ライフサイエンスコーパス: enteropathy

1 rates intestinal radiation injury (radiation enteropathy).

2 logic tests for celiac disease (seronegative enteropathy).

3 adin and reduced markers of inflammation and enteropathy.

4 of, and, ideally, assess interventions for, enteropathy.

5 viously carried out studies on environmental enteropathy.

6 ong-term effects and avoid chronic allograft enteropathy.

7 rization of the pathogenesis of HIV-mediated enteropathy.

8 extrudates were formulated to combat celiac enteropathy.

9 due to severe noninflammatory protein-losing enteropathy.

10 cluding HAART-related adverse events and HIV enteropathy.

11 c disease is a diet-induced, T cell-mediated enteropathy.

12 spected to have an associated protein-losing enteropathy.

13 and histologic evidence of gluten-sensitive enteropathy.

14 ly includes a minority of patients with mild enteropathy.

15 d disease-specific antibodies in addition to enteropathy.

16 itis, motility, malabsorption, and radiation enteropathy.

17 y drug users develop subclinical small bowel enteropathy.

18 ations of nonsteroidal antiinflammatory drug enteropathy.

19 the key factor in staphylococcal enterotoxin enteropathy.

20 to be correlated with the degree (extent) of enteropathy.

21 c event, and 1 (2%) developed protein-losing enteropathy.

22 ular NSAID users may be prone to small bowel enteropathy.

23 or hypoalbuminemia are useful clues to NSAID enteropathy.

24 ellular targets underlying Glafenine-induced enteropathy.

25 ovesicular rash and is often associated with enteropathy.

26 t the disease arises secondary to hereditary enteropathy.

27 ora in the small intestine and NSAID-induced enteropathy.

28 esents an in vitro model of gluten-sensitive enteropathy.

29 d Bob activation is a plausible cause of HIV enteropathy.

30 contributes substantially to the associated enteropathy.

31 rotein is feasible and ameliorates radiation enteropathy.

32 role for drug adduct formation in diclofenac enteropathy.

33 some infection was primarily associated with enteropathy.

34 ths and no patients developed protein losing enteropathy.

35 hunting, thromboembolism, and protein-losing enteropathy.

36 from LMICs who are at risk of environmental enteropathy.

37 ntrol mice or those exhibiting NSAID-induced enteropathy.

38 l small intestine in a murine model of NSAID enteropathy.

39 been additionally diagnosed with celiac-like enteropathy.

40 CeD) is a common gluten-sensitive autoimmune enteropathy.

41 as revealed as an AIE-75-positive autoimmune enteropathy.

42 with HIV status, and also with environmental enteropathy.

43 ymphocytes that may also play a role in some enteropathies.

44 syndrome (SBS) (59%), PNDD (14%), congenital enteropathies (10%), chronic intestinal pseudo-obstructi

45 , lymphoproliferation (52.4%), noninfectious enteropathy (23.1%), opportunistic infections (15.7%), a

46 (32%), thrombosis (12%), and protein-losing enteropathy (8%).

47 Acquired enteropathy affecting both gut structure and function li

48 nclusion disease (MVID), a lethal hereditary enteropathy affecting neonates.

49 unction, such as environmental (or tropical) enteropathy, affects zinc absorption, losses, and homeos

50 nfortunately these molecules cause a gastric enteropathy after chronic dosing in rats.

51 Coeliac disease is an immune-mediated enteropathy against dietary gluten present in wheat, rye

52 a case of EATL complicating adult autoimmune enteropathy (AIE).

53 is factor (TNF) in graft-versus-host disease enteropathy, an adenoviral vector encoding a TNF inhibit

54 cations for the pathogenesis of TNF-mediated enteropathies and chronic inflammatory diseases of the i

55 ions, and 1 patient each with protein-losing enteropathy and ascites.

56 microbial component, including environmental enteropathy and chronic colitis-associated colorectal ca

57 c has the potential to resolve environmental enteropathy and clear bacterial pathogens, we aimed to a

58 ritize interventions to reduce environmental enteropathy and diarrhea.

59 eric viral infections may contribute to AIDS enteropathy and disease progression.

60 ome, which may contribute to AIDS-associated enteropathy and disease progression.

