ライフサイエンスコーパス: epidermal keratinocyte

1 dy was to determine the role of mitoStat3 in epidermal keratinocytes.

2 tes the proliferation and differentiation of epidermal keratinocytes.

3 kin tissue environment has limited effect on epidermal keratinocytes.

4 ostasis, we conditionally knocked out SRF in epidermal keratinocytes.

5 rganotypic three-dimensional cultures of rat epidermal keratinocytes.

6 normal human fibroblasts and neonatal human epidermal keratinocytes.

7 g-term ex vivo expansion of functional human epidermal keratinocytes.

8 ted the effect of Mg(2+) on TRPV3 in primary epidermal keratinocytes.

9 ages, and myelomonocytic cells as well as in epidermal keratinocytes.

10 n HSV-1 infection and their interaction with epidermal keratinocytes.

11 re produced by placenta, adrenal glands, and epidermal keratinocytes.

12 ar signals mediating cell differentiation in epidermal keratinocytes.

13 o cellular components of the skin, including epidermal keratinocytes.

14 , and modulating TGF-beta signaling in mouse epidermal keratinocytes.

15 als for UVB irradiation-induced apoptosis in epidermal keratinocytes.

16 further elaborate the desmosome proteome in epidermal keratinocytes.

17 ontact or were physically separated from the epidermal keratinocytes.

18 del, correlating with K17 induction in basal epidermal keratinocytes.

19 e also attenuated by chrysin pretreatment of epidermal keratinocytes.

20 l regulator that promotes differentiation in epidermal keratinocytes.

21 ortant roles in the survival and motility of epidermal keratinocytes.

22 ance of proliferation and differentiation in epidermal keratinocytes.

23 ted in cSCC cells compared with normal human epidermal keratinocytes.

24 adhesion molecule expression and function in epidermal keratinocytes.

25 lammatory skin and in the differentiation of epidermal keratinocytes.

26 EP2 to the plasma membrane of differentiated epidermal keratinocytes.

27 et of the TNF alpha-regulated genes in human epidermal keratinocytes.

28 d the expression of endogenous TRPC in human epidermal keratinocytes.

29 CCL20 and antimicrobial peptide release from epidermal keratinocytes.

30 C/EBPalpha is abundantly expressed in mouse epidermal keratinocytes.

31 cells responded more quickly to therapy than epidermal keratinocytes.

32 a) is an important regulator of apoptosis in epidermal keratinocytes.

33 results were obtained by using normal human epidermal keratinocytes.

34 ory neurons but are highly expressed in skin epidermal keratinocytes.

35 kappa B (NF-kappaB) pathways in normal human epidermal keratinocytes.

36 omplex regulation of Col7a1 transcription in epidermal keratinocytes.

37 s the predominant species expressed in basal epidermal keratinocytes.

38 imus block activation of calcineurin/NFAT in epidermal keratinocytes.

39 T1 was predominantly nuclear in normal basal epidermal keratinocytes.

40 al differentiation and hyperproliferation of epidermal keratinocytes.

41 d that p63 promotes glycolytic metabolism in epidermal keratinocytes.

42 ependent fashion with predominant effects on epidermal keratinocytes.

43 ons is an enhancer element that is active in epidermal keratinocytes.

44 yaluronan synthase (HAS) activation in human epidermal keratinocytes.

45 cutaneous IL-33 reduces REG3A expression in epidermal keratinocytes.

46 xygen species and extensive apoptosis of the epidermal keratinocytes.

47 dermal junction and accompanying necrosis of epidermal keratinocytes.

48 is a water and glycerol channel expressed in epidermal keratinocytes.

49 intermediate filament (IF) network of basal epidermal keratinocytes.

50 of intercellular adhesions between mammalian epidermal keratinocytes.

51 nt directional migration in undifferentiated epidermal keratinocytes.

52 mice lacking p53-mediated POMC induction in epidermal keratinocytes.

