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2 hown to be manipulated during infection with enteropathogenic and enterohaemorrhagic Escherichia coli
3 n infections with the attaching and effacing enteropathogenic and enterohaemorrhagic Escherichia coli
7 uman and animal enteric pathogens, including enteropathogenic and enterohaemorrhagic Escherichia coli
9 secreted by the type III secretion system of enteropathogenic and enterohemorrhagic Escherichia coli
12 lasia and colitis and is used as a model for enteropathogenic and enterohemorrhagic Escherichia coli
14 ression of the type III secretion systems of enteropathogenic and enterohemorrhagic Escherichia coli
16 lence of the closely related human pathogens enteropathogenic and enterohemorrhagic Escherichia coli
17 oC SboC and SeoC are homologues of EspJ from enteropathogenic and enterohemorrhagic Escherichia coli
18 hing and effacing mouse pathogen that models enteropathogenic and enterohemorrhagic Escherichia coli
19 rocyte effacement-encoded regulator (Ler) of enteropathogenic and enterohemorrhagic Escherichia coli
21 ses resembling those of humans infected with enteropathogenic and enterohemorrhagic Escherichia coli.
22 cus of enterocyte effacement (LEE), a PAI of enteropathogenic and enterohemorrhagic Escherichia coli.
23 e occasions within the different lineages of enteropathogenic and enterohemorrhagic Escherichia coli.
24 ntium is a natural mouse pathogen related to enteropathogenic and enterohemorrhagic Escherichia coli.
26 e investigated the effects of nonpathogenic, enteropathogenic, and probiotic bacteria on the dynamics
27 sing chaperone HdeA promotes the survival of enteropathogenic bacteria during transit through the har
29 P are less prevalent and are mainly found in enteropathogenic bacteria, where they play key roles in
34 IL-1 superfamily members, yet their role in enteropathogenic bacterial infection remains poorly defi
37 t successful establishment in the gut by the enteropathogenic bacterium Salmonella enterica serovar T
40 and histological data characterizing a novel enteropathogenic BEC strain, NB, detected in fecal speci
42 ater secretion, antibiotic prescription, and enteropathogenic colonization, each of which involves an
43 Porcine epidemic diarrhea virus (PEDV) is an enteropathogenic coronavirus causing lethal watery diarr
45 hazard ratio [HR] 1.9; 0.99-3.5) and typical enteropathogenic E coli (HR 2.6; 1.6-4.1) in infants age
47 Shigella, ST-ETEC, Cryptosporidium, typical enteropathogenic E coli) can substantially reduce the bu
48 leal bacteria (P = .03) when challenged with enteropathogenic E coli, and were protected from immune
49 that cause significant human disease are the enteropathogenic E. coli (EPEC) and enterotoxigenic E. c
50 compared these TIR interactions in EHEC and enteropathogenic E. coli (EPEC) and found that five inte
51 Detailed analysis of the H-NST proteins from enteropathogenic E. coli (EPEC) and uropathogenic E. col
52 Enterotoxigenic Escherichia coli (ETEC) and enteropathogenic E. coli (EPEC) are common causes of dia
53 nterohemorrhagic Escherichia coli (EHEC) and enteropathogenic E. coli (EPEC) are enteric bacterial pa
54 beled Shiga toxin-producing E. coli; whereas enteropathogenic E. coli (EPEC) are LEE+ and often carry
55 (Stx)-producing Escherichia coli (STEC) and enteropathogenic E. coli (EPEC) are the major foodborne
60 EC O157:H7 serotype arose from its ancestor, enteropathogenic E. coli (EPEC) O55:H7 (sorbitol ferment
61 ely, new isolates continue to be mistyped as enteropathogenic E. coli (EPEC) or enterohemorrhagic E.
63 menal bacteria, (3) prevent the adherence of enteropathogenic E. coli (EPEC) to epithelial monolayers
64 detect the role of phenotypic variability in enteropathogenic E. coli (EPEC), an important human path
65 s enterohemorrhagic Escherichia coli (EHEC), enteropathogenic E. coli (EPEC), and Citrobacter rodenti
67 er enterohemorrhagic E. coli (EHEC), typical enteropathogenic E. coli (EPEC), or atypical EPEC, depen
68 coli (EAEC), enterotoxigenic E. coli (ETEC), enteropathogenic E. coli (EPEC), Shigella spp., Campylob
71 15:H-) and compared it with those from human enteropathogenic E. coli (EPEC, O127:H6) and enterohemor
72 i (in 21.2% and 8.5%, respectively), typical enteropathogenic E. coli (in 18.0% and 8.3%, respectivel
74 on was found between norovirus GII + typical enteropathogenic E. coli (OR = 0.09, 95% CI: 0.01, 0.95)
75 osubstrate, also significantly decreased the enteropathogenic E. coli -associated decrease in transep
76 a diverse array of clonal groups, including enteropathogenic E. coli 2 (EPEC 2), enterohemorrhagic E
77 coli [EAEC], enterotoxigenic E. coli [ETEC], enteropathogenic E. coli [EPEC], and Shiga-toxigenic E.
