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1 ated by levels of the specific enzyme marker eosinophil peroxidase.
2 that is dependent on the presence of active eosinophil peroxidase.
3 g and internalizing both myeloperoxidase and eosinophil peroxidase.
4 g the presence of DNA traps colocalized with eosinophil peroxidase.
5 id not change inflammatory markers IL-16 and eosinophil peroxidase.
6 d significant enrichments in eosinophils and eosinophil peroxidase.
7 10 secretory tissues stained only weakly for eosinophil peroxidase.
8 Western blotting to evaluate the presence of eosinophil peroxidase (a marker of eosinophil degranulat
9 in vitro and in vivo led to degranulation of eosinophil peroxidase, a granule protein whose enzymatic
11 d fsp-1) and bronchial alveolar lavage fluid eosinophil peroxidase activity differentially increased
12 s the synthesis of OVA-specific IgE and skin eosinophil peroxidase activity in mice with ongoing skin
14 e endometrium and smaller effects on uterine eosinophil peroxidase activity than nafoxidine, tamoxife
17 n peroxidases, including myeloperoxidase and eosinophil peroxidase, all of which exhibit strong seque
18 (especially eosinophils), lower activity of eosinophil peroxidase, an increase in IgG blocking antib
19 in the bronchoalveolar lavage fluid, reduced eosinophil peroxidase and B-hexosaminidase activity, inc
20 the reactive brominating species produced by eosinophil peroxidase and by activated eosinophils, resu
21 ctivated eosinophils degranulate and release eosinophil peroxidase and leukotriene C(4) in a dose-dep
22 9 likewise reduced extracellular deposits of eosinophil peroxidase and tenascin C, the effects not se
23 ressed eosinophil-related genes, such as the eosinophil peroxidase and the major basic protein, but d
24 orrelated with lower mRNA expression of Epx (eosinophil peroxidase) and Prg2 (major basic protein) as
25 tured eotaxin-3, major basic protein 1, EDN, eosinophil peroxidase, and Charcot-Leyden crystal protei
27 he chemoattractant eotaxin and the cytotoxic eosinophil peroxidase, and exhibit muscle eosinophilia.
28 nd transcripts encoding major basic protein, eosinophil peroxidase, and GATA-1, -2, and -3 to an exte
30 eptides, neuropeptides, major basic protein, eosinophil peroxidase, and many US Food and Drug Adminis
31 antly induced expression of Siglec-F, CD11c, eosinophil peroxidase, and other markers typical for act
38 Reactive brominating species produced by eosinophil peroxidase attacked the plasmalogen vinyl eth
39 l surface markers, as well as the release of eosinophil peroxidase by eosinophils in the bronchial mu
41 cificity studies show that only MPO, but not eosinophil peroxidase, can highly activate these agents,
42 ct of eosinophils is based on the release of eosinophil peroxidase causing impaired reactive oxygen s
43 alpha-chain, and transcripts encoding mouse eosinophil peroxidase, CCR3, the IL-3/IL-5/GM-CSF recept
45 nophil degranulation (assessed as submucosal eosinophil peroxidase deposition by immunohistochemistry
46 ls, bronchoalveolar lavage eosinophilia, and eosinophil peroxidase deposition in bronchial mucosa.