61 r human X-linked neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome (IPEX; MIM 30493

62 developments in the area of acute radiation enteropathy and examine the current state of knowledge r

63 d allergic enterocolitis with protein-losing enteropathy and had low birth weight.

64 s to be the predominant isoform in radiation enteropathy and may be more important in the mechanisms

65 IV/pigtailed macaque model of HIV to examine enteropathy and microbial translocation.

66 of Gla KO mice provides a model for study of enteropathy and neuropathy in Fabry disease.

67 nents of the small intestinal microbiota and enteropathy and offer a rationale for developing therapi

68 ding death, heart transplant, protein losing enteropathy and plastic bronchitis.

69 profound congenital sensorineural deafness, enteropathy and renal tubular dysfunction.

70 Enteropathy and small-intestinal ulcers are common adver

71 despite reducing biomarkers of environmental enteropathy and the prevalence of pathogenic intestinal

72 etes (T1D), but a direct causal link between enteropathy and triggering of autoimmunity is yet to be

73 Enteric infections, enteropathy and undernutrition in early childhood are pr

74 This could be a mechanism of SIV-associated enteropathy and viral pathogenesis.

75 s herpetiformis (DH) have a gluten-sensitive enteropathy and while on gluten-containing diets have el

76 iated with the development of protein-losing enteropathy and with decreased survival.

77 by immune dysregulation, polyendocrinopathy, enteropathy and X-linked inheritance (MIM 304930).

78 correlated with all parameters of radiation enteropathy and, after adjusting for radiation dose and

79 sive means of detecting and monitoring NSAID enteropathy (and possibly other gastrointestinal mucosal

80 entified 5 children with polyendocrinopathy, enteropathy, and dermatitis reminiscent of IPEX syndrome

81 munity, polyclonal lymphocytic infiltration, enteropathy, and lymphoid malignancy.

82 ve regurgitation, arrhythmia, protein-losing enteropathy, and plastic bronchitis are potential compli

83 t presented with lymphedema, protein loosing enteropathy, and sclerosing cholangitis and was diagnose

84 Immune dysregulation, polyendocrinopathy, enteropathy, and X-linked inheritance (IPEX) is one of a

85 d by immune-dysfunction, polyendocrinopathy, enteropathy, and X-linked inheritance.

86 se immune dysregulation, polyendocrinopathy, enteropathy, and X-linked syndrome (IPEX).

87 ave immunodysregulation, polyendocrinopathy, enteropathy, and X-linked syndrome.

88 alabsorption caused by chronic environmental enteropathy are associated with growth faltering seen in

89 inal translocation of microbial products and enteropathy as well as alterations in gut bacterial comm

90 trast enhanced ultrasound (CEUS) findings in enteropathy associated T-cell lymphoma (EATL) complicati

91 Enteropathy associated T-cell lymphoma (EATL) is a rare

92 ory coeliac disease (RCD) type I and II, and enteropathy associated T-cell lymphoma (EATL).

93 he intestinal microbiota might contribute to enteropathy associated with use of nonsteroidal anti-inf

94 Enteropathy-associated T-cell lymphoma (EATL) is a compl

95 Enteropathy-associated T-cell lymphoma (EATL) is a rare

96 Enteropathy-associated T-cell lymphoma (EATL) is a rare

97 Enteropathy-associated T-cell lymphoma (EATL) is a rare

98 The refractory state itself and enteropathy-associated T-cell lymphoma (EATL) were the m

99 tions of celiac disease that may progress to enteropathy-associated T-cell lymphoma (EATL).

100 r magnetic resonance enterography to exclude enteropathy-associated T-cell lymphoma and ulcerative je

101 ac disease) appears to be a manifestation of enteropathy-associated T-cell lymphoma in most cases.

102 tion-to-treat analysis in 252 nodal PTCL and enteropathy-associated T-cell lymphoma patients (excludi

103 negative anaplastic large cell lymphoma, and enteropathy-associated T-cell lymphoma were enrolled.

104 Enteropathy-associated T-cell lymphoma, an often fatal c

105 ties, with the exception of 2 of 10 cases of enteropathy-associated T-cell lymphoma.

106 patient with the characteristic phenotype of enteropathy-associated T-cell lymphoma.