78 athotypes (enteroaggregative E. coli [EAEC], enteropathogenic E. coli [EPEC], enterotoxigenic E. coli
79 ntial regulation of protein kinase C-zeta by enteropathogenic E. coli and enterohemorrhagic E. coli m
80 tion of tight junctions after infection with enteropathogenic E. coli and enterohemorrhagic E. coli.
81 and lt for enterotoxigenic E. coli, eaeA for enteropathogenic E. coli and Shiga toxin-producing E. co
83 ithelial tight junctions, but the effects of enteropathogenic E. coli are more profound than those of
85 and probably contributes to the survival of enteropathogenic E. coli during the intestinal inflammat
86 activated luxS in three E. coli backgrounds: enteropathogenic E. coli E2348-69, and enterohaemorrhagi
87 found that uropathogenic E. coli CFT073 and enteropathogenic E. coli E2348/69 occupy intestinal nich
90 fall into any of the four classical EHEC and enteropathogenic E. coli groups but instead was closely
96 We constructed a tightly inducible clone of enteropathogenic E. coli O127:H6 lifA for affinity purif
98 g the O157:H7 serotype and are found in some enteropathogenic E. coli O55:H7 strains but are absent f
99 intestinal epithelial monolayers infected by enteropathogenic E. coli or enterohemorrhagic E. coli we
100 stal structure of the intimin-Tir complex of enteropathogenic E. coli predicts that each of these fou
102 esolution crystal structures of Gmm from the enteropathogenic E. coli strain O128: the structure of t
104 employed two proteins, intimin and tir, from enteropathogenic E. coli that are critical to the bacter
105 equenced pathovars, enterotoxigenic E. coli, enteropathogenic E. coli, and enteroaggregative E. coli.
106 fficile, enteroaggregative Escherichia coli, enteropathogenic E. coli, and enterotoxigenic E. coli),
107 O1, enterotoxigenic Escherichia coli (ETEC), enteropathogenic E. coli, Campylobacter jejuni, and Giar
108 ral pathogenic strains of E. coli, including enteropathogenic E. coli, enterohemorrhagic E. coli, ent
109 a enterocolitica, enteroaggregative E. coli, enteropathogenic E. coli, enterotoxigenic E. coli, Shiga
110 c from EAEC and Shigella flexneri, EspC from enteropathogenic E. coli, EspP from enterohemorrhagic E.
111 aggregative Escherichia coli, Campylobacter, enteropathogenic E. coli, rotavirus, and Entamoeba histo
112 ichia coli, particularly enterotoxigenic and enteropathogenic E. coli, rotavirus, Giardia lamblia, an
119 ded enterotoxigenic Escherichia coli (ETEC), enteropathogenic E.coli (EPEC), Listeria monocytogenes,
120 three CPE-positive type C EN strains caused enteropathogenic effects in rabbit small intestinal loop
121 ing that both CPB and CPE are needed for the enteropathogenic effects of CN3758 MDS lysate supernatan
123 nsequently, CPE and CPB contributions to the enteropathogenic effects of MDS lysate supernatants of C
124 t or reversing the cpb mutation restored the enteropathogenic effects of MDS lysate supernatants.
126 ell as lower proportions of enterotoxigenic, enteropathogenic, enterohemorrhagic, and commensal E. co
131 The human pathogens enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC), as we
132 ith SadA from Salmonella enterica, EhaG from enteropathogenic Escherichia coli (EHEC), and UpaG from
133 eotide sequence was determined for pMAR7, an enteropathogenic Escherichia coli (EPEC) adherence facto
134 of attaching and effacing pathogens such as enteropathogenic Escherichia coli (EPEC) and Citrobacter
136 It had been suggested that the flagella of enteropathogenic Escherichia coli (EPEC) and enterohemor
142 on of several important virulence factors in enteropathogenic Escherichia coli (EPEC) and reduced EPE
143 ues and fluids in response to infection with enteropathogenic Escherichia coli (EPEC) and Shiga-toxig
149 The type IV bundle-forming pili (BFP) of enteropathogenic Escherichia coli (EPEC) are required fo
150 ude two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and Esp
157 lifA, for lymphocyte inhibitory factor A) in enteropathogenic Escherichia coli (EPEC) encoding a prot
158 The attaching and effacing (A/E) pathogen enteropathogenic Escherichia coli (EPEC) forms character
160 The type III secretion system (TTSS) of enteropathogenic Escherichia coli (EPEC) has been associ
161 yte effacement (LEE) pathogenicity island of enteropathogenic Escherichia coli (EPEC) has not been de
184 The plasmid-encoded Per regulatory locus of enteropathogenic Escherichia coli (EPEC) is generally co
186 diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
190 rial pathogens like Salmonella, Shigella and enteropathogenic Escherichia coli (EPEC) is the transloc
191 initial steps in biofilm development, and in enteropathogenic Escherichia coli (EPEC) it is mediated
194 Although the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) mediates microc
195 ttens that were presumptively diagnosed with enteropathogenic Escherichia coli (EPEC) on the basis of
200 Production of type IV bundle-forming pili by enteropathogenic Escherichia coli (EPEC) requires BfpB,