53 In epidermal keratinocytes a syndecan-TRPC4 complex control

54 rough specific toll-like receptors (TLRs) in epidermal keratinocytes, a DFE-induced murine atopic der

55 In response to UVB damage, epidermal keratinocytes activate a specific repair pathw

56 , the major autoantigen in PV, cause loss of epidermal keratinocyte adhesion, resulting in blisters a

57 le to attenuate DeltaNp63alpha expression in epidermal keratinocytes after such stress.

58 Although recent studies have shown that epidermal keratinocytes also participate in sensory tran

59 date the role of IL-6 in skin wound healing, epidermal keratinocyte and dermal fibroblast cells were

60 L2 and of IFNgamma-induced CCL8 and CXL10 by epidermal keratinocytes and (3) thereby limited the recr

61 Normal human epidermal keratinocytes and 3D raft treatment with SR-A

62 and MARCO reduced SNA uptake in normal human epidermal keratinocytes and 3D rafts after topical appli

63 the mechanism of SNA uptake in normal human epidermal keratinocytes and 3D skin equivalents.

64 us (VZV) is a skin-tropic virus that infects epidermal keratinocytes and causes chickenpox.

65 9 null) mice, normal proliferation occurs in epidermal keratinocytes and corneal basal cells.

66 t STRA6 is constitutively expressed in human epidermal keratinocytes and dermal fibroblasts and is re

67 hich inactivates cortisol), was expressed in epidermal keratinocytes and dermal fibroblasts in human

68 ted Smad1/5/8 are expressed in human primary epidermal keratinocytes and dermal fibroblasts.

69 lysis using RNA isolated from cultured human epidermal keratinocytes and dermal fibroblasts.

70 xpression of 12 435 genes were determined in epidermal keratinocytes and dermal fibroblasts.

71 x steroids by primary cultures of human skin epidermal keratinocytes and dermal fibroblasts.

72 mobility group box 1 (HMGB1) from UV-damaged epidermal keratinocytes and driven by Toll-like receptor

73 The present study demonstrates that epidermal keratinocytes and fibroblast cells express sig

74 (ERK) activation in both normal human adult epidermal keratinocytes and five of seven SCCHN cell lin

75 ed the role of several coactivators in human epidermal keratinocytes and found that, although these c

76 It is mainly found in epidermal keratinocytes and has been hypothesized to be

77 proapoptotic and proinflammatory actions on epidermal keratinocytes and has been implicated in the p

78 3 channel (TRPV3) is abundantly expressed in epidermal keratinocytes and has important roles in senso

79 ng cyclin-dependent kinase 4 (Cdk4) in human epidermal keratinocytes and human mammary epithelial cel

80 a transporter-directed RNAi screen in human epidermal keratinocytes and identified SLC22A20 (OAT6) a

81 elective regulation of TSLP transcription in epidermal keratinocytes and IEC.

82 leading to the accumulation of mutations in epidermal keratinocytes and immunosuppression, which con

83 TRPV4 ion channels function in epidermal keratinocytes and in innervating sensory neuro

84 d that C/EBPalpha is induced in normal human epidermal keratinocytes and in the epidermis of human su

85 zed by IgG autoantibodies (AuAbs) binding to epidermal keratinocytes and inducing this devastating di

86 roliferation and aberrant differentiation of epidermal keratinocytes and inflammation.

87 cation is the final differentiative step for epidermal keratinocytes and involves dramatic cell conde

88 ncrease in H2O2 was observed in normal human epidermal keratinocytes and its production was inhibited

89 lammation-induced activation of Axl in human epidermal keratinocytes and LCs.

90 ied indeed a functioning GFRP/GTPCHI axis in epidermal keratinocytes and melanocytes in the cytosol,

91 of 0.05-J/cm(2) reduced melanin pigments in epidermal keratinocytes and melanocytes, compared to unt

92 rolled by skin-specific factors derived from epidermal keratinocytes and not by resident dendritic ce

93 reted cysteine-rich protein, is expressed by epidermal keratinocytes and sebocytes and serves as an a

94 , and functional approaches in cocultures of epidermal keratinocytes and sensory neurons.

