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1 ed rats and mice, and in mice with an intact enterohepatic circulation.
2 al CFTR(inh)-172 accumulation facilitated by enterohepatic circulation.
3 a measurement (one patient) is suggestive of enterohepatic circulation.
4 ll intestine and only in animals with intact enterohepatic circulation.
5 olate cotransporting polypeptide, within the enterohepatic circulation.
6 e quantity of bile salts fluxing through the enterohepatic circulation.
7 ossing biological membranes, and clearing by enterohepatic circulation.
8 lysis and limited access of L -Tyr-CA to the enterohepatic circulation.
9 l absorption, and rat-human extrapolation of enterohepatic circulation.
10 nal selective FXR reactivation normalized BA enterohepatic circulation along with up-regulation of in
11 transformation enzyme activities, changes in enterohepatic circulation, altered bioavailability of en
13 -uptake of bile acids, thus interrupting the enterohepatic circulation and reducing the total bile ac
14 urnal variation has been demonstrated in the enterohepatic circulation and the gut microbiota, existi
16 results suggest that systemic alterations in enterohepatic circulation, as well as host and microbiot
17 n hepatocytes and maintained in vivo through enterohepatic circulation between the liver and small in
18 tabolites into bile as well as a slowdown of enterohepatic circulation (bile acid recirculation) of b
19 findings triggered us to study the liver and enterohepatic circulation (EHC) following intra-amniotic
20 acid (BA) synthesis and transport within the enterohepatic circulation has revealed potential targets
21 ials, and the related feedback mechanisms in enterohepatic circulation, have been considered targets
22 from enterocytes of the small intestine into enterohepatic circulation in response to bile-induced FX
25 bile salts, a critical determinant of their enterohepatic circulation, is mediated primarily by the
26 ects of bile acids on tissues outside of the enterohepatic circulation may be of major pathophysiolog
28 We analyzed expressions of factors mediating enterohepatic circulation of BA using ileal and colonic
29 r, they suggest a potential role for altered enterohepatic circulation of BAs in improving insulin se
30 tic BA uptake machinery maintains a (slower) enterohepatic circulation of BAs, although it is occasio
33 ver bile acid compositions via the disturbed enterohepatic circulation of bile acids and the disturba
34 n of bile acids, a rate-limiting step in the enterohepatic circulation of bile acids and transactivat
37 l diarrhea, steatorrhea, interruption of the enterohepatic circulation of bile acids, and reduced pla
38 hASBT, SLC10A2) plays a critical role in the enterohepatic circulation of bile acids, as well as in c
46 oduct is likely to play an essential role in enterohepatic circulation of bile acids; further charact
47 transporter (ASBT, SLC10A2) facilitates the enterohepatic circulation of bile salts and plays a key
49 nse of the transport process involved in the enterohepatic circulation of bile salts to obstructive c
56 ted charcoal, to interrupt enterovascular or enterohepatic circulations, offers benefit compared with
58 However, the lead compound 1a suffered from enterohepatic circulation, preventing further developmen
59 gation of bile acids entering liver from the enterohepatic circulation rather than in de novo bile ac
61 oea (BAD) can occur due to disruption to the enterohepatic circulation such as following cholecystect
62 ofluids, and several tissues involved in the enterohepatic circulation were measured and compared to