戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  (11 of 20 patients; P < 0.001 versus distal enthesis).
2 maturation of the tendon-to-bone attachment (enthesis).
3 the CESS on the mechanical properties of the enthesis.
4 stions about the healing capabilities of the enthesis.
5 traction to elicit in vivo adaptation of the enthesis.
6 between the tendon and cartilage ends of the enthesis.
7 okines have also been described in the human enthesis.
8  bone across a specialized tissue called the enthesis.
9  utero eventually populate the entire mature enthesis.
10  synovial invasion (pannus formation) of the enthesis.
11  were present on the soft tissue side of the enthesis.
12 t the presence of small blood vessels at the enthesis.
13 uch as enthesopathies, which localize to the enthesis.
14  promoted chondrogenesis in joint cavity and enthesis.
15  the structure and function of the postnatal enthesis.
16 muscle forces from tendon to bone across the enthesis.
17 e periodontal ligament (PDL) functions as an enthesis, a connective tissue attachment that dissipates
18                             For example, the enthesis, a fibrocartilage tissue that connects tendon t
19                                          The enthesis, a fibrocartilaginous tissue connecting tendon
20                                          The enthesis, a fibrocartilaginous transition between tendon
21                     However, injuries to the enthesis, a hypocellular and avascular tissue, often res
22 yet little is known if and how the postnatal enthesis adapts to increased loading.
23 tion and materials can be used to repair the enthesis after injury.
24 eness to Ihh, from the developing tendon and enthesis altered the differentiation of enthesis progeni
25 ng defective bone morphometry underlying the enthesis and decreased mechanical properties.
26 cuss the known biology of IL-23 at the human enthesis and highlight the remarkable emerging story of
27 s specialized ECM organization at the tendon enthesis and its ability to transmit force.
28 mmatory arthritis that primarily affects the enthesis and may culminate in bony ankylosis of the spin
29 sites: the proximal and distal halves of the enthesis and the adjacent calcaneal superior tuberosity.
30 nt inflammatory changes in ligament, tendon, enthesis, and adjacent bone in the DIP joint disease of
31 t of musculoskeletal tissues such as tendon, enthesis, and bone relies on proliferation and different
32 ctor mediating inflammation in the synovium, enthesis, and bone.
33 ssing cells were ensconced in healthy murine enthesis, and other extraskeletal anchorage points inclu
34 occur at the tendon-to-bone interface (i.e., enthesis) area, with a high prevalence for the elderly p
35 bined ultrasonography of the Achilles tendon enthesis at different stages of spondylarthritis (SpA) w
36 lysis to describe IC subtypes present in the enthesis before, during, and after mineralization, and t
37                                          The enthesis bone and soft tissues have radically different
38 t in general, especially the hypothesis that enthesis-bone architecture may be important in disease i
39  presence of activated hedgehog signaling in enthesis cells early in the healing process may enhance
40 ated: (1) the significant alterations of the enthesis composition and structure occur from the newbor
41                                          The enthesis connects tendon and bone, two vastly different
42 sed the number of Hh-responsive cells in the enthesis, demonstrating that responsiveness to Hh is mod
43 he necessity of Gli1-lineage cells for mouse enthesis development and healing, we then examined their
44 oading, the biological factors that modulate enthesis development are poorly understood.
45  presence, and role of immune cells (ICs) in enthesis development is not known.
46               Loss of muscle loading impairs enthesis development, yet little is known if and how the
47 ired for tissue maturation, homeostasis, and enthesis development.
48 t the potential role of FGF signaling during enthesis development.
49            A better understanding of how the enthesis develops will lead to more effective therapies
50 cx) and Sox9, directly control production of enthesis ECM components.
51 iption of a direct target of Scxa, Col1a, in enthesis ECM is regulated by the ratio of scxa to sox9a
52                     Regional organization of enthesis extracellular matrix (ECM) generates difference
53 onstrate that Hh signaling within developing enthesis fibrocartilage cells is required for enthesis f
54 ascular invasion, which occurs along rows of enthesis fibrocartilage cells.
55 nd they form by endochondral ossification of enthesis fibrocartilage.
56                                          The enthesis for many tendons consists of a mineralized grad
57 population of resident lymphoid cells at the enthesis for the first time offers an explanation for th
58 ling in these cells led to defects in tendon enthesis formation by P56, including defective bone morp
59 ow mechanical stimulation impacts tendon and enthesis formation during development, as well as tendon
60 nthesis fibrocartilage cells is required for enthesis formation.
61              Current clinical treatments for enthesis healing remain limited.
62 ent of Gli1+ cells and hedgehog signaling in enthesis healing, Gli1 expression was examined via linea
63 mice to study the effectiveness of early-age enthesis healing.
64 y and impaired maturation of Achilles tendon enthesis in Sparc (-/-) mice.
