions can lead to changes in enterohepatic recycling of androgens.
146 xperienced transaminitis, revealing enhanced enterohepatic recycling of deglucuronidated tacrine in t
147 or based oral delivery of GLP-1 gene through enterohepatic recycling pathways of bile acids.
148                      In this process called "enterohepatic recycling", only 5% of the bile acid pool
149 intestinal microflora, are absorbed, undergo enterohepatic recycling, and reach circulating concentra
150    This review introduces the key factors in enterohepatic recycling, especially the mechanism of bil
151 fects of CR relate to functional recovery of enterohepatic signaling through the bile salt-FGF19 axis
152 nfluence the gut environment in ICP to alter enterohepatic signalling.
153                It is highly expressed in the enterohepatic system, where it senses bile acid levels t
154 ward, protective pathway operative in murine enterohepatic tissues wherein FXR induces AKR1B7 to deto
155 acids can cause toxicity and inflammation in enterohepatic tissues(2).
156  and inflammation by acting predominantly in enterohepatic tissues, but also in peripheral organs.
157 ed by bile acids and abundantly expressed in enterohepatic tissues, plays a crucial role in maintaini
158 d receptors that are highly expressed in the enterohepatic tissues.
159 ably, despite the broad knowledge of the FXR enterohepatic transcriptional activity, the molecular me
160 criptionally regulate their biosynthesis and enterohepatic transport.

 
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