107 me clonally expanded in refractory sprue and enteropathy-associated T-cell lymphoma.

108 of refractory celiac disease and to exclude enteropathy-associated T-cell lymphoma.

109 fections and their associated complications, enteropathy, autoimmunity, and lymphoproliferative disor

110 Enteropathy, autoimmunity, granulomas, and splenomegaly

111 data supporting the notion of protein-losing enteropathy being a consequence of severe iron deficienc

112 fections, lymphoproliferation, autoimmunity, enteropathy, bronchiectasis, increased risk of lymphoma,

113 is and management of patients with suspected enteropathy, but negative results from serologic tests f

114 the nutritional components of environmental enteropathy by analyzing the specific metabolic and gut-

115 ly infection the viruses themselves cause an enteropathy by heretofore undetermined mechanisms.

116 n reduced faecal biomarkers of environmental enteropathy (calprotectin, myeloperoxidase, alpha1-antit

117 t interactions, anorexia, and protein-losing enteropathy can all contribute to the protein-calorie ma

118 and excluded, a unique entity known as AIDS enteropathy can be diagnosed.

119 results are encouraging, and protein-losing enteropathy can be expected to resolve.

120 Although AIDS enteropathy can pose a diagnostic challenge so too does

121 o safely reduce the risk of small-intestinal enteropathy caused by ASA.

122 Coeliac disease (CD), an enteropathy caused by cereal gluten ingestion, is charac

123 disease is a complex, polygenic inflammatory enteropathy caused by exposure to dietary gluten that oc

124 damage, lymphangiectasia, and protein-losing enteropathy caused by overactivation of the complement s

125 The gluten-sensitive enteropathy celiac disease is tightly associated with th

126 et of autoantibodies in the gluten-sensitive enteropathy celiac disease.

127 (TG2) are a hallmark of the gluten-sensitive enteropathy celiac disease.

128 Lymphocytic enteropathy (celiac disease and lymphocytic duodenosis)

129 mmunodeficiency-associated and environmental enteropathies, celiac disease, inflammatory bowel diseas

130 In addition to the characteristic enteropathy, celiac disease is associated with various e

131 , angiopathic thrombosis, and protein-losing enteropathy (CHAPLE) is an ultra-rare genetic disorder c

132 disease is a gluten-induced immune-mediated enteropathy characterized by a specific genetic genotype

133 Coeliac disease is a common enteropathy characterized by an increased mortality main

134 eliac disease is a gluten-induced autoimmune enteropathy characterized by the presence of tissue tran

135 l lymphangiectasia (PIL) is a protein-losing enteropathy characterized by tortuous and dilated lymph

136 me, rejection particularly chronic allograft enteropathy continues to be a major barrier to long-term

137 a for prevention and treatment of radiation, enteropathy could become a reality and would be of subst

138 Syndromic congenital tufting enteropathy (CTE) is a life-threatening recessive human

139 Congenital tufting enteropathy (CTE) is a rare autosomal recessive diarrhea

140 Congenital tufting enteropathy (CTE) is a severe autosomal recessive human

141 etes (T1D) often experience gastrointestinal enteropathy (DE) of unclear etiology.

142 ly have intestinal symptoms, termed diabetic enteropathy (DE), though its etiology is unknown.

143 MEDNIK syndrome (mental retardation, enteropathy, deafness, peripheral neuropathy, ichthyosis

144 nd histopathological injury during radiation enteropathy development.

145 e from a patient with attaching-and-effacing enteropathy displayed the localized adherence attaching-

146 intestine likely plays a role in intestinal enteropathy during HIV infection.

147 Environmental enteropathy (EE) impairs the gut's absorptive capacity a

148 Environmental enteropathy (EE) is an asymptomatic abnormality of small

149 Environmental enteropathy (EE) refers to villus blunting, reduced abso

150 re collectively referred to as environmental enteropathy (EE).

151 is model, we found that Salmonella-inflicted enteropathy elicits parallel blooms of the pathogen and

152 genital syndrome characterized by autoimmune enteropathy, endocrinopathy, dermatitis, and other autoi

153 a useful drug in the treatment of autoimmune enteropathy, even in patients who have not responded to

154 tinal permeability with diarrhea, called HIV enteropathy, even without enteric opportunistic infectio

155 and allergic manifestations including severe enteropathy, food allergies, atopic dermatitis, hyper-Ig

156 ctocolitis (FPIAP), and food protein-induced enteropathy (FPE).