95 nal diacylglycerol/phorbol ester receptor in epidermal keratinocytes and suggest that activation of t

96 hat HVEM and LTbetaR were expressed on human epidermal keratinocytes and that LIGHT could directly pr

97 that AQP3 has a pro-differentiative role in epidermal keratinocytes and that PLD2 activity is necess

98 sly shown that RasGRP1 is expressed in mouse epidermal keratinocytes and that transgenic mice overexp

99 itin sulfate proteoglycan 4) was detected in epidermal keratinocytes and the cardiac conduction syste

100 creased SIRT1 protein levels in normal human epidermal keratinocytes and the immortalized keratinocyt

101 Fas ligand (FasL) causes apoptosis of epidermal keratinocytes and triggers the appearance of s

102 pholipase-A2 type X (sPLA2-X) is released by epidermal keratinocytes and we have shown that lysophosp

103 of Agouti transcripts and protein in primary epidermal keratinocytes, and BMP signaling positively re

104 --a stimulus of angiogenesis--is produced by epidermal keratinocytes, and elevated levels have been f

105 of wound repair, predominantly expressed in epidermal keratinocytes, and peaked in the subsequent pr

106 these cells fail to transdifferentiate into epidermal keratinocytes, and there was no improvement in

107 potent chemotactic and mitogenic factor for epidermal keratinocytes, and these properties are centra

108 sions, and migration and that differentiated epidermal keratinocytes are a component of the stem cell

109 em cells in Terc(-/-) mice, and normal human epidermal keratinocytes are also ALT-positive.

110 Epidermal keratinocytes are complex cells that create a

111 Epidermal keratinocytes are particularly suitable candid

112 higher levels of endogenous bcl-2 protein in epidermal keratinocytes as assessed by immunoblotting an

113 alterations in growth and differentiation of epidermal keratinocytes, as well as a marked increase in

114 city accompanied by MIF release in mouse ear epidermal keratinocytes, as well as in human keratinocyt

115 We confirmed AIRE expression in HaCaT epidermal keratinocytes, as well as its interaction with

116 Incubation of normal human epidermal keratinocytes at 4(o)C or with sodium azide pr

117 nal in TPA-induced shedding of TNFalpha from epidermal keratinocytes; (b) PKCepsilon-mediated signals

118 Notch1-deficient epidermal keratinocytes become progressively hyperplasti

119 the transcriptional activity of NFkappaB in epidermal keratinocytes, both under basal treatment and

120 s cell carcinoma (SCC) are both derived from epidermal keratinocytes but are phenotypically diverse.

121 physiology of the cornified cell envelope in epidermal keratinocytes but may reside in the challenges

122 inally thought of as a disorder primarily of epidermal keratinocytes, but is now recognised as one of

123 , the results indicate that HSV enters human epidermal keratinocytes, but not neurons, by a low-pH, e

124 We show how the epidermal keratinocyte can direct hMSC differentiation s

125 intrinsic sensory transduction mechanisms in epidermal keratinocytes can directly elicit AP firing in

126 Smad7 induction in epidermal keratinocytes caused an increase in keratinocy

127 ltaNp63alpha overexpression in primary human epidermal keratinocytes causes decreased cell proliferat

128 During differentiation, epidermal keratinocytes cease to express integrins, but

129 A treatment was observed in the immortalized epidermal keratinocyte cell line NHEK-HPV, whereas PMA d

130 NT5E was also located on epidermal keratinocytes, cells of the dermis, and on noc

131 isplayed increased DMBA-induced apoptosis in epidermal keratinocytes compared to wild-type mice.

132 Unirradiated normal human epidermal keratinocytes contained 1.49 (+/- 0.11) 8-oxo-

133 Conversely, epidermal keratinocytes contained less stromal Igfbp4 an

134 ting bacterial invasion of canine progenitor epidermal keratinocytes (CPEK).

