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1 nterest for ITMS analysis: o-nitrophenol and ephedrine.
2 times worsened, and where tried responded to ephedrine.
3 as increased when nicotine was combined with ephedrine.
4 iated by GABAB receptors were reduced 28% by ephedrine.
5 atecholamines were measured before and after ephedrine.
6 = albuterol > salmeterol > dobutamine > or = ephedrine.
7 ioselective separation of metamphetamine and ephedrine.
8 albuterol, 2.5% for dobutamine, and 1.1% for ephedrine.
9 ine >/= fenoterol > albuterol > dobutamine > ephedrine.
10 weight, n = 9), or a mixture of nicotine and ephedrine (0.8 mg/kg of body weight and 5 mg/kg of body
11  of fraction of receptors lost/h) 0.082 with ephedrine, 0.250 with dobutamine, 0.148 with albuterol,
12                                              Ephedrine (100-1000 microM) slowed spontaneous firing an
13   Caffeine (10 mg/kg of body weight, n = 4), ephedrine (5 mg/kg of body weight, n = 4), nicotine (0.8
14 +/- 0.82) was not appreciably different from ephedrine (5.0 +/- 0.96) and significantly less than tha
15 rug treatment period, the monkeys were given ephedrine (6 mg) and caffeine (50 mg) orally three times
16 For example, herbal preparations may contain ephedrine (a drug), which might alter heart rate so that
17    All plasma catecholamines increased after ephedrine administration.
18  Technology (NIST) with certified values for ephedrine alkaloids, synephrine, caffeine, and selected
19                    The use of salbutamol and ephedrine alone or combined with pyridostigmine or 3,4-D
20 h control rats; more than either nicotine or ephedrine alone.
21 mediated by a postsynaptic mechanism because ephedrine also reduced baclofen-induced hyperpolarizatio
22 ction: see text] Very practical synthesis of ephedrine analogues in high yields and enantiopurity was
23 S)-2 with biologically active amines (1R,2S)-ephedrine and (S)-dapoxetine were also investigated by (
24 ine phosphinite ligands (AMPP*) derived from ephedrine and bearing a P-chirogenic center either at th
25                        The administration of ephedrine and caffeine (E+C) has been proposed to promot
26 ls comparing placebo with ephedrine (n = 5), ephedrine and caffeine (n = 12), ephedra (n = 1), and ep
27                            Use of ephedra or ephedrine and caffeine is associated with increased risk
28                                              Ephedrine and ephedra promote modest short-term weight l
29 omethane precursor: stimulant drugs, such as ephedrine and methamphetamine, and certain antidepressan
30 delta(2)H values were distinct from those of ephedrine and methylamphetamine samples of a semi-synthe
31                 Increasing concentrations of ephedrine and pseudoephedrine also increased the number
32                                              Ephedrine and pseudoephedrine are stimulant drugs whose
33 strial routes used to produce the precursors ephedrine and pseudoephedrine by measurement of stable i
34 moselectivity for simultaneous separation of ephedrine and pseudoephedrine containing multiple chiral
35 termediate formation via the chlorination of ephedrine and pseudoephedrine enantiomers.
36 gative electrospray and the diastereoisomers ephedrine and pseudoephedrine in positive mode.
37              The channel blocking effects of ephedrine and pseudoephedrine might reduce the synaptic
38 ne, methamphetamine, MDMA, MDA), ephedrines (ephedrine and pseudoephedrine), antidepressant venlafaxi
39 produce high amounts of alkaloids, primarily ephedrine and pseudoephedrine, which have been utilized
40 ing 100 microM (D,L)-arginine, 10 microM (-)-ephedrine, and 10 microM lomefloxacin.
41 ar analytes including ethambutol, isoniazid, ephedrine, and gemcitabine in biological matrices was fu
42 eutical compounds include DOPA, amphetamine, ephedrine, and penicillamine.
43                                     Mahuang, ephedrine, and related alkaloids have not benefited phys
44 hat biologically relevant central effects of ephedrine are more likely to be mediated either by non-d
45  excess (>95% ee in most cases) by way of an ephedrine-based oxazaphospholidine borane complex.
46 gest that patients should avoid nicotine and ephedrine before undergoing (18)F-FDG PET to minimize (1
47              By preparation of 30 samples of ephedrine by the "fully synthetic" industrial process an
48                                              Ephedrine can significantly facilitate the initial stage
49 in, whereas the weak agonists dobutamine and ephedrine caused only 3-4-fold increases, similar to lev
50   Taken together, these results suggest that ephedrine causes hyperpolarization and suppresses GABAB
51 ased (7.9-fold for nicotine and 3.7-fold for ephedrine), compared to the control rats.