157 pecimens from patients with gluten-sensitive enteropathy (GSE) (obtained during gluten challenge) as

158 Classically, enteropathy has been identified histopathologically.

159 tinal pathogens and associated environmental enteropathy has been proposed to explain this problem.

160 ept of extraintestinal presentations without enteropathy has only recently become accepted.

161 es with the clinical manifestations of NSAID enteropathy, however, remains unknown.

162 s of heart failure death were protein-losing enteropathy (HR, 7.1; P=0.0043), single morphologically

163 oody diarrhea, malabsorption, protein-losing enteropathy, hypoalbuminemia, and failure to thrive.

164 idosis presenting with severe protein-losing enteropathy, hypoalbuminemia, and proximal myopathy who

165 ficant problem worldwide, with environmental enteropathy implicated as a contributing factor.

166 n gastrointestinal tract, eventually causing enteropathies in predisposed individuals.

167 el syndrome in 12 patients (27%), intestinal enteropathy in 20 patients (44%), and motility disorder

168 NO may therefore be an important mediator of enteropathy in both Th1- and Th2-inducing conditions.

169 The enteropathy in complicated SAM did not respond to either

170 Enteropathy in HIV infection is not eliminated with comb

171 a role in the pathogenesis of GI disease and enteropathy in HIV-infected people.

172 ibute to the development of NSAID-associated enteropathy in human beings.

173 liac disease is the most common food-induced enteropathy in humans, with a prevalence of approximatel

174 wasting syndrome characterized by severe SIV enteropathy in the absence of opportunistic infections.

175 TNF inhibition decreases the severity of enteropathy in the DBA/2 --> B6D2F1 murine model of colo

176 marker and supports a role for environmental enteropathy in the pathogenesis of growth shortfall.

177 es in adults and children with environmental enteropathy in Zambia and Tanzania over 24 y, alongside

178 ear swelling (in model 1), gluten-dependent enteropathy (in model 3), and body weight loss (in model

179 a) model of diarrhea, causing characteristic enteropathies, including inflammation, necrosis, and col

180 rated structural manifestations of radiation enteropathy, including radiation injury score (6.5 +/- 0

181 iac disease (CD), a T helper 1 cell-mediated enteropathy induced by gluten.

182 l administration of anti-CD3 ameliorated the enteropathy induced by intraperitoneal injection of the

183 present controlled study, the effect of the enteropathy induced by this organism on the retinal vasc

184 Enteropathy is a frequent complication of diclofenac and

185 Nonsteroidal antiinflammatory drug enteropathy is a stepwise process involving direct mucos

186 NSAID enteropathy is a stepwise process involving direct mucos

187 Tropical enteropathy is an asymptomatic villous atrophy of the sm

188 Autoimmune enteropathy is characterized by chronic secretory diarrh

189 cosal IL-10 in SIV/HIV infection in inducing enteropathy is not well understood.

190 ng the pathophysiology and diagnosis of this enteropathy is the difficulty of obtaining information a

191 luten-free diet, and for whom no etiology of enteropathy is ultimately identified, should be treated

192 The molecular basis of protein-losing enteropathy is unknown.

193 HIV-associated enteropathy is well-documented in chronic HIV-1 infectio

194 Childhood gut dysfunction (enteropathy) is common in resource-poor environments.

195 Celiac disease (CD), or gluten-sensitive enteropathy, is a common multifactorial disorder resulti

196 Celiac Sprue, or gluten-sensitive enteropathy, is an inheritable human disease of the smal

197 Coeliac disease, or gluten-sensitive enteropathy, is only one aspect of a range of possible m

198 sociated autoimmune disease gluten-sensitive enteropathy, leaving little room for a differential envi

199 oimmunity and alopecia, a condition we named enteropathy-lymphocytopenia-alopecia.