135 Consistent with in vivo results, epidermal keratinocytes cultured from (Epid)CaR(-/-) mic

136 ction of ODC activity did not rescue primary epidermal keratinocyte cultures isolated from ODCER2 mic

137 the culmination of signals exchanged between epidermal keratinocytes, dermal fibroblasts and leukocyt

138 Although epidermal keratinocyte development and differentiation p

139 Epidermal keratinocytes dialogue with sensory neurons th

140 mber, functions as an important regulator of epidermal keratinocyte differentiation and survival.

141 order chromatin remodeling in the context of epidermal keratinocyte differentiation in the skin.

142 The expression of epidermal keratinocyte differentiation markers was affec

143 Calcium induces epidermal keratinocyte differentiation, but the mechanis

144 Proper epidermal keratinocyte differentiation, which is necessa

145 kinase-independent activity is required for epidermal keratinocyte differentiation.

146 idermal equivalent model that mimics in vivo epidermal keratinocyte differentiation.

147 In cultures of normal human epidermal keratinocytes dioxin accelerates cell differen

148 ologous or allogeneic dermal fibroblasts and epidermal keratinocytes directly into an injured area, r

149 In human skin, epidermal keratinocytes do not express IGF-1, and hence

150 As epidermal keratinocytes do not express integrin alpha(v)

151 domain (TRADD)-dependent TNFR1 signaling in epidermal keratinocytes drives skin inflammation in Shar

152 cluding bone and cartilage, but not in basal epidermal keratinocytes, exhibit normal epidermal differ

153 Mice overexpressing soluble VEGFR3 in epidermal keratinocytes exhibited hypoplastic cutaneous

154 holipid classes was measured in normal human epidermal keratinocytes exposed to cumene hydroperoxide

155 Terminally differentiating epidermal keratinocytes express a large number of struct

156 Epidermal keratinocytes express at least six CYP4F enzym

157 ere was a paracellular barrier defect in rat epidermal keratinocytes expressing a Ppp2r2a siRNA.

158 g, the dermal fibroblast has advantages over epidermal keratinocytes for delivering C7 to DEB patient

159 Epidermal keratinocytes form a structural and immune bar

160 The initial events that specify epidermal keratinocytes from ectodermal progenitor cells

161 ts on matched primary dermal fibroblasts and epidermal keratinocytes from human donors on three bioma

162 POMC was secreted by both human epidermal keratinocytes (from 5 healthy donors) and matc

163 phenotype through the transgenic delivery of epidermal keratinocyte growth factors or inflammatory me

164 induced skin cancer using immortalized human epidermal keratinocyte (HaCaT) cells through repetitive

165 212 murine cell line keratinocytes and human epidermal keratinocytes (HEK), express cell-surface GITR

166 sion was evaluated in cytokine-treated human epidermal keratinocytes (HEK)s, murine PAM 212 cell line

167 vels of miR-205 and miR-184 in primary human epidermal keratinocytes (HEKs) and corneal epithelial ke

168 Elevating FIH-1 levels in primary human epidermal keratinocytes (HEKs) and human corneal epithel

169 variants that are expressed in primary human epidermal keratinocytes (HEKs) and the biochemical activ

170 ions of select target genes in primary human epidermal keratinocytes (HEKs) using chromatin immunopre

171 Human epidermal keratinocytes (HEKs) were used to assess if QD

172 the cell cycle, in mouse epidermis and human epidermal keratinocytes (HEKs).

173 e growth arrest and differentiation of human epidermal keratinocytes (HEKs).

174 th layered autologous dermal fibroblasts and epidermal keratinocytes in a hydrogel carrier showed rap

175 nking activities in cultured human and mouse epidermal keratinocytes in a modified in situ assay.

176 crease in integrin alpha9beta1 expression in epidermal keratinocytes in cutaneous and corneal wounds.