52                     Herbal ecstasy refers to ephedrine-containing preparations.
53       The only exceptions are E. sinica- and ephedrine-containing supplements, which have been associ
54                There is no regulation of the ephedrine content of available herbal ecstasy products.
55                                          The ephedrine derived imidazolidin-2-one 21 was allowed to r
56                                     However, ephedrine did not reduce fast synaptic potentials mediat
57 d as a function of coupling efficiency, with ephedrine, dobutamine, albuterol, fenoterol, and epineph
58 ctivation of the sympathetic nervous system, ephedrine does not stimulate BAT in humans.
59 tive phase permits isolating >50% of the (+)-ephedrine enantiomer as target compound in only 4 min.
60 e (amphetamine, methamphetamine, MDMA, MDA), ephedrines (ephedrine and pseudoephedrine), antidepressa
61 xyglucose PET-CT in every volunteer, whereas ephedrine failed to stimulate BAT.
62 ubs, including methylenedioxymethamfetamine, ephedrine, gamma-hydroxybutyrate; gamma-butyrolactone, 1
63  The beta-adrenergic agonists salbutamol and ephedrine have proven to be effective as therapies for h
64 clear explanation for a beneficial effect of ephedrine in endplate diseases.
65              The combination of nicotine and ephedrine increased the uptake 12.0-fold, compared with
66                                              Ephedrine increases ischemia-dependent arrhythmias at do
67 othesis that the sympathomimetic activity of ephedrine increases the risk of lethal arrhythmias.
68    Fetal genotype may increase resistance to ephedrine-induced acidosis.
69                                         Both ephedrine-induced hyperpolarization and inhibition of ba
70                                     Although ephedrine is a centrally active stimulant, its effect on
71                   The sympathomimetic amine, ephedrine, is used to augment physical performance and a
72 ed results for trials comparing placebo with ephedrine (n = 5), ephedrine and caffeine (n = 12), ephe
73                     To assess the effects of ephedrine on cardiac autonomic control, baroreceptor ref
74                       To study the effect of ephedrine on dopamine-containing cells, current-clamp mi
75 trials of efficacy and safety of ephedra and ephedrine on weight loss or athletic performance; disagr
76 n 24 hours prior to an adverse event or that ephedrine or an associated product was found in blood or
77 chloride were synthesized from either from l-ephedrine or d-pseudoephedrine hydrochloride in reaction
78 is of methylamphetamine hydrochloride from l-ephedrine or d-pseudoephedrine hydrochloride via reducti
79 etamine is often prepared from the precursor ephedrine or pseudoephedrine, which in turn are obtained
80              This analysis suggests that (1) ephedrine or salbutamol is the first choice of treatment
81       The magnitude of treatment effect with ephedrine or salbutamol seems to increase gradually, pea
82 se inhibitor, 65 of 69 patients who received ephedrine or salbutamol, 18 of 29 who were given 3,4-dia
83 ts/min no drug vs. 218 +/- 26 beats/min with ephedrine, p = 0.03).
84     Resolution of fluoroiodoacetic acid with ephedrine provides either enantiomeric form of the elect
85 plexes of seven model drugs (atenolol, DOPA, ephedrine, pseudoephedrine, isoproterenol, norepinephrin
86 al myasthenic syndromes respond favorably to ephedrine, pseudoephedrine, or albuterol.
87  We have shown here that chlorination of the ephedrine/pseudoephedrine compounds occurs via inversion
88  the aim of linking the intermediates to the ephedrine/pseudoephedrine starting materials.
89 olunteers, we found that the sympathomimetic ephedrine raised blood pressure, heart rate, and energy
90                            Conversion of ten ephedrine samples to methylamphetamine showed that this
91                                              Ephedrine significantly (P<.01) increased vaginal pulse
92                                  Ephedra and ephedrine sometimes are used for weight loss or enhanced
93                                   The use of ephedrine-substituted quaternary ammonium binaphthyl sal
94  measured following administration of either ephedrine sulfate (50 mg) or placebo in a randomized, do
95 irst empirical examination of the effects of ephedrine sulfate, an alpha- and beta-adrenergic agonist
96        In the rats injected with nicotine or ephedrine, the mean uptake of (18)F-FDG, in percentage i
97  Despite perfusion with AMPT, the ability of ephedrine to cause hyperpolarization was restored after
98 her after 30 s of myocardial ischemia during ephedrine treatment (204 +/- 25 beats/min no drug vs. 21
99 had increased ventricular arrhythmias during ephedrine treatment (p = 0.01) and four had VF.
100 ve animals with increased arrhythmias during ephedrine treatment had none during a third exercise and
101  infarction were retested after five days of ephedrine use (Xenadrine, 0.4 mg/kg/day orally).
102 studies were controlled trials of ephedra or ephedrine used for weight loss or athletic performance a
103 ible case reports documented that ephedra or ephedrine was consumed within 24 hours prior to an adver
104                                              Ephedrine was helpful in half of the patients.
105 athletic performance were found; 7 trials of ephedrine were too heterogeneous to synthesize.

 
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