200 This enteropathy may appear at any age and is characterized b

201 us, suggesting that EE with superimposed HIV enteropathy may be a distinct pathophysiological conditi

202 HIV enteropathy may result from direct effects of HIV on gas

203 Myosin-5B malfunction causes the congenital enteropathy microvillus inclusion disease, underlining i

204 nic (model 2), and the gliadin memory T-cell enteropathy (model 3) models of celiac disease, intraven

205 endothelial cells and in vivo in a radiation enteropathy mouse model confirm that genes involved in N

206 1), thrombocyte counts (n = 1), and chronic enteropathy (n = 1).

207 s trigger celiac disease (CD), an autoimmune enteropathy occurring in genetically susceptible persons

208 ole in the pathogenesis of acute and chronic enteropathies of dogs, including idiopathic inflammatory

209 wed characteristics typical of proliferative enteropathies of other animals, i.e., intracellular colo

210 Enteropathy of the small intestine developed in gnotobio

211 completed the study, 119 (73%) had tropical enteropathy on enrollment (L:M > 0.10).

212 Patients with enteropathy or intestinal mucosal inflammation (associat

213 Children with enteropathy or intestinal mucosal inflammation are at gr

214 findings (eg, heart failure, protein-losing enteropathy, or new arrhythmias), and somatic growth.

215 In mice with enteropathy, oral anti-CD3 reduced levels of inflammator

216 HEV seropositivity was associated with worse enteropathy (P = .05 and P = .005, respectively).

217 significantly shorter than patients without enteropathy (P = 0.007).

218 severe complications such as protein-losing enteropathy, plastic bronchitis, and peripheral edema th

219 Heart Association III/IV, or protein-losing enteropathy/plastic bronchitis) 20 years after Fontan wa

220 chronic effusions (n = 4) or protein-losing enteropathy (PLE) (n = 5) after lateral tunnel-type Font

221 kedown was required in 3% and protein-losing enteropathy (PLE) developed in 6%.

222 Patients with protein-losing enteropathy (PLE) fail to maintain intestinal epithelial

223 Patients with protein-losing enteropathy (PLE) following the Fontan operation have a

224 Protein-losing enteropathy (PLE) is a rare syndrome of gastrointestinal

225 Protein-losing enteropathy (PLE), characterized by loss of proteins in

226 Protein-losing enteropathy (PLE), the loss of plasma proteins through t

227 fficiency, and development of protein-losing enteropathy (PLE).

228 Protein-losing enteropathy (present in 34 patients) resolved in all who

229 ac disease (CD) is an increasingly diagnosed enteropathy (prevalence, 1:200-1:300) that is induced by

230 Both experimental enteropathies produced mucosal damage, although injury w

231 Although environmental enteropathy reduces microbial translocation, it does so

232 crolimus (FK506) in patients with autoimmune enteropathy refractory to steroids and cyclosporine.

233 The histology of tropical enteropathy resembles that seen in small-bowel bacterial

234 ight to crypt depth, video capsule endoscopy enteropathy score, enzyme-linked immune absorbent spot,

235 year-old boy presented with a protein-losing enteropathy secondary to a hypertrophic gastropathy.

236 howed that in those patients with ataxia and enteropathy, separate antibody populations react with th

237 gative patients with an identified cause for enteropathy should be treated accordingly; a follow-up b

238 nifestation (67%), followed by growth delay, enteropathy, skin disease, pulmonary disease, endocrinop

239 ercise capacity, arrhythmias, protein-losing enteropathy, somatic growth retardation, neo-aortic valv

240 ogenesis of tissue-specific inflammation and enteropathies such as CD.

241 and SCL in cats share features with chronic enteropathies such as IBD and monomorphic epitheliotropi

242 Celiac disease (CD) is a gluten-sensitive enteropathy that develops in genetically susceptible ind

243 medications in the world, yet they induce an enteropathy that is associated with high morbidity and m

244 , angiopathic thrombosis, and protein-losing enteropathy (the CHAPLE syndrome) is caused by abnormal

245 After ruling out other seronegative enteropathies, the diagnosis can be confirmed by both cl

246 sal gene expression contribute to intestinal enteropathy, the role of small noncoding RNAs, specifica

247 entricular failure, cyanosis, protein-losing enteropathy, thromboembolism, and dysrhythmias often lea

248 g-term follow-up of patients with autoimmune enteropathy treated with tacrolimus is currently availab