177 nstream signaling is strongly induced within epidermal keratinocytes in cutaneous psoriatic lesions,

178 for effects on human dermal fibroblasts and epidermal keratinocytes in monolayer culture, and for ef

179 Here we report that epidermal keratinocytes in psoriatic lesions are charact

180 eta(-/-) mice exhibit increased apoptosis in epidermal keratinocytes in response to carcinogen treatm

181 ar function of Merlin in AJ establishment in epidermal keratinocytes in vitro and confirm that a role

182 Furthermore, in normal human epidermal keratinocytes in vitro, treatment with Staphyl

183 e antiproliferative effects on human primary epidermal keratinocytes in vitro.

184 nds, we compared VEGF production by oral and epidermal keratinocytes in vitro.

185 alpha9beta1 is seen in basal and suprabasal epidermal keratinocytes in wounds.

186 e transcription factor, AHR, in normal human epidermal keratinocytes increased AHR binding in the gen

187 Knockdown of 11beta-HSD1 in human epidermal keratinocytes increased the production of thym

188 in a variety of cell lines and primary human epidermal keratinocytes, indicating that the protein was

189 ith loss of Connexin 26 and Connexin 30 from epidermal keratinocyte intercellular junctions and accum

190 Here we show that attenuating PTEN in epidermal keratinocytes is a predisposing factor for UVB

191 he sonic hedgehog (shh) signaling pathway in epidermal keratinocytes is a primary event leading to th

192 otype whereby intercellular adhesion between epidermal keratinocytes is disrupted, adversely affectin

193 How p63 regulates metabolism in epidermal keratinocytes is incompletely understood, and

194 The stem cell potential of human epidermal keratinocytes is retained in vitro but lost ov

195 esults not only reveal how UV irradiation of epidermal keratinocytes is sensed by the innate immune s

196 utaneous wounds, the number of proliferating epidermal keratinocytes is significantly reduced in K14-

197 sults indicate that the inhibition of JNK in epidermal keratinocytes is sufficient to initiate their

198 tingly, the prolonged expression of Snail in epidermal keratinocytes is sufficient to recapitulate ea

199 neage-committed human cells, including human epidermal keratinocytes, JARID2 predominantly exists as

200 (PKC) isoforms have been associated with the epidermal keratinocyte (KC) granular layer differentiati

201 ripheral blood mononuclear cells (PBMCs) and epidermal keratinocytes (KC) from patients with psoriasi

202 Primary epidermal keratinocytes (KC) from TG mice exhibit signif

203 Epidermal keratinocytes (KCs) and cannabinoid (CB) recep

204 During malignant transformation in skin, epidermal keratinocytes (KCs) frequently acquire the cap

205 und that IFN-beta was abundantly produced by epidermal keratinocytes (KCs) in psoriasis and during wo

206 The nucleus of epidermal keratinocytes (KCs) is a complex and highly co

207 ) that are in close contact with surrounding epidermal keratinocytes (KCs).

208 odermal progenitor cell specification to the epidermal keratinocyte lineage.

209 Inhibition of this pathway in epidermal keratinocytes may account, in part, for the th

210 sufficient retinoic acid synthesis by keloid epidermal keratinocytes may contribute to the pathogenes

211 Epidermal keratinocytes migrate through the epidermis up

212 Normal epidermal keratinocyte morphology is also retained.

213 of cSCC cell lines (n = 8) and normal human epidermal keratinocytes (n = 11) with real-time quantita

214 tutively expressed in cultured normal canine epidermal keratinocytes (NCEKs), and its expression leve

215 easible to discriminate between normal human epidermal keratinocytes (NHEK) and dermal fibroblasts (N

216 n peroxide (H(2)O(2)) levels in normal human epidermal keratinocytes (NHEK) and melanocytes (mel) wer

217 ific keratins K1, K10 and K2 in normal human epidermal keratinocytes (NHEK) and two important cell li

218 melanin effectively prevented neonatal human epidermal keratinocytes (NHEK) from G2/M phase arrest un

219 ifferentiated or differentiated normal human epidermal keratinocytes (NHEK) in culture, but is dramat

