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1 that adds this residue was purified from pea epicotyls.
2 applied to one side of a carborundum-abraded epicotyls.
3 ts from homogenates of green leaf, etiolated epicotyl and cotyledon, and root tissues of pea were poo
4 d to cotyledons were transferred through the epicotyl and into the collection medium.
5   The initial rate H+/ATP coupling ratios of epicotyl and the nitrate-sensitive fruit proton pumping
6 uples H+ pumping from ATP hydrolysis both in epicotyls and in nitrate-sensitive fruit V-ATPases.
7 US expression was limited to the cotyledons, epicotyl, and root elongation zone.
8 abundant in the apical region of hypocotyls, epicotyls, and roots.
9 n fruit tonoplasts, unlike those of seedling epicotyls, contain nitrate-insensitive H+-ATPase activit
10      Substantial vascular exudation from pea epicotyls could be obtained without tissue damage at 0.5
11 s mutant the temperature-dependent arrest of epicotyl development is due to a failure of normal leaf
12 d, reconstituted V-ATPases of both fruit and epicotyl exhibited similar inhibitor profiles, except th
13                            XET activity from epicotyl extracts used nonfucosylated seed amyloid xylog
14  with a structural role of the former during epicotyl growth where efficient association with cellulo
15                                   Unlike the epicotyl H(+)-ATPase, the fruit H(+)-ATPase activity was
16                             In addition, the epicotyl H+ pumping activity was inactivated by oxidatio
17                                   Unlike the epicotyl inhibitors, and resistant to oxidation.
18       We conclude that the 45Ca2+ fed to pea epicotyls occurs largely in the cell wall, and that auxi
19 displaced through the elongation zone of the epicotyl of etiolated pea (Pisum sativum L. var Alaska)
20 rapid growth and most abundant in elongating epicotyls of etiolated seedlings.
21                        Protein extracts from epicotyls or germinating cotyledons, in which XET1 or NX
22  isolated from nasturtium (Tropaeolum majus) epicotyl RNA.
23           We show for pea (Pisum sativum L.) epicotyl sections that this hypothesis is incorrect.
24                                         When epicotyl segments (6 mm long, 4 mm from the apical hook)
25 te H+ efflux, increase Ca2+ release from pea epicotyl segments, whereas cycloheximide, which inhibits
26                               Hypocotyls and epicotyls show higher accumulation of pmeu1 transcripts
27 on in all vegetative tissues examined (root, epicotyl, stem, and leaf) except in germinating cotyledo
28 onset of curvature, on the upper side of the epicotyl, there was a conspicuous transient increase in
29 fication of embryogenic protoplast cells and epicotyl tissues.
30                     Cold inactivation of the epicotyl V-ATPase by nitrate ( > or = 100 mM) was correl
31  H+-leakage indicated that the reconstituted epicotyl V-ATPase exhibited twice as much intrinsic unco
32 sac V-ATPases appeared thicker than those of epicotyl V-ATPases in electron micrographs.
33  is lower than that of nitrate-sensitive and epicotyl V-ATPases.
34 e of intact pea (Pisum sativum L. cv Alaska) epicotyls was examined by gel-filtration chromatography.
35 ly, Agrobacterium-mediated transformation of epicotyls was unsuccessful when a constitutive promoter
36  by tonoplast vesicles from lemon fruits and epicotyls were compared.
37 r H+-ATPases (V-ATPases) of lemon fruits and epicotyls were detergent-solubilized, purified by column
38                  Seven day old etiolated pea epicotyls were loaded symmetrically with 3H-indole 3-ace
39 d etiolated pea (Pisum sativum L. cv Alaska) epicotyls with 3 molar LiCl.