47 3 receptors, whereas airway eosinophilia and eosinophil peroxidase deposition were blunted but not el
48 Furthermore, 2-BrHDA production elicited by eosinophil peroxidase-derived reactive brominating speci
49 s and activated eosinophils, indicating that eosinophil peroxidase did not contribute to luminol-BLI
50 peroxidase, a naturally dimeric protein, and eosinophil peroxidase do not undergo H(2)O(2)-dependent
51 double knock-out mice (major basic protein-1/eosinophil peroxidase dual gene deletion) show that eosi
53 oteins eosinophil cationic protein (ECP) and eosinophil peroxidase (EPO) (P < .05), while IFABP was p
54 hiocyanate (SCN(-)) compete for oxidation by eosinophil peroxidase (EPO) and H(2)O(2), yielding, resp
57 the discovery that myeloperoxidase (MPO) and eosinophil peroxidase (EPO) can generate nitrotyrosine v
59 nate (SCN(-)) is the preferred substrate for eosinophil peroxidase (EPO) in fluids of physiologic hal
64 otection was unimpaired in mice deficient in eosinophil peroxidase (EPO) or major basic protein 1 (MB
65 dy, we generated knockout mice deficient for eosinophil peroxidase (EPO) to assess the role(s) of thi
68 tic infections and many forms of cancer, and eosinophil peroxidase (EPO), a secreted hemoprotein, pla
72 in (MBP), eosinophil cationic protein (ECP), eosinophil peroxidase (EPO), and eosinophil-derived neur
73 dies demonstrate that myeloperoxidase (MPO), eosinophil peroxidase (EPO), and lactoperoxidase (LPO),
74 inophil-derived neurotoxin (EDN or RNase 2), eosinophil peroxidase (EPO), and major basic protein-1 (
75 anule products major basic protein (MBP) and eosinophil peroxidase (EPO), it was determined that eosi
76 ith either isolated myeloperoxidase (MPO) or eosinophil peroxidase (EPO), plasma levels of halides (C
77 3-chlorotyrosine (ClY), selective markers of eosinophil peroxidase (EPO)- and myeloperoxidase-catalyz
82 s were quantified for levels of eosinophils, eosinophil peroxidase (EPX) immunohistochemical staining
83 was to validate a novel ELISA-based assay of eosinophil peroxidase (EPX) in sputum as a rapid and rel
86 was to compare nasal, pharyngeal, and sputum eosinophil peroxidase (EPX) levels with induced sputum e
87 and extracellular granules were detected by eosinophil peroxidase (EPX) staining and MCs by KIT stai
89 ty that halogenated nucleobases generated by eosinophil peroxidase exert cytotoxic and mutagenic effe
91 Zn-SOD to physiologically relevant levels of eosinophil peroxidase-generated reactive brominating spe
93 ngerprint" for proteins modified through the eosinophil peroxidase-H(2)O(2) system in the presence of
97 dase-H2O2-Cl(-)- Br(-) system but not by the eosinophil peroxidase-H2O2-Cl(-)-Br(-) system, indicatin
106 ing the classic eosinophil granule proteins (eosinophil peroxidase, major basic protein, the ribonucl
107 oxidase enzymes, such as myeloperoxidase and eosinophil peroxidase, may play a fundamental role in re
108 oteolytic reactions detected the presence of eosinophil peroxidase, MBP, and fibrin alpha-, beta-, an
109 in, eosinophil-derived neurotoxin (EDN), and eosinophil peroxidase measurement and to evaluate the us
112 ength and branching were not associated with eosinophil peroxidase nor with demographics such as age
113 egulation of immunity was not dependent upon eosinophil peroxidase or major basic protein 1 and did n
114 t 5-bromouracil could be generated by either eosinophil peroxidase or myeloperoxidase, which preferen
115 hese results indicate that HOBr generated by eosinophil peroxidase oxidizes uracil to 5-bromouracil.
117 In addition to TNF secretion, release of eosinophil peroxidase promoted colitis identifying direc
118 correlated to eosinophil counts (r = 0.691), eosinophil peroxidase (r = 0.738), and TGF-beta (r = 0.5
119 bstrate specificities of myeloperoxidase and eosinophil peroxidase regarding chloride and bromide.
122 reactive brominating species produced by the eosinophil peroxidase system of activated eosinophils at
125 Reactive brominating species produced by eosinophil peroxidase target the vinyl ether bond of pla
126 -linking inhibited GM-CSF-induced release of eosinophil peroxidase, TNF-alpha, and IL-8 (n = 7-8) but
128 (lysoPAF) promote degranulation (release of eosinophil peroxidase) via a mechanism that is independe
131 rated that 5-bromodeoxycytidine generated by eosinophil peroxidase was taken up by cultured cells and
132 e reduced the exploratory end point of nasal eosinophil peroxidase week-12 ratio to baseline in the 1
134 hil granule proteins major basic protein and eosinophil peroxidase were more frequently detected in t
135 e branch points, substance P expression, and eosinophil peroxidase were quantified within each airway
136 ophils and contain extracellular deposits of eosinophil peroxidase, which uses hydrogen peroxide as a