201 tion of type IV bundle-forming pili (BFP) by enteropathogenic Escherichia coli (EPEC) requires the pr
205 and virulence-associated -components in the enteropathogenic Escherichia coli (EPEC) strain E2348/69
206 gative transfer system identified in O119:H2 enteropathogenic Escherichia coli (EPEC) strain MB80 by
209 n, CesT, serves a chaperone function for the enteropathogenic Escherichia coli (EPEC) translocated in
215 Here, we report that the bacterial pathogen enteropathogenic Escherichia coli (EPEC) uses the type I
216 Outer membrane intimin directs attachment of enteropathogenic Escherichia coli (EPEC) via its Tir rec
218 to inhibit attachment of microcolony-forming enteropathogenic Escherichia coli (EPEC) was investigate
226 ns and actin polymerization, the hallmark of enteropathogenic Escherichia coli (EPEC), enterohemorrha
227 es, collected semimonthly, were screened for enteropathogenic Escherichia coli (EPEC), enterotoxigeni
229 processes as well as actin-based motility of enteropathogenic Escherichia coli (EPEC), vaccinia, and
230 e factor in two groups of enteric pathogens: enteropathogenic Escherichia coli (EPEC), which is a maj
236 mmon organisms detected by the GI panel were enteropathogenic Escherichia coli (EPEC, n = 21), norovi
237 eal pathogens in the post-GI PCR cohort were enteropathogenic Escherichia coli (n = 14, 8%), noroviru
238 eal pathogens in the post-GI PCR cohort were enteropathogenic Escherichia coli (n=14, 8%), norovirus
239 -to-severe diarrhoea associated with typical enteropathogenic Escherichia coli among children aged 6-
240 nding cis-complemented derivatives of rabbit enteropathogenic Escherichia coli and compared their abi
241 derstanding of the molecular pathogenesis of enteropathogenic Escherichia coli and enterohemorrhagic
243 related clinically important human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
245 n secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion fro
248 Using the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli as a model Tfp system,
249 angstrom resolution cryo-EM structure of the enteropathogenic Escherichia coli ATPase EscN in complex
250 We purified the PulE homologue BfpD of the enteropathogenic Escherichia coli bundle-forming pilus (
252 gens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli disarm host cells by i
253 rt the 1.9 A resolution crystal structure of enteropathogenic Escherichia coli GfcC, a periplasmic pr
255 cted with Citrobacter rodentium, a model for enteropathogenic Escherichia coli infection in humans, t
256 acter rodentium infection, a mouse model for enteropathogenic Escherichia coli infection, Hvem-/- mic
257 secretion system effector protein NleE from enteropathogenic Escherichia coli plays a key role in th
258 orum sensing to TTS in enterohemorrhagic and enteropathogenic Escherichia coli show that quorum sensi
259 entium uses virulence factors similar to the enteropathogenic Escherichia coli to produce attaching a
262 zyme in the interaction between the host and enteropathogenic Escherichia coli(EPEC) and Shiga-toxige
264 cellular (S. Typhimurium) and extracellular (enteropathogenic Escherichia coli) enteric pathogens, vi
265 perone-delivered to the translocase, EscV in enteropathogenic Escherichia coli, and cross it in stric
266 bacter rodentium is the rodent equivalent of enteropathogenic Escherichia coli, and it causes colitis
270 n 24 months with MSD, infection with typical enteropathogenic Escherichia coli, enterotoxigenic E col
271 and Campylobacter coli, Cryptosporidium spp, enteropathogenic Escherichia coli, heat-stable enterotox
272 rodentium, a murine model pathogen for human enteropathogenic Escherichia coli, predominantly coloniz
273 how that important other pathogens including enteropathogenic Escherichia coli, Shigella flexneri, an
280 d that extrinsic stress signals generated by enteropathogenic infection are epistatic to the hypoplas
283 in, the major proinflammatory determinant of enteropathogenic Salmonella, which was found to be glyco
285 ce factor-depleted E. coli C600 strains with enteropathogenic Shiga-toxin negative E. coli O26:H11, a
286 eagenic Escherichia coli (enteroaggretative, enteropathogenic), Shigella, and Campylobacter were the
287 the plague bacillus Yersinia pestis and two enteropathogenic species, Yersinia pseudotuberculosis an
289 not clear why these avian coronaviruses are enteropathogenic, whereas other closely related avian co
290 ed to mount a robust immune response against enteropathogenic Y. pseudotuberculosis by promoting Th17
291 es) do not display altered susceptibility to enteropathogenic Yersinia compared to wild-type mice.
293 plasmids (pCD in Yersinia pestis and pYV in enteropathogenic Yersinia pseudotuberculosis and Yersini
295 es have demonstrated that TTSS expression in enteropathogenic Yersinia spp. also inhibits the respira
296 oonotic pathogen hepatitis E virus (HEV) and enteropathogenic Yersinia spp. were analyzed in parallel