220 yme was detected in cultures of normal human epidermal keratinocytes (NHEK) in response to Ca(2+)-ind

221 owed that EGCG treatment of the normal human epidermal keratinocytes (NHEK) inhibits ultraviolet (UV)

222 ation of DNA ligase in cultured normal human epidermal keratinocytes (NHEK) on exposure to the DNA-da

223 how that a single low-dose exposure of human epidermal keratinocytes (NHEK) to an FS20 light source i

224 Rapidly proliferating normal human epidermal keratinocytes (NHEK) were infected with retrov

225 TLR4 mRNA expression in primary normal human epidermal keratinocytes (NHEK).

226 Here, we used normal human epidermal keratinocytes (NHEKs) to determine the effects

227 ration and apoptosis studies in normal human epidermal keratinocytes (NHEKs) were conducted by MTT (3

228 , in a dose-dependent manner in normal human epidermal keratinocytes (NHEKs).

229 ing sites using ChIP-on-chip in normal human epidermal keratinocytes (NHEKs).

230 al vein endothelial cells (HUVECs) and human epidermal keratinocytes (NHEKs).

231 ytes during wound repair and in normal human epidermal keratinocytes (NHEKs).

232 eek for 8 weeks induced p53 mutations in the epidermal keratinocytes of 100% of the mice.

233 was significantly elevated (17-fold) in the epidermal keratinocytes of 7,12-dimethylbenz[a]anthracen

234 are intrinsic to cutaneous neurons and that epidermal keratinocytes only modulate this transduction.

235 In human HaCaT and primary epidermal keratinocytes, photodynamic induction of apopt

236 Hair regenerated from epidermal keratinocyte progenitor cells isolated from mu

237 carcinogen-treated transgenic skin, whereas epidermal keratinocyte proliferation in vitro was not af

238 No significant differences in TPA-induced epidermal keratinocyte proliferation were observed in C/

239 and displayed no detectable abnormalities in epidermal keratinocyte proliferation, differentiation, o

240 ion of COX-2 with either inhibitor decreased epidermal keratinocyte proliferation.

241 differentiation in monolayer cultures of rat epidermal keratinocytes (REKs), but high PpIX signals we

242 cycle exit and differentiation of suprabasal epidermal keratinocytes require nuclear IkappaB kinase a

243 homeostasis and terminal differentiation of epidermal keratinocytes required for formation of the sk

244 differentiation-mitosis checkpoint in human epidermal keratinocytes, resulting in impaired cell divi

245 in the maturation and activity of ADAM17 in epidermal keratinocytes, resulting in significantly upre

246 X receptor (RXR)-alpha and RXR-beta in mouse epidermal keratinocytes (RXR-alphabeta(ep-/-)) or a topi

247 factor-2 in skin of mice lacking RXRalpha in epidermal keratinocytes (RXRalpha(ep-/-) mice), which in

248 s bacterial supernatant-treated normal human epidermal keratinocytes, S1PR1 knockdown reduced IL36G,

249 xpressed only by thymic epithelial cells and epidermal keratinocytes, Skint-1 drives specifically the

250 We found that in epidermal keratinocytes, soy PG inhibited TLR2 and TLR4

251 T pathway as new molecular targets of CsA in epidermal keratinocytes, suggesting a previously unknown

252 ression of CTIP2 was ablated specifically in epidermal keratinocytes, suggests that CTIP2 functions i

253 Fra-1 and Fra-2) that are key controllers of epidermal keratinocyte survival and differentiation, and

254 integration site 1) as a regulator of human epidermal keratinocyte survival.