65  important for functional development of the enthesis, including its postnatal mineralization.
66 n the healing process may enhance healing of enthesis injuries by mimicking developmental processes.
67 ansplantation of Gli1-lineage cells to mouse enthesis injuries improved healing, demonstrating their
68 lead to new therapeutic strategies for adult enthesis injuries.
69 ibrocartilage healing across multiple animal enthesis injury models, including mice, rats, rabbits, a
70 lized collagen scaffold effectively promotes enthesis injury repair, demonstrating potential for clin
71        The development and maturation of the enthesis involve cellular processes likened to an arrest
72                                          The enthesis is a transitional tissue between tendon and bon
73                                          The enthesis is prone to injury due to mechanical demands, a
74 tendon-bone interface otherwise known as the enthesis, is crucial to the movement of the skeleton.
75 cells, to disturbances in the ratio of M1/M2 enthesis macrophages, to CD8 + T cell mediated inflammat
76                                              Enthesis microdamage (fissuring) as well as vascular and
77 esence of inflammatory cells and evidence of enthesis microdamage.
78 ions between ICs and MCs may be important to enthesis mineralization, and suggest that progenitor MCs
79 don and enthesis (Scx-Cre) and characterized enthesis morphology as well as mechanical properties in
80 maller, with altered bone shape and impaired enthesis morphology, morphometry, and organization.
81                                              Enthesis morphometry, histology, and collagen alignment
82 ry chondrogenesis in the mandibular adductor enthesis of duck versus quail.
83  secondary cartilage at the insertion (i.e., enthesis) of the mandibular adductor muscles on the lowe
84 ns unclear whether IL-23 acts locally at the enthesis or distally on circulating cell populations.
85  was to demonstrate the applicability of the enthesis organ concept to other insertion sites.
86                            The concept of an enthesis organ is of general significance in understandi
87 ts there were also inflammatory cells in the enthesis organ itself.
88                  Finally, the concept of an 'enthesis organ' may explain the magnetic resonance imagi
89                   The presence and extent of enthesis organs was evaluated at each site in serial sec
90 ll soft tissue components of the entheses or enthesis organs were examined.
91                                    Articular enthesis organs were found at 14 entheses, including the
92                               Extraarticular enthesis organs were seen at 2 sites, the biceps brachii
93 e to which other insertions form comparable "enthesis organs" has not been established.
94                                       In all enthesis organs, sesamoid and/or periosteal fibrocartila
95                                   The tendon enthesis originates from a specific pool of hedgehog-act
96 evelop when ossification at one point on the enthesis outstrips that on either side.
97 the development of treatments for tendon and enthesis pathologies.
98  and enthesis altered the differentiation of enthesis progenitor cells, resulting in significantly re
99           Six resident cell types, including enthesis progenitors and mineralizing chondrocytes, were
100 d micro-organisms, should be assessed at the enthesis rather than in the synovium.
101 emonstrating their therapeutic potential for enthesis regeneration.
102 entage of inorganic content as scaffolds for enthesis repair.
103 ditionally knockout Fgf9 in mouse tendon and enthesis (Scx-Cre) and characterized enthesis morphology
104         Ion channel inhibition did not alter enthesis secondary cartilage but caused bone to form in
105                 Thus, our study reveals that enthesis secondary cartilage forms through mechanisms th
106 bling that of quail, including an absence of enthesis secondary cartilage.
107         An electron microscopic study of the enthesis site-where the extraocular muscle tendon insert
108 isease patterns in SpA are related to normal enthesis structure and biomechanics.
109              Here, we studied adaptations in enthesis structure and function in response to increased
110 usion, deletion of Scx led to impairments in enthesis structure, which translated into impaired funct
111                                              Enthesis tensile mechanical properties were also assesse
112  uncovered a fibrous architecture within the enthesis that controls trade-offs between strength and t
113 Hh-responsive cells in the developing murine enthesis that were distinct from tendon fibroblasts and
114 cause of the relative inaccessibility of the enthesis, the inflammatory, microbiologic, and immunolog
115  were noted on the soft tissue side of every enthesis; the most common changes were cell clustering a
116 issues, we identified putative mechanisms of enthesis toughening in a mouse model and then manipulate
117 he cortical shell is an integral part of the enthesis, transferring load to an extensive skeletal reg
118 spatial expression of Fgf9 in the tendon and enthesis using in situ hybridization.
119  On MRI, edema within the soft tissue at the enthesis was evident in both groups.
120 ere present almost exclusively at the distal enthesis, were evident in patients with early SpA and in
121 ration of proinflammatory mediators from the enthesis, whereas the synovitis of rheumatoid arthritis
122                    This also led to a weaker enthesis with increased collagen damage in young tendon
123 iomechanical alterations of the rotator cuff enthesis with maturation and aging in a mouse model.

 
Page Top