コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 g colonization factor antigen I (CFA/I) from enterotoxigenic Escherichia coli.
2 s an important virulence factor expressed by enterotoxigenic Escherichia coli.
3 cally secreted onto the surface of wild type enterotoxigenic Escherichia coli.
4 essing colonization factor Ag I (CFA/I) from enterotoxigenic Escherichia coli.
5 detoxified form of the heat-labile toxin of enterotoxigenic Escherichia coli.
6 ppendages found on the surface of strains of enterotoxigenic Escherichia coli.
7 Although FoodNet surveillance does not cover enterotoxigenic Escherichia coli, a common travel-associ
9 ization factor antigen I fimbriae (CFA/I) of enterotoxigenic Escherichia coli and is thought to be es
11 , particularly for noroviruses, rotaviruses, enterotoxigenic Escherichia coli, and Campylobacter jeju
12 n (nCT) and the heat-labile toxin 1 (nLT) of enterotoxigenic Escherichia coli are AB5-type enterotoxi
13 olerae and heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli are notorious for their
17 ted from infection with the enteric pathogen enterotoxigenic Escherichia coli by maternal natural IgG
18 (3) across all targets including heat-stable enterotoxigenic Escherichia coli, Campylobacter jejuni,
20 olerae toxin-coregulated pilus (TCP) and the enterotoxigenic Escherichia coli CFA/III pilus each medi
22 i-inflammatory Salmonella vaccine expressing enterotoxigenic Escherichia coli colonization factor Ag
25 ercooked scallops (three outbreaks caused by enterotoxigenic Escherichia coli), eggs (two outbreaks c
27 e with the heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli elicited the spectrum o
28 were due to rotavirus, Cryptosporidium spp, enterotoxigenic Escherichia coli encoding heat-stable to
29 ng likely etiologies were rotavirus (21.1%), enterotoxigenic Escherichia coli encoding heat-stable to
30 ity against influenza, shigellosis, cholera, enterotoxigenic Escherichia coli, enteropathogenic E. co
31 the last 20 years; Campylobacter (8.36%) and Enterotoxigenic Escherichia coli (ETEC) (7.15%) were the
32 were positive for Shigella sonnei (n = 66), enterotoxigenic Escherichia coli (ETEC) (n = 31) or nega
34 ubstrates: heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli (ETEC) and cholera toxi
35 luated to detect Shigella spp (Shigella) and enterotoxigenic Escherichia coli (ETEC) and determine th
39 dity, and childhood stunting attributable to enterotoxigenic Escherichia coli (ETEC) and shigella inf
41 neri 2a strain CVD 1203 as a live vector for enterotoxigenic Escherichia coli (ETEC) antigens is repo
45 used to detect Shigella spp. (Shigella) and enterotoxigenic Escherichia coli (ETEC) are complex or e
46 CS1 is the prototype of a class of pili of enterotoxigenic Escherichia coli (ETEC) associated with
53 ation factor antigen I (CFA/I), archetype of enterotoxigenic Escherichia coli (ETEC) Class 5 fimbriae
60 fic activity against colonization factors of enterotoxigenic Escherichia coli (ETEC) could provide pa
62 ity of a recombinant subunit vaccine against enterotoxigenic Escherichia coli (ETEC) delivered by TCI
63 ype of eight genetically related fimbriae of enterotoxigenic Escherichia coli (ETEC) designated class
66 of an animal model that closely approximates enterotoxigenic Escherichia coli (ETEC) disease in human
67 id vectors, we designed SC608 to express the enterotoxigenic Escherichia coli (ETEC) fimbrial subunit
70 nd heat-labile (LT) enterotoxins produced by enterotoxigenic Escherichia coli (ETEC) have been docume
73 n (LT) provides a colonization advantage for enterotoxigenic Escherichia coli (ETEC) in vivo, we hypo
95 oral vaccine against both Shigella spp. and enterotoxigenic Escherichia coli (ETEC) is being develop
102 antigen expressed in nearly 20% of clinical enterotoxigenic Escherichia coli (ETEC) isolates, is com
103 with clinical and epidemiologic features of enterotoxigenic Escherichia coli (ETEC) occurred among p
104 safety and immunogenicity of an oral, killed enterotoxigenic Escherichia coli (ETEC) plus cholera tox