255 Following UV exposure, epidermal keratinocytes synthesize proopiomelanocortin (

256 ls of VEGF protein and mRNA were observed in epidermal keratinocytes than in oral keratinocytes after

257 e to microbial component-mimicking agents in epidermal keratinocytes that form the physical barrier o

258 tive correlations included the morphology of epidermal keratinocytes, the appearance of nests of nevu

259 ling after a calcium switch in primary human epidermal keratinocytes, the loss of this receptor did n

260 During terminal differentiation of the epidermal keratinocytes, the nucleus undergoes a program

261 During terminal differentiation of the epidermal keratinocytes, the nucleus undergoes programme

262 in-specific factor(s) derived primarily from epidermal keratinocytes, thereby providing a mechanism f

263 alpha-2-glycoprotein is normally produced by epidermal keratinocytes, these studies raise the possibi

264 ifferentiation and inhibits proliferation in epidermal keratinocytes through interaction with the vit

265 tips, where they are transferred to adjacent epidermal keratinocytes through pathways that involve mi

266 ore, we propose that loss of cell contact in epidermal keratinocytes through reactive oxygen species-

267 ns resulting from exposure of cultured human epidermal keratinocytes to arsenite and antimonite in co

268 expression of KLK5 and KLK7 in normal human epidermal keratinocytes to better understand how these m

269 fic IQGAP requirement in vivo, we engineered epidermal keratinocytes to express individual IQGAP prot

270 epidermal growth factor (EGF) stimulation of epidermal keratinocytes to initiate the cell migration c

271 ession of multiple host-defense molecules in epidermal keratinocytes to promote healing.

272 se characterized by reduced adherence of the epidermal keratinocytes to the underlying dermis, and is

273 Furthermore, epidermal keratinocytes treated with p38 MAPK inhibitors

274 itochondrial apoptotic signaling pathways in epidermal keratinocytes, triggering apoptosis and afford

275 In transgenic mice overexpressing Bmx in epidermal keratinocytes, tumors induced by a two-stage c

276 ed transgenic mice that overexpress TRPV3 in epidermal keratinocytes under the control of the keratin

277 Surface epithelial cells, such as the epidermal keratinocyte, undergo a process of terminal ce

278 The major cell type of the epidermis, the epidermal keratinocyte, undergoes a carefully choreograp

279 We demonstrate here that in epidermal keratinocytes, unliganded heterodimers of vita

280 Epidermal keratinocytes, unlike other cells, express man

281 tional responses to ultraviolet radiation in epidermal keratinocytes using microarray chips containin

282 geted the overexpression of RasGRP1 to basal epidermal keratinocytes using the keratin 5 promoter.

283 ion in ultraviolet-B-irradiated normal human epidermal keratinocytes was also assessed and correlated

284 rehensively the JNK-regulated genes in human epidermal keratinocytes, we compared the transcriptional

285 16) E6 and E7 gene-immortalized normal human epidermal keratinocytes, we demonstrated intracellular a

286 In epidermal keratinocytes, we observed higher proportions

287 ma on human skin cells, primary normal human epidermal keratinocytes were exposed to ultraviolet-B ra

288 Immortalized HaCaT and adult human epidermal keratinocytes were used as a model and the eff

289 recently shown expression of RasGRP1 in the epidermal keratinocytes where it can mediate Ras activat

290 integrin alpha3beta1 is highly expressed in epidermal keratinocytes, where it regulates both cell-au

291 f CXCL14 was significantly down-regulated in epidermal keratinocytes with age.

292 demonstrated that treatment of normal human epidermal keratinocytes with cis-UCA resulted in increas

293 We show that stimulation of epidermal keratinocytes with EGF, but not with other gro

294 To address this issue, we treated human epidermal keratinocytes with five EFNAs individually and

295 day revealed a functional circadian clock in epidermal keratinocytes with hundreds of transcripts reg

296 as altered differentiation and activation of epidermal keratinocytes with increased expression of hai

297 fibroblasts led to increased invasiveness of epidermal keratinocytes with pre-malignant features.

298 However, direct treatment of normal human epidermal keratinocytes with S1P increased the expressio

299 e presence of functional opioid receptors on epidermal keratinocytes, with attendant regulation of ke

300 tured human skin, or isolated cultured human epidermal keratinocytes, with triiodothyronine (100 pmol