249 Celiac disease is an enteropathy triggered by dietary gluten found in wheat,

250 Celiac disease is an immune-mediated enteropathy triggered by gliadin, a component of the gra

251 Celiac disease (CD) is an immune-mediated enteropathy triggered by gluten in genetically susceptib

252 Celiac disease (CD) is an immune-mediated enteropathy triggered by the ingestion of gluten-contain

253 Celiac disease (CD) is an autoimmune enteropathy triggered in susceptible individuals by the

254 treatment of 31 patients with a diagnosis of enteropathy-type intestinal T-cell lymphoma treated at t

255 The risk is highest for enteropathy-type T-cell lymphoma (ETL) and B-cell lympho

256 nts believed to have celiac disease or other enteropathies unrelated to gluten, but negative results

257 s, showed that in addition to protein-losing enteropathy, ventricular indexed end-diastolic volume >1

258 Radiation enteropathy was also associated with sustained shifts in

259 In the ileum and colon, enteropathy was associated with increased caspase-3 stai

260 A previously undescribed enterococcal enteropathy was associated with preretinal neovasculariz

261 ith clinical parameters only, protein-losing enteropathy was associated with transplantation-free sur

262 Gluten-sensitive enteropathy was found in 24%.

263 yanosis was present in 7, and protein-losing enteropathy was present in 4.

264 ium and the causative agent of proliferative enteropathy, was developed using an Original Space Bag i

265 In contrast the patient with an autoimmune enteropathy, was HLA-DQ9/DQ6-positive, also arguing agai

266 To identify candidate etiologies for AIDS enteropathy, we used next-generation sequencing to defin

267 ss, and physiological changes resembling HIV enteropathy were previously found in the HT-29 intestina

268 Environmental enteropathy, which is linked to undernutrition and chron

269 ncy virus (SIV) infection is associated with enteropathy, which likely contributes to AIDS progressio

270 Three patients with diagnosed autoimmune enteropathy who continued to have intractable diarrhea d

271 DDs) are a collection of rare, heterogeneous enteropathies with early onset and often severe outcomes

272 nclusion disease (MVID) is a rare intestinal enteropathy with an onset within a few days to months af

273 iarrhea caused by early-onset protein-losing enteropathy with primary intestinal lymphangiectasia, ed

274 The clinical effects of gluten-sensitive enteropathy with villous atrophy limited to the duodenal

275 ents we detect a primary immunodeficiency or enteropathy, with clinical consequences (XIAP, CYBA, SH2

276 e and immunodysregulation polyendocrinopathy enteropathy X-linked (IPEX) patients.

277 PEX (immune dysregulation polyendocrinopathy enteropathy X-linked) syndrome patient with a FOXP3 muta

278 nt of immunodysregulation polyendocrinopathy enteropathy X-linked-like syndrome.

279 of immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) and IPEX-related disorders.

280 Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome is a devastating a

281 Immune-dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome is a lethal diseas

282 Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome is a rare, fatal a

283 th immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, associated with a

284 ith immunodysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, the human disease

285 de immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, which is caused b

286 he immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome.

287 se immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX).

288 Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX; OMIM 304930) syndrome is a

289 he immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) and found them to

290 of immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) with or without FO

291 ome (immune dysfunction, polyendocrinopathy, enteropathy, X-linked syndrome) in humans.

292 EX (immunodysregulation, polyendocrinopathy, enteropathy, X-linked syndrome) mutations.

293 the immune dysregulation polyendocrinopathy, enteropathy, X-linked syndrome, or X-linked autoimmunity

294 nd immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome.

295 me; immunodysregulation, polyendocrinopathy, enteropathy, X-linked syndrome; Crohn's disease; and fam

296 X (immune dysregulation, polyendocrinopathy, enteropathy, X-linked) patients.

297 X (immune dysregulation, polyendocrinopathy, enteropathy, X-linked) patients.

298 X (immune dysregulation, polyendocrinopathy, enteropathy, X-linked), caused by a lack of regulatory T

299 nd immune dysregulation, polyendocrinopathy, enteropathy, X-linked-like syndrome.

300 d an immune dysregulation-polyendocrinopathy-enteropathy-X-linked (IPEX)-like phenotype for STAT1 mut