111 coded on an apparent pathogenicity island of enterotoxigenic Escherichia coli (ETEC) strain H10407, m
120 mber of serologically distinct pili found in enterotoxigenic Escherichia coli (ETEC) strains associat
122 B/c mice by intranasal (i.n.) inoculation of enterotoxigenic Escherichia coli (ETEC) strains H10407 (
128 olonize the small intestine is essential for enterotoxigenic Escherichia coli (ETEC) to cause diarrhe
130 specific polymerase chain reaction (PCR) for enterotoxigenic Escherichia coli (ETEC) toxins after cha
132 terotoxin (LT) is retained on the surface of enterotoxigenic Escherichia coli (ETEC) via an interacti
133 er membrane protein NlpA is repressed by the enterotoxigenic Escherichia coli (ETEC) virulence regula
134 uster of the CS18 (PCFO20) fimbriae of human enterotoxigenic Escherichia coli (ETEC) was found to inc
138 for many Gram-negative pathogens, including enterotoxigenic Escherichia coli (ETEC), a major cause o
139 stinal colonization and diarrheal disease by enterotoxigenic Escherichia coli (ETEC), an E. coli path
141 virus, sapovirus, astrovirus, and rotavirus, enterotoxigenic Escherichia coli (ETEC), Campylobacter s
143 olunteer challenges with Vibrio cholerae O1, enterotoxigenic Escherichia coli (ETEC), enteropathogeni
145 rototype hybrid vaccine against Shigella and enterotoxigenic Escherichia coli (ETEC), the genes encod
146 ing challenge in developing vaccines against enterotoxigenic Escherichia coli (ETEC), the most common
147 ections caused by the gram-negative bacteria enterotoxigenic Escherichia coli (ETEC), Vibrio cholerae
148 or the detection of Yersinia enterocolitica, enterotoxigenic Escherichia coli (ETEC), Vibrio, and Ple
156 cter jejuni, Salmonella spp., Shigella spp., enterotoxigenic Escherichia coli [ETEC], Shiga toxin-pro
159 s) were fused to a rotavirus enterotoxin and enterotoxigenic Escherichia coli fimbrial antigen genes
160 otein was used to isolate DNA fragments from enterotoxigenic Escherichia coli genomic DNA that carry
163 sed Organ-Chips (Intestine-Chips) exposed to enterotoxigenic Escherichia coli heat-stable enterotoxin
164 itive stool samples also tested positive for enterotoxigenic Escherichia coli, indicating that dual i
166 , and an inverse relationship exists between enterotoxigenic Escherichia coli infections producing th
168 Heat-labile enterotoxin (LT), produced by enterotoxigenic Escherichia coli, is a close relative of
170 f HRV against acute diarrhea associated with enterotoxigenic Escherichia coli; it was 4.0% (95% CI, -
172 The tip adhesin FasG of the 987P fimbriae of enterotoxigenic Escherichia coli mediates two distinct a
174 igs infected with hemolytic F4(+) strains of enterotoxigenic Escherichia coli often develop septicemi
175 lobacter, Salmonella, and Vibrio species and enterotoxigenic Escherichia coli), only 24% were suscept
177 four pathogens: rotavirus, Cryptosporidium, enterotoxigenic Escherichia coli producing heat-stable t
178 ns (PAFs) shows that Shigella (PAF, 11%) and enterotoxigenic Escherichia coli producing labile toxin
180 g colonization factor antigen I (CFA/I) from enterotoxigenic Escherichia coli results in the rapid on
181 n transposon mutants from a single colony of enterotoxigenic Escherichia coli, Salmonella typhimurium
182 enteric pathogens include Ascaris, Giardia, enterotoxigenic Escherichia coli, Shigella, and Campylob
183 detection rates >20% were found for each of enterotoxigenic Escherichia coli, Shigella, Campylobacte
185 CS1 pili are important virulence factors of enterotoxigenic Escherichia coli strains associated with
187 serologically distinct pili associated with enterotoxigenic Escherichia coli that cause diarrhoea in
190 Entamoeba histolytica, Salmonella enterica, enterotoxigenic Escherichia coli, Vibrio cholerae, Campy
191 here is no evidence that Rns, a regulator of enterotoxigenic Escherichia coli virulence genes, respon
192 ain, which degrades enterocyte receptors for enterotoxigenic Escherichia coli, was shown in an experi
193 mpylobacter, and heat-stable toxin-producing enterotoxigenic Escherichia coli were the leading causes