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1 is in the adult brain via maintenance of the ependyma.
2 cilia of epithelial cells in the trachea and ependyma.
3 s expression on the luminal membranes of the ependyma.
4 vessels to terminate immediately beneath the ependyma.
5 es, and cellular necrosis was evident in the ependyma.
6 minating in bulbs immediately underneath the ependyma.
7 hannel Na(x) expressed by astrocytes and the ependyma.
8 induced signals were present in ventricular ependyma.
9 glia, especially astrocytes and ventricular ependyma.
10 ry epithelium, as well as choroid plexus and ependyma.
11 layer V and by glia in the white matter and ependyma.
12 rs reside underneath the lateral ventricular ependyma.
15 nin-1/CD133, is exclusively localized to the ependyma, although not all ependymal cells are CD133(+).
16 er of SVZ astrocytes interpolated within the ependyma and established contact with the ventricle.
18 -17, is expressed in adult rats and mice by ependyma and SVZ cells with long basal processes, and in
19 neumoniae can be detrimental to the ciliated ependyma and that antipneumolysin antibody may have a th
20 al nuclei within the hypothalamus and in the ependyma and the circumventricular organs that act as an
21 e, pia mater, and aspects of the ventricular ependyma and was relatively low within areas of white ma
22 dentified in radial glia, mature astrocytes, ependyma, and choroid plexus epithelium, but not in neur
23 lt CNS (retina, cerebellum, cerebral cortex, ependyma, and choroid) as well as the adult kidney epith
25 rked enhancement throughout her meninges and ependyma, and TTR amyloid deposition was confirmed by me
26 e show that astrocytes integrated within the ependyma are dividing, BrdU-labeled astrocytes that shar
27 on direct ependymal injury, indicating that ependyma are not a major source of endogenous neural ste
28 cerebral injection, subjacent to ventricular ependyma, as well as scattered in cerebral white and gra
29 bencephalon and spinal cord were seen in the ependyma, but many labeled cells were found in nonependy
30 al mitosis in the rhombencephalon was in the ependyma, but this finding was not true in the spinal co
31 ld-type pneumococci caused disruption to the ependyma, but this was not observed in rats infected wit
33 We find here that connexin signaling in the ependyma changes after injury of the adult spinal cord,
37 in mouse brain in the meninges, ventricular ependyma, circumventricular organs, along the vasculatur
40 est (26%) in subventricular locations at the ependyma/CSF boundary and least with increasing distance
41 density increased by over 50% at 2 mm at the ependyma/CSF boundary and only by 15% at 10 mm and this
45 rush injuries across the entire spinal cord, ependyma-derived progeny remained local, did not migrate
47 lytic cycle genes within the brain stem, the ependyma (EP), containing the limbic and cortical areas,
50 astrocyte foot processes, glia limitans and ependyma, facilitates water movement into and out of the
55 an 86% decline in total NSCs/mm(2) of intact ependyma in 2-year old versus 3-month-old mice, with few
56 y was detected in the ciliated region of the ependyma in the central canal from early postnatal devel
58 ccessory hypothalamic neurosecretory nuclei, ependyma lining the ventricles and choroid plexus which
61 t vimentin-immunopositive processes from the ependyma of the adjacent surface of the third ventricle.
63 reactive cells were found exclusively in the ependyma of the basal region of the lateral ventricles (
64 r, a spike of mitotic activity occurs in the ependyma of the rhombencephalon and throughout the spina
66 n the gene product after transduction of the ependyma, reduces Abeta plaque deposition, neurodegenera
68 cible deletion of Foxj1-Ank3 from mature SVZ ependyma resulted in dramatic depletion of neurogenesis.
70 n of rAAV4betagal resulted again in striking ependyma-specific expression of transgene, with a notabl
71 epithelium of the choroid plexus and in the ependyma, such as asymmetrical cell shape and size, misp
73 undetectable in OX-42-positive cells in the ependyma, the external capsule, choroid plexus, and meni
74 served that, as they incorporated within the ependyma, they took on antigenic and morphologic charact
77 neglected in the study of meningitis is the ependyma, which has been identified as a location of adu
78 particularly those of the choroid plexus and ependyma, which play critical roles in producing cerebro
79 r, baseline gliogenesis occurs mainly in the ependyma with substantial contribution by nonependymal a